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1. Full Text Tumour suppressor RNF43 is a stem-cell E3 ligase that induces endocytosis of Wnt receptors
by Koo, B.K and Spit, M and Jordens, I and Low, T.Y and Stange, D.E and van de Wetering, M and van Es, J.H and Mohammed, S and Heck, A.J.R and Maurice, M.M and Clevers, H
Nature (London), ISSN 0028-0836, 2012, Volume 488, Issue 7413, pp. 665 - 669
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Biological and medical sciences | Multiple tumors. Solid tumors. Tumors in childhood (general aspects) | Medical sciences | Tumors | Oncogene Proteins - genetics | Receptors, Wnt - antagonists & inhibitors | Cell Proliferation | Receptors, G-Protein-Coupled - metabolism | Humans | Ubiquitin - metabolism | Paneth Cells - pathology | Stem Cells - cytology | Receptors, Wnt - metabolism | Stem Cells - metabolism | DNA-Binding Proteins - deficiency | Adenoma - metabolism | DNA-Binding Proteins - metabolism | Endocytosis | Ubiquitination | Lysosomes - metabolism | Stem Cells - enzymology | Tumor Suppressor Proteins - deficiency | Organoids - metabolism | Tumor Suppressor Proteins - genetics | HEK293 Cells | Colorectal Neoplasms - metabolism | Paneth Cells - metabolism | Tumor Suppressor Proteins - metabolism | Organoids - pathology | Oncogene Proteins - metabolism | Organoids - cytology | Ubiquitin-Protein Ligases - metabolism | Frizzled Receptors - metabolism | DNA-Binding Proteins - genetics | beta Catenin - metabolism | Animals | Wnt Signaling Pathway - drug effects | Adenoma - pathology | Ubiquitin-Protein Ligases - deficiency | Oncogene Proteins - deficiency | Mice | Receptors, G-Protein-Coupled - genetics | Colorectal Neoplasms - pathology | Ubiquitin-Protein Ligases - genetics | Ubiquitin | Colorectal cancer | Physiological aspects | Development and progression | Research | Gene expression | Risk factors | Proteins | Epidermal growth factor | Mutation | Kinases | Rodents | Stem cells | Index Medicus
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2. Full Text Distinct ATOH1 and Neurog3 requirements define tuft cells as a new secretory cell type in the intestinal epithelium
by Gerbe, F and van Es, J.H and Makrini, L and Brulin, B and Mellitzer, G and Robine, S and Romagnolo, B and Shroyer, N.F and Bourgaux, J.F and Pignodel, C and Clevers, H and Jay, P
The Journal of cell biology, ISSN 0021-9525, 2011, Volume 192, Issue 5, pp. 767 - 780
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Intestinal Mucosa - metabolism | Adenocarcinoma - pathology | Humans | Adenoma - metabolism | Intestinal Mucosa - cytology | Intestine, Large - metabolism | Adenocarcinoma - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Cell Differentiation | Membrane Proteins - metabolism | Intestine, Large - cytology | Protein-Serine-Threonine Kinases - metabolism | Biomarkers - metabolism | SOX9 Transcription Factor - metabolism | Basic Helix-Loop-Helix Transcription Factors - physiology | Nerve Tissue Proteins - physiology | Basic Helix-Loop-Helix Transcription Factors - genetics | Intestinal Neoplasms - metabolism | Mice, Inbred C57BL | Biomarkers - analysis | Nerve Tissue Proteins - genetics | Intestinal Neoplasms - pathology | Nerve Tissue Proteins - metabolism | Animals | Intestine, Small - cytology | Cyclooxygenase 2 - metabolism | Adenoma - pathology | Mice | Cyclooxygenase 1 - metabolism | Intestine, Small - metabolism | Isomerases - metabolism | Proteins | Intestines | Research | Chemical properties | Epithelium | Cells | Scanning electron microscopy | Enzymes | Identification | Anatomy & physiology | Stem cells | Index Medicus | Life Sciences | Cancer
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3. Full Text Yap-dependent reprogramming of Lgr5+ stem cells drives intestinal regeneration and cancer
by Gregorieff, Alex and Liu, Yu and Inanlou, Mohammad R and Khomchuk, Yuliya and Wrana, Jeffrey L
Nature (London), ISSN 0028-0836, 10/2015, Volume 526, Issue 7575, pp. 715 - 718
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Intestinal Mucosa - metabolism | Neoplastic Stem Cells - cytology | Receptors, G-Protein-Coupled - metabolism | Intestines - radiation effects | Male | Stem Cells - cytology | Phosphoproteins - metabolism | Stem Cells - metabolism | Colonic Neoplasms - metabolism | Regeneration - radiation effects | Adenoma - metabolism | Neoplastic Stem Cells - metabolism | Organoids - metabolism | Neoplastic Stem Cells - pathology | Female | Epiregulin - metabolism | Protein-Serine-Threonine Kinases - metabolism | Wnt Signaling Pathway | Disease Models, Animal | Radiation, Ionizing | Cell Differentiation - radiation effects | ErbB Receptors - metabolism | Paneth Cells - cytology | Stem Cells - radiation effects | Homeostasis - radiation effects | Cell Survival - radiation effects | Animals | Cell Transformation, Neoplastic | Adaptor Proteins, Signal Transducing - deficiency | Colonic Neoplasms - pathology | Phosphoproteins - deficiency | Adenoma - pathology | Mice | Paneth Cells - radiation effects | Adaptor Proteins, Signal Transducing - metabolism | Cell Cycle Proteins | Intestines - cytology | Index Medicus
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4. Full Text The Genomic Landscape of Renal Oncocytoma Identifies a Metabolic Barrier to Tumorigenesis
by Joshi, Shilpy and Tolkunov, Denis and Aviv, Hana and Hakimi, Abraham A and Yao, Ming and Hsieh, James J and Ganesan, Shridar and Chan, Chang S and White, Eileen
Cell reports (Cambridge), ISSN 2211-1247, 12/2015, Volume 13, Issue 9, pp. 1895 - 1908
cancer metabolism | cancer genomics | lysosome | mitochondria | oncocytoma | Oncocytoma | Mitochondria | Cancer metabolism | Cancer genomics | Lysosome | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Cyclin D1 - metabolism | AMP-Activated Protein Kinases - metabolism | Kidney Neoplasms - genetics | Adenoma, Oxyphilic - pathology | Kidney - pathology | Humans | Transcriptome | Carcinoma, Renal Cell - genetics | Adenoma, Oxyphilic - genetics | Male | Kidney Neoplasms - metabolism | Autophagy - drug effects | Tumor Suppressor Protein p53 - genetics | DNA Copy Number Variations | Kidney - metabolism | Lysosomes - metabolism | Adenoma, Oxyphilic - metabolism | Female | Lysosome-Associated Membrane Glycoproteins - metabolism | Carcinoma, Renal Cell - pathology | Metformin - pharmacology | Tumor Suppressor Protein p53 - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Carcinoma, Renal Cell - metabolism | Sequence Analysis, RNA | Cyclin D1 - genetics | Cathepsins - metabolism | Cell Transformation, Neoplastic | Golgi Apparatus - metabolism | Kidney Neoplasms - pathology | AMP-Activated Protein Kinases - genetics | Karyotype | Index Medicus
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5. Full Text Reciprocal regulation of gastrointestinal homeostasis by SHP2 and STAT-mediated trefoil gene activation in gp130 mutant mice
by Tebbutt, Niall C and Giraud, Andrew S and Waring, Paul and Jenkins, Brendan and Malki, Sina and Alderman, Barbara M and Ernst, Matthias and Heath, Joan K and Inglese, Melissa and Clay, Fiona J and Grail, Dianne and Hollande, Frédéric
Nature medicine, ISSN 1078-8956, 10/2002, Volume 8, Issue 10, pp. 1089 - 1097
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Protein Tyrosine Phosphatase, Non-Receptor Type 11 | Membrane Glycoproteins - metabolism | Transcriptional Activation | Peptides - genetics | Neuropeptides | Protein Tyrosine Phosphatases - metabolism | Wound Healing | Antigens, CD - genetics | Antigens, CD - metabolism | Growth Substances - genetics | Peptides - metabolism | Digestive System Physiological Phenomena | Protein Phosphatase 2 | Interleukin-6 - metabolism | Cytokine Receptor gp130 | Colon - pathology | Interleukin-11 - metabolism | Mice, Transgenic | Spleen - cytology | Colon - metabolism | STAT1 Transcription Factor | Mice | Intestinal Mucosa - pathology | Intestinal Mucosa - metabolism | Homeostasis | Stomach Neoplasms - metabolism | Stomach Neoplasms - pathology | Stomach - metabolism | Adenoma - metabolism | DNA-Binding Proteins - metabolism | Muscle Proteins | Trefoil Factor-3 | Trefoil Factor-2 | Cell Line | Stomach - pathology | Gene Expression Regulation | Intracellular Signaling Peptides and Proteins | Membrane Glycoproteins - genetics | Proteins - genetics | Animals | Proteins - metabolism | Spleen - metabolism | Mucins | Stomach - cytology | Adenoma - pathology | Signal Transduction - physiology | Trans-Activators - metabolism | Growth Substances - metabolism | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
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6. Full Text Immunohistochemical, genetic and clinical characterization of sporadic aldosterone-producing adenomas
by Monticone, Silvia and Castellano, Isabella and Versace, Karine and Lucatello, Barbara and Veglio, Franco and Gomez-Sanchez, Celso E and Williams, Tracy A and Mulatero, Paolo
Molecular and cellular endocrinology, ISSN 0303-7207, 08/2015, Volume 411, Issue C, pp. 146 - 154
KCNJ5 | CACNA1D | CYP11B1 immunohistochemistry | ATP1A1 | CYP11B2 immunohistochemistry | Aldosterone producing adenoma | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Sodium-Potassium-Exchanging ATPase - genetics | Plasma Membrane Calcium-Transporting ATPases - genetics | Adenoma - genetics | Humans | Hyperaldosteronism - pathology | Middle Aged | Adrenal Cortex - metabolism | Male | Hyperaldosteronism - metabolism | Adrenal Cortex - pathology | Adenoma - metabolism | Plasma Membrane Calcium-Transporting ATPases - metabolism | Aldosterone - metabolism | Adrenocortical Adenoma - genetics | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Adult | Female | Cytochrome P-450 CYP11B2 - metabolism | Hyperaldosteronism - genetics | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | Adrenocortical Adenoma - pathology | Adrenocortical Adenoma - metabolism | Sodium-Potassium-Exchanging ATPase - metabolism | Calcium Channels, L-Type - genetics | Steroid 11-beta-Hydroxylase - metabolism | Adenoma - pathology | Calcium Channels, L-Type - metabolism | Genetic aspects | Corticosteroids | Aldosterone | Steroids | Index Medicus | ATP2B3
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7. Full Text Increased formate overflow is a hallmark of oxidative cancer
by Meiser, Johannes and Schuster, Anne and Pietzke, Matthias and Voorde, Johan Vande and Athineos, Dimitris and Oizel, Kristell and Burgos-Barragan, Guillermo and Wit, Niek and Dhayade, Sandeep and Morton, Jennifer P and Dornier, Emmanuel and Sumpton, David and Mackay, Gillian M and Blyth, Karen and Patel, Ketan J and Niclou, Simone P and Vazquez, Alexei
Nature communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1368 - 12
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Methotrexate - pharmacology | Intestinal Mucosa - metabolism | Adenoma - genetics | Gene Expression Regulation, Neoplastic | Mammary Glands, Animal - virology | Male | Mammary Neoplasms, Experimental - metabolism | Mammary Neoplasms, Experimental - genetics | Adenoma - metabolism | Isoenzymes - metabolism | Antimetabolites, Antineoplastic - pharmacology | Female | Mammary Tumor Virus, Mouse - pathogenicity | Intestinal Neoplasms - genetics | Formates - metabolism | Mammary Neoplasms, Experimental - drug therapy | Tumor Microenvironment - drug effects | Glycine Hydroxymethyltransferase - genetics | Intestines - pathology | Oxidation-Reduction | Isoenzymes - genetics | Mammary Glands, Animal - pathology | Intestinal Neoplasms - metabolism | Mice, Inbred C57BL | Mitochondria - metabolism | Mitochondria - drug effects | Glycine Hydroxymethyltransferase - metabolism | Serine - metabolism | Intestinal Neoplasms - pathology | Animals | Mammary Glands, Animal - metabolism | Formates - pharmacology | Adenoma - pathology | Cell Line, Tumor | Mice | Mammary Neoplasms, Experimental - virology | Phenotypes | Mitochondria | Animal models | Intestine | Oxidative metabolism | Serine | Catabolism | Mammary gland | Tumor cell lines | Metabolism | Overflow | Cancer | Index Medicus
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8. Full Text K⁺ Channel Mutations in Adrenal Aldosterone-Producing Adenomas and Hereditary Hypertension
by Murim Choi and Ute I. Scholl and Peng Yue and Peyman Björklund and Bixiao Zhao and Carol Nelson-Williams and Weizhen Ji and Yoonsang Cho and Aniruddh Patel and Clara J. Men and Elias Lolis and Max V. Wisgerhof and David S. Geller and Shrikant Mane and Per Hellman and Gunnar Westin and Göran Åkerström and Wenhui Wang and Tobias Carling and Richard P. Lifton
Science (American Association for the Advancement of Science), ISSN 0036-8075, 2/2011, Volume 331, Issue 6018, pp. 768 - 772
Depolarization | Hypertension | Somatic mutation | Cell growth | Hyperaldosteronism | REPORTS | Loss of heterozygosity | Adenoma | Genetic mutation | Sequencing | Tumors | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Fundamental and applied biological sciences. Psychology | Classical genetics, quantitative genetics, hybrids | Human | Biological and medical sciences | Cell physiology | Molecular and cellular biology | Genetics of eukaryotes. Biological and molecular evolution | Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes | Adrenal Glands - pathology | Potassium - metabolism | Cell Proliferation | Humans | Hyperaldosteronism - pathology | Hyperplasia | Protein Multimerization | Zona Glomerulosa - pathology | Male | Hyperaldosteronism - metabolism | Sodium - metabolism | Aldosterone - metabolism | Adrenocortical Adenoma - genetics | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Female | Hypertension - genetics | Adrenal Cortex Neoplasms - metabolism | Cell Line | G Protein-Coupled Inwardly-Rectifying Potassium Channels - chemistry | Hyperaldosteronism - genetics | Mutant Proteins - genetics | Mutant Proteins - metabolism | Zona Glomerulosa - metabolism | Adrenal Cortex Neoplasms - pathology | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | Adrenocortical Adenoma - pathology | Adrenocortical Adenoma - metabolism | Hypertension - metabolism | Adrenal Cortex Neoplasms - genetics | Mutant Proteins - chemistry | Mutation | Index Medicus
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9. Subclinical adenomas in postmortem pituitaries: Classification and correlations to clinical data
by Buurman, Hilke and Saeger, Wolfgang
European journal of endocrinology, ISSN 0804-4643, 05/2006, Volume 154, Issue 5, pp. 753 - 758
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology
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10. Multiple aberrant hormone receptors in Cushing's syndrome
by El Ghorayeb, Nada and Bourdeau, Isabelle and Lacroix, André
European journal of endocrinology, ISSN 0804-4643, 10/2015, Volume 173, Issue 4, pp. M45 - M60
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Gene Expression | Receptors, G-Protein-Coupled - metabolism | Humans | Receptors, LH - metabolism | Receptors, Vasopressin - metabolism | Receptors, Glucagon - metabolism | Cushing Syndrome - metabolism | Receptors, Gastrointestinal Hormone - metabolism | Adenoma - metabolism | Receptor, Melanocortin, Type 2 - metabolism | Adrenal Gland Neoplasms - metabolism | Receptors, Serotonin, 5-HT4 - metabolism | Receptors, Adrenergic, beta - metabolism | Cyclic AMP - metabolism
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