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Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2011, Volume 31, Issue 29, pp. 10506 - 10515
The mechanisms subserving the ability of glucocorticoid signaling within the medial prefrontal cortex (mPFC) to terminate stress-induced activation of the... 
INHIBITION | STRIA TERMINALIS | RAT BASOLATERAL AMYGDALA | PYRAMIDAL CELLS | CORTEX | PITUITARY-ADRENOCORTICAL AXIS | ADRENAL AXIS | HPA AXIS | NEGATIVE FEEDBACK | NEUROSCIENCES | BED NUCLEUS | Long-Term Synaptic Depression - genetics | Corticosterone - pharmacology | gamma-Aminobutyric Acid - metabolism | Male | Long-Term Synaptic Depression - drug effects | Freezing Reaction, Cataleptic - drug effects | Patch-Clamp Techniques - methods | Arachidonic Acids - metabolism | Prefrontal Cortex - drug effects | Piperidines - pharmacology | Pyramidal Cells - physiology | Stress, Psychological - metabolism | Pyramidal Cells - drug effects | Electric Stimulation - methods | Endocannabinoids | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Disease Models, Animal | Pyrazoles - pharmacology | Microscopy, Electron, Transmission | Stress, Psychological - drug therapy | Mice, Inbred C57BL | Rats | Glycerides - metabolism | Prefrontal Cortex - cytology | Rats, Sprague-Dawley | Mice, Inbred ICR | Hormone Antagonists - pharmacology | Mice, Knockout | Stress, Psychological - pathology | Animals | Signal Transduction - drug effects | Prefrontal Cortex - metabolism | Freezing Reaction, Cataleptic - physiology | Signal Transduction - physiology | Mice | Receptor, Cannabinoid, CB1 - deficiency | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | In Vitro Techniques | Mifepristone - pharmacology
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 07/2010, Volume 114, Issue 1, pp. 259 - 270
J. Neurochem. (2010) 114, 259–270. Acute administration of atomoxetine and methylphenidate, attention‐deficit/hyperactivity disorder (ADHD) drugs, activates... 
methylphenidate | striatum | atomoxetine | dopamine | mice | prefrontal cortex | Striatum | Mice | Dopamine | Prefrontal cortex | Atomoxetine | Methylphenidate | SUBSTANCE-ABUSE | DEFICIT HYPERACTIVITY DISORDER | FIRING PATTERN | BIOCHEMISTRY & MOLECULAR BIOLOGY | FRONTAL-CORTEX | REPEATED METHAMPHETAMINE | NEUROSCIENCES | VENTRAL TEGMENTAL AREA | NUCLEUS-ACCUMBENS | ATTENTION-DEFICIT/HYPERACTIVITY DISORDER | WORKING-MEMORY PERFORMANCE | FREELY-MOVING RATS | Motor Activity - drug effects | Male | Corpus Striatum - metabolism | Extracellular Space - metabolism | Prefrontal Cortex - drug effects | Atomoxetine Hydrochloride | Neurons - metabolism | Dopamine - metabolism | Methylphenidate - administration & dosage | Dopamine Uptake Inhibitors - pharmacology | Proto-Oncogene Proteins c-fos - metabolism | Methylphenidate - pharmacology | Mice, Inbred ICR | Dopamine Uptake Inhibitors - administration & dosage | Animals | Microdialysis | Norepinephrine - metabolism | Propylamines - administration & dosage | Serotonin - metabolism | Prefrontal Cortex - metabolism | Adrenergic Uptake Inhibitors - administration & dosage | Corpus Striatum - drug effects | Adrenergic Uptake Inhibitors - pharmacology | Propylamines - pharmacology | Biogenic Monoamines - metabolism | Child development | Serotonin | Neurons | Analysis | Phenols | Universities and colleges | Methylphenidate hydrochloride | Noradrenaline | Brain | Adrenal glands | Rodents | Cells
Journal Article
Journal Article
Journal of Pain, ISSN 1526-5900, 2008, Volume 9, Issue 1, pp. 28 - 36
Abstract Hypogonadotrophic hypogonadism is characteristically induced in men by intrathecal, transdermal, or sustained-action opioids. Although women receiving... 
Anesthesia & Perioperative Care | Pain Medicine | DHEAS | gonadotrophins | testosterone | pain/drug therapy | amenorrhea | Hypogonadism | opioids | estradiol | CORTICOTROPIN-RELEASING HORMONE | MENSTRUAL-CYCLE | DEHYDROEPIANDROSTERONE-SULFATE | GONADOTROPIN-SECRETION | SEXUAL FUNCTION | NEUROSCIENCES | CLINICAL NEUROLOGY | CIGARETTE-SMOKING | hypogonadism | ANDROGEN | FREE TESTOSTERONE | METHADONE-MAINTENANCE | POSTMENOPAUSAL WOMEN | Humans | Middle Aged | Administration, Cutaneous | Hypothalamo-Hypophyseal System - secretion | Adrenal Cortex - secretion | Analgesics, Opioid - adverse effects | Gonadotropins - metabolism | Hypogonadism - metabolism | Pain - drug therapy | Adult | Female | Hypogonadism - physiopathology | Ovary - drug effects | Adrenal Cortex - drug effects | Gonadal Steroid Hormones - secretion | Estradiol - secretion | Hypothalamo-Hypophyseal System - physiopathology | Adrenal Cortex Diseases - chemically induced | Estradiol - metabolism | Hypothalamo-Hypophyseal System - drug effects | Gonadal Steroid Hormones - metabolism | Androgens - secretion | Administration, Oral | Adrenal Cortex Diseases - physiopathology | Gonadotropins - secretion | Down-Regulation - drug effects | Adrenal Cortex Diseases - metabolism | Down-Regulation - physiology | Adrenal Cortex - physiopathology | Hypogonadism - chemically induced | Delayed-Action Preparations - adverse effects | Analgesics, Opioid - administration & dosage | Ovary - secretion | Aged | Androgens - metabolism | Ovary - physiopathology | Women | Testosterone | Care and treatment | Dehydroepiandrosterone | Pain | Opioids
Journal Article
Progress in Neuropsychopharmacology & Biological Psychiatry, ISSN 0278-5846, 10/2012, Volume 39, Issue 1, pp. 112 - 119
Stress and the resulting increase in glucocorticoid levels have been implicated in the pathophysiology of depressive disorders. We investigated the effects of... 
Depression | Restraint stress | Anxiety | Prefrontal cortex | Brain-derived neurotrophic factor (BDNF) | Glucocorticoid receptor | ACTIVATION | MESSENGER-RNA EXPRESSION | BDNF | ABNORMALITIES | TIANEPTINE | PSYCHIATRY | CHRONIC MILD STRESS | ADRENAL AXIS | NEUROSCIENCES | CLINICAL NEUROLOGY | HIPPOCAMPUS | POTENTIATION | CORTICOSTERONE LEVELS | PHARMACOLOGY & PHARMACY | Depression - physiopathology | Rats, Wistar | Prefrontal Cortex - physiopathology | Male | Receptors, Glucocorticoid - metabolism | Receptors, Nerve Growth Factor - metabolism | Brain-Derived Neurotrophic Factor - pharmacology | Depression - metabolism | Prefrontal Cortex - drug effects | Restraint, Physical - methods | Stress, Psychological - metabolism | Anxiety - complications | Depression - complications | Brain-Derived Neurotrophic Factor - metabolism | Anxiety - metabolism | Disease Models, Animal | Restraint, Physical - psychology | Stress, Psychological - blood | Anxiety - blood | Down-Regulation | Restraint, Physical - physiology | Glucocorticoids - blood | Anxiety - physiopathology | Rats | Behavior, Animal - physiology | Animals | Prefrontal Cortex - metabolism | Glutamic Acid - metabolism | Receptor, trkB - metabolism | Stress, Psychological - complications | Depression - blood | Stress, Psychological - physiopathology | Corticosteroids | Analysis | Depression, Mental | Liquid chromatography | Stress (Psychology) | Glutamate | Steroids | Cortex (prefrontal) | Glutamic acid | Brain-derived neurotrophic factor | Glucocorticoids | Sucrose | Food intake | Glucocorticoid receptors | High-performance liquid chromatography | Body weight gain | Stress | Western blotting
Journal Article