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Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 12510 - 11
Rearrangements of the anaplastic lymphoma kinase (ALK) gene in non-small cell lung cancer (NSCLC) represent a novel molecular target in a small subset of... 
GENE FUSIONS | SPITZ TUMORS | ASSAY | ANAPLASTIC LYMPHOMA-KINASE | MULTIDISCIPLINARY SCIENCES | FISH | CRIZOTINIB | RET FUSIONS | INFLAMMATORY MYOFIBROBLASTIC TUMORS | REARRANGEMENTS | MULTIPLEX | Lung Neoplasms - drug therapy | Oncogene Proteins, Fusion - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Humans | Lung Neoplasms - metabolism | Middle Aged | Male | Antineoplastic Agents - therapeutic use | RNA, Messenger - metabolism | Case-Control Studies | Anaplastic Lymphoma Kinase - genetics | Adenocarcinoma of Lung - genetics | Adenocarcinoma of Lung - drug therapy | Anaplastic Lymphoma Kinase - metabolism | Female | High-Throughput Nucleotide Sequencing - instrumentation | High-Throughput Nucleotide Sequencing - methods | Dynactin Complex - genetics | Lung Neoplasms - genetics | Gene Expression | Carcinoma, Non-Small-Cell Lung - surgery | Crizotinib - therapeutic use | Carcinoma, Non-Small-Cell Lung - genetics | RNA, Messenger - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | In Situ Hybridization, Fluorescence | Adenocarcinoma of Lung - surgery | Dynactin Complex - metabolism | Golgi Matrix Proteins - genetics | Oncogene Proteins, Fusion - genetics | Lung Neoplasms - surgery | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Adenocarcinoma of Lung - metabolism | Neoplasm Staging | Golgi Matrix Proteins - metabolism | Immunohistochemistry | Transcription | Lung cancer | Fluorescence in situ hybridization | Non-small cell lung carcinoma | Kinases | Lymphoma | Protein-tyrosine kinase | Tumors | Life Sciences
Journal Article
Cell Metabolism, ISSN 1550-4131, 04/2018, Volume 27, Issue 4, pp. 935 - 943.e4
Journal Article
International Journal of Cancer, ISSN 0020-7136, 08/2018, Volume 143, Issue 4, pp. 931 - 943
IFN‐γ plays a crucial role in anti‐tumor responses and also induces expression of PD‐L1, a well‐established inhibitor of anti‐tumor immune function.... 
IFNG | PI3K | STAT1 | lung adenocarcinoma | PD‐L1 | PD-L1 | TARGET | CELLS | ACTIVATION | PHOSPHORYLATION | TUMOR-DEVELOPMENT | EGFR | THERAPY | INTERFERON-GAMMA | ONCOLOGY | PATHWAY | RESISTANCE | Phosphorylation | Prognosis | Adenocarcinoma of Lung - pathology | Humans | Lung Neoplasms - metabolism | Transcriptome | Tumor Microenvironment | Lung Neoplasms - pathology | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - metabolism | STAT1 Transcription Factor - metabolism | Adenocarcinoma of Lung - genetics | Janus Kinase 2 - metabolism | Transcription, Genetic | Adenocarcinoma of Lung - immunology | Proto-Oncogene Proteins c-akt - metabolism | STAT3 Transcription Factor - metabolism | Lung Neoplasms - genetics | T-Lymphocytes, Cytotoxic - immunology | Signal Transduction | Interferon-gamma - physiology | B7-H1 Antigen - metabolism | Lung Neoplasms - immunology | Cell Line, Tumor | Adenocarcinoma of Lung - metabolism | Adenocarcinoma | Lung cancer | Crosstalk | Cytotoxicity | AKT protein | Lymphocytes T | Signal transduction | Pathways | Lymphocytes | Immunotherapy | Janus kinase 2 | Cell cycle | Bioindicators | Inhibition | Stat1 protein | Sensitivity analysis | Immune response | Tumor cells | Gene expression | Patients | Molecular chains | 1-Phosphatidylinositol 3-kinase | Signaling | Biomarkers | Interferon | Combinatorial analysis | Cancer | Tumors
Journal Article
Journal Article
Cancer Science, ISSN 1347-9032, 12/2018, Volume 109, Issue 12, pp. 3783 - 3793
The p53‐inducible gene 3 ( PIG 3) is one of the p53‐induced genes at the onset of apoptosis, which plays an important role in cell apoptosis and DNA damage... 
migration | p53‐inducible gene 3 | invasion | focal adhesion kinase | non‐small cell lung cancer | non-small cell lung cancer | p53-inducible gene 3 | PIG3 | APOPTOSIS | FAK | ONCOLOGY | PROHIBITIN | CANCER STATISTICS | INHIBITOR | PHASE-I | EXPRESSION | BINDING | Neoplasm Transplantation | Up-Regulation | Phosphorylation | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Humans | Lung Neoplasms - metabolism | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Intracellular Signaling Peptides and Proteins - metabolism | Adenocarcinoma of Lung - genetics | Adult | Female | Focal Adhesion Kinase 1 - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | A549 Cells | Signal Transduction | Neoplasm Invasiveness | Proto-Oncogene Proteins - genetics | Lymphatic Metastasis | Animals | Proto-Oncogene Proteins pp60(c-src) - metabolism | Cell Line, Tumor | Aged | Mice | Adenocarcinoma of Lung - metabolism | Cell Movement | Adenocarcinoma | Squamous cell carcinoma | RNA | Genes | Genomics | Development and progression | Metastasis | Lung cancer, Non-small cell | Education parks | School facilities | Analysis | Genetic aspects | Tumor proteins | Cancer | p53 Protein | DNA damage | Lung cancer | Clinical trials | Genomes | Kinases | Cancer therapies | Lymph nodes | Metastases | Cell adhesion & migration | Growth factors | Deoxyribonucleic acid--DNA | Lymphatic system | Small cell lung carcinoma | Therapeutic applications | Non-small cell lung carcinoma | Lung carcinoma | Gene expression | Biomarkers | Mutation | Focal adhesion kinase | Paxillin | Binding sites | Cell migration | Apoptosis | Tumors | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2018, Volume 13, Issue 5, p. e0197402
S100A16 is a conserved member of the S100 protein family in mammals. Its upregulation was observed in many tumors and is related to malignant transformation.... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | METHYLATION | INVASION | PROTEIN | AMPLIFICATION | CANCER GENOME ATLAS | BIOMARKER | PATHWAY | MULTIDISCIPLINARY SCIENCES | CHEMORESISTANCE | Immunohistochemistry | Multivariate Analysis | S100 Calcium Binding Protein A6 - metabolism | Prognosis | Lung Neoplasms - mortality | Humans | Lung Neoplasms - metabolism | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Gene Expression Profiling | Adenocarcinoma of Lung - mortality | DNA Methylation | Biomarkers, Tumor - metabolism | Female | Retrospective Studies | Carcinoma, Non-Small-Cell Lung - metabolism | Cell Cycle Proteins - metabolism | Kaplan-Meier Estimate | Gene Dosage | Carcinoma, Non-Small-Cell Lung - mortality | Disease-Free Survival | Cell Line, Tumor | CpG Islands | Aged | Adenocarcinoma of Lung - metabolism | Adenocarcinoma | Genetic aspects | Lung cancer, Non-small cell | Methylation | Health aspects | EF hand proteins | Transformation | Copy number | Lung cancer | Genomes | Tissue analysis | Multivariate analysis | Data mining | Tissues | Cancer therapies | Gene sequencing | Proteins | S100 protein | Cell growth | DNA methylation | Indicators | Deoxyribonucleic acid--DNA | CpG islands | Cell survival | Regression analysis | Gene expression | Survival | Survival analysis | Lungs | Correlation analysis | Medical prognosis | Epigenetics | Cancer | Tumors | Deoxyribonucleic acid | DNA
Journal Article
CELL DEATH & DISEASE, ISSN 2041-4889, 02/2018, Volume 9, Issue 2, pp. 244 - 9
Chemotherapy is a standard treatment for non-small-cell lung cancer (NSCLC). However, the dose-limiting toxicity of drugs and the development of... 
GEFITINIB | PROFILES | BMI-1 | STATISTICS | RESISTANCE | SELF-RENEWAL | TUMORIGENESIS | TUMOR-INITIATING CELLS | CELL BIOLOGY | Lung Neoplasms - drug therapy | Adenocarcinoma of Lung - pathology | Neoplastic Stem Cells - drug effects | Humans | Lung Neoplasms - metabolism | Gene Expression Regulation, Neoplastic | Spheroids, Cellular - pathology | Drugs, Chinese Herbal - pharmacology | Lung Neoplasms - pathology | MicroRNAs - metabolism | NF-kappa B - metabolism | Apoptotic Protease-Activating Factor 1 - genetics | Carcinogenesis - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Adenocarcinoma of Lung - genetics | Neoplastic Stem Cells - metabolism | Adenocarcinoma of Lung - drug therapy | Neoplastic Stem Cells - pathology | Female | MicroRNAs - agonists | Spheroids, Cellular - drug effects | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | A549 Cells | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Spheroids, Cellular - metabolism | Carcinogenesis - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Apoptotic Protease-Activating Factor 1 - metabolism | Carcinogenesis - drug effects | Carcinogenesis - pathology | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | NF-kappa B - genetics | Tumor Burden - drug effects | Mice, Nude | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Carcinoma, Non-Small-Cell Lung - drug therapy | Adenocarcinoma of Lung - metabolism | Antineoplastic Agents, Phytogenic - pharmacology | Proto-Oncogene Proteins c-bcl-2 - genetics | Drug Resistance, Neoplasm - drug effects | Medicinal plants | Cell proliferation | Adenocarcinoma | Toxicity | MiRNA | Chemoresistance | Non-small cell lung carcinoma | Drug resistance | Drug development | Cancer therapies | Chemotherapy | Lungs | Stem cells | Tumorigenesis | Cell migration
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2018, Volume 128, Issue 4, pp. 1268 - 1282
Journal Article
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2018, Volume 143, Issue 12, pp. 3169 - 3180
Claudins are a family of transmembrane proteins integral to the structure and function of tight junctions (TJ). Disruption of TJ and alterations in claudin... 
AKT | xenograft | IGF‐1 receptor | YAP/TAZ | lung alveolar epithelial type II cells | IGF-1 receptor | TARGET | PHOSPHORYLATION | JUNCTION PROTEIN CLAUDIN-7 | HIPPO PATHWAY | YAP | TAZ | ONCOLOGY | GROWTH | INHIBITS CELL-PROLIFERATION | EXPRESSION | Lung Neoplasms - genetics | Claudins - genetics | Claudins - physiology | Promoter Regions, Genetic | Receptor, IGF Type 1 - metabolism | Phosphorylation | Cell Proliferation | Proto-Oncogene Proteins c-yes - metabolism | Adenocarcinoma of Lung - pathology | Neoplasm Invasiveness | Humans | Lung Neoplasms - metabolism | Lung Neoplasms - pathology | Intracellular Signaling Peptides and Proteins - metabolism | Blotting, Western | Mice, Knockout | DNA Methylation | Neoplasm Metastasis | Adenocarcinoma of Lung - genetics | Animals | Signal Transduction - physiology | Mice | Adenocarcinoma of Lung - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Adenocarcinoma | Genetic research | Metastasis | Methylation | Analysis | Membrane proteins | Cell proliferation | Transcription | Xenotransplantation | AKT protein | Insulin-like growth factors | Malignancy | Kinases | Inactivation | Metastases | Proteins | Xenografts | DNA methylation | Alveoli | Growth factors | Deactivation | Tight junctions | Invasiveness | siRNA | Insulin | Yes-associated protein | Signaling | Lungs | Tumor suppressor genes | Disruption | Cell migration | Cancer | Tumors | Structure-function relationships
Journal Article