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FEBS Journal, ISSN 1742-464X, 05/2016, Volume 283, Issue 9, pp. 1689 - 1700
Intervertebral discs ( IVD s) provide stability and flexibility to the spinal column; however, IVD s, and in particular the nucleus pulposus ( NP ), undergo a... 
intervertebral disc degeneration | Smad | BMP | mesenchymal stem cells | nucleus pulposus | BMP7 | REGENERATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MESENCHYMAL STEM-CELLS | TRANSPLANTATION | CROSS-TALK | LUMBAR-SPINE | IN-VITRO | NUCLEUS PULPOSUS CELLS | DISEASE | GROWTH | DIFFERENTIATION | RNA, Small Interfering - genetics | Bone Morphogenetic Protein 7 - genetics | Humans | Extracellular Matrix - metabolism | Intervertebral Disc Degeneration - metabolism | Intervertebral Disc Degeneration - therapy | Collagen Type II - metabolism | Intervertebral Disc Degeneration - pathology | Lentivirus - metabolism | Glucuronosyltransferase - genetics | Mesenchymal Stromal Cells - cytology | Smad1 Protein - genetics | Lentivirus - genetics | Intervertebral Disc - pathology | Chondrocytes - metabolism | Disease Models, Animal | Intervertebral Disc Degeneration - genetics | SOX9 Transcription Factor - metabolism | Chondrocytes - pathology | Rabbits | Genetic Vectors - chemistry | Signal Transduction | Bone Marrow Cells - cytology | Glycosaminoglycans - metabolism | Keratin-8 - genetics | Gene Expression Regulation | Genetic Vectors - metabolism | Mesenchymal Stromal Cells - metabolism | Intervertebral Disc - metabolism | Aggrecans - metabolism | Smad1 Protein - antagonists & inhibitors | Aggrecans - genetics | Collagen Type II - genetics | Keratin-19 - genetics | Animals | Glucuronosyltransferase - metabolism | Keratin-8 - metabolism | Bone Morphogenetic Protein 7 - metabolism | Smad1 Protein - metabolism | Keratin-19 - metabolism | Extracellular Matrix - pathology | SOX9 Transcription Factor - genetics | Bone Marrow Cells - metabolism | Mesenchymal Stem Cell Transplantation | RNA, Small Interfering - metabolism | Physiological aspects | Bone morphogenetic proteins | Cell differentiation | Analysis | Stem cells | Extracellular matrix | Spine | Rodents | Keratins | Therapy | Surgical implants | Smad protein | Mesenchyme | Collagen (type II) | Sox9 protein | Medical services | Differentiation (biology) | Electrochemical machining | Homeostasis | Nucleus pulposus | Remodeling | Intervertebral discs | Nuclei | Flexibility | Bone marrow | Degeneration | Effectiveness | Stability | Markers | Feasibility studies | Implantation | Columns (process) | Survival | Diseases | Overexpression | Disks | Feasibility | Chondroitin sulfate | Sulfate | Aggrecan | Viability | Bone morphogenetic protein 7 | Index Medicus
Journal Article
Nature Neuroscience, ISSN 1097-6256, 03/2012, Volume 15, Issue 3, pp. 414 - 422
Cortical plasticity is most evident during a critical period in early life, but the mechanisms that restrict plasticity after the critical period are poorly... 
ADULT VISUAL-CORTEX | PARVALBUMIN | EXPERIENCE-DEPENDENT PLASTICITY | INHIBITORY NEURONS | RAT FRONTAL-CORTEX | SULFATE | CRITICAL PERIOD | CENTRAL-NERVOUS-SYSTEM | PERINEURONAL NETS | NEUROSCIENCES | OCULAR DOMINANCE PLASTICITY | Action Potentials - genetics | Chondroitin Sulfates - genetics | Chondroitin Sulfates - metabolism | Age Factors | Amphetamines - pharmacology | Humans | Visual Pathways - metabolism | Cercopithecus aethiops | Gene Expression Regulation, Developmental - genetics | Otx Transcription Factors - metabolism | Parvalbumins - metabolism | Receptors, N-Acetylglucosamine - metabolism | Visual Cortex - physiology | Neuronal Plasticity - genetics | Neuronal Plasticity - physiology | Time Factors | Up-Regulation - physiology | Functional Laterality - physiology | Action Potentials - drug effects | Gene Expression Regulation, Developmental - physiology | Animals, Newborn | Sulfotransferases - genetics | Visual Cortex - cytology | Vesicular Glutamate Transport Protein 2 - metabolism | Bacterial Proteins - genetics | Cells, Cultured | Chondroitin Sulfate Proteoglycans - metabolism | Mice, Transgenic | Up-Regulation - genetics | Aggrecans - metabolism | Electroporation - methods | Nerve Tissue Proteins - metabolism | Sensory Deprivation - physiology | Patch-Clamp Techniques | Animals | Bacterial Proteins - metabolism | Luminescent Proteins - genetics | Mice | Visual Cortex - drug effects | In Vitro Techniques | Plant Lectins - metabolism | Astrocytes - metabolism | Luminescent Proteins - metabolism | Physiological aspects | Cerebral cortex | Research | Sulfates | Chondroitin | Index Medicus
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2015, Volume 24, Issue 1, pp. 146 - 157
Summary Objective Mechanical signals control key cellular processes in articular cartilage. Previously we have shown that mechanical compression is an... 
Rheumatology | Mechanosensitivity | Cartilage | Signal transduction | TGF-beta | Smad2/3P signaling | Ageing | CHONDROCYTES | TENSILE PROPERTIES | RHEUMATOLOGY | GROWTH-FACTOR-BETA | IN-VITRO | AGE-RELATED-CHANGES | GENE-EXPRESSION | EXTRACELLULAR-MATRIX | OSTEOARTHRITIS | PROMOTER | ORTHOPEDICS | GLYCATION END-PRODUCTS | Receptors, Transforming Growth Factor beta - genetics | Transforming Growth Factor beta1 - metabolism | Gene Expression Profiling | Smad3 Protein - metabolism | Cartilage, Articular - physiology | Collagen Type II - metabolism | Smad3 Protein - genetics | Aging - genetics | Heparan Sulfate Proteoglycans - genetics | Plasminogen Activator Inhibitor 1 - metabolism | Cartilage, Articular - metabolism | Cattle | Bone Morphogenetic Protein 2 - metabolism | Smad2 Protein - genetics | Plasminogen Activator Inhibitor 1 - genetics | Protein-Serine-Threonine Kinases - metabolism | Bone Morphogenetic Protein 2 - genetics | Extracellular Matrix | Signal Transduction | Protein-Serine-Threonine Kinases - genetics | Smad2 Protein - metabolism | Osteoarthritis - metabolism | Transforming Growth Factor beta1 - genetics | Aggrecans - metabolism | Aggrecans - genetics | Pressure | Collagen Type II - genetics | Fibronectins - metabolism | Osteoarthritis - genetics | Animals | Aging - physiology | Receptors, Transforming Growth Factor beta - metabolism | Heparan Sulfate Proteoglycans - metabolism | Connective Tissue Growth Factor - genetics | Fibronectins - genetics | Connective Tissue Growth Factor - metabolism | Aging - metabolism | Index Medicus
Journal Article
Matrix Biology, ISSN 0945-053X, 2011, Volume 30, Issue 2, pp. 145 - 153
Aggrecan degradation in articular cartilage occurs predominantly through proteolysis and has been attributed to the action of members of the matrix... 
MMP | ADAMTS | Interglobular domain | Chondroitin-sulfate-2 region | Aggrecan catabolism | RHEUMATOID-ARTHRITIS | PROTEOLYTIC CLEAVAGE | CLEAVAGE SITE | ACTIVATION MECHANISMS | LARGE AGGREGATING PROTEOGLYCAN | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | HUMAN ARTICULAR-CARTILAGE | MATRIX METALLOPROTEINASES | OSTEOARTHRITIC CARTILAGE | ONCOSTATIN-M | Matrix Metalloproteinases, Secreted - metabolism | Interleukin-1beta - pharmacology | Phenylmercuric Acetate - pharmacology | Matrix Metalloproteinase 12 - genetics | Cartilage - drug effects | Oncostatin M - pharmacology | Matrix Metalloproteinase 9 - metabolism | Matrix Metalloproteinase 7 - metabolism | Cattle | Isoenzymes - metabolism | Matrix Metalloproteinase 8 - metabolism | Gelatinases - metabolism | Matrix Metalloproteinase 1 - genetics | Recombinant Proteins - metabolism | Endopeptidases - metabolism | Matrix Metalloproteinase 8 - genetics | Peptide Fragments - metabolism | Isoenzymes - genetics | Matrix Metalloproteinase 2 - metabolism | Matrix Metalloproteinase 3 - metabolism | Aggrecans - metabolism | Matrix Metalloproteinase 12 - metabolism | Cartilage - metabolism | Aggrecans - genetics | ADAM Proteins - metabolism | Enzyme Precursors - metabolism | Matrix Metalloproteinase 7 - genetics | Animals | Endopeptidases - genetics | Matrix Metalloproteinases, Secreted - genetics | Phenylmercuric Acetate - analogs & derivatives | ADAM Proteins - genetics | Cartilage - enzymology | Matrix Metalloproteinase 1 - metabolism | Matrix Metalloproteinase 3 - genetics | Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 11/2013, Volume 210, Issue 12, pp. 2569 - 2582
Journal Article
Archives of Oral Biology, ISSN 0003-9969, 02/2019, Volume 98, pp. 17 - 25
To investigate the changes in insulin-like growth factor-1 (IGF-1) expression levels in the degenerative mandibular condylar cartilage. Thirty-six rats were... 
Temporomandibular joint | Insulin-like growth factor-1 (IGF-1) | Articular cartilage | Chondrocyte | Osteoarthritis | Dental occlusion | IGF-I | PERICELLULAR MATRIX | ALKALINE-PHOSPHATASE | BINDING-PROTEINS | CHONDROCYTE HYPERTROPHY | ARTICULAR-CARTILAGE | TEMPOROMANDIBULAR-JOINT | DENTISTRY, ORAL SURGERY & MEDICINE | DEGRADATION | PLATE | EXPRESSION | Receptor, IGF Type 1 - metabolism | Up-Regulation | Alkaline Phosphatase - metabolism | Glucuronidase - metabolism | Malocclusion - complications | Gene Expression Profiling | ADAMTS5 Protein - metabolism | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Insulin-Like Growth Factor Binding Protein 3 - metabolism | Mandibular Condyle - pathology | Tissue Inhibitor of Metalloproteinase-3 - metabolism | Female | Insulin-Like Growth Factor Binding Protein 5 - metabolism | Cartilage Diseases - etiology | Disease Models, Animal | Chondrocytes - pathology | Rats | Matrix Metalloproteinase 3 - metabolism | ADAMTS4 Protein - metabolism | Aggrecans - metabolism | Rats, Sprague-Dawley | Matrix Metalloproteinase 13 - metabolism | Gene Expression Regulation, Enzymologic | Cartilage, Articular - pathology | Collagen - metabolism | Animals | Temporomandibular Joint Disorders - etiology | Mandibular Condyle - metabolism | Proliferating Cell Nuclear Antigen - metabolism | Insulin-Like Growth Factor I - metabolism | Index Medicus | Dentistry
Journal Article
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 09/2007, Volume 101, Issue 6, pp. 581 - 589
The aberrant differentiation of pericytes along the adipogenic, chondrogenic, and osteogenic lineages may contribute to the development and progression of... 
Vascular disease | Pericytes | Wnt signaling | Chondrogenesis | Differentiation | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | differentiation | MESENCHYMAL PROGENITORS | pericytes | ARTERY WALL | CHONDROCYTE DIFFERENTIATION | BETA | STEM | ATHEROSCLEROTIC PLAQUE DEVELOPMENT | IN-VITRO | vascular disease | CALCIFICATION | PERIPHERAL VASCULAR DISEASE | chondrogenesis | HEMATOLOGY | EXPRESSION | LDL-Receptor Related Proteins - metabolism | Transcription, Genetic - drug effects | Chondrogenesis - drug effects | Chondrogenesis - genetics | Adipogenesis - drug effects | RNA, Messenger - metabolism | Wnt Proteins - metabolism | Transforming Growth Factor beta3 - metabolism | Collagen Type II - metabolism | TCF Transcription Factors - metabolism | Wnt Proteins - genetics | Cattle | Lithium Chloride - pharmacology | Adipogenesis - genetics | Transduction, Genetic | High Mobility Group Proteins - metabolism | Glycosaminoglycans - metabolism | Pericytes - metabolism | Cells, Cultured | Frizzled Receptors - metabolism | Proteoglycans - metabolism | Vascular Diseases - genetics | Lipid Metabolism | Signal Transduction - genetics | Vascular Diseases - physiopathology | Aggrecans - metabolism | beta Catenin - metabolism | beta Catenin - genetics | Transcription Factors - metabolism | Animals | Signal Transduction - drug effects | Wnt3 Protein | SOX9 Transcription Factor | Vascular Diseases - metabolism | Index Medicus
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 07/2017, Volume 42, Issue 4, pp. 1277 - 1293
Background/Aims: The aim of this study was to investigate the influence of Cx43-and Smad-mediated TGF-beta/BMP signaling pathway on the differentiation of bone... 
Smad1 | BMP2 | Bone marrow mesenchymal stem cells | Cartilage differentiation | Cx43 | Osteoblast differentiation | AMINO-PROPEPTIDE | DEFECTS | PHYSIOLOGY | CHONDROGENIC DIFFERENTIATION | CELL BIOLOGY | UMBILICAL-CORD | GENE-EXPRESSION | ARTICULAR CHONDROCYTES | EXTRACELLULAR-MATRIX | MORPHOGENETIC PROTEINS | STROMAL CELLS | BMSCS | Osteopontin - genetics | Chondrocytes - cytology | Rats, Wistar | Chondrogenesis - genetics | Osteocalcin - genetics | Osteopontin - metabolism | Rats, Nude | Core Binding Factor Alpha 1 Subunit - metabolism | Collagen Type II - metabolism | Transfection | Bone Morphogenetic Protein 2 - metabolism | Injections, Subcutaneous | Mesenchymal Stem Cells - cytology | Cartilage - cytology | Smad1 Protein - genetics | Adenoviridae - genetics | Cell Differentiation | Mesenchymal Stem Cells - metabolism | Chondrocytes - metabolism | Osteogenesis - genetics | Connexin 43 - genetics | Bone Morphogenetic Protein 2 - genetics | Connexin 43 - metabolism | Genetic Vectors - chemistry | Signal Transduction | Bone Marrow Cells - cytology | Osteocalcin - metabolism | Gene Expression Regulation | Genetic Vectors - metabolism | Rats | Aggrecans - metabolism | Cartilage - metabolism | Aggrecans - genetics | Collagen Type II - genetics | Animals | Transforming Growth Factor beta - genetics | Smad1 Protein - metabolism | Adenoviridae - metabolism | Primary Cell Culture | Transforming Growth Factor beta - metabolism | Bone Marrow Cells - metabolism | Core Binding Factor Alpha 1 Subunit - genetics | Mesenchymal Stem Cell Transplantation | Biomedical materials | Tissue engineering | Stem cells | Cell cycle | Bone marrow | Arthritis | Gene expression | Potassium | Index Medicus
Journal Article
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 05/2015, Volume 67, Issue 5, pp. 1240 - 1249
Journal Article