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Oncogene, ISSN 0950-9232, 12/2011, Volume 30, Issue 50, pp. 4977 - 4989
Familial adenomatous polyposis (FAP) is caused by germline mutations in the adenomatous polyposis coli (APC) gene. Two promoters, 1A and 1B, have been... 
Q-PCR | mutation | promoter | familial adenomatous ployposis | APC | METHYLATION | HYPERMETHYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | BETA-CATENIN | CLINICAL CHARACTERIZATION | REGION | CELL BIOLOGY | GERM-LINE MUTATIONS | ONCOLOGY | COLORECTAL-CANCER | GENETICS & HEREDITY | COLI GENE | EXPRESSION | TRUNCATING MUTATIONS | Adenomatous Polyposis Coli Protein - genetics | Intestinal Mucosa - metabolism | Adenomatous Polyposis Coli Protein - biosynthesis | Adenoma - genetics | Humans | Middle Aged | Gene Silencing | Male | Adenomatous Polyposis Coli - pathology | Promoter Regions, Genetic - genetics | Adenoma - metabolism | Adenomatous Polyposis Coli - metabolism | Sweden | Alleles | Adenoma - pathology | Germ-Line Mutation | Adult | Female | Registries | Aged | Adenomatous Polyposis Coli - genetics | Gene Expression Regulation, Neoplastic - genetics | Intestinal Mucosa - pathology | Oncology | Mutation | Genomics | Cancer | Transcription | Familial adenomatous polyposis | Gene regulation | Mucosa | adenomatous polyposis coli protein | Adenoma | Gene deletion | Blood | Promoters | Gene silencing | adenomatous polyposis coli | Models | Tumorigenesis | Original | Q–PCR | pathology | Dermatologi och venereologi | Dermatology and Venereal Diseases | Genetic | Adenomatous Polyposis Coli Protein | genetics | biosynthesis | Intestinal Mucosa | Promoter Regions | Adenomatous Polyposis Coli | Gene Expression Regulation | metabolism | Cancer and Oncology | Neoplastic | Cancer och onkologi
Journal Article
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 03/2016, Volume 22, Issue 5, pp. 1284 - 1294
Journal Article
Cancer Research, ISSN 0008-5472, 02/2003, Volume 63, Issue 3, pp. 728 - 734
Journal Article
Journal Article
Gynecologic Oncology, ISSN 0090-8258, 2015, Volume 139, Issue 1, pp. 30 - 39
Abstract Objective To validate our earlier observation that 11 chemoresistance-associated mRNAs are molecular markers of poor overall survival in ovarian... 
Hematology, Oncology and Palliative Medicine | Obstetrics and Gynecology | Tumor heterogeneity | Chemotherapy response | Ovarian serous carcinoma | Survival | Quantitative PCR | Metastases | MANAGEMENT | MARKER | ANTICANCER DRUG-RESISTANCE | GUIDELINES | OBSTETRICS & GYNECOLOGY | BCL-2 | ONCOLOGY | BAG3 | S100A4 | PROTEINS | Prognosis | Humans | Middle Aged | Cystadenocarcinoma, Serous - metabolism | Annexin A2 - biosynthesis | Cystadenocarcinoma, Serous - genetics | RNA, Messenger - metabolism | Cystadenocarcinoma, Serous - drug therapy | Ovarian Neoplasms - genetics | S100 Proteins - genetics | Mitogen-Activated Protein Kinase 3 - biosynthesis | Aged, 80 and over | Apoptosis Regulatory Proteins - genetics | Adult | Female | Ovarian Neoplasms - metabolism | S100 Proteins - biosynthesis | Ovarian Neoplasms - drug therapy | Adenomatous Polyposis Coli Protein - genetics | Apoptosis Regulatory Proteins - biosynthesis | Gene Expression | Adenomatous Polyposis Coli Protein - biosynthesis | Mitogen-Activated Protein Kinase 3 - genetics | RNA, Messenger - genetics | Drug Resistance, Neoplasm - genetics | Annexin A2 - genetics | Adaptor Proteins, Signal Transducing - genetics | Adaptor Proteins, Signal Transducing - biosynthesis | Aged | Biomarkers, Tumor - genetics | Biomarkers, Tumor - biosynthesis | Genetic aspects | Carcinoma | Genes | Cancer
Journal Article
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 2014, Volume 124, Issue 5, pp. 1899 - 1913
Journal Article
Cancer Science, ISSN 1347-9032, 03/2015, Volume 106, Issue 3, pp. 217 - 226
Genetic, physiological and environmental factors are implicated in colorectal carcinogenesis. Mutations in the mutL homolog 1 ( MLH 1 ) gene, one of the DNA... 
Colon carcinogenesis | Mlh1 | inflammation | radiation | Lynch syndrome | Inflammation | Radiation | LYNCH-SYNDROME | MICROSATELLITE INSTABILITY | ULCERATIVE-COLITIS | SITE MUTATION | HEREDITARY COLORECTAL-CARCINOMA | BOWEL-DISEASE | ONCOLOGY | DNA-MISMATCH-REPAIR | MOUSE MODEL | CANCER-RISK | SODIUM-INDUCED COLITIS | Inflammation - chemically induced | MutL Protein Homolog 1 | Colonic Neoplasms - genetics | Tumor Suppressor Protein p53 - biosynthesis | Adenocarcinoma - chemically induced | Colonic Neoplasms - chemically induced | Male | DNA Mismatch Repair - genetics | Carcinogenesis - radiation effects | beta Catenin - biosynthesis | Colitis - chemically induced | Nuclear Proteins - deficiency | Female | Adenocarcinoma - genetics | Nuclear Proteins - genetics | Disease Models, Animal | Radiation, Ionizing | Adenomatous Polyposis Coli Protein - genetics | Dextran Sulfate - pharmacology | Cyclin-Dependent Kinase Inhibitor p21 - biosynthesis | Adenomatous Polyposis Coli Protein - biosynthesis | Carcinogenesis - immunology | Mice, Inbred C57BL | Carcinogenesis - genetics | Inflammation - immunology | Mice, Knockout | Animals | Adaptor Proteins, Signal Transducing - deficiency | Adaptor Proteins, Signal Transducing - genetics | Mice | Immunohistochemistry | Dextran | Ionizing radiation | Colon cancer | Gastrointestinal diseases | Colitis | Sulfates | Tumor proteins | Carcinogenesis | Drinking water | Laboratories | p53 Protein | Colorectal carcinoma | Colorectal cancer | Polyposis coli | Cancer therapies | DNA repair | Risk factors | Lymphocytes | Mismatch repair | Sodium sulfate | DNA methylation | Deoxyribonucleic acid--DNA | MLH1 protein | Environmental factors | Polyposis | Inflammatory bowel disease | Sodium | Mutation | Tumors | Index Medicus | Original
Journal Article
BMC Cancer, ISSN 1471-2407, 12/2015, Volume 15, Issue 1, p. 457
Journal Article