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by Shungin, Dmitry and Winkler, Thomas W and Croteau-Chonka, Damien C and Ferreira, Teresa and Locke, Adam E and Mägi, Reedik and Strawbridge, Rona J and Pers, Tune H and Fischer, Krista and Justice, Anne E and Workalemahu, Tsegaselassie and Wu, Joseph M. W and Buchkovich, Martin L and Heard-Costa, Nancy L and Roman, Tamara S and Drong, Alexander W and Song, Ci and Gustafsson, Stefan and Day, Felix R and Esko, Tonu and Fall, Tove and Kutalik, Zoltán and Luan, Jian’an and Randall, Joshua C and Scherag, André and Vedantam, Sailaja and Wood, Andrew R and Chen, Jin and Fehrmann, Rudolf and Karjalainen, Juha and Kahali, Bratati and Liu, Ching-Ti and Schmidt, Ellen M and Absher, Devin and Amin, Najaf and Anderson, Denise and Beekman, Marian and Bragg-Gresham, Jennifer L and Buyske, Steven and Demirkan, Ayse and Ehret, Georg B and Feitosa, Mary F and Goel, Anuj and Jackson, Anne U and Johnson, Toby and Kleber, Marcus E and Kristiansson, Kati and Mangino, Massimo and Mateo Leach, Irene and Medina-Gomez, Carolina and Palmer, Cameron D and Pasko, Dorota and Pechlivanis, Sonali and Peters, Marjolein J and Prokopenko, Inga and Stančáková, Alena and Ju Sung, Yun and Tanaka, Toshiko and Teumer, Alexander and Van Vliet-Ostaptchouk, Jana V and Yengo, Loïc and Zhang, Weihua and Albrecht, Eva and Ärnlöv, Johan and Arscott, Gillian M and Bandinelli, Stefania and Barrett, Amy and Bellis, Claire and Bennett, Amanda J and Berne, Christian and Blüher, Matthias and Böhringer, Stefan and Bonnet, Fabrice and Böttcher, Yvonne and Bruinenberg, Marcel and Carba, Delia B and Caspersen, Ida H and Clarke, Robert and Warwick Daw, E and Deelen, Joris and Deelman, Ewa and Delgado, Graciela and Doney, Alex S. F and Eklund, Niina and Erdos, Michael R and Estrada, Karol and Eury, Elodie and Friedrich, Nele and Garcia, Melissa E and Giedraitis, Vilmantas and Gigante, Bruna and Go, Alan S and Golay, Alain and Grallert, Harald and Grammer, Tanja B and Gräßler, Jürgen and Grewal, Jagvir and Groves, Christopher J and Haller, Toomas and Hallmans, Goran and ... and The PAGE Consortium and The International Endogene Consortium and The MAGIC Investigators and The ReproGen Consortium and The CKDGen Consortium and The ADIPOGen Consortium and The ICBP and The CARDIOGRAMplusC4D Consortium and The GENIE Consortium and The GLGC and The GEFOS Consortium and The MuTHER Consortium and The LifeLines Cohort Study and PAGE Consortium and LifeLines Cohort Study and CARDIOGRAMplusC4D Consortium and MuTHER Consortium and ICBP and ADIPOGEN Consortium and CKDGen Consortium and GEFOS Consortium and Int Endogene Consortium and GENIE Consortium and MAGIC Investigators and ReproGen Consortium and GLGC and ADIPOGen Consortium and International Endogene Consortium and Göteborgs universitet and Gothenburg University and Institutionen för medicin, avdelningen för invärtesmedicin och klinisk nutrition and Sahlgrenska Academy and Centre for Bone and Arthritis Research and Sahlgrenska akademin and Institute of Medicine, Department of Internal Medicine and Clinical Nutrition
Nature (London), ISSN 1476-4687, 2015, Volume 518, Issue 7538, pp. 187 - 196
Body fat distribution is a heritable trait and a well-established predictor of adverse metabolic outcomes, independent of overall adiposity. To increase our... 
ABDOMINAL ADIPOSITY | METAANALYSIS | VARIANTS | MULTIDISCIPLINARY SCIENCES | COMMON SNPS | GLYCEMIC TRAITS | RISK | FALSE DISCOVERY | GENOME-WIDE ASSOCIATION | ADIPOGENIC DIFFERENTIATION | SEXUAL-DIMORPHISM | Body Mass Index | Genome-Wide Association Study | Age Factors | Neovascularization, Physiologic - genetics | Epigenesis, Genetic | Humans | Male | Continental Population Groups - genetics | Sex Characteristics | Obesity - genetics | Europe - ethnology | Genome, Human - genetics | Adipose Tissue - metabolism | Insulin - metabolism | Models, Biological | Adipocytes - metabolism | Insulin Resistance - genetics | Polymorphism, Single Nucleotide - genetics | Female | Body Fat Distribution | Transcription, Genetic - genetics | Adipogenesis - genetics | Waist-Hip Ratio | Quantitative Trait Loci - genetics | Adipose tissues | Quantitative trait loci | Genetic research | Genetic aspects | Research | Metabolism | Health aspects | Studies | Body mass index | Genealogy | Body fat | Insulin resistance | Genetics | Genomes | Abdomen | Meta-analysis | Life Sciences | Adipocytes/metabolism Adipogenesis/genetics Adipose Tissue/metabolism Age Factors Body Fat Distribution Body Mass Index Continental Population Groups/genetics Epigenesis, Genetic Europe/ethnology Female Genome, Human/genetics Genome-Wide Association Study Humans Insulin/metabolism Insulin Resistance/genetics Male Models, Biological Neovascularization, Physiologic/genetics Obesity/genetics Polymorphism, Single Nucleotide/genetics Quantitative Trait Loci/genetics Sex Characteristics Transcription, Genetic/genetics Waist-Hip Ratio | Folkhälsovetenskap, global hälsa, socialmedicin och epidemiologi | Endokrinologi och diabetes | Public Health, Global Health, Social Medicine and Epidemiology | Endocrinology and Diabetes
Journal Article
by Li, A and Hu, Y and Liu, X and Zhao, L and Tian, Q and Du, M
Journal of Animal Science, ISSN 0021-8812, 12/2018, Volume 96, Issue suppl_3, pp. 245 - 245
Abstract Long non-coding RNAs (lncRNAs) have been revealed to play key role in the adipogenesis. Recently, a number of lncRNAs have been identified in adipose... 
cattle | lncRNA | adipogenesis | Abstracts
Journal Article
by Jo, Y and Peng, D and Kim, W and Lee, J and Lee, H
Journal of Animal Science, ISSN 0021-8812, 12/2018, Volume 96, Issue suppl_3, pp. 448 - 448
Abstract The objective of this study was to determine the effects of vitamin A (VA) as a supplementation to the diet of cows in late pregnancy on tissue... 
vitamin A | adipogenesis | calf | Abstracts
Journal Article
Journal of Animal Science, ISSN 0021-8812, 12/2018, Volume 96, Issue suppl_3, pp. 348 - 349
Abstract It is unknown how dairy heifer skeletal muscle responds to increased pre-weaning ME intake. Therefore, the objective was to determine how elevated ME... 
Muscle | Abstracts | Transcriptome | Adipogenesis
Journal Article
Journal Article
Journal Article
Endocrinology (Philadelphia), ISSN 1945-7170, 2017, Volume 158, Issue 10, pp. 3109 - 3125
Developmental exposure to environmental factors has been linked to obesity risk later in life. Nuclear receptors are molecular sensors that play critical roles... 
ENDOCRINE-DISRUPTING CHEMICALS | OBESITY | DNA METHYLATION | PRENATAL EXPOSURE | ENDOCRINOLOGY & METABOLISM | FAT | DIFFERENTIATION | OBESOGEN TRIBUTYLTIN | ADIPOGENESIS | ORGANOTIN COMPOUNDS | ADIPOCYTE | Trialkyltin Compounds - pharmacology | Enhancer of Zeste Homolog 2 Protein - genetics | Gene Expression - drug effects | Adipogenesis - drug effects | Mice, Inbred C57BL | Adipocytes - cytology | Retinoid X Receptors - physiology | Sequence Analysis, RNA - veterinary | Adipogenesis - physiology | Cell Differentiation - genetics | PPAR gamma - physiology | Animals | Endocrine Disruptors - pharmacology | Cell Differentiation - drug effects | Retinoid X Receptors - drug effects | Mesenchymal Stem Cells - cytology | Chromatin - physiology | Gene Knockdown Techniques - veterinary | Obesity - etiology | Epigenesis, Genetic - drug effects | Mice | Adipogenesis - genetics | Chromatin - drug effects | Endocrine disruptors | Chromatin | Adipose tissue | Mesenchyme | Stem cell transplantation | Homology | Activation | Genomes | Receptors | Biomedical materials | Allografts | Cell fate | Biocompatibility | Tributyltin | Health risks | Exposure | Environmental factors | Retinoid X receptors | Gene expression | Nuclear receptors | Chemicals | Lysine | Skin & tissue grafts | Stem cells | Rosiglitazone | Receptor mechanisms | Differentiation | Methylation | Antifouling substances
Journal Article