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The Journal of Clinical Endocrinology & Metabolism, ISSN 1945-7197, 2013, Volume 98, Issue 12, pp. 4826 - 4833
Journal Article
Medicine (Baltimore), ISSN 0025-7974, 2017, Volume 96, Issue 15, p. e6614
In this study, we analyze and discuss the treatments of postoperative nasal complications after endonasal transsphenoidal resection of pituitary neoplasms... 
nasal | complications | pituitary neoplasms | endonasal transsphenoidal resection | SURGERY | MEDICINE, GENERAL & INTERNAL | EXPERIENCE | OUTCOMES | OF-THE-LITERATURE | ADENOMAS | LESIONS | Epistaxis - etiology | Epistaxis - therapy | Nasal Lavage - methods | Pituitary Neoplasms - surgery | Aftercare - methods | Postoperative Complications - etiology | Rhinitis, Atrophic - etiology | Tissue Adhesions - etiology | Nose - surgery | Humans | Middle Aged | Paranasal Sinuses - physiopathology | Male | Nasal Mucosa - pathology | Sphenoid Sinusitis - therapy | Recovery of Function | Young Adult | Nose Diseases - etiology | Olfactory Nerve Diseases - etiology | Nasal Septal Perforation - therapy | Adult | Female | Retrospective Studies | Rhinitis, Atrophic - therapy | Sphenoid Sinus - surgery | Child | Cerebrospinal Fluid Rhinorrhea - etiology | Nose Diseases - therapy | Nose - injuries | Natural Orifice Endoscopic Surgery - adverse effects | Postoperative Complications - therapy | Sphenoid Sinusitis - etiology | Nasal Surgical Procedures - methods | Olfactory Nerve Diseases - therapy | Tissue Adhesions - therapy | Natural Orifice Endoscopic Surgery - methods | Adolescent | Cerebrospinal Fluid Rhinorrhea - therapy | Aged | Nasal Septal Perforation - etiology | Usage | Care and treatment | Excision (Surgery) | Complications | Surgery
Journal Article
Annals of Epidemiology, ISSN 1047-2797, 2009, Volume 19, Issue 1, pp. 58 - 69
Purpose The mucosa of the small intestine encompasses about 90% of the luminal surface area of the digestive system, but only 2% of the total annual... 
Internal Medicine | Adenocarcinoma | Molecular Biology | Cancer Epidemiology | Celiac Disease | Crohn Disease | Non-Hodgkin Lymphoma | Neuroendocrine Tumor | Gastrointestinal Stromal Tumor | Bile Acid Metabolism | Microbial Flora | RISK-FACTORS | CROHNS-DISEASE | ADENOMA-CARCINOMA SEQUENCE | COLON CARCINOGENESIS | MEDITERRANEAN LYMPHOMA | CELIAC-DISEASE | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | INFLAMMATORY-BOWEL-DISEASE | COLORECTAL-CANCER | POPULATION-BASED COHORT | GASTROINTESTINAL STROMAL TUMORS | United States - epidemiology | Intestinal Mucosa - metabolism | Gastrointestinal Stromal Tumors - epidemiology | Prevalence | Intestine, Small - pathology | Adenoma - epidemiology | Adenocarcinoma - etiology | Humans | Neuroendocrine Tumors - epidemiology | Cytokines - physiology | Gastrointestinal Stromal Tumors - etiology | Incidence | Colorectal Neoplasms - etiology | Inflammation - metabolism | Colorectal Neoplasms - epidemiology | Intestine, Small - microbiology | Risk Factors | Adenoma - etiology | Intestinal Neoplasms - etiology | Intestinal Neoplasms - epidemiology | Intestinal Mucosa - microbiology | Lymphoma, Non-Hodgkin - epidemiology | Inflammation - etiology | Intestine, Small - metabolism | Neuroendocrine Tumors - etiology | Adenocarcinoma - epidemiology | Intestinal Mucosa - pathology | Lymphoma, Non-Hodgkin - etiology | Epidemiology | Crohn's disease
Journal Article
Gut, ISSN 0017-5749, 06/2017, Volume 66, Issue 6, pp. 1138 - 1153
Key physiological functions of the liver, including glucose and lipid metabolism, become disturbed in the setting of non-alcoholic fatty liver disease (NAFLD)... 
Cardiovascular Disease | Diabetes Mellitus | Nonalcoholic Steatohepatitis | CORONARY-ARTERY CALCIUM | BONE-MINERAL DENSITY | HEPATIC INSULIN-RESISTANCE | GAMMA-GLUTAMYL-TRANSFERASE | COLORECTAL ADENOMA | TERM-FOLLOW-UP | CHRONIC KIDNEY-DISEASE | GASTROENTEROLOGY & HEPATOLOGY | TYPE-2 DIABETES-MELLITUS | INDEPENDENT RISK-FACTOR | ADIPOSE-TISSUE | Colorectal Neoplasms - epidemiology | Cardiovascular Diseases - etiology | Prevalence | Cytokines - metabolism | Renal Insufficiency, Chronic - etiology | Humans | Risk Factors | Gastrointestinal Microbiome | Adipokines - metabolism | Non-alcoholic Fatty Liver Disease - physiopathology | Osteoporosis - etiology | Diabetes Mellitus, Type 2 - epidemiology | Incidence | Non-alcoholic Fatty Liver Disease - complications | Renal Insufficiency, Chronic - epidemiology | Colorectal Neoplasms - etiology | Animals | Diet | Cardiovascular Diseases - epidemiology | Diabetes Mellitus, Type 2 - etiology | Nutritional Status | Osteoporosis - epidemiology | Causes of | Inflammatory bowel diseases | Liver diseases | Research | Cytokines | Cardiovascular diseases | Adipose tissue | Colorectal cancer | Cardiovascular disease | Metabolic syndrome | Risk factors | Alcohol use | Liver cancer | Glucose metabolism | Ultrasonic imaging | Fatty liver | Atherosclerosis | Tumor necrosis factor-TNF | Lipid metabolism | Diabetes mellitus (non-insulin dependent) | Hypertension | Obesity | Kidneys | Mortality | Diabetes mellitus | Vitamin deficiency | Histology | Inflammation | Dietary intake | Meta-analysis | Steatosis | Studies | Genetic variance | Phospholipase | Coronary vessels | Insulin resistance | Bone mineral density | Diabetes | Kidney diseases
Journal Article
British Journal of Cancer, ISSN 0007-0920, 06/2017, Volume 117, Issue 1, pp. 41 - 50
Background: Some highly penetrant familial cancer syndromes exhibit elevated leukaemia risk, and there is evidence for familial clustering of lung cancer and... 
familial clustering | lung cancer | high-energy beam | ionising radiation | myeloid leukaemia | space radiation exposure | mice | BRCA2 MUTATION CARRIERS | CATALYTIC SUBUNIT | EFFECTIVENESS RBE VALUES | BIOLOGICAL EFFECTIVENESS | ATOMIC-BOMB SURVIVORS | RETROSPECTIVE COHORT | BREAST-CANCER | LUNG-CANCER | SPACE RADIATION | ONCOLOGY | MYELOID-LEUKEMIA | Carcinoma, Hepatocellular - mortality | Harderian Gland | Neoplasms - etiology | Gamma Rays - adverse effects | Adenoma - genetics | Adenoma - mortality | Lung Neoplasms - mortality | Adenocarcinoma - etiology | Leukemia, Myeloid - genetics | Sebaceous Gland Neoplasms - etiology | Liver Neoplasms, Experimental - mortality | Neoplastic Syndromes, Hereditary - etiology | Neoplastic Syndromes, Hereditary - mortality | Neoplasms, Radiation-Induced - mortality | Neoplasms, Radiation-Induced - genetics | Lung Neoplasms - etiology | Neoplasms - genetics | Carcinoma, Hepatocellular - genetics | Adenocarcinoma - genetics | Carcinoma, Hepatocellular - etiology | Liver Neoplasms, Experimental - genetics | Neoplastic Syndromes, Hereditary - genetics | Leukemia, Myeloid - mortality | Radiation, Ionizing | Lung Neoplasms - genetics | Sebaceous Gland Neoplasms - genetics | Proportional Hazards Models | Neoplasms - mortality | Adenoma - etiology | Liver Neoplasms, Experimental - etiology | Leukemia, Myeloid - etiology | Animals | Sebaceous Gland Neoplasms - mortality | Mice | Adenocarcinoma - mortality | Translational Therapeutics
Journal Article
TURKISH JOURNAL OF HEMATOLOGY, ISSN 1300-7777, 2016, Volume 33, Issue 1, pp. 66 - 70