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1. Full Text RB loss in resistant EGFR mutant lung adenocarcinomas that transform to small-cell lung cancer
by Niederst, Matthew J and Sequist, Lecia V and Poirier, John T and Mermel, Craig H and Lockerman, Elizabeth L and Garcia, Angel R and Katayama, Ryohei and Costa, Carlotta and Ross, Kenneth N and Moran, Teresa and Howe, Emily and Fulton, Linnea E and Mulvey, Hillary E and Bernardo, Lindsay A and Mohamoud, Farhiya and Miyoshi, Norikatsu and VanderLaan, Paul A and Costa, Daniel B and Jänne, Pasi A and Borger, Darrell R and Ramaswamy, Sridhar and Shioda, Toshi and Iafrate, Anthony J and Getz, Gad and Rudin, Charles M and Mino-Kenudson, Mari and Engelman, Jeffrey A
Nature communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, pp. 6377 - 6377
Lung Neoplasms - drug therapy | Recurrence | Adenocarcinoma - pathology | Adenocarcinoma of Lung | Humans | Lung Neoplasms - metabolism | ErbB Receptors - genetics | Gene Expression Regulation, Neoplastic | bcl-X Protein - genetics | Lung Neoplasms - pathology | Small Cell Lung Carcinoma - drug therapy | Small Cell Lung Carcinoma - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Adenocarcinoma - metabolism | Adenocarcinoma - genetics | Antineoplastic Agents - pharmacology | Lung - metabolism | Gefitinib | ErbB Receptors - deficiency | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Lung - pathology | Aniline Compounds - pharmacology | ErbB Receptors - antagonists & inhibitors | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Retinoblastoma Protein - deficiency | Small Cell Lung Carcinoma - genetics | Sulfonamides - pharmacology | Adenocarcinoma - drug therapy | Disease Progression | Drug Resistance, Neoplasm - genetics | Small Cell Lung Carcinoma - pathology | Afatinib | Retinoblastoma Protein - genetics | Erlotinib Hydrochloride - pharmacology | Lung - drug effects | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Quinazolines - pharmacology | bcl-X Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Drug Resistance, Neoplasm - drug effects | Index Medicus
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2. Full Text The TRAIL-Induced Cancer Secretome Promotes a Tumor-Supportive Immune Microenvironment via CCR2
by Hartwig, Torsten and Montinaro, Antonella and von Karstedt, Silvia and Sevko, Alexandra and Surinova, Silvia and Chakravarthy, Ankur and Taraborrelli, Lucia and Draber, Peter and Lafont, Elodie and Arce Vargas, Frederick and El-Bahrawy, Mona A and Quezada, Sergio A and Walczak, Henning
Molecular cell, ISSN 1097-2765, 02/2017, Volume 65, Issue 4, pp. 730 - 742.e5
cytokine | microenvironment | TRAIL | MDSC | TRAIL-R | tumor | CCL2 | FADD | CCR2 | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Adenocarcinoma - pathology | Cell Proliferation | Adenocarcinoma of Lung | Humans | Lung Neoplasms - metabolism | Caspase 8 - metabolism | Tumor Microenvironment | Lung Neoplasms - pathology | Receptors, TNF-Related Apoptosis-Inducing Ligand - metabolism | Caspase 8 - genetics | Adenocarcinoma - metabolism | Transfection | RNA Interference | Time Factors | Female | Adenocarcinoma - genetics | Chemokine CCL2 - metabolism | Receptors, TNF-Related Apoptosis-Inducing Ligand - genetics | Macrophages - immunology | Carcinoma, Non-Small-Cell Lung - pathology | Fas-Associated Death Domain Protein - genetics | Lung Neoplasms - genetics | A549 Cells | Cytokines - metabolism | Macrophages - pathology | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Adenocarcinoma - immunology | HCT116 Cells | Mice, Inbred C57BL | Carcinoma, Non-Small-Cell Lung - metabolism | Fas-Associated Death Domain Protein - metabolism | Tumor Burden | Mice, SCID | TNF-Related Apoptosis-Inducing Ligand - metabolism | Carcinoma, Non-Small-Cell Lung - immunology | Lung Neoplasms - immunology | Macrophages - metabolism | Phenotype | Receptors, CCR2 - metabolism | Animals | HeLa Cells | Metastasis | Macrophages | Cell death | Cancer | Lung cancer | Index Medicus
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3. Full Text The Notch ligand Jagged2 promotes lung adenocarcinoma metastasis through a miR-200 - Dependent pathway in mice
by Yang, Yanan and Ahn, Young-Ho and Gibbons, Don L and Zang, Yi and Lin, Wei and Thilaganathan, Nishan and Alvarez, Cristina A and Moreira, Daniel C and Creighton, Chad J and Gregory, Philip A and Goodall, Gregory J and Kurie, Jonathan M
The Journal of clinical investigation, ISSN 0021-9738, 04/2011, Volume 121, Issue 4, pp. 1373 - 1385
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | GATA3 Transcription Factor - genetics | Adenocarcinoma - pathology | Receptors, Notch - metabolism | Epithelial-Mesenchymal Transition - physiology | Humans | Lung Neoplasms - metabolism | Glycoproteins - metabolism | Lung Neoplasms - pathology | MicroRNAs - metabolism | Gene Expression Profiling | Mice, 129 Strain | Epithelial-Mesenchymal Transition - genetics | Antigens, CD - metabolism | Adenocarcinoma - metabolism | Peptides - metabolism | Jagged-2 Protein | Adenocarcinoma - genetics | Membrane Proteins - metabolism | GATA3 Transcription Factor - metabolism | Lung Neoplasms - genetics | Signal Transduction | Membrane Proteins - genetics | AC133 Antigen | Adenocarcinoma - secondary | Animals | Cell Line, Tumor | Ligands | Mice | MicroRNAs - genetics | Adenocarcinoma | Genetic aspects | Metastasis | Research | Index Medicus | Abridged Index Medicus
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4. Full Text Estimating Survival in Patients With Lung Cancer and Brain Metastases: An Update of the Graded Prognostic Assessment for Lung Cancer Using Molecular Markers (Lung-molGPA)
by Sperduto, Paul W and Yang, T. Jonathan and Beal, Kathryn and Pan, Hubert and Brown, Paul D and Bangdiwala, Ananta and Shanley, Ryan and Yeh, Norman and Gaspar, Laurie E and Braunstein, Steve and Sneed, Penny and Boyle, John and Kirkpatrick, John P and Mak, Kimberley S and Shih, Helen A and Engelman, Alex and Roberge, David and Arvold, Nils D and Alexander, Brian and Awad, Mark M and Contessa, Joseph and Chiang, Veronica and Hardie, John and Ma, Daniel and Lou, Emil and Sperduto, William and Mehta, Minesh P
JAMA oncology, ISSN 2374-2437, 11/2016, Volume 3, Issue 6, pp. 827 - 831
Life Sciences & Biomedicine | Oncology | Science & Technology | ErbB Receptors - metabolism | Prognosis | Adenocarcinoma of Lung | Lung Neoplasms - mortality | Humans | Male | Combined Modality Therapy | Lung Neoplasms - therapy | Receptor Protein-Tyrosine Kinases - metabolism | Carcinoma, Non-Small-Cell Lung - mortality | Brain Neoplasms - secondary | Carcinoma, Non-Small-Cell Lung - therapy | Carcinoma, Non-Small-Cell Lung - secondary | Adenocarcinoma - therapy | Anaplastic Lymphoma Kinase | Epidemiologic Methods | Brain Neoplasms - therapy | Biomarkers, Tumor - metabolism | Female | Aged | Retrospective Studies | Brain Neoplasms - mortality | Adenocarcinoma - mortality | Index Medicus | Brief Report | Online First | Research
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5. Full Text NRF2 regulates serine biosynthesis in non-small cell lung cancer
by DeNicola, Gina M and Chen, Pei-Hsuan and Mullarky, Edouard and Sudderth, Jessica A and Hu, Zeping and Wu, David and Tang, Hao and Xie, Yang and Asara, John M and Huffman, Kenneth E and Wistuba, Ignacio I and Minna, John D and DeBerardinis, Ralph J and Cantley, Lewis C
Nature genetics, ISSN 1061-4036, 12/2015, Volume 47, Issue 12, pp. 1475 - 1481
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | RNA, Small Interfering - genetics | NF-E2-Related Factor 2 - antagonists & inhibitors | Cell Proliferation | Lung Neoplasms - mortality | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Serine - biosynthesis | Immunoenzyme Techniques | Adenocarcinoma - metabolism | Carcinoma, Non-Small-Cell Lung - secondary | Biomarkers, Tumor - metabolism | NF-E2-Related Factor 2 - genetics | Adenocarcinoma - genetics | Tumor Cells, Cultured | Real-Time Polymerase Chain Reaction | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | RNA, Messenger - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Carcinoma, Non-Small-Cell Lung - mortality | Adenocarcinoma - secondary | Blotting, Western | Xenograft Model Antitumor Assays | Animals | NF-E2-Related Factor 2 - metabolism | Biomarkers, Tumor - genetics | Mice | Adenocarcinoma - mortality | Apoptosis | Transcription factors | Cell metabolism | Development and progression | Genetic aspects | Genetic transcription | Lung cancer, Non-small cell | Properties | Health aspects | Medical research | Enzymes | Lung cancer | Biosynthesis | RNA polymerase | Metabolism | Gene expression | Experiments | Metabolites | Scientific imaging | Tumorigenesis | Labeling | Bioinformatics | Mass spectrometry | Index Medicus
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6. Full Text Detection of known and novel ALK fusion transcripts in lung cancer patients using next-generation sequencing approaches
by Vendrell, Julie A and Taviaux, Sylvie and Béganton, Benoît and Godreuil, Sylvain and Audran, Patricia and Grand, David and Clermont, Estelle and Serre, Isabelle and Szablewski, Vanessa and Coopman, Peter and Mazières, Julien and Costes, Valérie and Pujol, Jean-Louis and Brousset, Pierre and Rouquette, Isabelle and Solassol, Jérôme
Scientific reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 12510 - 11
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Lung Neoplasms - drug therapy | Oncogene Proteins, Fusion - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | Lung Neoplasms - metabolism | Middle Aged | Male | Antineoplastic Agents - therapeutic use | RNA, Messenger - metabolism | Case-Control Studies | Anaplastic Lymphoma Kinase - genetics | Adenocarcinoma of Lung - genetics | Adenocarcinoma of Lung - drug therapy | Anaplastic Lymphoma Kinase - metabolism | Female | High-Throughput Nucleotide Sequencing - instrumentation | High-Throughput Nucleotide Sequencing - methods | Dynactin Complex - genetics | Lung Neoplasms - genetics | Gene Expression | Carcinoma, Non-Small-Cell Lung - surgery | Crizotinib - therapeutic use | Carcinoma, Non-Small-Cell Lung - genetics | RNA, Messenger - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | In Situ Hybridization, Fluorescence | Adenocarcinoma of Lung - surgery | Dynactin Complex - metabolism | Golgi Matrix Proteins - genetics | Oncogene Proteins, Fusion - genetics | Lung Neoplasms - surgery | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Adenocarcinoma of Lung - metabolism | Neoplasm Staging | Golgi Matrix Proteins - metabolism | Immunohistochemistry | Transcription | Lung cancer | Fluorescence in situ hybridization | Non-small cell lung carcinoma | Kinases | Lymphoma | Protein-tyrosine kinase | Tumors | Index Medicus | Life Sciences
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7. Full Text Stearoyl-CoA-desaturase 1 regulates lung cancer stemness via stabilization and nuclear localization of YAP/TAZ
by Noto, A and De Vitis, C and Pisanu, M.E and Roscilli, G and Ricci, Giulia and Catizone, A and Sorrentino, G and Chianese, G and Taglialatela-Scafati, O and Trisciuoglio, D and Del Bufalo, D and Di Martile, M and Di Napoli, A and Ruco, L and Costantini, S and Jakopin, Z and Budillon, A and Melino, G and Del Sal, G and Ciliberto, G and Mancini, R
Oncogene, ISSN 0950-9232, 2017, Volume 36, Issue 32, pp. 4573 - 4584
Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Immunohistochemistry | Wnt3A Protein - metabolism | Adenocarcinoma - pathology | Prognosis | Adenocarcinoma of Lung | Lung Neoplasms - mortality | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Male | Intracellular Signaling Peptides and Proteins - metabolism | Neoplasm Proteins - metabolism | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Adenocarcinoma - metabolism | Cell Nucleus - metabolism | Neoplastic Stem Cells - metabolism | HEK293 Cells | Female | Inhibitor of Apoptosis Proteins - metabolism | Protein Stability | Fatty Acids - metabolism | Protein-Serine-Threonine Kinases - metabolism | Down-Regulation | Stearoyl-CoA Desaturase - antagonists & inhibitors | Survivin | Stearoyl-CoA Desaturase - genetics | Axin Signaling Complex - metabolism | Stearoyl-CoA Desaturase - metabolism | Primary Cell Culture | Adaptor Proteins, Signal Transducing - metabolism | Adenocarcinoma - mortality | Care and treatment | Monounsaturated fatty acids | Research | Gene expression | Lung cancer | Stem cells | Adenocarcinoma | Cell survival | Transcription | Data processing | Desaturase | Fatty acids | Survival | Yes-associated protein | β-catenin | Localization | Bioinformatics | Index Medicus
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8. Full Text Nkx2-1 Represses a Latent Gastric Differentiation Program in Lung Adenocarcinoma
by Snyder, Eric L and Watanabe, Hideo and Magendantz, Margaret and Hoersch, Sebastian and Chen, Tiffany A and Wang, Diana G and Crowley, Denise and Whittaker, Charles A and Meyerson, Matthew and Kimura, Shioko and Jacks, Tyler
Molecular cell, ISSN 1097-2765, 04/2013, Volume 50, Issue 2, pp. 185 - 199
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Thyroid Nuclear Factor 1 | Adenocarcinoma - pathology | Cell Proliferation | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Lung Neoplasms - metabolism | Transcriptional Activation | Gene Expression Regulation, Neoplastic | Transcriptome | Lung Neoplasms - pathology | Stomach - metabolism | Mutation, Missense | Respiratory Mucosa - pathology | Adenocarcinoma - metabolism | Pulmonary Alveoli - metabolism | Cell Differentiation | Lung - metabolism | Nuclear Proteins - genetics | Binding Sites | Hyperplasia - metabolism | Proto-Oncogene Proteins p21(ras) - metabolism | Hepatocyte Nuclear Factor 3-beta - metabolism | Hepatocyte Nuclear Factor 4 - metabolism | Lung - pathology | Transcription Factors - physiology | Pulmonary Alveoli - pathology | Stomach - pathology | Mice, Transgenic | Nuclear Proteins - metabolism | Hepatocyte Nuclear Factor 4 - genetics | Transcription Factors - genetics | Tumor Burden | Organ Specificity | Hepatocyte Nuclear Factor 3-alpha - metabolism | Transcription Factors - metabolism | Animals | Cell Transformation, Neoplastic | Protein Binding | Mice | Nuclear Proteins - physiology | Respiratory Mucosa - metabolism | Adenocarcinoma | Chromosomal proteins | Lung cancer | Genomics | Tumors | Index Medicus
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9. Full Text LKB1 and KEAP1/NRF2 Pathways Cooperatively Promote Metabolic Reprogramming with Enhanced Glutamine Dependence in KRAS-Mutant Lung Adenocarcinoma
by Galan-Cobo, Ana and Sitthideatphaiboon, Piyada and Qu, Xiao and Poteete, Alissa and Pisegna, Marlese A and Tong, Pan and Chen, Pei-Hsuan and Boroughs, Lindsey K and Rodriguez, Mirna L. M and Zhang, Winter and Parlati, Francesco and Wang, Jing and Gandhi, Varsha and Skoulidis, Ferdinandos and DeBerardinis, Ralph J and Minna, John D and Heymach, John
Cancer research (Chicago, Ill.), ISSN 0008-5472, 07/2019, Volume 79, Issue 13, pp. 3251 - 3267
Life Sciences & Biomedicine | Oncology | Science & Technology | Cell Proliferation | Oxidative Stress | Adenocarcinoma of Lung - pathology | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Glutamine - metabolism | Lung Neoplasms - pathology | Cellular Reprogramming | Kelch-Like ECH-Associated Protein 1 - genetics | Adenocarcinoma of Lung - genetics | Adenosine Triphosphate - metabolism | Biomarkers, Tumor - metabolism | Female | NF-E2-Related Factor 2 - genetics | Tumor Cells, Cultured | Protein-Serine-Threonine Kinases - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Protein-Serine-Threonine Kinases - genetics | Glutaminase - metabolism | Kelch-Like ECH-Associated Protein 1 - metabolism | Mice, SCID | Xenograft Model Antitumor Assays | Animals | Energy Metabolism | Metabolic Networks and Pathways | Mice, Nude | NF-E2-Related Factor 2 - metabolism | Mice, Inbred NOD | Biomarkers, Tumor - genetics | Mice | Mice, Inbred BALB C | Mutation | Adenocarcinoma of Lung - metabolism | Apoptosis | Index Medicus
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