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PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e89227
Open vascular reconstructions frequently fail due to the development of recurrent disease or intimal hyperplasia (IH). This paper reports a novel drug delivery... 
TGF-BETA | DENDRITIC CELLS | PERIVASCULAR DELIVERY | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MUSCLE-CELL PROLIFERATION | NEOINTIMAL HYPERPLASIA | RE-ENDOTHELIALIZATION | GENE-TRANSFER | PLGA NANOPARTICLES | DRUG-ELUTING STENTS | Carotid Arteries - drug effects | Carotid Arteries - metabolism | Muscle, Smooth, Vascular - metabolism | Ribosomal Protein S6 Kinases - metabolism | Myocytes, Smooth Muscle - pathology | Male | Adventitia - metabolism | Phosphorylation - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Hyperplasia - metabolism | Muscle, Smooth, Vascular - drug effects | Cell Survival - drug effects | Rats | Adventitia - drug effects | Rats, Sprague-Dawley | Hyperplasia - pathology | Sirolimus - administration & dosage | Muscle, Smooth, Vascular - pathology | Animals | Adventitia - pathology | Carotid Arteries - pathology | Cell Proliferation - drug effects | Nanoparticles - administration & dosage | Drug Delivery Systems - methods | Drugs | Nanoparticles | Drug delivery systems | Hostages | Rapamycin | Drug approval | Vehicles | TOR protein | Phosphorylation | Disease | Angioplasty | Hyperplasia | Smooth muscle | Materials science | Drug delivery | Kinases | Polylactide-co-glycolide | Biomedical materials | Cell growth | Surgery | Inhibition | Carotid artery | Stents | Muscles | Thrombosis | Restenosis | Injury prevention | Hospitals | Science programs | Poly(lactide-co-glycolide) | Veins & arteries
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2013, Volume 33, Issue 10, pp. 2397 - 2406
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 11/2014, Volume 307, Issue 10, pp. H1419 - H1429
Oral all-trans retinoic acid (atRA) has been shown to reduce the formation of neointimal hyperplasia; however, the dose required was 30 times the... 
smooth muscle cell proliferation and differentiation | nitric oxide | restenosis | endothelium/vascular | peripheral vascular disease | MIGRATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | PROLIFERATION | TRANS-RETINOIC ACID | POLYTETRAFLUOROETHYLENE | VASCULAR INJURY | INHIBITION | SMOOTH-MUSCLE-CELLS | DIFFERENTIATION | ACUTE PROMYELOCYTIC LEUKEMIA | ALL-TRANS | Recurrence | Muscle, Smooth, Vascular - metabolism | Apoptosis - drug effects | Hyperplasia | Male | Carotid Stenosis - metabolism | Drug Carriers | Adventitia - metabolism | Dose-Response Relationship, Drug | Citrates - chemistry | Neointima | Time Factors | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Carotid Artery Injuries - pathology | Disease Models, Animal | Fibroblasts - metabolism | Muscle, Smooth, Vascular - drug effects | Tretinoin - administration & dosage | Macrophages - pathology | Carotid Artery, Common - metabolism | Carotid Stenosis - therapy | Cells, Cultured | Membranes, Artificial | Adventitia - drug effects | Carotid Stenosis - pathology | Rats, Sprague-Dawley | Carotid Artery Injuries - therapy | Animals | Carotid Artery, Common - drug effects | Fibroblasts - drug effects | Carotid Artery, Common - pathology | Macrophages - drug effects | Polymers - chemistry | Adventitia - pathology | Cell Proliferation - drug effects | Vascular Cell Adhesion Molecule-1 - metabolism | Physiological aspects | Stenosis | Health aspects | Citrates | Vascular Biology and Microcirculation | endothelium | vascular
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2017, Volume 37, Issue 11, pp. 2102 - 2102
OBJECTIVE—The role of TGF-β (transforming growth factor-β) signaling in abdominal aortic aneurysm (AAA) formation is controversial. Others reported that... 
aneurysm | smooth muscle | SERVICES TASK-FORCE | ATHEROSCLEROSIS | RECEPTOR | mice | macrophages | TGF-beta | DISRUPTION | angiotensin II | ANEURYSM MOUSE MODEL | PERIPHERAL VASCULAR DISEASE | aorta | SMOOTH-MUSCLE-CELLS | APOE(-/-) MICE | HEMATOLOGY | E-DEFICIENT MICE | PROMOTES | PROGRESSION | Protein-Serine-Threonine Kinases - deficiency | Tunica Media - pathology | Aortic Aneurysm, Abdominal - chemically induced | Receptors, Transforming Growth Factor beta - genetics | Aortic Aneurysm, Abdominal - prevention & control | Muscle, Smooth, Vascular - metabolism | Transforming Growth Factor beta1 - antagonists & inhibitors | Transforming Growth Factor beta2 - metabolism | Aortic Aneurysm, Thoracic - prevention & control | Transforming Growth Factor beta1 - metabolism | Male | Aortic Aneurysm, Thoracic - chemically induced | Transforming Growth Factor beta3 - metabolism | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | Adventitia - metabolism | Receptor, Transforming Growth Factor-beta Type II | Aortic Aneurysm, Thoracic - pathology | Tunica Media - metabolism | Transforming Growth Factor beta - antagonists & inhibitors | Female | Aorta, Abdominal - pathology | Aorta, Thoracic - drug effects | Vascular Remodeling - drug effects | Angiotensin II | Aorta, Abdominal - metabolism | Dilatation, Pathologic | Aorta, Thoracic - pathology | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Severity of Illness Index | Genetic Predisposition to Disease | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Aorta, Thoracic - metabolism | Transforming Growth Factor beta3 - antagonists & inhibitors | Mice, Knockout | Antibodies - pharmacology | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Signal Transduction - drug effects | Aortic Aneurysm, Thoracic - metabolism | Transforming Growth Factor beta2 - antagonists & inhibitors | Adventitia - pathology | Transforming Growth Factor beta - metabolism | Receptors, Transforming Growth Factor beta - deficiency | growth factor | Animal Models of Human Disease | Vascular Disease | Genetically Altered and Transgenic Models | Aortic Dissection | genetically altered mice
Journal Article
Molecular Medicine, ISSN 1076-1551, 10/2015, Volume 21, Issue S1, pp. S13 - S18
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2014, Volume 9, Issue 9, p. e107888
Multiple clinical studies show that arterial stiffness, measured as pulse wave velocity (PWV), precedes hypertension and is an independent predictor of... 
ALL-CAUSE | INDEPENDENT PREDICTOR | DIETARY-SODIUM | PULSE-WAVE VELOCITY | MULTIDISCIPLINARY SCIENCES | DISEASE | CARDIOVASCULAR MORTALITY | ATHEROSCLEROSIS | VASCULAR STIFFNESS | MICE | PREVALENCE | Carotid Arteries - drug effects | Pulse Wave Analysis | Sodium, Dietary - adverse effects | Stroke - diagnostic imaging | Tunica Media - drug effects | Endothelium, Vascular - drug effects | Male | Stroke - physiopathology | Stroke - genetics | Adventitia - metabolism | Tunica Media - metabolism | Ultrasonography | Female | Blood Pressure - drug effects | Epigenesis, Genetic - drug effects | Rats, Inbred Dahl | Aorta - physiopathology | Hypertension - genetics | Genetic Predisposition to Disease | Aorta - drug effects | Rats | Adventitia - drug effects | Hypertension - physiopathology | Phenotype | Animals | Carotid Arteries - physiopathology | Endothelium, Vascular - metabolism | Vascular Stiffness - drug effects | Stroke (Disease) | Proteins | Hypertension | Epigenetic inheritance | Genes | Blood pressure | Gene expression | Regulators | Salts | Syngeneic grafts | Change detection | Smooth muscle | Cardiovascular disease | Blood | Risk factors | Rodents | Cell adhesion | Aorta | Extracellular matrix | Stiffness | Carotid artery | Stroke | Mortality | Health risks | Muscles | Histology | Molecular modelling | Sodium | Hormone replacement therapy | Wave velocity | Widening | Cardiovascular diseases | Apoptosis | Kidney transplantation | Structure-function relationships
Journal Article
Hypertension, ISSN 0194-911X, 08/2018, Volume 72, Issue 4, pp. 881 - 888
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2016, Volume 36, Issue 7, pp. 1367 - 1376
Objective-Vascular smooth muscle cells (VSMCs) are believed to dedifferentiate and proliferate in response to vessel injury. Recently, adventitial progenitor... 
Myocardin | c-Myb genes | Cell differentiation | Smooth muscle cells | Progenitor cells | ARTERIAL INJURY | cell differentiation | STEM-CELLS | ATHEROSCLEROSIS | progenitor cells | NEOINTIMA FORMATION | VEIN GRAFTS | IDENTIFICATION | VESSEL WALL | OLIGONUCLEOTIDES | smooth muscle cells | myocardin | PERIPHERAL VASCULAR DISEASE | ACCUMULATION | HEMATOLOGY | EXPRESSION | Vascular Remodeling | Muscle, Smooth, Vascular - metabolism | Humans | Transcriptional Activation | Myocytes, Smooth Muscle - pathology | Proto-Oncogene Proteins c-myb - genetics | Adventitia - injuries | Stem Cells - metabolism | Adventitia - metabolism | Transfection | Time Factors | Ataxin-1 - metabolism | HEK293 Cells | Trans-Activators - genetics | Transcription, Genetic | Carotid Artery Injuries - genetics | Myocytes, Smooth Muscle - drug effects | Nuclear Proteins - genetics | Binding Sites | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Transforming Growth Factor beta1 - pharmacology | Carotid Artery Injuries - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Promoter Regions, Genetic | Signal Transduction | Carotid Artery, Common - metabolism | Mice, Inbred C57BL | Genotype | Mice, Transgenic | Nuclear Proteins - metabolism | Adventitia - drug effects | Proto-Oncogene Proteins c-myb - metabolism | Muscle, Smooth, Vascular - injuries | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Cell Differentiation - drug effects | Stem Cells - drug effects | Stem Cells - pathology | Carotid Artery, Common - pathology | Trans-Activators - metabolism | Adventitia - pathology | Cell Proliferation - drug effects | Index Medicus
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 07/2016, Volume 39, Issue 2, pp. 721 - 739
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2014, Volume 34, Issue 8, pp. 1704 - 1715
OBJECTIVE—Pulmonary hypertension (PH) is a progressive disease arising from remodeling and narrowing of pulmonary arteries (PAs) resulting in high pulmonary... 
Adventitia | Fibroblast | NADPH oxidase | Pulmonary artery | pulmonary artery | ACTIVATION | adventitia | PROLIFERATION | FIBROBLASTS | NAD(P)H OXIDASE | fibroblast | CHRONIC HYPOXIA | MODELS | INFLAMMATION | NOX4 | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | MOLECULAR-MECHANISMS | Up-Regulation | Antihypertensive Agents - pharmacology | Fibroblasts - enzymology | Cell Proliferation | Reactive Oxygen Species - metabolism | Humans | Monocrotaline | NADPH Oxidases - metabolism | Pyrroles | Extracellular Matrix - metabolism | Male | Dose-Response Relationship, Drug | Extracellular Matrix - genetics | Transfection | Time Factors | HEK293 Cells | Adventitia - enzymology | NADPH Oxidases - genetics | Hypertrophy, Right Ventricular - pathology | Hypertension, Pulmonary - drug therapy | Indoles | Hypertension, Pulmonary - enzymology | Disease Models, Animal | Signal Transduction | Mice, Inbred C57BL | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Rats | Familial Primary Pulmonary Hypertension | Hypertension, Pulmonary - genetics | Hypoxia - complications | Adventitia - drug effects | NADPH Oxidase 4 | Pulmonary Artery - drug effects | Fibroblasts - pathology | Hypertrophy, Right Ventricular - enzymology | Rats, Sprague-Dawley | Pulmonary Artery - enzymology | Animals | Hypertrophy, Right Ventricular - prevention & control | Adventitia - pathology | Mice | Hypertension, Pulmonary - etiology | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Cell Movement | Index Medicus
Journal Article
Alcoholism: Clinical and Experimental Research, ISSN 0145-6008, 12/2017, Volume 41, Issue 12, pp. 2051 - 2065
Background Cell and molecular mechanisms mediating the cardiovascular effects of alcohol are not fully understood. Our aim was to determine the effect of... 
Stem Cells | Alcohol | Hedgehog | Stem Cell Antigen‐1 | Atherosclerosis | Progenitor Cells | MODERATE | POPULATION | CONTRIBUTE | ISCHEMIC-HEART-DISEASE | PROLIFERATION | SUBSTANCE ABUSE | Stem Cell Antigen-1 | IN-VITRO | ADVENTITIA | SMOOTH-MUSCLE-CELLS | EXPRESSION | CONSUMPTION | Carotid Arteries - drug effects | Hedgehog Proteins - drug effects | Stem Cells - immunology | Gene Expression - drug effects | Cell Proliferation - physiology | Membrane Proteins - immunology | Mice, Transgenic | Hedgehog Proteins - physiology | Animals | Patched-1 Receptor - biosynthesis | Antigens, Ly - immunology | Carotid Arteries - physiopathology | Cell Differentiation - drug effects | Veratrum Alkaloids - pharmacology | Stem Cells - physiology | Vascular Remodeling - physiology | Cell Proliferation - drug effects | Mice | Vascular Remodeling - drug effects | Zinc Finger Protein Gli2 - biosynthesis | Antigens | Analysis | Stem cells | Fluorescence | Genetic engineering | Gene expression | Differentiation (biology) | Transgenic | Stem cell transplantation | Smooth muscle | Remodeling | Alcohols | Allografts | Ataxin | Rodents | Inhibition | Carotid artery | Expansion | Injury analysis | Thickening | Ethanol | Muscles | Blood vessels | Patched protein | Polymerase chain reaction | Signaling | Molecular modelling | Hedgehog protein | Green fluorescent protein | Cells (biology)
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 81, pp. 96 - 106
Abstract Abdominal aortic aneurysm (AAA) is a serious vascular disease with high mortality. Our previous study suggested that hyperhomocysteinemia (HHcy)... 
Cardiovascular | Mitochondrial reactive oxygen species | Hyperhomocysteinemia | Inflammasome | Abdominal aortic aneurysm | Macrophage | HOMOCYSTEINE | APOPTOSIS | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | NLRP3 INFLAMMASOME | MODEL | BETA | CELL BIOLOGY | ENDOVASCULAR REPAIR | DYSFUNCTION | STRESS | RNA, Small Interfering - genetics | Aortic Aneurysm, Abdominal - chemically induced | Aortic Aneurysm, Abdominal - prevention & control | Folic Acid - pharmacology | Inflammasomes - metabolism | Reactive Oxygen Species - metabolism | Apolipoproteins E - deficiency | NLR Family, Pyrin Domain-Containing 3 Protein | Hyperhomocysteinemia - metabolism | Caspase 1 - metabolism | Male | Interleukin-1beta - genetics | Aortic Aneurysm, Abdominal - metabolism | Adventitia - metabolism | Inflammasomes - drug effects | Hyperhomocysteinemia - prevention & control | Interleukin-1beta - metabolism | Calcium Phosphates - adverse effects | Disease Models, Animal | Fibroblasts - metabolism | Gene Expression | Macrophages - pathology | Aortic Aneurysm, Abdominal - complications | Mice, Inbred C57BL | Carrier Proteins - antagonists & inhibitors | Rats | Mitochondria - metabolism | Adventitia - drug effects | Mitochondria - drug effects | Mitochondria - pathology | Fibroblasts - pathology | Mice, Knockout | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Caspase 1 - genetics | Apolipoproteins E - genetics | Fibroblasts - drug effects | Macrophages - drug effects | Adventitia - pathology | Mice | Primary Cell Culture | Macrophage Activation - drug effects | Hyperhomocysteinemia - chemically induced | Hyperhomocysteinemia - complications | RNA, Small Interfering - metabolism | Phosphates | Macrophages | Abdominal aneurysm | Analysis
Journal Article