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Diabetes, ISSN 0012-1797, 05/2016, Volume 65, Issue 5, pp. 1410 - 1423
Beige adipocytes emerge postnatally within the white adipose tissue in response to certain environmental cues, such as chronic cold exposure. Because of its... 
THERMOGENESIS | WHITE FAT | ACTIVATED PROTEIN-KINASE | ENDOCRINOLOGY & METABOLISM | HYPOTHALAMIC AMPK | PPAR-GAMMA | BROWN ADIPOSE-TISSUE | CAPSICUM-ANNUUM | CH-19 SWEET | ENERGY-METABOLISM | TRANSCRIPTIONAL COMPLEX | Adipocytes, Beige - drug effects | Transcription Factors - chemistry | Capsaicin - therapeutic use | Adipogenesis - drug effects | Capsaicin - chemistry | Male | Hydrogenation | Anti-Obesity Agents - therapeutic use | Adrenergic beta-2 Receptor Agonists - pharmacology | Receptors, Adrenergic, beta-2 - chemistry | DNA-Binding Proteins - metabolism | Capsaicin - agonists | Adipocytes, Beige - pathology | Adrenergic beta-2 Receptor Agonists - therapeutic use | Anti-Obesity Agents - antagonists & inhibitors | Adrenergic beta-2 Receptor Antagonists - pharmacology | Anti-Obesity Agents - agonists | Cold Temperature | Obesity - chemically induced | Receptors, Adrenergic, beta-2 - genetics | Mice, Inbred C57BL | Cells, Cultured | Acclimatization | Adrenergic beta-2 Receptor Antagonists - toxicity | Capsaicin - analogs & derivatives | Mice, Transgenic | Adipocytes, Beige - cytology | Random Allocation | Transcription Factors - genetics | DNA-Binding Proteins - genetics | DNA-Binding Proteins - chemistry | Receptors, Adrenergic, beta-2 - metabolism | Obesity - metabolism | Obesity - pathology | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Animals | Signal Transduction - drug effects | Oxygen Consumption - drug effects | Obesity - prevention & control | Protein Stability - drug effects | Capsaicin - antagonists & inhibitors | Dietary Supplements | Energy Metabolism - drug effects | Adipocytes, Beige - physiology | Weight reducing preparations | Obesity | Complications and side effects | Fat cells | Dosage and administration | Research | Beta adrenoceptors | Pharmacology and Therapeutics
Journal Article
Journal of Chemical Information and Modeling, ISSN 1549-9596, 05/2015, Volume 55, Issue 5, pp. 1045 - 61
Journal Article
Kidney International, ISSN 0085-2538, 07/2017, Volume 92, Issue 1, pp. 101 - 113
Macrophage activation is increased in diabetes and correlated with the onset and progression of vascular complications. To identify drugs that could inhibit... 
macrophages | fibrosis | inflammation | diabetes | PROTEIN | PERIPHERAL-BLOOD | NEPHROPATHY | MONOCYTIC CELLS | BETA | ALPHA-TOCOPHEROL SUPPLEMENTATION | CYTOKINE PRODUCTION | INSULIN-RESISTANCE | UROLOGY & NEPHROLOGY | PLASMA-GLUCOSE | Tumor Necrosis Factor-alpha - metabolism | Diabetes Mellitus, Experimental - drug therapy | Phosphorylation | Diabetic Cardiomyopathies - metabolism | Kidney - pathology | Streptozocin | Humans | Diabetes Mellitus, Experimental - genetics | Male | NF-kappa B - metabolism | beta-Arrestin 2 - metabolism | Receptors, Adrenergic, beta-2 - drug effects | Adrenergic beta-2 Receptor Agonists - pharmacology | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Protein Kinase C - metabolism | Myocardium - metabolism | Diabetic Cardiomyopathies - prevention & control | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Diabetic Nephropathies - prevention & control | Adrenergic beta-2 Receptor Antagonists - pharmacology | Kidney - drug effects | Anti-Inflammatory Agents - pharmacology | Receptors, Adrenergic, beta-2 - genetics | Diabetic Nephropathies - metabolism | Diabetic Nephropathies - chemically induced | THP-1 Cells | beta-Arrestin 2 - genetics | Diabetic Nephropathies - genetics | Myocardium - pathology | Rats, Sprague-Dawley | Receptors, Adrenergic, beta-2 - metabolism | Diabetic Cardiomyopathies - genetics | Rats, Zucker | Macrophages - metabolism | Animals | NF-KappaB Inhibitor alpha - metabolism | Fibrosis | Macrophages - drug effects | Diabetic Cardiomyopathies - chemically induced | Macrophage Activation - drug effects
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 02/2013, Volume 112, Issue 3, pp. 476 - 486
RATIONALE:Short-term β-adrenergic stimulation promotes contractility in response to stress but is ultimately detrimental in the failing heart because of... 
heart failure | cardiac progenitor cells | adrenergic regulation | adrenergic receptors | adult stem cells | PROTEIN-KINASE-A | CHRONIC HEART-FAILURE | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | DILATED CARDIOMYOPATHY | LEFT-VENTRICULAR DYSFUNCTION | FAILING HEART | GENE-EXPRESSION | MEDIATED DNA-SYNTHESIS | PERIPHERAL VASCULAR DISEASE | CYCLE PROGRESSION | BETA-ADRENERGIC RECEPTORS | HEMATOLOGY | Cyclin D1 - metabolism | Phosphorylation | Coculture Techniques | Humans | Male | G-Protein-Coupled Receptor Kinase 2 - metabolism | Receptors, Adrenergic, beta-2 - drug effects | Proto-Oncogene Proteins c-kit - metabolism | Stem Cells - metabolism | Adrenergic beta-2 Receptor Agonists - pharmacology | Dose-Response Relationship, Drug | Stem Cell Transplantation | Transfection | RNA Interference | Time Factors | Myocardial Infarction - pathology | Cell Death | Receptors, Adrenergic, beta-1 - drug effects | Nitric Oxide Synthase Type III - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Adrenergic beta-2 Receptor Antagonists - pharmacology | Disease Models, Animal | Myocardial Infarction - surgery | Cell Survival | Receptors, Adrenergic, beta-2 - genetics | Cells, Cultured | Receptors, Adrenergic, beta-1 - metabolism | Myocardial Infarction - metabolism | Receptors, Adrenergic, beta-2 - metabolism | Myocytes, Cardiac - pathology | Myocytes, Cardiac - transplantation | Animals | Myocytes, Cardiac - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Myocytes, Cardiac - metabolism | Stem Cells - drug effects | Stem Cells - pathology | Receptors, Adrenergic, beta-1 - genetics | Cell Proliferation - drug effects | Mice | Mitogen-Activated Protein Kinase 1 - metabolism
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