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PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e14062
Background: The cancer stem cell theory hypothesizes that cancers are perpetuated by cancer stem cells (CSC) or tumor initiating cells (TIC) possessing... 
COLON-CANCER | POPULATION | MARKER | MULTIDISCIPLINARY SCIENCES | GROWTH | HYALURONAN | PLURIPOTENCY | FLOW-CYTOMETRY | IDENTIFICATION | CD133(+) | TUMORS | Immunohistochemistry | Humans | Lung Neoplasms - metabolism | Middle Aged | Glycoproteins - metabolism | Peptides - genetics | Immunoblotting | Lung Neoplasms - pathology | Male | Transplantation, Heterologous | Gene Expression Profiling | Antigens, CD - genetics | Antigens, CD - metabolism | Neoplasms, Experimental - pathology | Octamer Transcription Factor-3 - genetics | Flow Cytometry | Peptides - metabolism | Neoplastic Stem Cells - metabolism | Hyaluronan Receptors - metabolism | Neoplasms, Experimental - genetics | Neoplastic Stem Cells - pathology | Female | Nuclear Proteins - genetics | Repressor Proteins - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Glycoproteins - genetics | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | AC133 Antigen | Hyaluronan Receptors - genetics | Animals | Polycomb Repressive Complex 1 | Mice, Nude | Octamer Transcription Factor-3 - metabolism | Cell Line, Tumor | Aged | Mice | Neoplasms, Experimental - metabolism | Squamous cell carcinoma | Lung cancer, Non-small cell | Analysis | Stem cells | Flow cytometry | Biotechnology | Laboratories | Heart surgery | Oct-4 protein | Lung cancer | Colorectal cancer | Stem cell transplantation | Proteins | Allografts | Epidermal growth factor | CD44 antigen | Xenografts | Cell cycle | CD34 antigen | Subpopulations | Cell survival | Tumor cells | Non-small cell lung carcinoma | Tumor cell lines | Cisplatin | Studies | Polymerase chain reaction | Pathology | Cytometry | Properties (attributes) | Medical prognosis | Cell lines | Biomarkers | In vivo methods and tests | Pluripotency | Tumors | Apoptosis | Cancer
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 2015, Volume 64, Issue 5, pp. 1444 - 1449
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 997 - 16
The molecular mechanisms that couple glycolysis to cancer drug resistance remain unclear. Here we identify an ATP-binding motif within the NADPH oxidase... 
NADPH OXIDASE 4 | PROTEIN | PYRUVATE-KINASE | REDOX REGULATION | PKM2 | MULTIDISCIPLINARY SCIENCES | ISOFORM EXPRESSION | PROMOTES | ATP-BINDING | CELL-DEATH | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Drug Resistance, Neoplasm | Male | Kidney Neoplasms - metabolism | Kidney - metabolism | Membrane Proteins - metabolism | Carcinoma, Renal Cell - drug therapy | Energy Metabolism - physiology | Kidney - drug effects | Carcinoma, Renal Cell - pathology | Cells, Cultured | Etoposide - pharmacology | Mitochondria - metabolism | Mitochondria - drug effects | Xenograft Model Antitumor Assays | Carcinoma, Renal Cell - metabolism | p300-CBP Transcription Factors - metabolism | NADPH Oxidase 4 - metabolism | Animals | Carrier Proteins - metabolism | Mice, Nude | Thyroid Hormones - metabolism | Kidney Neoplasms - pathology | Mice | Mice, Inbred BALB C | Antineoplastic Agents, Phytogenic - pharmacology | Kidney Neoplasms - drug therapy | Energy Metabolism - drug effects | Drugs | Cell culture | Energy metabolism | Reactive oxygen species | Syngeneic grafts | Oxidase | Lysosomes | Cytotoxicity | Pyruvic acid | Event-related potentials | Drug resistance | NAD(P)H oxidase | Coupling (molecular) | Mitochondria | Allosteric properties | NOX4 protein | Allografts | Xenografts | Drug metabolism | Acetylation | Pyruvate kinase | Cultures | Metabolism | Molecular modelling | Cell death | Glycolysis | ATP | Clear cell-type renal cell carcinoma | Cancer | Kidney transplantation
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 1931-857X, 08/2009, Volume 297, Issue 2, pp. 461 - 470
Recently, kidney fibrosis following transplantation has become recognized as a main contributor of chronic allograft nephropathy. In transplantation, transient... 
Ischemia-reperfusion | α-smooth muscle actin | Superoxide | Manganese(III) tetrakis(1-methyl-4-pyridyl) porphyrin | Interstitial fibrosis | alpha-smooth muscle actin | manganese(III) tetrakis(1-methyl-4-pyridyl) porphyrin | OXIDATIVE STRESS | ISCHEMIA/REPERFUSION INJURY | PHYSIOLOGY | REPERFUSION INJURY | ischemia-reperfusion | interstitial fibrosis | MAPK KINASE ACTIVATION | NITRIC-OXIDE | UROLOGY & NEPHROLOGY | INDUCED FUNCTIONAL INJURY | superoxide | SUPEROXIDE-DISMUTASE | URETERAL OBSTRUCTION | CHRONIC ALLOGRAFT NEPHROPATHY | IMMUNOSTIMULATED MACROPHAGES | Kidney - blood supply | Reactive Oxygen Species - metabolism | Kidney - pathology | Apoptosis - drug effects | S100 Calcium-Binding Protein A4 | Actins - metabolism | Male | Metalloporphyrins - pharmacology | Tyrosine - analogs & derivatives | Antigens, CD - metabolism | Kidney - metabolism | Time Factors | Lipid Peroxidation - drug effects | Reperfusion Injury - metabolism | Ischemia - pathology | Superoxide Dismutase - metabolism | Disease Models, Animal | Kidney - drug effects | Oxidation-Reduction | Reperfusion Injury - pathology | Ischemia - metabolism | S100 Proteins - metabolism | Antioxidants - pharmacology | Disease Progression | Hydrogen Peroxide - metabolism | Collagen - metabolism | Tyrosine - metabolism | Animals | Reperfusion Injury - prevention & control | Antigens, Differentiation, Myelomonocytic - metabolism | Fibrosis | Glucosephosphate Dehydrogenase - metabolism | HSP47 Heat-Shock Proteins - metabolism | Mice | Mice, Inbred BALB C | Oxidative Stress - drug effects | Proliferating Cell Nuclear Antigen - metabolism | Vitamin E - pharmacology
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e90883
Bone marrow-derived mesenchymal stem cells (BM-MSCs) have recently shown promise as a therapeutic tool in various types of chronic kidney disease (CKD) models.... 
EPITHELIAL-CELLS | MULTIDISCIPLINARY SCIENCES | HEPATOCYTE GROWTH-FACTOR | INTERSTITIAL FIBROSIS | IGA NEPHROPATHY | ANTIINFLAMMATORY PROTEIN TSG-6 | DIABETIC-NEPHROPATHY | TNF-ALPHA | CHRONIC KIDNEY-DISEASE | RAT REMNANT KIDNEY | STROMAL CELLS | Inflammation - chemically induced | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Interleukin-8 - genetics | Cell Adhesion Molecules - genetics | Epithelial Cells - metabolism | Coculture Techniques | Epithelial Cells - drug effects | Humans | Tumor Necrosis Factor-alpha - genetics | Actins - metabolism | Epithelial-Mesenchymal Transition - drug effects | NF-kappa B - metabolism | Fibrosis - metabolism | Actins - genetics | Inflammation - metabolism | Mesenchymal Stromal Cells - cytology | Hepatocyte Growth Factor - genetics | Chemokine CCL2 - metabolism | Chemokine CCL5 - metabolism | Interleukin-8 - metabolism | Fibrosis - prevention & control | Interleukin-6 - metabolism | Collagen Type IV - metabolism | Kidney Tubules, Proximal - pathology | Fibrosis - chemically induced | Interleukin-6 - genetics | Signal Transduction | Bone Marrow Cells - cytology | Gene Expression Regulation | Mesenchymal Stromal Cells - metabolism | Chemokine CCL2 - genetics | Epithelial Cells - pathology | Cell Adhesion Molecules - metabolism | Hepatocyte Growth Factor - metabolism | Albumins - pharmacology | NF-kappa B - genetics | Chemokine CCL5 - genetics | Kidney Tubules, Proximal - metabolism | Collagen Type IV - genetics | Inflammation - prevention & control | Fibrosis - pathology | Primary Cell Culture | Bone Marrow Cells - metabolism | Kidney Tubules, Proximal - drug effects | Prognosis | Chronic kidney failure | Collagen | Analysis | Stem cells | Albumin | Inflammation | Health aspects | Cell culture | Animal models | Nephrology | Mesenchyme | Syngeneic grafts | Epithelial cells | Paracrine signalling | Stem cell transplantation | Kinases | E-cadherin | Interleukin 6 | Proteins | Allografts | Rodents | Bone marrow | Collagen (type IV) | Cardiology | Growth factors | Interleukin 8 | Recombinant | NF-κB protein | Immunoglobulins | Deactivation | Hepatocyte growth factor | Tumor necrosis factor-α | Studies | Fibrosis | Kidney diseases | Diabetes | Proteinuria | Kidney transplantation
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2017, Volume 12, Issue 11, p. e0187637
Background Hypoxia preconditioning has been proven to be an effective method to enhance the therapeutic action of mesenchymal stem cells (MSCs). However, the... 
ISCHEMIA-REPERFUSION INJURY | SURVIVAL | EMBOLISM | DEXAMETHASONE | THERAPY | INCREASE | MULTIDISCIPLINARY SCIENCES | MICE | MARROW STROMAL CELLS | DELIVERY | Bronchoalveolar Lavage Fluid | Protein Carbonylation | Lung Injury - pathology | Capillaries - pathology | Glutathione - metabolism | Antioxidants - metabolism | Caspase 3 - metabolism | Male | NF-kappa B - metabolism | Pulmonary Embolism - pathology | Cell Hypoxia | Proto-Oncogene Proteins c-bcl-2 - metabolism | Lung Injury - therapy | Mesenchymal Stromal Cells - cytology | Leukocyte Count | Reperfusion Injury - complications | Biomarkers - metabolism | Pulmonary Embolism - complications | Reperfusion Injury - therapy | Capillaries - physiopathology | Thrombosis - physiopathology | Reperfusion Injury - pathology | Signal Transduction | Cytochromes c - metabolism | Organ Size | Thiobarbituric Acid Reactive Substances - metabolism | Rats, Sprague-Dawley | Hydrogen Peroxide - metabolism | Thrombosis - pathology | Intercellular Adhesion Molecule-1 - metabolism | Lung Injury - physiopathology | Animals | Reperfusion Injury - physiopathology | Cytosol - metabolism | Lung Injury - complications | Hemodynamics | Vascular Cell Adhesion Molecule-1 - metabolism | Mesenchymal Stem Cell Transplantation | Apoptosis | Mitogen-Activated Protein Kinases - metabolism | Peroxidase - metabolism | Pulmonary Embolism - physiopathology | Care and treatment | Stem cells | Physiological aspects | Research | Properties | Reperfusion injury | Risk factors | Cytochrome | Reactive oxygen species | Transplants & implants | Bcl-2 protein | Mesenchyme | Embolisms | Membrane permeability | Prostaglandin E2 | Stem cell transplantation | Critical care | Angiogenesis | Mitochondria | Reperfusion | Allografts | Pathways | Ischemia | Rodents | Bone marrow | Thromboembolism | Alveoli | Drug dosages | Injuries | Glutathione | Edema | NF-κB protein | Cytokines | Internal medicine | Rats | MAP kinase | Inflammation | Permeability | Thrombosis | Embolism | Medicine | Cytochrome c | Signaling | Lungs | Skin & tissue grafts | Interleukin 10 | Ventilation | Hypoxia | Clinical medicine | Laboratory animals | Preconditioning | Cell migration
Journal Article
European Journal of Immunology, ISSN 0014-2980, 02/2011, Volume 41, Issue 2, pp. 413 - 424
During infection, TLR agonists are released and trigger mature as well as differentiating innate immune cells. Early encounter with TLR agonists (R848; LPS)... 
Tolerance | STAT‐3 | TLR | PD‐L1 | PD-L1 | STAT-3 | MYCOBACTERIUM-TUBERCULOSIS | ALLOGRAFT-REJECTION | HUMAN DENDRITIC CELLS | IFN-GAMMA | TRANSPLANT TOLERANCE | IMMUNOLOGY | CUTTING EDGE | IMMUNE-RESPONSES | REGULATORY T-CELLS | B7 FAMILY | DIFFERENTIATION | T-Lymphocyte Subsets - immunology | Antigen-Presenting Cells - cytology | Immune Tolerance - physiology | Monocytes - cytology | Dendritic Cells - immunology | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | STAT Transcription Factors - metabolism | Monocytes - immunology | Antigens, CD - metabolism | Lipopolysaccharide Receptors - metabolism | Lymphocyte Culture Test, Mixed | T-Lymphocytes, Regulatory - immunology | Signal Transduction - immunology | Interleukin-4 - pharmacology | Interleukin-10 - metabolism | Histocompatibility Antigens Class II - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Interleukin-6 - metabolism | STAT3 Transcription Factor - metabolism | Antigen-Presenting Cells - metabolism | B7-H1 Antigen | Gene Expression Regulation - immunology | Antigen-Presenting Cells - drug effects | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Imidazoles - pharmacology | Antigen-Presenting Cells - immunology | Toll-Like Receptors - agonists | Monocytes - drug effects | Cell Differentiation - immunology | Mitogen-Activated Protein Kinase 3 - metabolism | Cell Differentiation - drug effects | Antigens, CD1 - metabolism | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Dendritic Cells - cytology | Protein Kinase Inhibitors - pharmacology | STAT Transcription Factors - antagonists & inhibitors | STAT3 Transcription Factor - antagonists & inhibitors | Mitogen-Activated Protein Kinase 1 - metabolism | Medical research | Rodents
Journal Article
ChemBioChem, ISSN 1439-4227, 07/2017, Volume 18, Issue 13, pp. 1317 - 1331
Human induced pluripotent stem‐cell‐derived cardiomyocytes (hiPSC CMs) may be used in regenerative medicine for individualized tissue transplants in the... 
cardiomyocytes | glycoproteins | sialic acids | human pluripotent stem cells | proteomics | Staining and Labeling - methods | Fucose - chemistry | Galactose - metabolism | Receptors, G-Protein-Coupled - metabolism | Induced Pluripotent Stem Cells - chemistry | Fucose - metabolism | Humans | Sialic Acids - chemistry | Acetylglucosamine - metabolism | Cell Membrane - chemistry | Receptor, EphA7 - metabolism | Polysaccharides - chemistry | Cell Differentiation | Cell Membrane - metabolism | Gastrins - genetics | Membrane Proteins - metabolism | Receptor, EphA7 - genetics | Receptor-Like Protein Tyrosine Phosphatases, Class 2 - genetics | Induced Pluripotent Stem Cells - cytology | Induced Pluripotent Stem Cells - metabolism | Carbohydrate Sequence | Myocytes, Cardiac - cytology | Membrane Proteins - genetics | Glycomics - methods | Ciliary Neurotrophic Factor Receptor alpha Subunit - genetics | Gene Expression Regulation | Sialic Acids - metabolism | Myocytes, Cardiac - chemistry | Gastrins - metabolism | Acetylglucosamine - chemistry | Polysaccharides - metabolism | Laminin - genetics | Galactose - chemistry | Receptor-Like Protein Tyrosine Phosphatases, Class 2 - metabolism | Myocytes, Cardiac - metabolism | Receptors, G-Protein-Coupled - genetics | Laminin - metabolism | Ciliary Neurotrophic Factor Receptor alpha Subunit - metabolism | Polysaccharides | Usage | Analysis | Stem cells | Glycoproteins | Organic acids | Membrane proteins | Gel electrophoresis | Tissue engineering | Capillary electrophoresis | Glycoprotein | Transplants | Cardiomyocytes | Marking | N-glycans | Patients | Cell surface | Regeneration (physiology) | Proteins | Medicine | Regeneration | Biomarkers | Electrophoresis | Bioindicators | Differentiation | Galactose | Pluripotency | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e98749
Skin-derived mesenchymal stem cells (SMSCs) can differentiate into the three embryonic germ layers. For this reason, they are considered a powerful tool for... 
MARKER | GERM-CELLS | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | PORCINE SKIN | GENE-EXPRESSION | OOCYTES | MICE | ANTI-MULLERIAN HORMONE | CHEMOTHERAPY | FERTILITY | Tumor Necrosis Factor-alpha - metabolism | LIM-Homeodomain Proteins - metabolism | Ovarian Follicle - physiopathology | Oogenesis - physiology | Homeodomain Proteins - metabolism | Male | Interferon-gamma - metabolism | Interleukin-1beta - metabolism | Female | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Ovarian Follicle - metabolism | Mesenchymal Stromal Cells - physiology | Disease Models, Animal | Ovary - metabolism | Primary Ovarian Insufficiency - metabolism | Mesenchymal Stromal Cells - metabolism | Mice, Transgenic | Transcription Factors - metabolism | Animals | Primary Ovarian Insufficiency - physiopathology | Mice | Skin - physiopathology | Transforming Growth Factor beta - metabolism | RNA-Binding Proteins - metabolism | Ovary - physiopathology | Chemotherapy | Analysis | Stem cells | Genetic engineering | Skin | Transforming growth factors | Health aspects | Ovarian cancer | Cancer | Health care | Flow cytometry | Mesenchyme | Body weight | Childrens health | Recovery of function | Fluorescence | Antibodies | Stem cell transplantation | Reproductive organs | Males | Oocytes | Interleukin 6 | Oogenesis | Reproduction | Allografts | Fertility | Rodents | Bone marrow | Busulfan | Damage | Interleukin 8 | Polyclonal antibodies | Reproductive systems | Cytokines | Research & development--R&D | Cloning | Medical treatment | Gynecology | Transgenic mice | Organs | Inflammation | Ovaries | Tumor necrosis factor-α | Mammals | Gene expression | Embryos | Medicine | Cyclophosphamide | γ-Interferon | Laboratory animals | Females | Apoptosis | Research & development | R&D
Journal Article
Journal of Controlled Release, ISSN 0168-3659, 09/2016, Volume 238, pp. 139 - 148
Therapeutic nanoparticles (NPs) approved for clinical use in solid tumor therapy provide only modest improvements in patient survival, in part due to... 
Nanoparticles | Intravital microscopy | Multiple particle tracking (MPT) | PEG density | Surface plasmon resonance (SPR) | Tumor tissue penetration | SOLID TUMORS | DEEP PENETRATION | CHEMISTRY, MULTIDISCIPLINARY | PHASE-III TRIAL | BREAST-CANCER | DELIVERY-SYSTEM | IN-VIVO | PHARMACOLOGY & PHARMACY | EXTRACELLULAR-MATRIX | HUMAN MUCUS | Neoplasms - metabolism | Nanoparticles - analysis | Humans | Polyethylene Glycols - analysis | Drug Carriers - analysis | Albumin-Bound Paclitaxel - administration & dosage | Antineoplastic Agents - administration & dosage | Polyglycolic Acid - analysis | Breast - metabolism | Breast Neoplasms - metabolism | Antineoplastic Agents - metabolism | Nanoparticles - metabolism | Surface Properties | Doxorubicin - analogs & derivatives | Female | Doxorubicin - metabolism | Polyglycolic Acid - metabolism | Diffusion | Lactic Acid - analysis | Doxorubicin - administration & dosage | Albumin-Bound Paclitaxel - metabolism | Polyethylene Glycols - metabolism | Drug Carriers - metabolism | Lactic Acid - metabolism | Proteoglycans - metabolism | Breast Neoplasms - drug therapy | Polyethylene Glycols - administration & dosage | Neoplasms - drug therapy | Particle Size | Collagen - metabolism | Animals | Breast - drug effects | Mice, Nude | Cell Line, Tumor | Mice | Laminin - metabolism | Drug Combinations | Drugs | Ethylene glycol | Drug delivery systems | Drug therapy | Drug approval | Tumors | Vehicles | Medical colleges | Cancer | multiple particle tracking (MPT) | tumor tissue penetration | surface plasmon resonance (SPR) | nanoparticles | intravital microscopy
Journal Article