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Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 09/2008, Volume 3, Issue 5, pp. 1430 - 1436
Background and objectives: Lowe syndrome is defined by congenital cataracts, mental retardation, and proximal tubulopathy and is due to mutations in OCRL.... 
OCULOCEREBRORENAL SYNDROME | HYPERCALCIURIA | GENE | HYPOPHOSPHATEMIC RICKETS | TRAFFICKING | UROLOGY & NEPHROLOGY | GLYCOSURIA | MUTATIONS | OCRL1 | FANCONI-SYNDROME | DENT-DISEASE | Oculocerebrorenal Syndrome - genetics | Lysosomal Storage Diseases - physiopathology | Amino Acid Metabolism, Inborn Errors - physiopathology | Humans | Acidosis, Renal Tubular - genetics | Child, Preschool | Oculocerebrorenal Syndrome - complications | Male | Renal Tubular Transport, Inborn Errors - genetics | Nephrocalcinosis - physiopathology | Albuminuria - physiopathology | Amino Acid Metabolism, Inborn Errors - genetics | Fanconi Syndrome - physiopathology | Adult | Female | Hypercalciuria - genetics | Fanconi Syndrome - genetics | Renal Tubular Transport, Inborn Errors - physiopathology | Child | Oculocerebrorenal Syndrome - physiopathology | Glycosuria - physiopathology | Kidney Tubules, Proximal - physiopathology | Phosphoric Monoester Hydrolases - genetics | Glomerular Filtration Rate | Glycosuria - genetics | Europe | Hypercalciuria - physiopathology | Lysosomal Storage Diseases - genetics | Proteinuria - genetics | Proteinuria - physiopathology | Albuminuria - genetics | Phenotype | Nephrocalcinosis - genetics | Hypophosphatemia, Familial - genetics | Adolescent | Hypophosphatemia, Familial - physiopathology | Acidosis, Renal Tubular - physiopathology | Mutation | Index Medicus | Hereditary Disease
Journal Article
Diabetes Care, ISSN 0149-5992, 08/2016, Volume 39, Issue Supplement 2, pp. S165 - S171
Diabetic nephropathy (DN) is the most common cause of end-stage renal disease worldwide. Blood glucose and blood pressure control reduce the risk of developing... 
GLUCOSE COTRANSPORTER 2 | DAPAGLIFLOZIN | CANAGLIFLOZIN | ENDOCRINOLOGY & METABOLISM | DOUBLE-BLIND | RENAL-DISEASE | GLOMERULAR HYPERFILTRATION | TYPE-2 | NEPHROPATHY | BLOOD-PRESSURE | EMPAGLIFLOZIN | Albuminuria - complications | Cardiovascular Diseases - prevention & control | Humans | Kidney Glomerulus - physiopathology | Diabetes Mellitus, Type 1 - complications | Glomerular Filtration Rate - physiology | Albuminuria - physiopathology | Glomerular Filtration Rate - drug effects | Diabetic Nephropathies - physiopathology | Glucosides - therapeutic use | Blood Pressure - drug effects | Blood Pressure - physiology | Benzhydryl Compounds - therapeutic use | Diabetic Nephropathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Kidney - physiopathology | Hypoglycemic Agents - therapeutic use | Kidney Failure, Chronic - physiopathology | Kidney - drug effects | Cardiovascular Diseases - metabolism | Diabetes Mellitus, Type 1 - physiopathology | Kidney Failure, Chronic - drug therapy | Kidney Glomerulus - drug effects | Risk Factors | Sodium-Glucose Transporter 2 Inhibitors | Diabetes Mellitus, Type 1 - drug therapy | Diabetes Mellitus, Type 2 - physiopathology | Albuminuria - drug therapy | Diabetes Mellitus, Type 1 - blood | Kidney Failure, Chronic - complications | Renin-Angiotensin System - drug effects | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Index Medicus
Journal Article
Diabetologia, ISSN 0012-186X, 3/2012, Volume 55, Issue 3, pp. 600 - 607
Endothelial dysfunction is important in the development of vascular complications in diabetes. Patients with type 2 diabetes have increased production of the... 
Type 2 diabetes | Human Physiology | Metabolic Diseases | Internal Medicine | Endothelin receptor antagonist | Bosentan | Medicine & Public Health | Endothelin-1 | Placebo | Randomised controlled trial | Endothelial dysfunction | FMD | Microalbuminuria | Endo-Pat | ATHEROSCLEROSIS | HUMANS | MECHANISMS | AMPLITUDE | INSULIN-RESISTANCE | DEPENDENT VASODILATATION | ENDOCRINOLOGY & METABOLISM | NITRIC-OXIDE | DYSFUNCTION | BLOCKADE | CORONARY-ARTERY-DISEASE | Albuminuria - complications | Microvessels - physiopathology | Peripheral Vascular Diseases - prevention & control | Humans | Middle Aged | Endothelium, Vascular - drug effects | Male | Hyperemia - prevention & control | Albuminuria - physiopathology | Diabetic Angiopathies - physiopathology | Diabetic Nephropathies - physiopathology | Brachial Artery - physiopathology | Female | Brachial Artery - drug effects | Diabetic Angiopathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Endothelin Receptor Antagonists | Severity of Illness Index | Double-Blind Method | Peripheral Vascular Diseases - physiopathology | Endothelium, Vascular - physiopathology | Microvessels - drug effects | Antihypertensive Agents - therapeutic use | Diabetic Nephropathies - complications | Diabetes Mellitus, Type 2 - physiopathology | Sulfonamides - therapeutic use | Hyperemia - physiopathology | Aged | Vasodilation - drug effects | Albumin | Care and treatment | Endothelin | Endothelium | Index Medicus
Journal Article
Journal Article
Nephrology Dialysis Transplantation, ISSN 0931-0509, 5/2008, Volume 23, Issue 5, pp. 1621 - 1627
Journal Article
Circulation, ISSN 0009-7322, 01/2018, Volume 137, Issue 2, pp. 119 - 129
BACKGROUND:Empagliflozin, a sodium-glucose cotransporter 2 inhibitor, reduced cardiovascular morbidity and mortality in patients with type 2 diabetes mellitus... 
Treatment outcome | Hospitalization | Kidney diseases | Mortality | Diabetes mellitus type 2 | Sodiumglucose transporter 2 | CARDIAC & CARDIOVASCULAR SYSTEMS | EVENTS | hospitalization | SAFETY | HEART-FAILURE | sodium-glucose transporter 2 | RISK | DEATH | MECHANISMS | treatment outcome | IMPACT | kidney diseases | mortality | PERIPHERAL VASCULAR DISEASE | diabetes mellitus, type 2 | SGLT2 INHIBITION | PROGRESSION | Prevalence | Cardiovascular Diseases - drug therapy | Humans | Middle Aged | Male | Cause of Death | Renal Insufficiency, Chronic - drug therapy | Albuminuria - physiopathology | Glomerular Filtration Rate - drug effects | Benzhydryl Compounds - adverse effects | Albuminuria - mortality | Time Factors | Cardiovascular Diseases - mortality | Renal Insufficiency, Chronic - mortality | Female | Glucosides - therapeutic use | Renal Insufficiency, Chronic - diagnosis | Benzhydryl Compounds - therapeutic use | Glucosides - adverse effects | Kidney - physiopathology | Cardiovascular Diseases - diagnosis | Sodium-Glucose Transporter 2 Inhibitors - therapeutic use | Cardiovascular Diseases - physiopathology | Kidney - drug effects | Sodium-Glucose Transporter 2 Inhibitors - adverse effects | Risk Factors | Treatment Outcome | Renal Insufficiency, Chronic - physiopathology | Diabetes Mellitus, Type 2 - diagnosis | Diabetes Mellitus, Type 2 - physiopathology | Diabetes Mellitus, Type 2 - mortality | Aged | Diabetes Mellitus, Type 2 - drug therapy | Type 2 diabetes | Care and treatment | Index Medicus | Abridged Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 06/2011, Volume 79, Issue 12, pp. 1331 - 1340
Journal Article
Diabetes, ISSN 0012-1797, 01/2014, Volume 63, Issue 1, pp. 291 - 299
Journal Article
American Journal of Obstetrics and Gynecology, ISSN 0002-9378, 2014, Volume 211, Issue 5, pp. 516.e1 - 516.e11
Journal Article
Kidney International, ISSN 0085-2538, 12/2010, Volume 78, Issue 11, pp. 1136 - 1153
The remnant kidney model in C57BL/6 mice does not develop progressive chronic kidney disease (CKD). In this study we modified the model to mimic features of... 
blood pressure | telemetry | glomerulosclerosis | interstitial fibrosis | angiotensin II | ARTERIAL-BLOOD-PRESSURE | LOW-BIRTH-WEIGHT | 9 GENE MYH9 | INDUCED HYPERTENSION | CARDIAC-HYPERTROPHY | UROLOGY & NEPHROLOGY | AFRICAN-AMERICANS | GROWTH-FACTOR | FOCAL SEGMENTAL GLOMERULOSCLEROSIS | CHRONIC-RENAL-FAILURE | MOLECULAR-MECHANISMS | Blood Pressure | Kidney Diseases - physiopathology | Antihypertensive Agents - pharmacology | Tetrazoles - pharmacology | Angiotensin II - administration & dosage | Kidney - pathology | Species Specificity | Hydralazine - pharmacology | Hypertension - drug therapy | Male | Mice, 129 Strain | Angiotensin II Type 1 Receptor Blockers - pharmacology | X-Ray Microtomography | Kidney Diseases - genetics | Hypertension - etiology | Anemia - physiopathology | Albuminuria - physiopathology | Nephrectomy | Time Factors | Glomerulonephritis - etiology | Hypertension - genetics | Glomerulonephritis - physiopathology | Anemia - etiology | Kidney - physiopathology | Disease Models, Animal | Genetic Predisposition to Disease | Infusion Pumps, Implantable | Kidney - drug effects | Albuminuria - etiology | Heart Diseases - physiopathology | Kidney Diseases - pathology | Kidney Diseases - prevention & control | Mice, Inbred C57BL | Desoxycorticosterone | Imidazoles - pharmacology | Sodium Chloride, Dietary | Telemetry | Disease Progression | Hypertension - pathology | Hypertension - physiopathology | Infusions, Subcutaneous | Heart Diseases - etiology | Animals | Fibrosis | Mice | Kidney Diseases - etiology | Blood Pressure Monitoring, Ambulatory | Chronic Disease | Index Medicus
Journal Article
Journal of Hypertension, ISSN 0263-6352, 07/2010, Volume 28, Issue 7, pp. 1422 - 1428
Journal Article