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Journal of International Medical Research, ISSN 0300-0605, 6/2019, Volume 47, Issue 6, pp. 2524 - 2532
Journal Article
Pancreatology, ISSN 1424-3903, 07/2019, Volume 19, Issue 5, pp. 781 - 785
An accessory middle colic artery (AMCA) is an aberrant artery feeding the splenic flexure of the colon. Little is known about the branching pattern of an AMCA.... 
Dorsal pancreatic artery | Accessary middle colic artery | Inferior pancreaticoduodenal artery | ADENOCARCINOMA | COLON | CT ANGIOGRAPHY | GASTROENTEROLOGY & HEPATOLOGY | CT imaging | Usage | Care and treatment | Pancreatic cancer | Spleen | Studies | Lymphatic system | Computed tomography | Surgery | Colorectal cancer | Colon | Pancreas | Patients | Dissection | Veins & arteries
Journal Article
Cancer Cell, ISSN 1535-6108, 03/2012, Volume 21, Issue 3, pp. 418 - 429
Pancreatic ductal adenocarcinomas (PDAs) are characterized by a robust fibroinflammatory response. We show here that this desmoplastic reaction generates... 
INTERSTITIAL FLUID PRESSURE | SOLID TUMORS | ONCOLOGY | COMPARING STANDARD PANCREATICODUODENECTOMY | HUMAN OSTEOSARCOMA XENOGRAFTS | RANDOMIZED-TRIAL | HEAD ADENOCARCINOMA | TUMOR VASCULATURE | EXTENDED LYMPHADENECTOMY | CANCER | BLOOD-PRESSURE | CELL BIOLOGY | Adenocarcinoma - pathology | Cell Adhesion Molecules - administration & dosage | Stromal Cells - pathology | Pancreatic Neoplasms - blood supply | Deoxycytidine - pharmacology | Hyaluronic Acid - physiology | Polyethylene Glycols - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Pancreatic Neoplasms - drug therapy | Deoxycytidine - therapeutic use | Stromal Cells - drug effects | Hyaluronoglucosaminidase - administration & dosage | Drug Evaluation, Preclinical | Hyaluronoglucosaminidase - therapeutic use | Tumor Microenvironment - drug effects | Animals, Genetically Modified | Deoxycytidine - administration & dosage | Pancreatic Neoplasms - pathology | Microvessels - drug effects | Adenocarcinoma - drug therapy | Carcinoma, Pancreatic Ductal - pathology | Extracellular Fluid - drug effects | Polyethylene Glycols - administration & dosage | Adenocarcinoma - blood supply | Hyaluronoglucosaminidase - pharmacology | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Hyaluronic Acid - metabolism | Mice | Carcinoma, Pancreatic Ductal - blood supply | Cell Adhesion Molecules - therapeutic use | Deoxycytidine - analogs & derivatives | Enzymes | Medical colleges | Care and treatment | Glycosaminoglycans | Oncology, Experimental | Research | Chemotherapy | Universities and colleges | Drug therapy | Hyaluronic acid | Public health | Tumors | Cancer
Journal Article
Journal Article
Annals of Oncology, ISSN 0923-7534, 5/2008, Volume 19, Issue 5, pp. 927 - 934
Arterial hypertension (HT) has been reported in all studies involving bevacizumab, an antiangiogenic agent designed to target vascular endothelial growth... 
Hypertension | Angiogenesis | Microcirculation | Endothelial function | Capillary rarefaction | endothelial function | LIES DOWNSTREAM | PERMEABILITY | angiogenesis | NITRIC-OXIDE SYNTHASE | KINASE | ANTIBODY | MECHANISMS | microcirculation | THERAPY | ONCOLOGY | capillary rarefaction | DRUGS | hypertension | ENDOTHELIAL GROWTH-FACTOR | Cholinergic Agents - administration & dosage | Cholinergic Agents - pharmacology | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Antibodies, Monoclonal - therapeutic use | Capillaries - drug effects | Endothelium, Vascular - drug effects | Male | Microscopy, Video | Microcirculation - drug effects | Bevacizumab | Antibodies, Monoclonal, Humanized | Iontophoresis | Colorectal Neoplasms - drug therapy | Hypertension - chemically induced | Forearm - blood supply | Angiogenesis Inhibitors - therapeutic use | Female | Blood Pressure - drug effects | Pilocarpine - administration & dosage | Angiogenesis Inhibitors - adverse effects | Diabetes Mellitus, Type 2 - complications | Colorectal Neoplasms - blood supply | Antibodies, Monoclonal - pharmacology | Endothelium, Vascular - physiopathology | Angiogenesis Inhibitors - pharmacology | Laser-Doppler Flowmetry | Adenocarcinoma - drug therapy | Adenocarcinoma - secondary | Adenocarcinoma - blood supply | Diabetes Mellitus, Type 2 - physiopathology | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Neovascularization, Pathologic - drug therapy | Pilocarpine - pharmacology | Fingers - blood supply | Aged | Vasodilation - drug effects
Journal Article
Cancer Research, ISSN 0008-5472, 09/2013, Volume 73, Issue 17, pp. 5402 - 5415
AKT signaling is constitutively activated in various cancers, due in large part to loss-of-function in the PTEN and PHLPP phosphatases that act as tumor... 
SURVIVAL | OVEREXPRESSION | ACTIVATION | ONCOLOGY | KINASE | RESISTANCE | INHIBITORS | MICRORNAS | NF-KAPPA-B | EXPRESSION | MTOR | Adenocarcinoma - pathology | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Phosphoprotein Phosphatases - metabolism | Gene Expression Regulation, Neoplastic | Neovascularization, Pathologic | 3' Untranslated Regions - genetics | Lung Neoplasms - pathology | MicroRNAs - metabolism | NF-kappa B - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Immunoenzyme Techniques | Carcinoma, Adenosquamous - blood supply | PTEN Phosphohydrolase - antagonists & inhibitors | Forkhead Transcription Factors - metabolism | Female | Adenocarcinoma - genetics | Lung - metabolism | Tumor Cells, Cultured | Proto-Oncogene Proteins c-akt - metabolism | Lung Neoplasms - blood supply | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | PTEN Phosphohydrolase - genetics | Lung - pathology | Carcinoma, Adenosquamous - pathology | Signal Transduction | Carcinoma, Non-Small-Cell Lung - blood supply | Carcinoma, Non-Small-Cell Lung - genetics | PTEN Phosphohydrolase - metabolism | Phosphoprotein Phosphatases - antagonists & inhibitors | Carcinoma, Adenosquamous - genetics | Blotting, Western | Adenocarcinoma - blood supply | Animals | Phosphoprotein Phosphatases - genetics | Mice, Nude | Mice | Mice, Inbred BALB C | MicroRNAs - genetics | Forkhead Box Protein O3 | Apoptosis
Journal Article
Journal Article
Journal Article
Gut, ISSN 0017-5749, 01/2013, Volume 62, Issue 1, pp. 112 - 120
Objective Pancreatic ductal adenocarcinoma (PDA) is characterised by stromal desmoplasia and vascular dysfunction, which critically impair drug delivery. This... 
INTERSTITIAL FLUID PRESSURE | CELLS | THERAPY | SOLID TUMORS | MICROENVIRONMENT | PERMEABILITY | DUCTAL ADENOCARCINOMA | GASTROENTEROLOGY & HEPATOLOGY | GEMCITABINE | CHEMOTHERAPY | ENDOTHELIAL GROWTH-FACTOR | Immunohistochemistry | Cell Adhesion Molecules - administration & dosage | Tissue Array Analysis | Pancreatic Neoplasms - blood supply | Antineoplastic Agents - administration & dosage | Hyaluronic Acid - physiology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Drug Delivery Systems | Pancreatic Neoplasms - drug therapy | Hyaluronoglucosaminidase - administration & dosage | Carcinoma, Pancreatic Ductal - mortality | Cell Adhesion Molecules - pharmacology | Pancreatic Neoplasms - mortality | Doxorubicin - administration & dosage | Deoxycytidine - administration & dosage | Kaplan-Meier Estimate | Mice, Transgenic | Treatment Outcome | Biomarkers, Tumor - physiology | Recombinant Proteins - pharmacology | Recombinant Proteins - administration & dosage | Hyaluronoglucosaminidase - pharmacology | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Carcinoma, Pancreatic Ductal - physiopathology | Mice | Carcinoma, Pancreatic Ductal - blood supply | Pancreatic Neoplasms - physiopathology | Drug Resistance, Neoplasm - physiology | Deoxycytidine - analogs & derivatives | Drug Resistance, Neoplasm - drug effects | Care and treatment | Blood-vessels | Pancreatic cancer | Patient outcomes | Physiological aspects | Models | Research | Hyaluronic acid | Tumors | Studies | Angiogenesis | Medical research | Medical prognosis | Cytotoxicity | Extracellular matrix | Permeability | Kinases | Vascular endothelial growth factor | stem cells | cell death | cancer vaccines | hyaluronan | epithelial cell growth | cancer immunobiology | drug delivery | matrix | epithelial cell adhesion | cancer genetics | carcinogenesis | pancreatic fibrosis | pharmacology | pharmacokinetics | tumour microenvironment | cancer | fibrosis | pancreatic disease | Original | 1506 | pancreatic tumours | extracellular matrix | oxidative stress
Journal Article
Journal Article