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Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 09/2018, Volume 354, pp. 153 - 175
The Adverse Outcome Pathways (AOPs) are designed to provide mechanistic understanding of complex biological systems and pathways of toxicity that result in... 
Regulatory Toxicology | Adverse Outcome Pathway | Developmental Neurotoxicity | NEURONAL APOPTOSIS | SIGNALING PATHWAYS | HIPPOCAMPAL GLUTAMATE | NEUROTROPHIC FACTOR | FACTOR MESSENGER-RNA | SUBUNIT COMPOSITION | POSTNATAL LEAD-EXPOSURE | SYNAPTIC PLASTICITY | PROGRAMMED CELL-DEATH | PHARMACOLOGY & PHARMACY | TOXICOLOGY | LONG-TERM POTENTIATION | Memory - drug effects | Memory Disorders - physiopathology | Neurons - pathology | Receptors, N-Methyl-D-Aspartate - antagonists & inhibitors | Age Factors | Learning - drug effects | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Child, Preschool | Memory Disorders - metabolism | Infant | Learning Disorders - physiopathology | Brain - growth & development | Brain - metabolism | Neurotoxicity Syndromes - psychology | Learning Disorders - psychology | Adult | Child Behavior - drug effects | Child Development - drug effects | Neurons - metabolism | Neurons - drug effects | Child | Infant, Newborn | Memory Disorders - psychology | Risk Assessment | Excitatory Amino Acid Antagonists - toxicity | Adverse Outcome Pathways | Brain - drug effects | Neurotoxicity Syndromes - etiology | Learning Disorders - chemically induced | Animals | Adolescent | Neurotoxicity Syndromes - physiopathology | Neurotoxicity Syndromes - metabolism | Learning Disorders - metabolism | Memory Disorders - chemically induced | Methyl aspartate | Brain | Cell death | Aspartate | Children | Glutamate | Epidemiology | Index Medicus
Journal Article
Particle and Fibre Toxicology, ISSN 1743-8977, 02/2016, Volume 13, Issue 1, p. 11
Several experimental studies have shown that carbon nanotubes (CNT) can induce respiratory effects, including lung fibrosis. The cellular and molecular events... 
Carbon nanotube | AOP | Mechanisms | Epithelial cell | Fibroblast | Lung fibrosis | Macrophage | OXIDATIVE STRESS | PULMONARY TOXICITY | INFLAMMASOME ACTIVATION | CROCIDOLITE ASBESTOS | FIBROGENIC RESPONSE | INHALATION EXPOSURE | RAT LUNG | IN-VITRO | EPITHELIAL-CELLS | FIBROTIC RESPONSE | TOXICOLOGY | Lung - pathology | Epithelial Cells - metabolism | Macrophages - pathology | Risk Assessment | Epithelial Cells - drug effects | Humans | Epithelial Cells - pathology | Pulmonary Fibrosis - pathology | Fibroblasts - pathology | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Fibroblasts - drug effects | Lung - drug effects | Cell Communication - drug effects | Inflammation Mediators - metabolism | Inhalation Exposure - adverse effects | Pulmonary Fibrosis - chemically induced | Macrophages - drug effects | Nanotubes, Carbon - adverse effects | Lung - metabolism | Pulmonary Fibrosis - metabolism | Extracellular Matrix Proteins - metabolism | Fibroblasts - metabolism | Complications and side effects | Care and treatment | Patient outcomes | Fibrosis | Nanotubes | Development and progression | Respiratory tract diseases | Research | Diagnosis | Medical research | Neutrophils | Carbon | Studies | Occupational safety | Toxicology | Researchers | Microscopy | Collagen | Fibroblasts | Extracellular matrix | Tumor necrosis factor-TNF | Test methods | Localization | Growth factors
Journal Article
Environmental Toxicology and Pharmacology, ISSN 1382-6689, 02/2020, Volume 74, pp. 103301 - 103301
•Integrated environmental field survey on biomarkers, fish physiology & populations.•Identification of adverse outcome pathways of complex chemical... 
Biochemical & physiological biomarkers | Ecological studies | Micropollutants risk assessment | Adverse outcome pathways | Index Medicus | Life Sciences | Ecotoxicology | Toxicology
Journal Article