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American journal of respiratory cell and molecular biology, ISSN 1044-1549, 11/2015, Volume 53, Issue 5, pp. 664 - 675
We have previously demonstrated increased airway smooth muscle (ASM) mass and airway hyperresponsiveness in whole-life vitamin D-deficient female mice. In this... 
RNA sequencing | mouse model | inflammation | vitamin D | airway remodeling | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | CATENIN | CELL BIOLOGY | RESPONSES | RESPIRATORY SYSTEM | GROWTH | DISEASE | RESISTANCE | ASTHMA | CHECKPOINT | EXPRESSION | Receptor, Epidermal Growth Factor - genetics | Bronchial Hyperreactivity - immunology | E1A-Associated p300 Protein - genetics | Calgranulin A - genetics | Bronchial Hyperreactivity - genetics | Matrix Metalloproteinase 9 - genetics | Calgranulin A - immunology | Vitamin D Deficiency - immunology | Neutrophils - metabolism | Disease Models, Animal | Lymphocytes - metabolism | Vitamin D Deficiency - genetics | Signal Transduction | Respiratory Hypersensitivity - metabolism | Respiratory Hypersensitivity - immunology | Peptide Initiation Factors - immunology | Wnt Proteins - immunology | Respiratory Hypersensitivity - genetics | Lymphocytes - pathology | Mice | Bronchoalveolar Lavage Fluid - chemistry | Vitamin D Deficiency - metabolism | Vitamin D Deficiency - complications | E1A-Associated p300 Protein - immunology | Matrix Metalloproteinase 9 - immunology | NF-kappa B - immunology | Muscle, Smooth - immunology | Airway Remodeling - immunology | Lymphocytes - immunology | Wnt Proteins - genetics | Calgranulin B - immunology | Bronchial Hyperreactivity - metabolism | Female | Calgranulin B - genetics | Lung - metabolism | Neutrophils - pathology | Receptor, Epidermal Growth Factor - immunology | Lung - pathology | Gene Expression Regulation | Neutrophils - immunology | Respiratory Hypersensitivity - complications | Muscle, Smooth - metabolism | Bronchial Hyperreactivity - complications | Pregnancy | Peptide Initiation Factors - genetics | Animals | NF-kappa B - genetics | Airway Resistance - immunology | Muscle, Smooth - pathology | Lung - immunology | Studies | Data analysis | Vitamin D | Rodents | Vitamin deficiency | Smooth muscle | Software | Chronic obstructive pulmonary disease | Genetic engineering | Inflammation | Gene expression | Growth factors
Journal Article
Nature Reviews Immunology, ISSN 1474-1733, 12/2003, Volume 3, Issue 12, pp. 994 - 1003
Dendritic cells (DCs) are mainly known for their capacity to induce primary immune responses. Here, we propose that DCs are similarly essential for controlling... 
Models, Immunological | Animals | Allergens - immunology | Dendritic Cells - immunology | Humans | Airway Resistance - immunology | Asthma - immunology | Hypersensitivity - immunology | Mice
Journal Article
European Respiratory Journal, ISSN 0903-1936, 02/2012, Volume 39, Issue 2, pp. 439 - 445
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 5/2013, Volume 91, Issue 5, pp. 637 - 644
Hypoxia-inducible factor (HIF)-1α is a master regulator of inflammatory activities of myeloid cells, including neutrophils and macrophages. These studies... 
Human Genetics | Hypoxia inducible factor (HIF)-1α | Biomedicine | Allergic inflammation | Internal Medicine | Molecular Medicine | Chemotaxis | Asthma | Eosinophils | Airway hyperresponsiveness | MEDICINE, RESEARCH & EXPERIMENTAL | YC-1 | PHENOTYPES | MODEL | HYPOXIA | HIF-2-ALPHA | INFLAMMATION | DISEASE | GENETICS & HEREDITY | Hypoxia inducible factor (HIF)-1 alpha | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Bronchoalveolar Lavage Fluid - immunology | Interleukin-13 - immunology | Interleukin-5 - genetics | Eosinophils - immunology | Hypoxia-Inducible Factor 1, alpha Subunit - deficiency | Goblet Cells - immunology | Interleukin-13 - genetics | Asthma - immunology | Female | Bronchoalveolar Lavage Fluid - cytology | Eosinophils - pathology | Interleukin-4 - genetics | Basic Helix-Loop-Helix Transcription Factors - deficiency | Hypoxia-Inducible Factor 1, alpha Subunit - immunology | Ovalbumin | Lung - pathology | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Chemotaxis - drug effects | Asthma - chemically induced | Mice, Knockout | Indazoles - pharmacology | Interleukin-5 - immunology | Interleukin-4 - immunology | Animals | Airway Resistance - immunology | Basic Helix-Loop-Helix Transcription Factors - immunology | Mice | Goblet Cells - pathology | Asthma - pathology | Lung - immunology | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | Hypoxia inducible factor (HIF)-1 alpha; Asthma; Allergic inflammation; Eosinophils; Chemotaxis; Airway hyperresponsiveness | MEDICIN
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 08/2012, Volume 47, Issue 2, pp. 209 - 217
Little is known about the mechanisms of persistent airflow obstruction that result from chronic occupational endotoxin exposure. We sought to analyze the... 
Airway resistance | Dendritic cells | Macrophages | Endotoxin | Neutrophils | Inhalation | FUNGAL-INFECTION | IMMUNE-RESPONSE | LPS INHALATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | RETINOIC-ACID | ALVEOLAR MACROPHAGE ELIMINATION | CELL BIOLOGY | inhalation | macrophages | dendritic cells | RESPIRATORY SYSTEM | COTTON TEXTILE WORKERS | IN-VIVO | REGULATORY T-CELLS | CIGARETTE-SMOKE | MICE | airway resistance | neutrophils | endotoxin | Leukocytes, Mononuclear - metabolism | Up-Regulation | Bronchial Hyperreactivity - immunology | Dendritic Cells - immunology | Occupational Exposure | Bronchoalveolar Lavage Fluid - immunology | Male | Pulmonary Disease, Chronic Obstructive - pathology | Airway Resistance - genetics | Bronchial Hyperreactivity - genetics | Lipopolysaccharides - immunology | Antigen-Presenting Cells - pathology | Myeloid Cells - immunology | Pneumonia - immunology | Models, Animal | Myeloid Cells - drug effects | Interleukin-6 - metabolism | Neutrophils - metabolism | Pneumonia - genetics | Pulmonary Disease, Chronic Obstructive - genetics | Antigen-Presenting Cells - metabolism | Gene Expression | Leukocytes, Mononuclear - drug effects | Interleukin-6 - genetics | Macrophages - pathology | Neutrophils - drug effects | Bronchial Hyperreactivity - pathology | Macrophages - metabolism | Interleukin-10 - genetics | Lung - drug effects | Interleukin-6 - immunology | Lipopolysaccharides - pharmacology | Mice | Myeloid Cells - pathology | Pneumonia - metabolism | Pulmonary Disease, Chronic Obstructive - immunology | Interleukin-10 - immunology | Endotoxins - pharmacology | Airway Resistance - drug effects | Pneumonia - pathology | Dendritic Cells - pathology | Leukocytes, Mononuclear - immunology | Interleukin-10 - metabolism | Bronchial Hyperreactivity - metabolism | Dendritic Cells - drug effects | Lung - metabolism | Pulmonary Disease, Chronic Obstructive - metabolism | Dendritic Cells - metabolism | Macrophages - immunology | Neutrophils - pathology | Lung - pathology | Mice, Inbred C57BL | Neutrophils - immunology | Antigen-Presenting Cells - immunology | Leukocytes, Mononuclear - pathology | Animals | Endotoxins - immunology | Airway Resistance - immunology | Myeloid Cells - metabolism | Lung - immunology
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 02/2017, Volume 195, Issue 3, pp. 302 - 313
Journal Article