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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2010, Volume 107, Issue 22, pp. 9944 - 9949
Actomyosin contractility affects cellular organization within tissues in part through the generation of mechanical forces at sites of cell—matrix and cell—cell... 
Cell growth | Microinjections | Epithelial cells | Bowties | Cell adhesion | Cadherins | Adherens junctions | Focal adhesions | Physiological regulation | Endothelial cells | Mechanotransduction | Traction force | Myosin | Adherens junction | PDMS | CONTACTS | ACTIVATION | mechanotransduction | MULTIDISCIPLINARY SCIENCES | MYOSIN IIB | myosin | EXTENSION | FOCAL ADHESIONS | traction force | SHAPE | adherens junction | RAC | GROWTH | RHO | VASCULAR ENDOTHELIAL-CADHERIN | Myosin Type II - physiology | rhoA GTP-Binding Protein - administration & dosage | Cadherins - metabolism | Mechanotransduction, Cellular - drug effects | Humans | Green Fluorescent Proteins - genetics | rhoA GTP-Binding Protein - physiology | rhoA GTP-Binding Protein - genetics | Mechanotransduction, Cellular - physiology | Antigens, CD - genetics | Antigens, CD - metabolism | Endothelial Cells - ultrastructure | Thrombin - pharmacology | Lysophospholipids - pharmacology | rac GTP-Binding Proteins - physiology | Cadherins - genetics | Endothelial Cells - physiology | Recombinant Proteins - metabolism | Green Fluorescent Proteins - metabolism | Cells, Cultured | Recombinant Proteins - genetics | rho GTP-Binding Proteins - physiology | Recombinant Proteins - administration & dosage | Sphingosine - pharmacology | Biomechanical Phenomena | Sphingosine - analogs & derivatives | Adherens Junctions - physiology | Adherens Junctions - drug effects | Adherens Junctions - ultrastructure | Amino Acid Substitution | Endothelial Cells - drug effects | Junctional complexes (Epithelium) | Cell junctions | Properties | Proteins | Biochemistry | Cells | Cell adhesion & migration | Index Medicus | Biological Sciences | Physical Sciences
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 2012, Volume 196, Issue 5, pp. 641 - 652
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, pp. e101815 - e101815
Background and Objectives: Blood-brain barrier (BBB) dysfunction is an integral feature of neurological disorders and involves the action of multiple... 
MOUSE-BRAIN | OXIDATIVE STRESS | NAD(P)H OXIDASE | IN-VITRO | PROTEIN-KINASE | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | NECROSIS-FACTOR-ALPHA | VASCULAR ENDOTHELIAL-CELLS | TNF-ALPHA | NF-KAPPA-B | Tumor Necrosis Factor-alpha - metabolism | Reactive Oxygen Species - metabolism | Down-Regulation | Humans | Cells, Cultured | NADPH Oxidases - metabolism | Blood-Brain Barrier - drug effects | Gene Expression Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Interleukin-6 - pharmacology | Endothelial Cells - cytology | Adherens Junctions - drug effects | Interleukin-6 - metabolism | Endothelial Cells - drug effects | Tight Junctions - drug effects | Oxidases | Nervous system diseases | Tumor necrosis factor | Acetylcysteine | Genetic aspects | Superoxide | Permeability | Brain | Oxidative stress | Biotechnology | Reactive oxygen species | Oxidase | Membrane permeability | Superoxide dismutase | Activation | Kinases | Phosphatase | NAD(P)H oxidase | Interleukin 6 | Proteins | Antioxidants | Time dependence | Genotype & phenotype | Blood-brain barrier | Catalase | Rodents | Tumor necrosis factor-TNF | Capillaries | Drug dosages | Neurological disorders | Phenotypes | Cytokines | Tight junctions | Pulmonary arteries | siRNA | Inflammation | Preventive medicine | Tumor necrosis factor-α | Gene expression | Cadherin | Endothelial cells | Endothelium | Neurological diseases | Sodium | Microvasculature | Index Medicus
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 8/2015, Volume 52, Issue 1, pp. 664 - 678
The blood-brain barrier (BBB) symbolically represents the gateway to the central nervous system. It is a single layer of specialized endothelial cells that... 
A2A adenosine receptor | Neurology | Neurosciences | Biomedicine | Tight and adherens junctions | Blood-brain barrier | Neurobiology | Paracellular permeability | Cell Biology | Rho-GTPase | STRESS FIBERS | CELLS | ACTIN | TRANSPORTERS | RHO-GTPASES | NEUROSCIENCES | Stress Fibers - drug effects | Humans | Deoxycytidine - pharmacology | rhoA GTP-Binding Protein - metabolism | Antineoplastic Agents - therapeutic use | Receptors, Adenosine A2 - metabolism | Adherens Junctions - metabolism | Adenosine A2 Receptor Agonists - pharmacology | T-Lymphocytes - drug effects | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Phosphorylation - drug effects | Tight Junctions - drug effects | Tight Junctions - metabolism | Cell Line | Endothelial Cells - metabolism | Jurkat Cells | Mice, Inbred C57BL | Electric Impedance | Down-Regulation - drug effects | Blood-Brain Barrier - metabolism | Cell Movement - drug effects | Focal Adhesions - drug effects | Stress Fibers - metabolism | Animals | Signal Transduction - drug effects | T-Lymphocytes - cytology | Endothelial Cells - cytology | Cell Membrane Permeability - drug effects | Focal Adhesions - metabolism | Glioblastoma - pathology | Adherens Junctions - drug effects | Glioblastoma - drug therapy | Endothelial Cells - enzymology | Deoxycytidine - analogs & derivatives | Endothelial Cells - drug effects | Index Medicus
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 04/2015, Volume 6, Issue 1, pp. 6725 - 6725
Histamine-induced vascular leakage is an integral component of many highly prevalent human diseases, including allergies, asthma and anaphylaxis. Yet, how... 
SMOOTH-MUSCLE | PULMONARY-HYPERTENSION | CELL-JUNCTIONS | MULTIDISCIPLINARY SCIENCES | PROTEIN-COUPLED RECEPTORS | VE-CADHERIN | ADHERENS JUNCTIONS | KINASE INHIBITOR | MYOSIN-II | CANCER | ENDOTHELIAL PERMEABILITY | RNA, Small Interfering - genetics | Skin - metabolism | GTP-Binding Protein alpha Subunits, Gq-G11 - deficiency | Humans | Capillary Permeability - drug effects | Endothelium, Vascular - drug effects | Male | rhoA GTP-Binding Protein - genetics | Adherens Junctions - metabolism | rho-Associated Kinases - antagonists & inhibitors | Receptors, Histamine H1 - metabolism | rho-Associated Kinases - metabolism | Anaphylaxis - chemically induced | Receptors, Histamine H1 - genetics | Female | Skin - pathology | Amides - pharmacology | Phospholipase C beta - metabolism | GTP-Binding Protein alpha Subunits, Gq-G11 - genetics | Histamine - pharmacology | Signal Transduction | Endothelial Cells - metabolism | Anaphylaxis - pathology | Gene Expression Regulation | rho-Associated Kinases - genetics | Adherens Junctions - pathology | Mice, Knockout | Phospholipase C beta - genetics | Anaphylaxis - genetics | Animals | Endothelium, Vascular - metabolism | Adherens Junctions - drug effects | Endothelium, Vascular - pathology | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Anaphylaxis - metabolism | Endothelial Cells - pathology | Skin - drug effects | rhoA GTP-Binding Protein - deficiency | Endothelial Cells - drug effects | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Cellular and Molecular Life Sciences, ISSN 1420-682X, 3/2011, Volume 68, Issue 5, pp. 877 - 892
The apical junctional complex (AJC), composed of tight and adherens junctions, maintains epithelial barrier function. Since cigarette smoking and chronic... 
Life Sciences | Biochemistry, general | Life Sciences, general | Tight junctions | Adherens junctions | Chronic obstructive pulmonary disease | Airway epithelium | Cigarette smoking | Transcriptional regulation | Biomedicine general | Epithelial polarity | Cell Biology | BRONCHIAL EPITHELIUM | BIOCHEMISTRY & MOLECULAR BIOLOGY | MESENCHYMAL TRANSITION | CELL BIOLOGY | LUNG-CANCER | OBSTRUCTIVE PULMONARY-DISEASE | PROTEIN INTERACTIONS | GENE-EXPRESSION | TOBACCO-SMOKE | POLARITY PROTEINS | INTERCELLULAR-JUNCTIONS | Smoking - adverse effects | Transcription, Genetic - drug effects | Epithelial Cells - metabolism | Respiratory Mucosa - drug effects | Oligonucleotide Array Sequence Analysis | Epithelial Cells - drug effects | Humans | Cells, Cultured | Intercellular Junctions - metabolism | Epithelial Cells - ultrastructure | Gene Expression Profiling | Permeability | Pulmonary Disease, Chronic Obstructive - pathology | Down-Regulation - drug effects | Intercellular Junctions - genetics | Signal Transduction - drug effects | Intercellular Junctions - drug effects | Pulmonary Disease, Chronic Obstructive - metabolism | Pulmonary Disease, Chronic Obstructive - genetics | Lung diseases, Obstructive | Anopheles | Analysis | Epithelium | Gene expression | Cigarettes | Smoking | Molecular biology | Respiratory system | Cell adhesion & migration | Indexing in process | Index Medicus | cigarette smoking | epithelial polarity | transcriptional regulation | airway epithelium | tight junctions | adherens junctions | chronic obstructive pulmonary disease
Journal Article
JOURNAL OF CELL SCIENCE, ISSN 0021-9533, 08/2012, Volume 125, Issue 15, pp. 3636 - 3648
Contact inhibition ubiquitously exists in non-transformed cells that are in contact with neighboring cells. This phenomenon explains the poor regenerative... 
Contact inhibition | Kaiso | ORGAN SIZE CONTROL | E-CADHERIN EXPRESSION | HIPPO PATHWAY | CELL-ADHESION | Proliferation | BETA-CATENIN | Human corneal endothelium | P120-CATENIN | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | p120 catenin | MYOFIBROBLAST TRANSFORMATION | MEDIATED TRANSCRIPTIONAL REPRESSION | Endothelial-mesenchymal transformation | RHO-GTPASES | RNA, Small Interfering - genetics | Humans | Middle Aged | Fibroblast Growth Factor 2 - pharmacology | Cell Growth Processes - physiology | Epithelial-Mesenchymal Transition - drug effects | rhoA GTP-Binding Protein - metabolism | Wnt Proteins - metabolism | Contact Inhibition - drug effects | Young Adult | Catenins - metabolism | Cornea - drug effects | Transfection | rho-Associated Kinases - metabolism | Edetic Acid - pharmacology | Aged, 80 and over | Adult | Cornea - cytology | Transforming Growth Factor beta1 - pharmacology | Catenins - physiology | Endothelium, Corneal - cytology | Cell Growth Processes - drug effects | Cells, Cultured | Endothelium, Corneal - drug effects | Endothelium, Corneal - ultrastructure | Cell Adhesion - drug effects | Catenins - genetics | Contact Inhibition - physiology | Transcription Factors - metabolism | Mitosis - drug effects | Mitosis - physiology | Signal Transduction - drug effects | Adherens Junctions - physiology | Cell Adhesion - physiology | Adherens Junctions - drug effects | Adolescent | Aged | RNA, Small Interfering - administration & dosage | Index Medicus | Endothelial–mesenchymal transformation
Journal Article
BMC Biology, ISSN 1741-7007, 08/2010, Volume 8, Issue 1, pp. 11 - 11
Journal Article
Developmental Biology, ISSN 0012-1606, 01/2013, Volume 373, Issue 2, pp. 267 - 280
During lung development, proper epithelial cell arrangements are critical for the formation of an arborized network of tubes. Each tube requires a lumen, the... 
Scribble | Branching morphogenesis | Lumen | Polarity | PCP | Lung | LOCALIZATION | E6 PROTEINS | NEURAL-TUBE DEFECTS | DEVELOPMENTAL BIOLOGY | E-CADHERIN | BETA-CATENIN | MOUSE MUTANT | VANGL2 | DISRUPTION | BRANCHING-MORPHOGENESIS | PLANAR POLARITY | Epithelial Cells - metabolism | Receptors, G-Protein-Coupled - metabolism | Epithelial Cells - drug effects | Zonula Occludens-2 Protein - metabolism | rhoA GTP-Binding Protein - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Protein Transport - drug effects | Lung - cytology | Adherens Junctions - metabolism | Gene Knockdown Techniques | Epithelium - embryology | Protein Binding - drug effects | Mammals - embryology | Cell Membrane - metabolism | Cell Polarity - drug effects | Lung - metabolism | Epithelial Cells - cytology | Imaging, Three-Dimensional | Cell Membrane - drug effects | Tight Junctions - drug effects | Tight Junctions - metabolism | Epithelium - drug effects | Epithelium - metabolism | beta Catenin - metabolism | Nerve Tissue Proteins - metabolism | Animals | Models, Biological | Lung - drug effects | Adherens Junctions - drug effects | Cell Communication - drug effects | Morpholinos - pharmacology | Cytoskeleton - metabolism | Morphogenesis - drug effects | Lung - embryology | Mice | Cytoskeleton - drug effects | Animal genetics | Proteins | Developmental biology | Index Medicus | From the Society for Developmental Biology
Journal Article
Circulation Research, ISSN 0009-7330, 08/2012, Volume 111, Issue 6, pp. 739 - 749
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 04/2018, Volume 314, Issue 4, pp. H805 - H811
Endothelium-dependent, nitric oxide-mediated dilatation is impaired in aging arteries. The dysfunction reflects increased production of reactive oxygen species... 
Vascular-endothelial cadherin | Aging | Adherens junctions | Nitric oxide | Endothelium | endothelium | nitric oxide | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | PERMEABILITY | VASCULAR DYSFUNCTION | RELAXING FACTOR | INVOLVEMENT | adherens junctions | vascular-endothelial cadherin | DILATOR DYSFUNCTION | ADHESION | ENHANCEMENT | RESISTANCE | PERIPHERAL VASCULAR DISEASE | aging | Phosphorylation | Reactive Oxygen Species - metabolism | Age Factors | Cadherins - metabolism | Rats, Inbred F344 | Endothelium, Vascular - drug effects | Spin Labels | Metalloporphyrins - pharmacology | Adherens Junctions - metabolism | Antigens, CD - metabolism | Cyclic N-Oxides - pharmacology | Superoxides - metabolism | Tail - blood supply | src-Family Kinases - metabolism | Superoxides - antagonists & inhibitors | Vasodilator Agents - pharmacology | Endothelial Cells - metabolism | Antioxidants - pharmacology | Animals | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Adherens Junctions - drug effects | Vasodilation - drug effects | Endothelial Cells - drug effects | Physiological aspects | Vascular endothelium | Physiological research | Superoxide | Research | Arteries | Reactive oxygen species | Blocking antibodies | Tempol | Superoxide dismutase | Inactivation | Proteins | Lysates | Inhibition | Stretching | Deactivation | Aging (artificial) | Src protein | Clustering | Cadherin | Chemical compounds | Studies | Acetylcholine | Immunofluorescence | Veins & arteries | Manganese | Index Medicus | Rapid Report
Journal Article