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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2012, Volume 109, Issue 52, pp. 21510 - 21515
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 02/2003, Volume 278, Issue 6, pp. 3751 - 3761
Transforming growth factor-beta (TGF-beta) elicits cellular effects by activating specific Smad proteins that control the transcription of target genes.... 
SIGNAL-TRANSDUCTION | TGF-BETA | PROTEIN | EPITHELIAL-CELLS | CANCER CELLS | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | N-TERMINAL KINASE | GENE-EXPRESSION | C-JUN | JNK PATHWAY | Fibronectins - biosynthesis | Receptors, Transforming Growth Factor beta - genetics | Receptors, Transforming Growth Factor beta - chemistry | Humans | Protein-Serine-Threonine Kinases | Molecular Sequence Data | Receptors, Transforming Growth Factor beta - physiology | Adaptor Proteins, Vesicular Transport | DNA-Binding Proteins - metabolism | Base Sequence | X-Linked Inhibitor of Apoptosis Protein | Activin Receptors, Type I - physiology | Genes, Tumor Suppressor | Mink | Amino Acid Sequence | Mutagenesis, Site-Directed | Activin Receptors, Type I - metabolism | DNA Primers | Activin Receptors, Type I - genetics | Cell Division - physiology | Plasminogen Activator Inhibitor 1 - biosynthesis | Adaptor Proteins, Signal Transducing | Animals | Proteins - metabolism | Receptors, Transforming Growth Factor beta - metabolism | Smad Proteins | Protein Binding | Trans-Activators - metabolism | Enzyme Activation | Activin Receptors, Type I - chemistry | Tumor Suppressor Proteins | Mitogen-Activated Protein Kinases - metabolism | Proteins/metabolism | Receptors; Transforming Growth Factor beta/chemistry/genetics/metabolism/physiology | Trans-Activators/metabolism | Medical and Health Sciences | Medicin och hälsovetenskap | Adaptor Proteins; Vesicular Transport | DNA-Binding Proteins/metabolism | Fibronectins/biosynthesis | Research Support; Non-U.S. Gov't | Natural Sciences | Plasminogen Activator Inhibitor 1/biosynthesis | Cell Division/physiology | Mutagenesis; Site-Directed | Naturvetenskap | Mitogen-Activated Protein Kinases/metabolism | Activin Receptors; Type I/chemistry/genetics/metabolism/physiology
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2016, Volume 291, Issue 29, pp. 15093 - 15107
Journal Article
Nature medicine, ISSN 1078-8956, 11/2014, Volume 20, Issue 11, pp. 1301 - 1309
Some of the anti-neoplastic effects of anthracyclines in mice originate from the induction of innate and T cell-mediated anticancer immune responses. Here we... 
MEDICINE, RESEARCH & EXPERIMENTAL | DOUBLE-STRANDED-RNA | DENDRITIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNATE | RIG-I | TUMORS | CELL BIOLOGY | TOLL-LIKE RECEPTOR-3 | BREAST-CANCER | ANTICANCER CHEMOTHERAPY | GENE | IMMUNE-RESPONSES | Doxorubicin - therapeutic use | Breast Neoplasms - immunology | Humans | RNA, Messenger - metabolism | Adaptor Proteins, Vesicular Transport - metabolism | Interferon Type I - biosynthesis | Receptors, Pattern Recognition - metabolism | Neoplasm Metastasis | Anthracyclines - pharmacology | Interferon Type I - metabolism | Female | Neoadjuvant Therapy | Gene Expression Regulation, Neoplastic - drug effects | RNA - metabolism | Receptor, Interferon alpha-beta - metabolism | Mice, Inbred C57BL | RNA, Messenger - genetics | Treatment Outcome | Toll-Like Receptor 3 - metabolism | Breast Neoplasms - drug therapy | Animals | Breast Neoplasms - genetics | Signal Transduction - drug effects | Breast Neoplasms - pathology | Immunocompetence - drug effects | Anthracyclines - therapeutic use | Chemokine CXCL10 - metabolism | Myxovirus Resistance Proteins - metabolism | Doxorubicin - pharmacology | Anthracyclines | Care and treatment | Analysis | Physiological aspects | Interferon | Research | Cancer | Signal transduction | Chemotherapy | Breast cancer | Chemokines | Life Sciences | Immunology | Immunotherapy
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2011, Volume 108, Issue 50, pp. 20054 - 20059
Journal Article
Nature Immunology, ISSN 1529-2908, 08/2017, Volume 18, Issue 9, pp. 1025 - 1034
Journal Article
Journal Article
Journal Article
Journal of Immunology, ISSN 0022-1767, 05/2010, Volume 184, Issue 10, pp. 5842 - 5848
The adapter protein TRIF mediates signal transduction through TLR3 and TLR4, inducing production of type I IFNs and inflammatory cytokines. The present study... 
TUMOR-NECROSIS-FACTOR | MNK1 | DENDRITIC CELLS | AU-RICH ELEMENTS | PHOSPHORYLATION | PROTEIN-KINASE | BIOSYNTHESIS | INITIATION-FACTOR 4E | MECHANISMS | IMMUNOLOGY | IDENTIFICATION | Tumor Necrosis Factor-alpha - metabolism | Protein-Serine-Threonine Kinases - deficiency | Bone Marrow Cells - enzymology | Dendritic Cells - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | Adaptor Proteins, Vesicular Transport - genetics | Protein Biosynthesis - immunology | Intracellular Signaling Peptides and Proteins - metabolism | Tumor Necrosis Factor-alpha - deficiency | RNA, Messenger - metabolism | MAP Kinase Signaling System - immunology | Intracellular Signaling Peptides and Proteins - deficiency | RNA, Messenger - biosynthesis | MAP Kinase Signaling System - genetics | Time Factors | Bone Marrow Cells - immunology | p38 Mitogen-Activated Protein Kinases - metabolism | Dendritic Cells - metabolism | Protein-Serine-Threonine Kinases - metabolism | Adaptor Proteins, Vesicular Transport - physiology | p38 Mitogen-Activated Protein Kinases - physiology | Protein-Serine-Threonine Kinases - physiology | Mice, Inbred C57BL | RNA, Messenger - genetics | Cells, Cultured | Mice, Transgenic | Mice, Knockout | Animals | Dendritic Cells - enzymology | Mice | Intracellular Signaling Peptides and Proteins - physiology | Bone Marrow Cells - metabolism | Tumor Necrosis Factor-alpha - antagonists & inhibitors
Journal Article