Articles

Search Articles

Advanced search
Help

Catalogue

Articles

Databases

X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (3982) 3982
Publication (497) 497
Book Review (16) 16
Book Chapter (15) 15
Book / eBook (4) 4
Conference Proceeding (4) 4
Dissertation (3) 3
Magazine Article (1) 1
Newsletter (1) 1
Paper (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
male (2475) 2475
humans (2467) 2467
female (1839) 1839
index medicus (1831) 1831
albuminuria (1804) 1804
animals (1583) 1583
middle aged (1216) 1216
urology & nephrology (1002) 1002
adult (1001) 1001
rats (852) 852
kidney - pathology (850) 850
aged (771) 771
nephropathy (724) 724
risk factors (705) 705
microalbuminuria (690) 690
mice (677) 677
diabetes (673) 673
endocrinology & metabolism (646) 646
glomerular filtration rate (632) 632
albuminuria - etiology (615) 615
diabetic nephropathies - pathology (585) 585
proteinuria (578) 578
hypertension (573) 573
albuminuria - urine (570) 570
kidney glomerulus - pathology (506) 506
diabetic nephropathy (502) 502
albuminuria - pathology (477) 477
albuminuria - metabolism (448) 448
blood pressure (434) 434
disease (424) 424
nephrology (421) 421
kidney diseases (406) 406
albuminuria - physiopathology (381) 381
creatinine - urine (353) 353
prevalence (351) 351
disease progression (348) 348
diabetic nephropathies - physiopathology (347) 347
diabetes mellitus, type 2 - complications (346) 346
albuminuria - complications (339) 339
expression (339) 339
diabetic nephropathies (338) 338
disease models, animal (338) 338
mellitus (338) 338
kidney (333) 333
peripheral vascular disease (325) 325
blood-pressure (322) 322
adolescent (317) 317
risk (313) 313
kidney - metabolism (310) 310
kidney - physiopathology (308) 308
time factors (308) 308
mortality (307) 307
kidney - drug effects (303) 303
creatinine - blood (301) 301
albuminuria - diagnosis (299) 299
glomerular-filtration-rate (294) 294
research (285) 285
albumin (282) 282
analysis (281) 281
diabetic nephropathies - metabolism (279) 279
progression (275) 275
type 2 diabetes (275) 275
urine (271) 271
chronic kidney disease (269) 269
oxidative stress (269) 269
mice, inbred c57bl (266) 266
diabetes mellitus (261) 261
chronic kidney-disease (258) 258
physiology (255) 255
inflammation (254) 254
cross-sectional studies (250) 250
albuminuria - drug therapy (249) 249
kidneys (247) 247
research article (243) 243
biomarkers - urine (240) 240
albuminuria - prevention & control (236) 236
article (228) 228
diabetic nephropathies - etiology (227) 227
prospective studies (225) 225
albuminuria - epidemiology (223) 223
rats, sprague-dawley (223) 223
blood glucose - metabolism (220) 220
pathology (219) 219
urologic and male genital diseases (219) 219
diabetic nephropathies - prevention & control (218) 218
fibrosis (214) 214
medicine (213) 213
chronic kidney failure (211) 211
child (207) 207
medicine & public health (205) 205
medicine, research & experimental (205) 205
hypertension - physiopathology (204) 204
injury (203) 203
health aspects (202) 202
hypertension - complications (202) 202
diabetic nephropathies - drug therapy (200) 200
biomarkers (197) 197
blood pressure - drug effects (197) 197
prognosis (197) 197
biomarkers - blood (196) 196
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (3821) 3821
Japanese (51) 51
French (33) 33
Russian (26) 26
German (23) 23
Chinese (17) 17
Spanish (10) 10
Italian (7) 7
Polish (6) 6
Portuguese (2) 2
Slovak (2) 2
Czech (1) 1
Dutch (1) 1
Hungarian (1) 1
Romanian (1) 1
Ukrainian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


1. Full Text Renal albumin absorption in physiology and pathology
by Birn, H and Christensen, E.I
Kidney International, ISSN 0085-2538, 02/2006, Volume 69, Issue 3, pp. 440 - 449
Albumin is the most abundant plasmaprotein serving multiple functions as a carrier of metabolites, hormones, vitamins, and drugs, as an acid/base buffer, as... 
kidney | endocytosis | fibrosis | proximal tubule | failure | progression of chronic renal | Proximal tubule | Endocytosis | Failure | Kidney | Progression of chronic renal | Fibrosis | HEREDITARY MEGALOBLASTIC-ANEMIA | CUBILIN-MEDIATED ENDOCYTOSIS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | FACTOR-COBALAMIN RECEPTOR | OPOSSUM KIDNEY-CELLS | FACTOR-VITAMIN B-12 | PROXIMAL TUBULE CELLS | UROLOGY & NEPHROLOGY | BRUSH-BORDER EXPRESSION | NF-KAPPA-B | RECEPTOR-ASSOCIATED PROTEIN | Albuminuria - pathology | Kidney Diseases - physiopathology | Kidney Tubules - physiopathology | Glomerular Filtration Rate | Receptors, Albumin - physiology | Kidney - pathology | Low Density Lipoprotein Receptor-Related Protein-2 - physiology | Albumins - metabolism | Kidney Diseases - pathology | Humans | Kidney Tubules - physiology | Inflammation | Albuminuria - physiopathology | Kidney - metabolism | Absorption | Animals | Kidney - chemistry | Receptors, Cell Surface - physiology | Kidney Diseases - etiology | Kidney Tubules - pathology | Albuminuria - urine | Kidney - physiopathology
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
2. Full Text Apolipoprotein C‐I plays a role in the pathogenesis of glomerulosclerosis
by Bus, Pascal and Pierneef, Louise and Bor, Rosalie and Wolterbeek, Ron and van Es, Leendert A and Rensen, Patrick CN and de Heer, Emile and Havekes, Louis M and Bruijn, Jan A and Berbée, Jimmy F and Baelde, Hans J
The Journal of Pathology, ISSN 0022-3417, 04/2017, Volume 241, Issue 5, pp. 589 - 599
Diabetic nephropathy is the leading cause of end‐stage renal disease. Diabetic patients have increased plasma concentrations of apolipoprotein C‐I (apoCI), and... 
macrophages | apolipoprotein CI | glomerulopathy | inflammation | diabetes | DB/DB MICE | ATHEROSCLEROSIS | PATHOLOGY | KNOCKOUT MICE | ADHESION | ONCOLOGY | PROGRESSIVE DIABETIC-NEPHROPATHY | RENAL INJURY | LIPOPROTEINS | ASSOCIATION | TRANSGENIC MICE | Apolipoprotein C-I - metabolism | Diabetic Nephropathies - etiology | Humans | Macrophages, Peritoneal - pathology | Male | Apolipoprotein C-I - genetics | Brain - metabolism | Kidney Failure, Chronic - pathology | Myocardium - metabolism | Kidney Glomerulus - metabolism | Female | Lung - metabolism | Spleen - pathology | Albuminuria - pathology | Diabetic Nephropathies - pathology | Lung - pathology | Albuminuria - etiology | Cytokines - metabolism | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Gene Expression Regulation | Pancreas - pathology | Mice, Transgenic | Myocardium - pathology | Kidney Glomerulus - pathology | Pancreas - metabolism | Kidney Failure, Chronic - metabolism | Animals | Spleen - metabolism | Brain - pathology | Aged | Mice | Macrophages, Peritoneal - metabolism | Kidney Failure, Chronic - etiology | Fibronectins | Diabetics | Diabetic nephropathies | Development and progression | Transforming growth factors | Macrophages | Apolipoproteins | Chronic kidney failure | Collagen | Analysis | Von Willebrand factor | Bone morphogenetic proteins | Genetic engineering
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
3. Full Text Interleukin-6 is essential for glomerular immunoglobulin A deposition and the development of renal pathology in Cd37-deficient mice
by Rops, Angelique L.W.M.M and Jansen, Erik and van der Schaaf, Alie and Pieterse, Elmar and Rother, Nils and Hofstra, Julia and Dijkman, Henry B.P.M and van de Logt, Anne-Els and Wetzels, Jack and van der Vlag, Johan and van Spriel, Annemiek B
Kidney International, ISSN 0085-2538, 06/2018, Volume 93, Issue 6, pp. 1356 - 1366
Immunoglobulin A (IgA) nephropathy (IgAN), the most common glomerulonephritis worldwide, is characterized by IgA depositions in the kidney. Deficiency of CD37,... 
CD37 | IgA nephropathy | glomerulonephritis | IL-6 | CD151 | RECEPTOR | TETRASPANIN CD37 PROTECTS | B-CELL LYMPHOMA | DISEASE | MESANGIAL CELLS | UROLOGY & NEPHROLOGY | GROWTH-FACTOR | KIDNEY | Cell Proliferation | Humans | Glomerulonephritis, IGA - immunology | Kidney Glomerulus - physiopathology | Neutrophil Infiltration | Tetraspanins - blood | Antigens, CD - genetics | Case-Control Studies | Tetraspanins - deficiency | Antigens, Neoplasm - blood | Kidney Glomerulus - metabolism | Albuminuria - metabolism | Tetraspanins - genetics | Interleukin-6 - metabolism | B-Lymphocytes - metabolism | Glomerulonephritis, IGA - pathology | Disease Models, Animal | Antigens, Neoplasm - genetics | Genetic Predisposition to Disease | Immunoglobulin A - metabolism | Podocytes - metabolism | Interleukin-6 - genetics | Mice, Inbred C57BL | Interleukin-6 - deficiency | Kidney Glomerulus - pathology | Podocytes - pathology | Albuminuria - prevention & control | Mice, Knockout | Glomerulonephritis, IGA - prevention & control | Phenotype | Animals | B-Lymphocytes - immunology | Immunoglobulin A - immunology | Podocytes - immunology | Albuminuria - immunology | Glomerulonephritis, IGA - metabolism | Kidney Glomerulus - immunology | Glomerulonephritis | Flow cytometry | Renal function | Kidneys | Cytokines | Neutrophils | Tumor necrosis factor-α | Nitrogen | Immunoglobulin A | Experiments | Endothelial cells | Lipopolysaccharides | Interleukin 6 | Proteins | Pathology | Cell activation | Microscopy | Lymphocytes B | Rodents | Interleukin 10 | Renal failure | Tumor necrosis factor-TNF | Immune system
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
4. Full Text Kidney Proximal Tubular Epithelial-Specific Overexpression of Netrin-1 Suppresses Inflammation and Albuminuria through Suppression of COX-2-Mediated PGE2 Production in Streptozotocin-Induced Diabetic Mice
by Mohamed, Riyaz and Jayakumar, Calpurnia and Ranganathan, Punithavathi V and Ganapathy, Vadivel and Ramesh, Ganesan
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 6, pp. 1991 - 2002
Inflammation plays a key role in the development and progression of diabetic kidney disease; however, the role of the anti-inflammatory molecule netrin-1 in... 
Pathology | ISCHEMIA-REPERFUSION INJURY | CELLS | ACTIVATION | MONOCYTE CHEMOATTRACTANT | INHIBITION | INSULIN-RESISTANCE | PROTECTS KIDNEY | PROTEINURIA | PATHOLOGY | UNC5B RECEPTOR | NEPHROPATHY | Organ Specificity - drug effects | Albuminuria - complications | Inflammation - pathology | Leukocytes - pathology | Gene Expression - drug effects | Apoptosis - drug effects | Diabetes Mellitus, Experimental - enzymology | Dinoprostone - biosynthesis | Netrin-1 | Albuminuria - blood | Epithelial Cells - drug effects | Hyperglycemia - complications | NF-kappa B - metabolism | Nerve Growth Factors - metabolism | Inflammation - blood | Mesangial Cells - drug effects | Necrosis | Diabetes Mellitus, Experimental - blood | Inflammation - complications | Hyperglycemia - pathology | Diabetes Mellitus, Experimental - complications | Nerve Growth Factors - blood | Kidney Tubules, Proximal - enzymology | Albuminuria - pathology | Kidney Tubules, Proximal - pathology | Tumor Suppressor Proteins - metabolism | Transgenes - genetics | Cytokines - secretion | Epithelial Cells - pathology | Mice, Transgenic | Glucose - pharmacology | Mesangial Cells - metabolism | Albuminuria - enzymology | Cell Movement - drug effects | Animals | Tumor Suppressor Proteins - blood | Hyperglycemia - blood | Chickens | Cyclooxygenase 2 - metabolism | Epithelial Cells - enzymology | Mesangial Cells - pathology | Diabetes Mellitus, Experimental - pathology | Hyperglycemia - enzymology | Leukocytes - drug effects | Mice | Inflammation - enzymology | Kidney Tubules, Proximal - drug effects | Regular
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
5. Full Text Cerebral structural changes in diabetic kidney disease: African American-diabetes heart study MIND
by Sink, Kaycee M and Divers, Jasmin and Whitlow, Christopher T and Palmer, Nicholette D and Smith, S. Carrie and Xu, Jianzhao and Hugenschmidt, Christina E and Wagner, Benjamin C and Williamson, Jeff D and Bowden, Donald W and Maldjian, Joseph A and Freedman, Barry I
Diabetes Care, ISSN 0149-5992, 02/2015, Volume 38, Issue 2, pp. 206 - 212
OBJECTIVEAlbuminuria and reduced kidney function are associated with cognitive impairment. Relationships between nephropathy and cerebral structural changes... 
LESION VOLUME | BRAIN ATROPHY | DEMENTIA | MRI | ALBUMINURIA | ENDOCRINOLOGY & METABOLISM | COGNITIVE IMPAIRMENT | WHITE-MATTER HYPERINTENSITIES | MELLITUS | TYPE-2 | VOXEL-BASED MORPHOMETRY | Albuminuria - complications | Glycated Hemoglobin A - metabolism | Humans | Middle Aged | Atrophy - pathology | Male | Brain Diseases - ethnology | Renal Insufficiency, Chronic - complications | European Continental Ancestry Group - ethnology | Glomerular Filtration Rate - physiology | Diabetes Mellitus, Type 2 - ethnology | Cognition Disorders - etiology | Adult | Female | Cognition Disorders - ethnology | Diabetes Mellitus, Type 2 - complications | Atrophy - ethnology | Albuminuria - pathology | Diabetic Nephropathies - pathology | Cross-Sectional Studies | Cognition Disorders - pathology | Albuminuria - ethnology | Linear Models | Diabetic Nephropathies - ethnology | Hypertension - pathology | Renal Insufficiency, Chronic - pathology | African Americans - ethnology | Diabetic Nephropathies - complications | Magnetic Resonance Imaging | Brain Diseases - pathology | Renal Insufficiency, Chronic - ethnology | Brain Diseases - complications | Hypertension - ethnology | Brain - pathology | Hypertension - complications | Aged | Diabetes Mellitus, Type 2 - pathology | Blood Glucose - metabolism | Type 2 diabetes | Care and treatment | Analysis | African Americans | Kidney diseases | Health aspects | Risk factors | Glomerular filtration rate | Health Disparities in Diabetes
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
6. Full Text What is the mechanism of microalbuminuria in diabetes: a role for the glomerular endothelium?
by Satchell, S C and Tooke, J E
Diabetologia, ISSN 0012-186X, 5/2008, Volume 51, Issue 5, pp. 714 - 725
Microalbuminuria is an important risk factor for cardiovascular disease and progressive renal impairment. This holds true in the general population and... 
Podocyte | Glycocalyx | Medicine & Public Health | Human Physiology | Glomerular endothelial cell | Metabolic Diseases | Internal Medicine | Diabetes | Glomerular filtration barrier | Microalbuminuria | glycocalyx | glomerular filtration barrier | SIZE-SELECTIVITY | NITRIC-OXIDE SYNTHASE | ISCHEMIC-HEART-DISEASE | NECROSIS-FACTOR-ALPHA | STRUCTURAL DETERMINANTS | URINARY ALBUMIN EXCRETION | microalbuminuria | GROWTH-FACTOR VEGF | glomerular endothelial cell | podocyte | ENDOCRINOLOGY & METABOLISM | CARDIOVASCULAR-DISEASE | LOW-GRADE INFLAMMATION | ESSENTIAL-HYPERTENSION | diabetes | Albuminuria - pathology | Diabetic Nephropathies - pathology | Glomerular Filtration Rate | Diabetes Mellitus, Type 1 - physiopathology | Humans | Risk Factors | Diabetes Mellitus, Type 1 - pathology | Kidney Glomerulus - physiopathology | Hypoglycemia - pathology | Kidney Glomerulus - pathology | Albuminuria - physiopathology | Diabetes Mellitus, Type 2 - physiopathology | Diabetic Angiopathies - pathology | Diabetic Nephropathies - physiopathology | Hypoglycemia - physiopathology | Diabetes Mellitus, Type 2 - pathology | Endothelial Cells - pathology | Endothelial Cells - physiology | Serum Albumin - metabolism | Growth factors | Endothelium | Review
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
7. Full Text Podocyte detachment and reduced glomerular capillary endothelial fenestration promote kidney disease in type 2 diabetic nephropathy
by Weil, E. Jennifer and Lemley, Kevin V and Mason, Clinton C and Yee, Berne and Jones, Lois I and Blouch, Kristina and Lovato, Tracy and Richardson, Meghan and Myers, Bryan D and Nelson, Robert G
Kidney International, ISSN 0085-2538, 11/2012, Volume 82, Issue 9, pp. 1010 - 1017
Podocyte detachment and reduced endothelial cell fenestration and relationships between these features and the classic structural changes of diabetic... 
electron microscopy | endothelial cells | glomerular filtration rate | podocytes | diabetic glomerulopathy | NUMBER | INJURY | VEGF-A EXPRESSION | PROTEINURIA | MELLITUS | PIMA-INDIANS | MICROALBUMINURIA | UROLOGY & NEPHROLOGY | FILTRATION-RATE | DYSFUNCTION | STAGE RENAL-DISEASE | Kidney Glomerulus - blood supply | Capillaries - pathology | Humans | Middle Aged | Antihypertensive Agents - administration & dosage | Diabetic Nephropathies - drug therapy | Male | Endothelial Cells - ultrastructure | Kidney Glomerulus - ultrastructure | Adult | Female | Diabetes Mellitus, Type 2 - complications | Albuminuria - pathology | Diabetic Nephropathies - pathology | Capillaries - ultrastructure | Glomerular Filtration Rate | Kidney Glomerulus - pathology | Podocytes - pathology | Microscopy, Electron | Podocytes - ultrastructure | Biopsy | Albuminuria - drug therapy | Diabetes Mellitus, Type 2 - pathology | Indians, North American | Losartan - administration & dosage | Endothelial Cells - pathology
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
8. Full Text Reducing VEGF-B Signaling Ameliorates Renal Lipotoxicity and Protects against Diabetic Kidney Disease
by Falkevall, Annelie and Mehlem, Annika and Palombo, Isolde and Heller Sahlgren, Benjamin and Ebarasi, Lwaki and He, Liqun and Ytterberg, A. Jimmy and Olauson, Hannes and Axelsson, Jonas and Sundelin, Birgitta and Patrakka, Jaakko and Scotney, Pierre and Nash, Andrew and Eriksson, Ulf and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Institutionen för immunologi, genetik och patologi and Uppsala universitet and Vaskulärbiologi
Cell Metabolism, ISSN 1550-4131, 03/2017, Volume 25, Issue 3, pp. 713 - 726
Diabetic kidney disease (DKD) is the most common cause of severe renal disease, and few treatment options are available today that prevent the progressive loss... 
diabetic kidney disease | albuminuria | insulin signaling | podocytes | lipotoxicity | endothelial fatty acid transport | vascular endothelial growth factor B | renal steatosis | HEART | LIPID-METABOLISM | GROWTH-FACTOR-B | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | ACCUMULATION | NEPHROPATHY | PODOCYTE | ADIPOSE | CELL BIOLOGY | Albuminuria - complications | Antibodies, Neutralizing - administration & dosage | Dyslipidemias - complications | Kidney - pathology | Humans | Middle Aged | Diabetes Mellitus, Type 1 - metabolism | Male | Diabetes Mellitus, Type 1 - complications | Diabetes Mellitus, Type 2 - metabolism | Dyslipidemias - pathology | Young Adult | Kidney - metabolism | Fatty Acid Transport Proteins - metabolism | Gene Deletion | Adult | Female | Albuminuria - metabolism | Diabetes Mellitus, Experimental - complications | Diabetes Mellitus, Experimental - metabolism | Diabetic Nephropathies - prevention & control | Lipids - toxicity | Diabetes Mellitus, Type 2 - complications | Kidney - physiopathology | Disease Models, Animal | Albuminuria - pathology | Insulin - pharmacology | Diabetic Nephropathies - pathology | Kidney - drug effects | Podocytes - metabolism | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Diabetes Mellitus, Type 1 - pathology | Antibodies, Neutralizing - pharmacology | Podocytes - pathology | Disease Progression | Up-Regulation - drug effects | Animals | Podocytes - drug effects | Signal Transduction - drug effects | Vascular Endothelial Growth Factor B - metabolism | Diabetes Mellitus, Experimental - pathology | Dyslipidemias - metabolism | Aged | Diabetes Mellitus, Type 2 - pathology | Endothelial growth factors | Analysis | Stem cells | Development and progression | Mice | Kidney diseases | Fatty acids | Endothelium | Insulin | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
9. Full Text Amelioration of Long-Term Renal Changes in Obese Type 2 Diabetic Mice by a Neutralizing Vascular Endothelial Growth Factor Antibody
by Allan Flyvbjerg and Frederik Dagnæs-Hansen and An S. De Vriese and Bieke F. Schrijvers and Ronald G. Tilton and Ruth Rasch
Diabetes, ISSN 0012-1797, 10/2002, Volume 51, Issue 10, pp. 3090 - 3094
Amelioration of Long-Term Renal Changes in Obese Type 2 Diabetic Mice by a Neutralizing Vascular Endothelial Growth Factor Antibody Allan Flyvbjerg 1 ,... 
FACTOR VEGF | FACTOR-BETA | INHIBITION | HUMAN MESANGIAL CELLS | PERMEABILITY FACTOR | ENDOCRINOLOGY & METABOLISM | PREVENTION | RECEPTOR | ANGIOTENSIN-II | DYSFUNCTION | EXPRESSION | Diabetes Mellitus - pathology | Vascular Endothelial Growth Factor A | Liver - pathology | Diabetic Nephropathies - etiology | Vascular Endothelial Growth Factors | Hyperglycemia - complications | Insulin - blood | Basement Membrane - pathology | Endothelial Growth Factors - immunology | Albuminuria - physiopathology | Diabetic Nephropathies - physiopathology | Mice, Mutant Strains | Hyperglycemia - pathology | Immunoglobulin G - pharmacology | Diabetes Mellitus - etiology | Female | Hyperglycemia - physiopathology | Diabetes Mellitus, Type 2 - complications | Ketones - urine | Disease Models, Animal | Hyperinsulinism - physiopathology | Albuminuria - pathology | Eating | Obesity | Diabetic Nephropathies - pathology | Hyperinsulinism - complications | Albuminuria - etiology | Mice, Inbred C57BL | Organ Size | Blood Glucose | Myocardium - pathology | Creatinine - urine | Antibodies - pharmacology | Hyperinsulinism - pathology | Animals | Diabetes Mellitus, Type 2 - physiopathology | Diabetes Mellitus - physiopathology | Lymphokines - immunology | Mice | Diabetes Mellitus, Type 2 - pathology | Intercellular Signaling Peptides and Proteins - immunology | Physiological aspects | Care and treatment | Vascular endothelial growth factor | Diabetic nephropathies
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
10. Full Text The protective role of Smad7 in diabetic kidney disease: Mechanism and therapeutic potential
by Chen, Hai Yong and Huang, Xiao R and Wang, Wansheng and Li, Jin Hua and Heuchel, Rainer L and Chung, Arthur C.K and Lan, Hui Yao
Diabetes, ISSN 0012-1797, 02/2011, Volume 60, Issue 2, pp. 590 - 601
OBJECTIVE-Although Smad3 has been considered as a downstream mediator of transforming growth factor-beta (TGF-beta) signaling in diabetes complications, the... 
CELLULAR BASIS | STRUCTURAL-FUNCTIONAL RELATIONSHIPS | UBIQUITIN LIGASE | ENDOCRINOLOGY & METABOLISM | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | EXTRACELLULAR-MATRIX | MICE | TGF-BETA RECEPTOR | GENE-TRANSFER | RENAL INJURY | NEPHROPATHY | Immunohistochemistry | Inflammation - pathology | Diabetes Mellitus, Experimental - genetics | Male | Fibrosis - metabolism | Inflammation - metabolism | Kidney Glomerulus - metabolism | Albuminuria - metabolism | Diabetes Mellitus, Experimental - metabolism | Smad7 Protein - genetics | Cytokines - genetics | Smad7 Protein - metabolism | Albuminuria - pathology | Gene Transfer Techniques | Diabetic Nephropathies - pathology | Fibrosis - genetics | Cytokines - metabolism | Signal Transduction | Diabetic Nephropathies - metabolism | Rats | Blood Glucose | Diabetic Nephropathies - genetics | Kidney Glomerulus - pathology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Mice, Knockout | Albuminuria - genetics | Animals | Analysis of Variance | Diabetes Mellitus, Experimental - pathology | Inflammation - genetics | Mice | Fibrosis - pathology | Complications and side effects | Diabetic nephropathies | Physiological aspects | Development and progression | Genetic aspects | Diabetes | Research | Transforming growth factors | Risk factors | Complications
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights