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Journal of Clinical Immunology, ISSN 0271-9142, 5/2008, Volume 28, Issue S1, pp. 34 - 41
Associations between systemic lupus erythematosus (SLE) and primary immunodeficiencies (PIDs) were analyzed to gain insight into the physiopathology of SLE.... 
Medical Microbiology | agammaglobulinemia | chronic granulomatous disease | Internal Medicine | ALPS | Biomedicine | Immunology | Systemic lupus erythematosus | primary immunodeficiencies | IPEX | Infectious Diseases | complement deficiencies | APECED | Chronic granulomatous disease | Primary immunodeficiencies | Agammaglobulinemia | Complement deficiencies | IMMUNOLOGICAL FEATURES | chronic gramilomatous disease | HYPER-IGM SYNDROME | CHRONIC GRANULOMATOUS-DISEASE | WISKOTT-ALDRICH-SYNDROME | systemic lupus erythernatosus | X-LINKED AGAMMAGLOBULINEMIA | COMMON VARIABLE IMMUNODEFICIENCY | COMPLEMENT DEFICIENCY | IMMUNOLOGY | CANDIDIASIS-ECTODERMAL DYSTROPHY | AUTOANTIBODY PRODUCTION | T-CELLS | Autoimmunity | Forkhead Transcription Factors - immunology | Humans | Immunoglobulins - genetics | CD40 Ligand - immunology | Lupus Erythematosus, Systemic - immunology | Complement System Proteins - genetics | Female | Immunologic Deficiency Syndromes - immunology | Transcription Factors - immunology | Disease Susceptibility | Self Tolerance | Lupus Erythematosus, Systemic - etiology | Transcription Factors - genetics | Forkhead Transcription Factors - genetics | Immunoglobulins - deficiency | Polymorphism, Genetic | Animals | CD40 Ligand - genetics | Complement System Proteins - deficiency | Immunologic Deficiency Syndromes - physiopathology | Lupus Erythematosus, Systemic - genetics | Immunologic Deficiency Syndromes - genetics | Mice | Lupus Erythematosus, Systemic - physiopathology | Lupus | Autoantigens | Immunoproteins
Journal Article
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2011, Volume 129, Issue 3, pp. 787 - 793.e6
Background CD27 is a lymphocyte costimulatory molecule that regulates T-cell, natural killer (NK) cell, B-cell, and plasma cell function, survival, and... 
Allergy and Immunology | phenotype | B cell | T cell | EBV | CD27 | immunodeficiency | hypogammaglobulinemia | natural killer cell | viremia | CD70 | T-CELL | IMMUNOLOGY | HEMOPHAGOCYTIC LYMPHOHISTIOCYTOSIS | B-CELLS | MEMORY | ALLERGY | XIAP DEFICIENCY | GENERATION | DIFFERENTIATION | EXPRESSION | EPSTEIN-BARR-VIRUS | Gram-Positive Bacterial Infections - immunology | Anemia, Aplastic - virology | Humans | Male | Gram-Positive Bacterial Infections - virology | Severe Combined Immunodeficiency - physiopathology | Young Adult | Severe Combined Immunodeficiency - virology | T-Lymphocytes - metabolism | Fatal Outcome | Epstein-Barr Virus Infections - genetics | Gram-Positive Bacterial Infections - genetics | Herpesvirus 4, Human - pathogenicity | B-Lymphocytes - pathology | T-Lymphocytes - pathology | Gram-Positive Bacterial Infections - complications | Anemia, Aplastic - complications | Anemia, Aplastic - genetics | B-Lymphocytes - metabolism | Siblings | Epstein-Barr Virus Infections - physiopathology | Gram-Positive Bacterial Infections - physiopathology | Epstein-Barr Virus Infections - virology | Epstein-Barr Virus Infections - immunology | Immunity, Humoral - genetics | Cells, Cultured | Herpesvirus 4, Human - immunology | Viremia - genetics | Severe Combined Immunodeficiency - immunology | Tumor Necrosis Factor Receptor Superfamily, Member 7 - genetics | Mutation - genetics | Agammaglobulinemia - etiology | Anemia, Aplastic - physiopathology | Severe Combined Immunodeficiency - complications | Severe Combined Immunodeficiency - genetics | B-Lymphocytes - immunology | Viremia - virology | Pedigree | Epstein-Barr Virus Infections - complications | T-Lymphocytes - immunology | Viremia - immunology | Consanguinity | Anemia, Aplastic - immunology | Antigens | Genes | Staphylococcus aureus infections | T cells | Diseases | Virus diseases | Lymphocytes | Virus diseases in children | Medical genetics | Cross infection | Children | Tetanus | Nosocomial infections | Mitogens | Ligands | Infections | T cell receptors | Family medical history | Patients | Age | Viral infections | Fever | Immune system | CD27 antigen | Immunoglobulins | Cell survival | Viremia | Aplastic anemia | Immunodeficiency | Hypogammaglobulinemia | Lymphocytes T | Costimulator | Infection | Lymphocytes B | Plasma cells | Sepsis | Mutation | Natural killer cells | Differentiation | NK cell
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2016, Volume 11, Issue 7, p. e0159607
Acalabrutinib (ACP-196) is a second-generation inhibitor of Bruton agammaglobulinemia tyrosine kinase (BTK) with increased target selectivity and potency... 
SURVIVAL | CLL | ACTIVATION | BRUTON TYROSINE KINASE | ONCOLOGY | MULTIDISCIPLINARY SCIENCES | IBRUTINIB | PCI-32765 | CHRONIC LYMPHOCYTIC-LEUKEMIA | GERMINAL CENTER | ANTIGEN | Lethargy - physiopathology | Protein-Tyrosine Kinases - metabolism | Diarrhea - physiopathology | Humans | Pyrazines - administration & dosage | Male | Antineoplastic Agents - administration & dosage | Protein Kinase Inhibitors - adverse effects | Weight Loss - drug effects | Diarrhea - chemically induced | Anorexia - physiopathology | Benzamides - administration & dosage | Protein-Tyrosine Kinases - genetics | Antineoplastic Agents - adverse effects | Female | B-Lymphocytes - pathology | Drug Evaluation, Preclinical | Benzamides - adverse effects | Disease Models, Animal | Lymphoma, Large B-Cell, Diffuse - drug therapy | Drug Administration Schedule | Lymphoma, Large B-Cell, Diffuse - enzymology | Lethargy - chemically induced | Lymphoma, Large B-Cell, Diffuse - mortality | B-Lymphocytes - drug effects | Disease-Free Survival | Protein Kinase Inhibitors - administration & dosage | Vomiting - chemically induced | Animals | B-Lymphocytes - immunology | Agammaglobulinaemia Tyrosine Kinase | Dogs | Pyrazines - adverse effects | Cell Line, Tumor | Anorexia - chemically induced | Lymphoma, Large B-Cell, Diffuse - veterinary | Vomiting - physiopathology | Protein-Tyrosine Kinases - antagonists & inhibitors | Antimitotic agents | Care and treatment | Safety and security measures | Dosage and administration | Non-Hodgkin's lymphomas | Research | Antineoplastic agents | Cell proliferation | Cell culture | Flow cytometry | Veterinary colleges | Anorexia | Leukemia | Cytotoxicity | Malignancy | Kinases | Veterinary medicine | Vomiting | Quality | Inhibition | Protein-tyrosine kinase | Tyrosine | Internal medicine | Hematology | Agammaglobulinemia | Diarrhea | Gene expression | Lymphoma | Bruton's tyrosine kinase | Inhibitors | Lymphocytes B | Lethargy | Lymphomas | Cancer | B-cell lymphoma
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 11/2017, Volume 190, Issue 2, pp. 226 - 234
Summary Many patients with primary immunodeficiency (PID) who have antibody deficiency develop progressive lung disease due to underlying subclinical infection... 
primary immunodeficiency disease | antibody deficiency | subclinical infection | lung disease | monitoring | BACTERIA | BRONCHIECTASIS | COMMON VARIABLE IMMUNODEFICIENCY | IMMUNE-DEFICIENCY | IMMUNOLOGY | CHILDREN | PRIMARY HYPOGAMMAGLOBULINEMIA | LUNG-DISEASE | VIRUSES | Spirometry | Asymptomatic Infections - epidemiology | Humans | Male | Adult | Female | Retrospective Studies | Immunologic Deficiency Syndromes - immunology | Child | Immunologic Deficiency Syndromes - therapy | Immunologic Deficiency Syndromes - complications | Lung Diseases - diagnosis | Europe | Lung Diseases - prevention & control | Medical Records | Immunoglobulin G - therapeutic use | Ambulatory Care | Agammaglobulinemia - physiopathology | Lung Diseases - immunology | Immunologic Deficiency Syndromes - physiopathology | Common Variable Immunodeficiency - physiopathology | Immunization, Passive | Respiratory System - physiopathology | Lung Diseases - complications | Immunoglobulins - therapeutic use | Practice Guidelines as Topic | Surveys | Medical research | Immunoglobulin G | Lung diseases | Medicine, Experimental | Medical records | Pulmonary function tests | Chest | Respiratory function | Common variable immunodeficiency | Subclinical infection | Microbiology | Laboratories | Complications | Antibodies | Infections | Patients | Laboratory tests | Sinus | Adults | Children | Health risk assessment | Monitoring | Original | Clinical Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Lungmedicin och allergi | Klinisk medicin | Respiratory Medicine and Allergy
Journal Article
Journal Article
Journal of Clinical Immunology, ISSN 0271-9142, 9/2010, Volume 30, Issue 5, pp. 734 - 745
Subcutaneous human IgG (SCIG) therapy in primary immunodeficiency (PID) offers sustained IgG levels throughout the dosing cycle and fewer adverse events (AEs)... 
serum IgG trough levels | Medical Microbiology | Biomedicine | Immunology | home infusion therapy | L-proline | Subcutaneous immunoglobulin (SCIG) | primary immunodeficiency | Internal Medicine | Infectious Diseases | local tolerability | PRIMARY ANTIBODY DEFICIENCIES | HUMAN INTRAVENOUS IMMUNOGLOBULIN | IGG | COMMON VARIABLE IMMUNODEFICIENCY | INFUSION | IMMUNOLOGY | REPLACEMENT THERAPY | QUALITY-OF-LIFE | HOME THERAPY | 10-PERCENT | IGIV-C | Common Variable Immunodeficiency - immunology | Agammaglobulinemia - immunology | Prospective Studies | Common Variable Immunodeficiency - drug therapy | Immunoglobulin G - blood | Humans | Middle Aged | Male | Common Variable Immunodeficiency - blood | Agammaglobulinemia - drug therapy | Genetic Diseases, X-Linked - drug therapy | Injections, Subcutaneous | Adult | Female | Protein Stability | Child | Genetic Diseases, X-Linked - physiopathology | Genetic Diseases, X-Linked - blood | Immunologic Factors - administration & dosage | Agammaglobulinemia - physiopathology | Genetic Diseases, X-Linked - immunology | Bacterial Infections - prevention & control | Agammaglobulinemia - blood | Immunoglobulins, Intravenous - therapeutic use | Adolescent | Common Variable Immunodeficiency - physiopathology | Aged | Immunologic Factors - adverse effects | Immunologic Factors - therapeutic use | Complications and side effects | Allergy | Bacterial infections | Immunoglobulin G | Immunodeficiency | Proline | Universities and colleges | Allergic reaction
Journal Article
Current Opinion in Allergy and Clinical Immunology, ISSN 1528-4050, 12/2013, Volume 13, Issue 6, pp. 589 - 595
PURPOSE OF REVIEWInborn errors of human natural killer (NK) cells may affect the development of these cells, their function, or both. There are two broad... 
Genetic diseases | Immunodeficiencies | Natural killer cells | genetic diseases | NK LYMPHOID DEFICIENCY | immunodeficiencies | SPORADIC MONOCYTOPENIA | SEVERE COMBINED IMMUNODEFICIENCY | RETICULAR DYSGENESIS | GROWTH-RETARDATION | IMMUNE-DEFICIENCY | ADENYLATE KINASE 2 | IMMUNOLOGY | MONOMAC SYNDROME | natural killer cells | ALLERGY | AUTOSOMAL-DOMINANT | ADENOSINE-DEAMINASE DEFICIENCY | Minichromosome Maintenance Complex Component 4 - genetics | Agammaglobulinemia - immunology | Adenylate Kinase - immunology | Killer Cells, Natural | Agammaglobulinemia - genetics | Humans | Minichromosome Maintenance Complex Component 4 - immunology | Mycobacterium Infections - immunology | Severe Combined Immunodeficiency - physiopathology | Janus Kinase 2 | Agammaglobulinemia - pathology | Virus Diseases - physiopathology | Mycobacterium Infections - genetics | Mycobacterium Infections - pathology | Severe Combined Immunodeficiency - pathology | Adenosine Deaminase - deficiency | Virus Diseases - genetics | GATA2 Transcription Factor - genetics | Janus Kinase 3 - immunology | Virus Diseases - immunology | Adenosine Deaminase - genetics | GATA2 Transcription Factor - immunology | Mycobacterium Infections - physiopathology | Severe Combined Immunodeficiency - immunology | Agammaglobulinemia - physiopathology | Severe Combined Immunodeficiency - genetics | Virus Diseases - pathology | Janus Kinase 3 - genetics | Adenylate Kinase - genetics | Adenosine Deaminase - immunology | Natural Killer cells | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Immunologi inom det medicinska området
Journal Article
Respiratory Medicine, ISSN 0954-6111, 2008, Volume 103, Issue 6, pp. 873 - 880
Journal Article