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by Zheng, Siyuan and Cherniack, Andrew D and Cherniack, Andrew D and Dewal, Ninad and Moffitt, Richard A and Moffitt, Richard A and Danilova, Ludmila and Murray, Bradley A and Murray, Bradley A and Lerario, Antonio M and Lerario, Antonio M and Else, Tobias and Knijnenburg, Theo A and Knijnenburg, Theo A and Ciriello, Giovanni and Kim, Hoon and Kim, Jaegil and Kim, Seungchan and Assie, Guillaume and Morozova, Olena and Akbani, Rehan and Shih, Juliann and Hoadley, Katherine A and Hoadley, Katherine A and Choueiri, Toni K and Choueiri, Toni K and Waldmann, Jens and Mete, Ozgur and Robertson, A. Gordon and Robertson, A. Gordon and Wu, Hsin-Ta and Wu, Ye and Wu, Junyuan and Wu, Hsin-Tu and Raphael, Benjamin J and Raphael, Benjamin J and Shao, Lina and Meyerson, Matthew and Demeure, Michael J and Demeure, Michael J and Beuschlein, Felix and Gill, Anthony J and Gill, Anthony J and Sidhu, Stan B and Sidhu, Stan B and Almeida, Madson Q and Almeida, Madson and Fragoso, Maria C.B.V and Cope, Leslie M and Cope, Leslie M and Kebebew, Electron and Habra, Mouhammed Amir and Habra, Mouhammed A and Whitsett, Timothy G and Whitsett, Timothy G and Bussey, Kimberly J and Bussey, Kimberly J and Rainey, William E and Rainey, William E and Asa, Sylvia L and Asa, Sylvia L and Bertherat, Jérôme and Fassnacht, Martin and Wheeler, David A and Wheeler, David A and Verhaak, Roel G.W and Verhaak, Roel G.W and Giordano, Thomas J and Giordano, Thomas J and Hammer, Gary D and Hammer, Gary D and Assié, Guillaume and Barisson Fragoso, Maria Candida and Benz, Christopher and Ally, Adrian and Balasundaram, Miruna and Bowlby, Reanne and Brooks, Denise and Butterfield, Yaron S.N and Carlsen, Rebecca and Dhalla, Noreen and Guin, Ranabir and Holt, Robert A and Jones, Corbin D and Jones, Steven J.M and Kasaian, Katayoon and Lee, Sandy and Lee, Darlene and Li, Haiyan I and Lim, Lynette and Ma, Yussanne and Marra, Marco A and Mayo, Michael and Moore, Richard A and Mungall, Karen and Mungall, Andrew J and Sadeghi, Sara and Schein, Jacqueline E and Sipahimalani, Payal and Tam, Angela and ... and Cancer Genome Atlas Research Network
Cancer Cell, ISSN 1535-6108, 05/2016, Volume 29, Issue 5, pp. 723 - 736
Journal Article
Annals of Oncology, ISSN 0923-7534, 01/2015, Volume 26, Issue 10, pp. 2119 - 2125
Journal Article
Science, ISSN 0036-8075, 2/2011, Volume 331, Issue 6018, pp. 768 - 772
Endocrine tumors such as aldosterone-producing adrenal adenomas (APAs), a cause of severe hypertension, feature constitutive hormone production and... 
Depolarization | Hypertension | Somatic mutation | Cell growth | Hyperaldosteronism | REPORTS | Loss of heterozygosity | Adenoma | Genetic mutation | Sequencing | Tumors | REMEDIABLE ALDOSTERONISM | HUMAN BREAST | COLORECTAL CANCERS | ADRENOCORTICAL-CELLS | MULTIDISCIPLINARY SCIENCES | CONDUCTION | GLOMERULOSA CELLS | IDENTIFICATION | MOLECULAR-BASIS | SELECTIVITY | POTASSIUM CHANNEL | Adrenal Glands - pathology | Potassium - metabolism | Cell Proliferation | Humans | Hyperaldosteronism - pathology | Hyperplasia | Protein Multimerization | Zona Glomerulosa - pathology | Male | Hyperaldosteronism - metabolism | Sodium - metabolism | Aldosterone - metabolism | Adrenocortical Adenoma - genetics | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Female | Hypertension - genetics | Adrenal Cortex Neoplasms - metabolism | Cell Line | G Protein-Coupled Inwardly-Rectifying Potassium Channels - chemistry | Hyperaldosteronism - genetics | Mutant Proteins - genetics | Mutant Proteins - metabolism | Zona Glomerulosa - metabolism | Adrenal Cortex Neoplasms - pathology | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | Adrenocortical Adenoma - pathology | Adrenocortical Adenoma - metabolism | Hypertension - metabolism | Adrenal Cortex Neoplasms - genetics | Mutant Proteins - chemistry | Mutation | Index Medicus | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 12/2011, Volume 96, Issue 12, pp. 3775 - 3784
Journal Article
European Journal of Endocrinology, ISSN 0804-4643, 12/2013, Volume 169, Issue 6, pp. 891 - 899
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 04/2012, Volume 97, Issue 4, pp. 1311 - 1319
Context: Mutations of the KCNJ5 gene have recently been identified in patients with aldosterone-producing adenomas (APA). Objective: Our objective was to... 
DIAGNOSIS | PLASMA | HYPERALDOSTERONISM | ENDOCRINOLOGY & METABOLISM | METANEPHRINES | PREVALENCE | HYPERTENSIVE PATIENTS | CORTISOL | Humans | Middle Aged | Molecular Sequence Data | Male | Adrenal Cortex Neoplasms - physiopathology | Cushing Syndrome - metabolism | Neoplasm Proteins - metabolism | RNA, Messenger - metabolism | Hyperaldosteronism - etiology | Base Sequence | Adrenocortical Adenoma - genetics | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Adult | Female | Retrospective Studies | Neoplasm Proteins - genetics | Adrenal Cortex Neoplasms - metabolism | Pheochromocytoma - metabolism | Cross-Sectional Studies | Genetic Association Studies | Cushing Syndrome - genetics | Japan | G Protein-Coupled Inwardly-Rectifying Potassium Channels - chemistry | Neoplasm Proteins - chemistry | Adrenal Cortex Neoplasms - pathology | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | Adrenocortical Adenoma - pathology | Adrenocortical Adenoma - metabolism | Adrenal Cortex Neoplasms - genetics | Adrenal Gland Neoplasms - metabolism | Pheochromocytoma - genetics | Aged | Adrenocortical Adenoma - physiopathology | Mutation | Adrenal Gland Neoplasms - genetics | Immunohistochemistry | Hospitals | Pheochromocytoma | Cell membranes | Cushing's syndrome | mRNA | Adenoma | Gene expression | Tumors | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Hypertension, ISSN 0194-911X, 11/2010, Volume 56, Issue 5, pp. 885 - 892
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 06/2017, Volume 102, Issue 6, pp. 1792 - 1796
Context: Whether primary aldosteronism (PA) is the consequence of a monoclonal or multiclonal process is unclear. Case Description: A 48-year-old man with... 
ADENOMA | COMMON | SOMATIC MUTATIONS | ENDOCRINOLOGY & METABOLISM | Immunohistochemistry | Neoplasms, Multiple Primary - surgery | Neoplasms, Multiple Primary - metabolism | Humans | Hyperaldosteronism - pathology | Middle Aged | Zona Glomerulosa - pathology | Male | Hyperaldosteronism - metabolism | Zona Reticularis - pathology | Adrenocortical Adenoma - genetics | Zona Fasciculata - pathology | Sulfotransferases - metabolism | Adrenal Cortex Neoplasms - metabolism | Cytochrome P-450 CYP11B2 - metabolism | Adrenocortical Adenoma - surgery | Neoplasms, Multiple Primary - genetics | Adrenal Cortex Neoplasms - surgery | Hyperaldosteronism - genetics | Adrenal Cortex Neoplasms - pathology | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | Neoplasms, Multiple Primary - pathology | Adrenocortical Adenoma - pathology | Adrenocortical Adenoma - metabolism | Adrenal Cortex Neoplasms - genetics | Calcium Channels, L-Type - genetics | Hyperaldosteronism - surgery | Steroid 17-alpha-Hydroxylase - metabolism | Adrenal glands | Cortex | Aldosterone synthase | Histology | Adenoma | Hydrocortisone | Aldosterone | Pigmentation | Nodules | Adrenalectomy | Heterogeneity | Histopathology | Computed tomography | Genetic analysis | Genetics | Steroidogenesis | Mutation | Tumors | Index Medicus | Abridged Index Medicus
Journal Article