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日本救急医学会雑誌, ISSN 0915-924X, 2009, Volume 20, Issue 2, pp. 67 - 76
目的:シベレスタットナトリウム水和物(シベレスタット)投与を早期に行うことの効果を検討すること。方法:シベレスタットを使用した様々な原疾患によるacute lung injury/acute respiratory distress... 
P/F比 | 好中球エラスターゼ阻害剤 | 生存率 | 投与時期
Journal Article
Biochimica et Biophysica Acta - Molecular Basis of Disease, ISSN 0925-4439, 10/2009, Volume 1792, Issue 10, pp. 941 - 953
Phospholipases A (PLA ) catalyse the cleavage of fatty acids esterified at the sn-2 position of glycerophospholipids. In acute lung injury-acute respiratory... 
PLA | Inhibitors of PLA | PAF-AH | Lung surfactant | Phospholipase A | ALI | ARDS | Index Medicus
Journal Article
Lung, ISSN 0341-2040, 12/2019, Volume 197, Issue 6, pp. 819 - 823
Lysozyme is an important component of the innate immune system and has roles in peptidoglycan cleavage of gram-positive organisms. Myeloid cells highly express... 
Acute lung injury | Pneumology/Respiratory System | LysMCre | Medicine & Public Health | Murine | Lysozyme | ALI | Medical research | Care and treatment | Lung cancer | Medicine, Experimental | Research | Gene expression
Journal Article
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 2009, Volume 1792, Issue 10, pp. 941 - 953
Phospholipases A 2 (PLA 2) catalyse the cleavage of fatty acids esterified at the sn-2 position of glycerophospholipids. In acute lung injury-acute respiratory... 
Phospholipase A 2 | PAF-AH | Lung surfactant | Inhibitors of PLA 2 | PLA 2 | ALI | ARDS
Journal Article
Journal Article
Journal Article
Inflammation, ISSN 0360-3997, 4/2007, Volume 30, Issue 1, pp. 14 - 27
The hypothesis that the expression of protease-activated receptors (PARs) protein is regulated at the level of transcription and that PAR isoforms, PAR-1,... 
Pathology | Medicine & Public Health | protease-activated receptor (PAR) | inflammation | Rheumatology | Internal Medicine | Pharmacology/Toxicology | coagulation | acute lung injury (ALI) | endotoxin | Inflammation | Acute lung injury (ALI) | Protease-activated receptor (PAR) | Endotoxin | Coagulation | CELLS | THROMBIN | TISSUE | SEPSIS | IMMUNOLOGY | CELL BIOLOGY | FIBRINOLYSIS | PROTEASE-ACTIVATED RECEPTORS | Blood Pressure | Immunohistochemistry | Respiratory Distress Syndrome, Adult - pathology | Tumor Necrosis Factor-alpha - metabolism | Receptors, Proteinase-Activated - metabolism | Epithelial Cells - metabolism | Respiratory Distress Syndrome, Adult - physiopathology | Rats, Wistar | Receptor, PAR-1 - metabolism | Receptor, PAR-2 - metabolism | Tumor Necrosis Factor-alpha - blood | Male | RNA, Messenger - metabolism | Receptors, Proteinase-Activated - genetics | Respiratory Distress Syndrome, Adult - metabolism | Lipopolysaccharides | Time Factors | Pulmonary Alveoli - metabolism | Transcription, Genetic | Lung - metabolism | Disease Models, Animal | Lung - pathology | Pulmonary Alveoli - pathology | Respiratory Distress Syndrome, Adult - chemically induced | Endothelial Cells - metabolism | Rats | Nitric Oxide Synthase Type II - blood | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Oxygen - blood | Animals | Receptors, Thrombin - metabolism | Macrophages, Alveolar - metabolism | Fibrin - metabolism | Nitric Oxide Synthase Type II - metabolism
Journal Article