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The Journal of Pathology, ISSN 0022-3417, 04/2017, Volume 241, Issue 5, pp. 589 - 599
Diabetic nephropathy is the leading cause of end‐stage renal disease. Diabetic patients have increased plasma concentrations of apolipoprotein C‐I (apoCI), and... 
macrophages | apolipoprotein CI | glomerulopathy | inflammation | diabetes | DB/DB MICE | ATHEROSCLEROSIS | PATHOLOGY | KNOCKOUT MICE | ADHESION | ONCOLOGY | PROGRESSIVE DIABETIC-NEPHROPATHY | RENAL INJURY | LIPOPROTEINS | ASSOCIATION | TRANSGENIC MICE | Apolipoprotein C-I - metabolism | Diabetic Nephropathies - etiology | Humans | Macrophages, Peritoneal - pathology | Male | Apolipoprotein C-I - genetics | Brain - metabolism | Kidney Failure, Chronic - pathology | Myocardium - metabolism | Kidney Glomerulus - metabolism | Female | Lung - metabolism | Spleen - pathology | Albuminuria - pathology | Diabetic Nephropathies - pathology | Lung - pathology | Albuminuria - etiology | Cytokines - metabolism | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Pancreas - pathology | Mice, Transgenic | Myocardium - pathology | Kidney Glomerulus - pathology | Pancreas - metabolism | Kidney Failure, Chronic - metabolism | Animals | Spleen - metabolism | Brain - pathology | Aged | Mice | Macrophages, Peritoneal - metabolism | Kidney Failure, Chronic - etiology | Fibronectins | Diabetics | Diabetic nephropathies | Development and progression | Transforming growth factors | Macrophages | Apolipoproteins | Chronic kidney failure | Collagen | Analysis | Von Willebrand factor | Bone morphogenetic proteins | Genetic engineering
Journal Article
Kidney International, ISSN 0085-2538, 06/2018, Volume 93, Issue 6, pp. 1356 - 1366
Immunoglobulin A (IgA) nephropathy (IgAN), the most common glomerulonephritis worldwide, is characterized by IgA depositions in the kidney. Deficiency of CD37,... 
CD37 | IgA nephropathy | glomerulonephritis | IL-6 | CD151 | RECEPTOR | TETRASPANIN CD37 PROTECTS | B-CELL LYMPHOMA | DISEASE | MESANGIAL CELLS | UROLOGY & NEPHROLOGY | GROWTH-FACTOR | KIDNEY | Cell Proliferation | Humans | Glomerulonephritis, IGA - immunology | Kidney Glomerulus - physiopathology | Neutrophil Infiltration | Tetraspanins - blood | Antigens, CD - genetics | Case-Control Studies | Tetraspanins - deficiency | Antigens, Neoplasm - blood | Kidney Glomerulus - metabolism | Albuminuria - metabolism | Tetraspanins - genetics | Interleukin-6 - metabolism | B-Lymphocytes - metabolism | Glomerulonephritis, IGA - pathology | Disease Models, Animal | Antigens, Neoplasm - genetics | Genetic Predisposition to Disease | Immunoglobulin A - metabolism | Podocytes - metabolism | Interleukin-6 - genetics | Mice, Inbred C57BL | Interleukin-6 - deficiency | Kidney Glomerulus - pathology | Podocytes - pathology | Albuminuria - prevention & control | Mice, Knockout | Glomerulonephritis, IGA - prevention & control | Phenotype | Animals | B-Lymphocytes - immunology | Immunoglobulin A - immunology | Podocytes - immunology | Albuminuria - immunology | Glomerulonephritis, IGA - metabolism | Kidney Glomerulus - immunology | Glomerulonephritis | Flow cytometry | Renal function | Kidneys | Cytokines | Neutrophils | Tumor necrosis factor-α | Nitrogen | Immunoglobulin A | Experiments | Endothelial cells | Lipopolysaccharides | Interleukin 6 | Proteins | Pathology | Cell activation | Microscopy | Lymphocytes B | Rodents | Interleukin 10 | Renal failure | Tumor necrosis factor-TNF | Immune system
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 6, pp. 1991 - 2002
Inflammation plays a key role in the development and progression of diabetic kidney disease; however, the role of the anti-inflammatory molecule netrin-1 in... 
Pathology | ISCHEMIA-REPERFUSION INJURY | CELLS | ACTIVATION | MONOCYTE CHEMOATTRACTANT | INHIBITION | INSULIN-RESISTANCE | PROTECTS KIDNEY | PROTEINURIA | PATHOLOGY | UNC5B RECEPTOR | NEPHROPATHY | Organ Specificity - drug effects | Albuminuria - complications | Inflammation - pathology | Leukocytes - pathology | Gene Expression - drug effects | Apoptosis - drug effects | Diabetes Mellitus, Experimental - enzymology | Dinoprostone - biosynthesis | Netrin-1 | Albuminuria - blood | Epithelial Cells - drug effects | Hyperglycemia - complications | NF-kappa B - metabolism | Nerve Growth Factors - metabolism | Inflammation - blood | Mesangial Cells - drug effects | Necrosis | Diabetes Mellitus, Experimental - blood | Inflammation - complications | Hyperglycemia - pathology | Diabetes Mellitus, Experimental - complications | Nerve Growth Factors - blood | Kidney Tubules, Proximal - enzymology | Albuminuria - pathology | Kidney Tubules, Proximal - pathology | Tumor Suppressor Proteins - metabolism | Transgenes - genetics | Cytokines - secretion | Epithelial Cells - pathology | Mice, Transgenic | Glucose - pharmacology | Mesangial Cells - metabolism | Albuminuria - enzymology | Cell Movement - drug effects | Animals | Tumor Suppressor Proteins - blood | Hyperglycemia - blood | Chickens | Cyclooxygenase 2 - metabolism | Epithelial Cells - enzymology | Mesangial Cells - pathology | Diabetes Mellitus, Experimental - pathology | Hyperglycemia - enzymology | Leukocytes - drug effects | Mice | Inflammation - enzymology | Kidney Tubules, Proximal - drug effects | Regular
Journal Article
Diabetes Care, ISSN 0149-5992, 02/2015, Volume 38, Issue 2, pp. 206 - 212
OBJECTIVEAlbuminuria and reduced kidney function are associated with cognitive impairment. Relationships between nephropathy and cerebral structural changes... 
LESION VOLUME | BRAIN ATROPHY | DEMENTIA | MRI | ALBUMINURIA | ENDOCRINOLOGY & METABOLISM | COGNITIVE IMPAIRMENT | WHITE-MATTER HYPERINTENSITIES | MELLITUS | TYPE-2 | VOXEL-BASED MORPHOMETRY | Albuminuria - complications | Glycated Hemoglobin A - metabolism | Humans | Middle Aged | Atrophy - pathology | Male | Brain Diseases - ethnology | Renal Insufficiency, Chronic - complications | European Continental Ancestry Group - ethnology | Glomerular Filtration Rate - physiology | Diabetes Mellitus, Type 2 - ethnology | Cognition Disorders - etiology | Adult | Female | Cognition Disorders - ethnology | Diabetes Mellitus, Type 2 - complications | Atrophy - ethnology | Albuminuria - pathology | Diabetic Nephropathies - pathology | Cross-Sectional Studies | Cognition Disorders - pathology | Albuminuria - ethnology | Linear Models | Diabetic Nephropathies - ethnology | Hypertension - pathology | Renal Insufficiency, Chronic - pathology | African Americans - ethnology | Diabetic Nephropathies - complications | Magnetic Resonance Imaging | Brain Diseases - pathology | Renal Insufficiency, Chronic - ethnology | Brain Diseases - complications | Hypertension - ethnology | Brain - pathology | Hypertension - complications | Aged | Diabetes Mellitus, Type 2 - pathology | Blood Glucose - metabolism | Type 2 diabetes | Care and treatment | Analysis | African Americans | Kidney diseases | Health aspects | Risk factors | Glomerular filtration rate | Health Disparities in Diabetes
Journal Article
Diabetologia, ISSN 0012-186X, 5/2008, Volume 51, Issue 5, pp. 714 - 725
Journal Article
Journal Article
Cell Metabolism, ISSN 1550-4131, 03/2017, Volume 25, Issue 3, pp. 713 - 726
Diabetic kidney disease (DKD) is the most common cause of severe renal disease, and few treatment options are available today that prevent the progressive loss... 
diabetic kidney disease | albuminuria | insulin signaling | podocytes | lipotoxicity | endothelial fatty acid transport | vascular endothelial growth factor B | renal steatosis | HEART | LIPID-METABOLISM | GROWTH-FACTOR-B | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | ACCUMULATION | NEPHROPATHY | PODOCYTE | ADIPOSE | CELL BIOLOGY | Albuminuria - complications | Antibodies, Neutralizing - administration & dosage | Dyslipidemias - complications | Kidney - pathology | Humans | Middle Aged | Diabetes Mellitus, Type 1 - metabolism | Male | Diabetes Mellitus, Type 1 - complications | Diabetes Mellitus, Type 2 - metabolism | Dyslipidemias - pathology | Young Adult | Kidney - metabolism | Fatty Acid Transport Proteins - metabolism | Gene Deletion | Adult | Female | Albuminuria - metabolism | Diabetes Mellitus, Experimental - complications | Diabetes Mellitus, Experimental - metabolism | Diabetic Nephropathies - prevention & control | Lipids - toxicity | Diabetes Mellitus, Type 2 - complications | Kidney - physiopathology | Disease Models, Animal | Albuminuria - pathology | Insulin - pharmacology | Diabetic Nephropathies - pathology | Kidney - drug effects | Podocytes - metabolism | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Diabetes Mellitus, Type 1 - pathology | Antibodies, Neutralizing - pharmacology | Podocytes - pathology | Disease Progression | Up-Regulation - drug effects | Animals | Podocytes - drug effects | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Diabetes Mellitus, Experimental - pathology | Dyslipidemias - metabolism | Aged | Diabetes Mellitus, Type 2 - pathology | Endothelial growth factors | Analysis | Stem cells | Development and progression | Mice | Kidney diseases | Fatty acids | Endothelium | Insulin | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Diabetes, ISSN 0012-1797, 10/2002, Volume 51, Issue 10, pp. 3090 - 3094
Amelioration of Long-Term Renal Changes in Obese Type 2 Diabetic Mice by a Neutralizing Vascular Endothelial Growth Factor Antibody Allan Flyvbjerg 1 ,... 
FACTOR VEGF | FACTOR-BETA | INHIBITION | HUMAN MESANGIAL CELLS | PERMEABILITY FACTOR | ENDOCRINOLOGY & METABOLISM | PREVENTION | RECEPTOR | ANGIOTENSIN-II | DYSFUNCTION | EXPRESSION | Diabetes Mellitus - pathology | Vascular Endothelial Growth Factor A | Liver - pathology | Diabetic Nephropathies - etiology | Vascular Endothelial Growth Factors | Hyperglycemia - complications | Insulin - blood | Basement Membrane - pathology | Endothelial Growth Factors - immunology | Albuminuria - physiopathology | Diabetic Nephropathies - physiopathology | Mice, Mutant Strains | Hyperglycemia - pathology | Immunoglobulin G - pharmacology | Diabetes Mellitus - etiology | Female | Hyperglycemia - physiopathology | Diabetes Mellitus, Type 2 - complications | Ketones - urine | Disease Models, Animal | Hyperinsulinism - physiopathology | Albuminuria - pathology | Eating | Obesity | Diabetic Nephropathies - pathology | Hyperinsulinism - complications | Albuminuria - etiology | Mice, Inbred C57BL | Organ Size | Blood Glucose | Myocardium - pathology | Creatinine - urine | Antibodies - pharmacology | Hyperinsulinism - pathology | Animals | Diabetes Mellitus, Type 2 - physiopathology | Diabetes Mellitus - physiopathology | Lymphokines - immunology | Mice | Diabetes Mellitus, Type 2 - pathology | Intercellular Signaling Peptides and Proteins - immunology | Physiological aspects | Care and treatment | Vascular endothelial growth factor | Diabetic nephropathies
Journal Article
Diabetes, ISSN 0012-1797, 02/2011, Volume 60, Issue 2, pp. 590 - 601
Journal Article