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American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 03/2012, Volume 302, Issue 6, pp. L521 - L529
Yang S, Banerjee S, de Freitas A, Cui H, Xie N, Abraham E, Liu G. miR-21 regulates chronic hypoxia-induced pulmonary vascular remodeling. Am J Physiol Lung... 
Smooth muscle cell | Pulmonary hypertension | Micro-ribonucleic acid | LUNG FIBROSIS | ACTIVATION | PHYSIOLOGY | smooth muscle cell | pulmonary hypertension | RECEPTOR | PROLIFERATION | CANCER | ARTERY ADVENTITIAL FIBROBLASTS | micro-ribonucleic acid | RESPIRATORY SYSTEM | SMOOTH-MUSCLE-CELLS | GROWTH-FACTOR | HYPERTENSION | EXPRESSION | Cyclin D1 - metabolism | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Humans | Myocytes, Smooth Muscle - pathology | bcl-X Protein - genetics | Hypertension, Pulmonary - physiopathology | Apoptosis - genetics | Muscle, Smooth, Vascular - physiopathology | Hypoxia - metabolism | Hypertrophy, Right Ventricular - physiopathology | Pulmonary Artery - metabolism | Cell Cycle Proteins - genetics | Lung - metabolism | Airway Remodeling - physiology | Hypertrophy, Right Ventricular - metabolism | Myocytes, Smooth Muscle - metabolism | Hypertrophy, Right Ventricular - genetics | Cell Line | Bone Morphogenetic Protein Receptors, Type II - genetics | Matrix Attachment Region Binding Proteins - metabolism | Ubiquitin-Protein Ligases - metabolism | Hypertension, Pulmonary - genetics | Up-Regulation - genetics | Hypertension, Pulmonary - metabolism | Hypoxia - complications | Lung - physiopathology | Pulmonary Artery - physiopathology | Bone Morphogenetic Protein Receptors, Type II - metabolism | Down-Regulation - genetics | Hypoxia - genetics | Animals | Cyclin D1 - genetics | Matrix Attachment Region Binding Proteins - genetics | Airway Remodeling - genetics | Hypoxia - physiopathology | Mice | MicroRNAs - genetics | Ubiquitin-Protein Ligases - genetics | bcl-X Protein - metabolism | Hypertension, Pulmonary - etiology | Physiological aspects | Bone morphogenetic proteins | MicroRNA | Research | Risk factors
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 3, pp. 769 - 780
Background Inflammation and oxidative stress play critical roles in patients with chronic obstructive pulmonary disease (COPD). Mitochondrial oxidative stress... 
Allergy and Immunology | airway hyperresponsiveness | antioxidant | proliferation | MitoQ | inflammation | mitochondria | airway smooth muscle | Ozone | oxidative stress | chronic obstructive pulmonary disease | ASTHMA FEATURES | MECHANISMS | IMMUNOLOGY | FRAGMENTATION | HYPERRESPONSIVENESS | BETA | RESPONSES | ALLERGY | ENERGY-METABOLISM | EXPRESSION | COPD | Respiratory System - pathology | Reactive Oxygen Species - metabolism | Humans | Middle Aged | Male | Pulmonary Disease, Chronic Obstructive - pathology | Bronchial Hyperreactivity - genetics | Ubiquinone - pharmacology | Muscle, Smooth - drug effects | Smoking - physiopathology | Organophosphorus Compounds - pharmacology | Myocytes, Smooth Muscle - drug effects | Pneumonia - genetics | Pulmonary Disease, Chronic Obstructive - genetics | Bronchial Hyperreactivity - chemically induced | Signal Transduction | Mitochondria - pathology | Smoking - metabolism | Bronchial Hyperreactivity - pathology | Reactive Oxygen Species - antagonists & inhibitors | Electron Transport Chain Complex Proteins - metabolism | Pulmonary Disease, Chronic Obstructive - chemically induced | Respiratory System - drug effects | Airway Remodeling - genetics | Mice | Oxidative Stress - drug effects | Bronchial Hyperreactivity - drug therapy | Myocytes, Smooth Muscle - pathology | Pneumonia - pathology | Membrane Potential, Mitochondrial - drug effects | Pneumonia - chemically induced | Adult | Female | Pulmonary Disease, Chronic Obstructive - metabolism | Myocytes, Smooth Muscle - metabolism | Ubiquinone - analogs & derivatives | Gene Expression Regulation | Hydrogen Peroxide - pharmacology | Electron Transport Chain Complex Proteins - genetics | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Muscle, Smooth - metabolism | Animals | Pneumonia - drug therapy | Respiratory System - metabolism | Aged | Muscle, Smooth - pathology | Oxidative stress | Care and treatment | Lung diseases, Obstructive | Inflammation | Pharmaceutical industry | Biomedical research | Disease | Laboratories | Mortality | Colleges & universities | Smooth muscle | Mitochondrial DNA | Metabolism | Defects | Antioxidants | Mitochondria | Hospitals | Biopsy | Chronic obstructive pulmonary disease | Apoptosis | Smoking | NO, Nitric oxide | ΔΨm, Mitochondrial membrane potential | ASM, Airway smooth muscle | NAC, N-acetylcysteine | RL, Lung resistance | BAL, Bronchoalveolar lavage | OCR, Oxygen consumption rate | dTPP, Decyltriphenylphosphonium bromide | AHR, Airway hyperresponsiveness | logPC100, Concentration of acetylcholine that increased lung resistance by 100 | GOLD, Global Initiative for Chronic Obstructive Lung Disease | ATP, Adenosine triphosphate | KC, Keratinocyte-derived cytokine | JC-1, 5,5′,6,6′-Tetrachloro-1,1′,3,3′-tetraethylbenzimidazolylcarbocyanine iodide | COPD, Chronic obstructive pulmonary disease | ROS, Reactive oxygen species | Mechanisms of Allergy and Clinical Immunology
Journal Article
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 01/2014, Volume 50, Issue 1, pp. 180 - 192
Journal Article
Cellular and Molecular Life Sciences, ISSN 1420-682X, 12/2017, Volume 74, Issue 23, pp. 4305 - 4314
Periostin is a protein that plays a key role in development and repair within the biological matrix of the lung. As a matricellular protein that does not... 
Life Sciences | Biomedicine, general | Biochemistry, general | Life Sciences, general | Matricellular protein | Periostin | Fibrosis | Extracellular matrix | Cell Biology | Asthma | Extracellularmatrix | MARROW-DERIVED FIBROCYTES | SURFACTANT PROTEIN-C | PERIPHERAL-BLOOD FIBROCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | CIRCULATING FIBROCYTES | IL-13-INDUCED PERIOSTIN | CELL BIOLOGY | GROWTH-FACTOR-BETA | SERUM PERIOSTIN | IDIOPATHIC PULMONARY-FIBROSIS | INTERSTITIAL PNEUMONIAS | Cell Adhesion Molecules - genetics | Humans | Extracellular Matrix - immunology | Pulmonary Fibrosis - genetics | Th2 Cells - immunology | Cell Adhesion Molecules - immunology | Airway Remodeling - immunology | Collagen Type I - genetics | Asthma - immunology | Cell Differentiation | Th2 Cells - pathology | Biomarkers - metabolism | Lung - pathology | Signal Transduction | Gene Expression Regulation | Pulmonary Fibrosis - immunology | Pulmonary Fibrosis - pathology | Fibroblasts - pathology | Asthma - genetics | Collagen Type I - immunology | Airway Remodeling - genetics | Fibroblasts - immunology | Asthma - pathology | Extracellular Matrix - pathology | Lung - immunology | Medical research | Collagen | Mortality | Medicine, Experimental | Development and progression | Secretion | Lung diseases | Organs | Mucus | Lymphocytes T | Remodeling | Patients | Morbidity | Respiratory tract | Proteins | Serum levels | Airway management | Pulmonary fibrosis | Lungs | Respiratory diseases | Biomarkers | Bioindicators | Adults | Aberration
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2014, Volume 192, Issue 8, pp. 3475 - 3487
Journal Article
Journal Article
Cell death & disease, ISSN 2041-4889, 08/2017, Volume 8, Issue 8, pp. e2998 - e2998
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2010, Volume 125, Issue 2, pp. S81 - S94
Journal Article
Journal of Immunology, ISSN 0022-1767, 07/2010, Volume 185, Issue 2, pp. 1205 - 1214
Journal Article
Clinical & Experimental Allergy, ISSN 0954-7894, 09/2016, Volume 46, Issue 9, pp. 1152 - 1161
Journal Article