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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2013, Volume 110, Issue 9, pp. 3223 - 3224
Expression of bone morphogenetic protein 4 (BMP4) in adipocytes of white adipose tissue (WAT) produces “white adipocytes” with characteristics of brown fat and... 
PNAS PLUS: AUTHOR SUMMARIES | Brown adipose tissue | Mitochondria | Metabolism | Adipocyte terminal differentiation | Adipocytes, Brown - metabolism | Bone Morphogenetic Protein 4 - genetics | Humans | Adipose Tissue, White - metabolism | Adipocytes, White - enzymology | Fatty Acid-Binding Proteins - metabolism | Bone Morphogenetic Protein 4 - metabolism | Mitochondria - ultrastructure | Thinness - metabolism | Thinness - pathology | Adipocytes, White - drug effects | Adipose Tissue, White - ultrastructure | Diet, High-Fat | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Adipose Tissue, Brown - ultrastructure | p38 Mitogen-Activated Protein Kinases - metabolism | Homeostasis - drug effects | Adipocytes, Brown - pathology | Insulin - pharmacology | Adipocytes, Brown - drug effects | Mice, Inbred C57BL | Mice, Transgenic | Mitochondria - metabolism | 3T3-L1 Cells | Activating Transcription Factor 2 - metabolism | Adipose Tissue, Brown - pathology | Gene Expression Regulation - drug effects | Organ Size - drug effects | Phenotype | Adipocytes, White - pathology | Animals | Oxygen Consumption - drug effects | Adipose Tissue, White - enzymology | Glucose - metabolism | Trans-Activators - metabolism | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Mice | Transcription Factors | Adipose Tissue, White - drug effects | Energy Metabolism - drug effects | OBESITY | MULTIDISCIPLINARY SCIENCES | Biological Sciences | metabolism | PNAS Plus | adipocyte terminal differentiation | brown adipose tissue | mitochondria
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2011, Volume 301, Issue 4, pp. 1425 - 1437
Thoracic perivascular adipose tissue (PVAT) is a unique adipose depot that likely influences vascular function and susceptibility to pathogenesis in obesity... 
Obesity | Lipid droplet proteins | Diabetes | Macrophages | Microarray | METABOLIC SYNDROME | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | RISK-FACTORS | LINKING OBESITY | PORCINE CORONARY-ARTERIES | microarray | macrophages | lipid droplet proteins | INSULIN-RESISTANCE | CARDIOVASCULAR-DISEASE | GENE-EXPRESSION | FAT | PPAR-GAMMA | PERIPHERAL VASCULAR DISEASE | diabetes | obesity | Immunohistochemistry | Inflammation - chemically induced | Adipose Tissue - physiology | Diet - adverse effects | Blood Vessels - ultrastructure | Male | Insulin - blood | Neutrophil Infiltration - physiology | Flow Cytometry | Microarray Analysis | Adipose Tissue, Brown - ultrastructure | Apoptosis Regulatory Proteins - biosynthesis | Microscopy, Electron, Transmission | Dietary Fats - adverse effects | Ion Channels - biosynthesis | Mice, Inbred C57BL | Adipose Tissue, Brown - physiology | Aorta, Thoracic - metabolism | Mitochondrial Proteins - biosynthesis | Reverse Transcriptase Polymerase Chain Reaction | Animals | Mitochondria, Heart - physiology | Adipose Tissue - ultrastructure | Inflammation - prevention & control | Mice | Uncoupling Protein 1 | Blood Vessels - physiology | Adipose tissues | Usage | DNA microarrays | Physiological aspects | Inflammation | Genetic aspects | Research | Gene expression | Energetics and Metabolism
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 3/2012, Volume 21, Issue 5, pp. 1124 - 1137
There is substantial evidence that impairment of peroxisome proliferator-activated receptor (PPAR)-γ-coactivator 1α (PGC-1α) levels and activity play an... 
RESPIRATORY-CHAIN | OXIDATIVE STRESS | COACTIVATOR | RECEPTOR-GAMMA | PGC-1-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | MUTANT HUNTINGTIN | GENETICS & HEREDITY | NULL MICE | NEURODEGENERATION | DYSFUNCTION | ADIPOSE-TISSUE | Neurons - pathology | Vacuoles - ultrastructure | Oxidative Stress | Huntington Disease - pathology | Transcriptional Activation | Bezafibrate - pharmacology | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | PPAR gamma - metabolism | Mitochondria - ultrastructure | Gliosis - pathology | Neuroprotective Agents - pharmacology | Muscle Fibers, Skeletal - ultrastructure | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Adipose Tissue, Brown - ultrastructure | Huntington Disease - drug therapy | Neuroprotective Agents - administration & dosage | Corpus Striatum - pathology | Disease Models, Animal | Mice, Transgenic | Survival Rate | Mitochondria - metabolism | Signal Transduction - genetics | Mitochondria - drug effects | Huntington Disease - metabolism | Bezafibrate - administration & dosage | Phenotype | Animals | Diet | Signal Transduction - drug effects | PPAR gamma - agonists | Huntington Disease - genetics | Mitochondria, Muscle - ultrastructure | Trans-Activators - metabolism | Adipose Tissue, Brown - drug effects | Mice | Mitochondria, Muscle - drug effects | Transcription Factors | Neuroprotection | Animal models | Transgenic mice | Muscles | Lipids | Huntington's disease | Atrophy | Diets | Mitochondria | Gliosis | Bezafibrate | Neostriatum | Glycolysis | Peroxisome proliferator-activated receptors | Adipose tissue (brown) | PGC-1 protein
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 08/2008, Volume 118, Issue 8, pp. 2808 - 2821
Journal Article
Particle and Fibre Toxicology, ISSN 1743-8977, 08/2013, Volume 10, Issue 1, pp. 43 - 43
Background: Inflammation and oxidative stress play critical roles in the pathogenesis of inhaled air pollutant-mediated metabolic disease. Inflammation in the... 
Oxidative stress | Ozone | Inflammation | Particulate matter | Epicardial adipose tissue | Perirenal adipose tissue | ATHEROSCLEROSIS | DIFFERENTIAL REGULATION | INSULIN-RESISTANCE | UNCOUPLING PROTEIN-1 | BROWN ADIPOCYTE | GENE-EXPRESSION | NITRIC-OXIDE | FAT | TOXICOLOGY | MEDIATORS | CORONARY-ARTERY-DISEASE | Particulate Matter - toxicity | Dietary Carbohydrates | Adipose Tissue, White - metabolism | Male | RNA, Messenger - metabolism | Kidney | Panniculitis - genetics | Time Factors | Adipokines - genetics | Adipose Tissue, White - ultrastructure | Adipose Tissue, Brown - ultrastructure | Inflammation Mediators - metabolism | Inhalation Exposure - adverse effects | Risk Assessment | Gene Expression Regulation | Ozone - toxicity | Adipokines - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Rats, Sprague-Dawley | Fructose | Pericardium | Panniculitis - chemically induced | Macrophages - metabolism | Animals | Macrophages - drug effects | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Oxidative Stress - drug effects | Panniculitis - pathology | Adipose Tissue, White - drug effects | Panniculitis - metabolism | Nitric Oxide Synthase Type II - metabolism | Adipose tissues | United States | Nitric oxide | Leptin | Air pollution | Cardiovascular diseases | Particles | Diseases | Outdoor air quality | Medical research | Rodents | Colleges & universities | Cardiovascular disease | Insulin
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2007, Volume 104, Issue 7, pp. 2366 - 2371
Journal Article
Endocrinology, ISSN 0013-7227, 01/2018, Volume 159, Issue 1, pp. 323 - 340
It is unknown how the lack of insulin receptor (IR)/insulinlike growth factor I receptor (IGFIR) in a tissue-specific manner affects brown fat development and... 
IGF-1 RECEPTORS | ENERGY-EXPENDITURE | ADIPOCYTES | PROTEIN | HORMONE | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | DYNAMICS | ABLATION | STRESS | ADIPOSE-TISSUE | Mitochondrial Dynamics | Metabolic Syndrome - etiology | Receptor, IGF Type 1 - metabolism | Adipose Tissue, Beige - pathology | Diet, High-Fat - adverse effects | Male | Metabolic Syndrome - metabolism | Mitochondria - ultrastructure | Atrophy | Adiposity | Hyperinsulinism - etiology | Receptor, Insulin - genetics | Adipose Tissue, Brown - ultrastructure | Metabolic Syndrome - physiopathology | Obesity - etiology | Weight Gain | Microscopy, Electron, Transmission | Adipose Tissue, Beige - ultrastructure | Mice, Inbred C57BL | Insulin Resistance | Obesity - physiopathology | Mitochondria - metabolism | Mitochondria - pathology | Organ Specificity | Receptor, IGF Type 1 - genetics | Adipose Tissue, Brown - pathology | Mice, Knockout | Obesity - metabolism | Obesity - pathology | Animals | Thermogenesis | Hypertriglyceridemia - etiology | Receptor, Insulin - metabolism | Adipose Tissue, Brown - metabolism | Metabolic Syndrome - pathology | Adipose Tissue, Beige - metabolism | Hyperinsulinemia | Adipose tissue | Transcription | Body fat | Body weight | Fission | High fat diet | Proteins | Receptors | Mitochondria | Uncoupling protein 1 | Insulin-like growth factor I receptors | Adipose tissue (brown) | Growth factors | Failure | Obesity | Metabolism | Insulin | Hypertriglyceridemia | Quality control | Cristae | Insulin resistance | Disruption | Metabolic disorders
Journal Article
Molecular Nutrition & Food Research, ISSN 1613-4125, 11/2017, Volume 61, Issue 11, pp. 1700261 - n/a