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The American Journal of Surgery, ISSN 0002-9610, 06/2018, Volume 215, Issue 6, pp. 1066 - 1067
FPC is an inherited, autosomal dominant disease characterized by a large number of adenomatous polyps in the colon/rectum early in life. The first historical... 
SURGERY | BLACK PATIENTS | Adenomatous Polyposis Coli - surgery | Genotype | Disease Management | Adenomatous Polyposis Coli - genetics | Humans | Digestive System Surgical Procedures - methods | Ostomy | Genetic counseling | Surveillance | Disease | Surgery | Colorectal cancer | Surgeons | Polyposis coli | Patients | Polyposis | Age
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 09/2018, Volume 38, Issue 17
Adenomatous polyposis coli (APC) is a key molecule to maintain cellular homeostasis in colonic epithelium by regulating cell-cell adhesion, cell polarity, and... 
APC | Armadillo repeats | Golgi fragmentation | Asef | Adenomatous polyposis coli | MIGRATION | PROTEIN | CHOLESTEROL | PHOSPHORYLATION | CRYSTAL-STRUCTURE | BIOCHEMISTRY & MOLECULAR BIOLOGY | armadillo repeats | CANCER | CELL BIOLOGY | EPITHELIAL-CELLS | adenomatous polyposis coli | COLORECTAL TUMOR-CELLS | MORPHOLOGY
Journal Article
by Wang, D and Zhang, Z and Li, YW and Xu, C and Yu, YJ and Li, MS and Chen, C and Zhang, XP
MEDICAL SCIENCE MONITOR, ISSN 1643-3750, 05/2019, Volume 25, pp. 3796 - 3803
Background: Familial adenomatous polyposis (FAP), which has a very high tendency of progression to colorectal cancer, is mainly caused by mutations of the... 
MEDICINE, RESEARCH & EXPERIMENTAL | Adenomatous Polyps | GERMLINE MUTATION | Pedigree | APC GENE | Adenomatous Polyposis Coli | Mutation | CANCER | CELL
Journal Article
Journal Article
Journal Article
Molecular Psychiatry, ISSN 1359-4184, 10/2014, Volume 19, Issue 10, pp. 1133 - 1142
Intellectual disabilities (IDs) and autism spectrum disorders link to human APC inactivating gene mutations. However, little is known about adenomatous... 
TUMOR-SUPPRESSOR APC | MENTAL-RETARDATION | PSYCHIATRY | BIOCHEMISTRY & MOLECULAR BIOLOGY | SYNAPTIC-TRANSMISSION | SPECTRUM DISORDERS | INTRACHROMOSOMAL INSERTION | BETA-CATENIN | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | CEREBRAL-CORTEX | INTERSTITIAL DELETION | RARE DE-NOVO | Autistic Disorder - pathology | Autistic Disorder - physiopathology | Cadherins - metabolism | Wnt Signaling Pathway - physiology | Male | Synapses - pathology | Excitatory Postsynaptic Potentials - physiology | Social Behavior | Long-Term Potentiation - physiology | Disease Models, Animal | Miniature Postsynaptic Potentials - physiology | Adenomatous Polyposis Coli Protein - genetics | Stereotyped Behavior - physiology | Cognition Disorders - physiopathology | Maze Learning - physiology | Pyramidal Cells - physiopathology | Synapses - physiology | Spatial Memory - physiology | Cognition Disorders - pathology | Dendritic Spines - physiology | Hippocampus - pathology | beta Catenin - metabolism | Mice, Knockout | Animals | Dendritic Spines - pathology | Adenomatous Polyposis Coli Protein - metabolism | Pyramidal Cells - pathology | Hippocampus - physiopathology | Psychological aspects | Proteins | Autism | Analysis | Physiological aspects | Cellular signal transduction | Genetic aspects | Polyposis, Familial | Research | Cognition disorders | Prevalence studies (Epidemiology)
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2015, Volume 10, Issue 10, p. e0141144
Myopia incidence in China is rapidly becoming a very serious sight compromising problem in a large segment of the general population. Therefore, delineating... 
REFRACTIVE ERROR | AXIAL LENGTH | PATHOLOGICAL COMPLICATIONS | OPTICAL COHERENCE TOMOGRAPHY | TYROSINE-HYDROXYLASE | MULTIDISCIPLINARY SCIENCES | OCULAR GROWTH | DOPAMINE SYNTHESIS | FORM-DEPRIVATION MYOPIA | RETINAL-PIGMENT EPITHELIUM | BRIEF PERIODS | Tyrosine 3-Monooxygenase - metabolism | Refraction, Ocular | Retina - metabolism | Humans | Male | Myopia - metabolism | Matrix Metalloproteinase 9 - metabolism | Collagen Type I - genetics | Vitreous Body - metabolism | Matrix Metalloproteinase 9 - genetics | Sclera - pathology | Disease Models, Animal | Adenomatous Polyposis Coli Protein - genetics | Collagen Type I - metabolism | Matrix Metalloproteinase 2 - metabolism | Sclera - metabolism | Tomography, Optical Coherence | Gene Expression Regulation | Myopia - pathology | Mice, Knockout | Myopia - genetics | Matrix Metalloproteinase 2 - genetics | Tyrosine 3-Monooxygenase - genetics | Animals | Vision, Ocular | Adenomatous Polyposis Coli Protein - metabolism | Vitreous Body - pathology | Mice | Retina - pathology | Cell proliferation | Biometrics | Photorefraction | Collagen (type I) | Laboratories | Pathogenesis | Familial adenomatous polyposis | Myopia | Collagens | Science | Retina | Tyrosine 3-monooxygenase | Polyposis coli | Incidence | Developmental stages | Rodents | Cell cycle | Adenomatous polyposis coli protein | Tyrosine | Dopamine | Complications | Hydroxylase | Optometry | Gene expression | College campuses | Gelatinase B | Gelatinase A | Adenomatous polyposis coli | Optical Coherence Tomography | Stem cells | Mutation
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