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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 05/2013, Volume 110, Issue 21, pp. 8555 - 8560
Cushing disease (CD) is a life-threatening disorder attributed to excess pituitary tumor-derived adrenocorticotrophic hormone (ACTH) and adrenal steroid... 
Cell growth | Phosphorylation | Pituitary neoplasms | Secretion | Plasmids | Cell lines | Pituitary ACTH hypersecretion | Mice | Orphan nuclear receptors | Tumors | MANAGEMENT | ACID | PHOSPHORYLATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | MODULATOR | EXPRESSION | Transcriptional Activation - genetics | Cell Proliferation | Receptors, Steroid - metabolism | Humans | Neoplasm Proteins - metabolism | ACTH-Secreting Pituitary Adenoma - therapy | Adrenocorticotropic Hormone - genetics | Pituitary ACTH Hypersecretion - genetics | ACTH-Secreting Pituitary Adenoma - genetics | Receptors, Thyroid Hormone - genetics | Response Elements - genetics | Corticosterone - secretion | Neoplasm Proteins - genetics | Pro-Opiomelanocortin - genetics | Pituitary ACTH Hypersecretion - therapy | Gene Expression Regulation, Neoplastic - genetics | Pituitary ACTH Hypersecretion - pathology | ACTH-Secreting Pituitary Adenoma - metabolism | Corticosterone - genetics | Adrenocorticotropic Hormone - secretion | Receptors, Thyroid Hormone - metabolism | Animals | Pituitary ACTH Hypersecretion - metabolism | Receptors, Steroid - genetics | Pro-Opiomelanocortin - biosynthesis | Cell Line, Tumor | ACTH-Secreting Pituitary Adenoma - pathology | Physiological aspects | Pituitary gland | Research | Hydrocortisone | Health aspects | Causes and theories of causation | Diseases | Rodents | Genetics | T cell receptors | Binding sites | Index Medicus | Biological Sciences
Journal Article
International Journal of Cancer, ISSN 0020-7136, 07/2018, Volume 143, Issue 1, pp. 139 - 150
Journal Article
Nature, ISSN 0028-0836, 01/2009, Volume 457, Issue 7229, pp. 603 - 607
Cancer stem cells are remarkably similar to normal stem cells: both self-renew, are multipotent and express common surface markers, for example, prominin 1... 
GENE | COLORECTAL-CANCER | POLYPOSIS | MULTIDISCIPLINARY SCIENCES | MOUSE | MUTATION | IDENTIFICATION | TUMOR-GROWTH | TUMORIGENESIS | PERIVASCULAR NICHE | HEMATOPOIETIC STEM | Neoplasm Transplantation | Neoplastic Stem Cells - cytology | Intestine, Small - pathology | Receptors, G-Protein-Coupled - metabolism | Adenoma - genetics | Glycoproteins - metabolism | Peptides - genetics | Transplantation, Heterologous | Stem Cells - cytology | Stem Cells - metabolism | Wnt Proteins - metabolism | Antigens, CD - genetics | Adenoma - metabolism | Antigens, CD - metabolism | Antigens, CD - analysis | Peptides - metabolism | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Intestinal Neoplasms - genetics | Biomarkers - metabolism | Glycoproteins - genetics | Intestinal Neoplasms - metabolism | Biomarkers - analysis | Cells, Cultured | Genes, Reporter - genetics | AC133 Antigen | beta Catenin - metabolism | beta Catenin - genetics | Intestinal Neoplasms - pathology | Cell Lineage | Animals | Intestine, Small - cytology | Cell Transformation, Neoplastic | Adenoma - pathology | Stem Cells - pathology | Mice | Glycoproteins - analysis | Mutation | Peptides - analysis | Genetic markers | Intestine, Small | Stem cells | Cancer cells | Genetic aspects | Diagnosis | Research | Properties | Health aspects | Cancer | Tissue | Brain | Microscopy | Skeletal system | Small intestine | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 2, pp. e14727 - e14727
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2011, Volume 6, Issue 10, pp. e25314 - e25314
Background: There are sparse data on genetic, epigenetic and vitamin D exposure in African Americans (AA) with colon polyp. Consequently, we evaluated serum... 
POPULATION | RECTAL-CANCER | ASSOCIATIONS | METAANALYSIS | BREAST-CANCER RISK | COLORECTAL-CANCER | D-RECEPTOR GENE | BIOLOGY | D DEFICIENCY | PREVENTION | CIRCULATING 25-HYDROXYVITAMIN-D | Colonic Neoplasms - genetics | Genetic Predisposition to Disease | Adenoma - genetics | Humans | Middle Aged | Risk Factors | Intercellular Signaling Peptides and Proteins - genetics | African Americans - genetics | Male | DNA Methylation - genetics | Promoter Regions, Genetic - genetics | Receptors, Calcitriol - genetics | Case-Control Studies | Vitamin D - blood | Adenoma - blood | Colonic Polyps - genetics | Colonic Neoplasms - blood | Polymorphism, Single Nucleotide - genetics | Female | Aged | Seasons | Odds Ratio | Colonic Polyps - blood | Epigenetic inheritance | Medical research | Genes | African Americans | Calcifediol | Liquid chromatography | B cells | Vitamin D | Medicine, Experimental | Genetic aspects | Single nucleotide polymorphisms | Alfacalcidol | Methylation | Mass spectrometry | Control methods | Homology | Population studies | Single-nucleotide polymorphism | Adenoma | Gene polymorphism | Risk factors | Confidence intervals | DNA methylation | Colon | Deoxyribonucleic acid--DNA | Statistical analysis | Multivariable control | Mass spectroscopy | Regression analysis | Risk analysis | Minority & ethnic groups | Serum levels | Tumor suppressor genes | Vitamin D receptors | Dkk1 protein | Health risk assessment | Polymorphism | Tumors | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, pp. e88126 - e88126
In pleomorphic adenomas of the salivary glands (PASG) recurrent chromosomal rearrangements affecting either 8q12 or 12q14 similar to 15 lead to an... 
MOLECULAR CHARACTERIZATION | STEM-CELLS | DNA-BINDING | TRANSCRIPTION FACTOR HMGA2 | HIGH-LEVEL EXPRESSION | FUSION TRANSCRIPTS | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | MESENCHYMAL TUMORS | SALIVARY-GLAND TUMORS | PLEOMORPHIC ADENOMAS | Uterine Neoplasms - genetics | Carcinoma, Papillary - genetics | Uterine Neoplasms - metabolism | Adenoma - genetics | Uterine Neoplasms - pathology | Humans | Leiomyoma - metabolism | DNA-Binding Proteins - genetics | Carcinoma, Papillary - pathology | Carcinoma, Papillary - metabolism | Adenoma - metabolism | DNA-Binding Proteins - metabolism | Leiomyoma - pathology | Thyroid Neoplasms - genetics | HMGA2 Protein - metabolism | HMGA2 Protein - genetics | Adenoma - pathology | Cell Line, Tumor | Female | Leiomyoma - genetics | Thyroid Neoplasms - metabolism | Thyroid Neoplasms - pathology | Chromosomal proteins | RNA | Genes | Thyroid diseases | Stem cells | Genetic aspects | Fibroblast growth factors | Genetic transcription | Tumors | Transcription factors | Adipose tissue | Pathogenesis | Glands | Activation | Insulin-like growth factors | Kinases | Chromosome rearrangements | Proteins | Transfection | Uterus | Genetics | Chromosomes | Thyroid | Fibroblast growth factor 1 | Salivary glands | Abnormalities | Benign | Fibroids | Gene expression | Thyroid cancer | Chromosome 8 | Index Medicus
Journal Article
Cancer, ISSN 0008-543X, 12/2014, Volume 120, Issue 23, pp. 3627 - 3634
Journal Article
Clinics in Colon and Rectal Surgery, ISSN 1531-0043, 12/2016, Volume 29, Issue 4, pp. 289 - 295
Abstract The genetic events involved in the transformation of normal colonic epithelium to neoplastic polyps to invasive carcinoma, as initially proposed by... 
Review Article | polyps | neoplasm | genetics | colorectal | MORTALITY | COLONOSCOPY | SURGERY | SIGMOIDOSCOPY | SERRATED LESIONS | PREVENTION | PREVALENCE | COLORECTAL-CANCER INCIDENCE | ADENOMA | GASTROENTEROLOGY & HEPATOLOGY | AGE
Journal Article
Familial Cancer, ISSN 1389-9600, 4/2017, Volume 16, Issue 2, pp. 303 - 309
We report on three brothers affected by pancreatic tumors, all due to different causes, including mutations associated with two different cancer predisposition... 
Biomedicine, general | Human Genetics | Familial pancreatic cancer | Familial adenomatous polyposis | APC gene | Double germline mutation | Epidemiology | Tumor disposition syndrome | Biomedicine | BRCA2 gene | Pancreatic cancer | Hereditary breast and ovarian cancer syndrome | Cancer Research | Hereditary pancreatic cancer | GENE-MUTATIONS | HEREDITARY PANCREATITIS | MICROSATELLITE INSTABILITY | ADENOCARCINOMA | PALB2 | ONCOLOGY | INCREASED RISK | PREDISPOSITION | COLORECTAL-CANCER | GENETICS & HEREDITY | PAPILLARY-MUCINOUS NEOPLASMS | Adenoma - surgery | Genetic Testing | Adenoma - genetics | Pancreatic Neoplasms - diagnosis | Colorectal Neoplasms - genetics | Humans | Middle Aged | Male | Genetic Counseling | Fatal Outcome | Genes, BRCA2 | Germ-Line Mutation | Adult | Female | Adenocarcinoma - genetics | Duodenal Neoplasms - surgery | Adenomatous Polyposis Coli - genetics | Adenoma - diagnosis | Genetic Predisposition to Disease | Pancreatic Neoplasms - surgery | Adenomatous Polyposis Coli - surgery | Cholangiopancreatography, Endoscopic Retrograde | Duodenal Neoplasms - genetics | Pancreatic Neoplasms - genetics | Duodenal Neoplasms - diagnosis | Adenocarcinoma - diagnosis | Breast Neoplasms - genetics | Esophageal Neoplasms - genetics | Pedigree | Aged | Proctocolectomy, Restorative | BRCA2 Protein - genetics | Anopheles | Gene mutations | Genes | Genetic research | Genetic aspects | Ovarian cancer | Index Medicus
Journal Article