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Biology of Blood and Marrow Transplantation, ISSN 1083-8791, 09/2017, Volume 23, Issue 9, pp. 1498 - 1508
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2016, Volume 11, Issue 2, pp. e0148881 - e0148881
Background The association of complement with the progression of acute T cell mediated rejection (ATCMR) is not well understood. We investigated the production... 
ALLOGRAFT-REJECTION | EPITHELIAL-CELLS | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | TRANSPLANT REJECTION | DECAY-ACCELERATING FACTOR | C3 GENE-EXPRESSION | PAROXYSMAL-NOCTURNAL HEMOGLOBINURIA | FACTOR-B GENE | ISCHEMIA-REPERFUSION | LOCALLY PRODUCED COMPLEMENT | Graft Survival - immunology | Humans | Middle Aged | Complement System Proteins - immunology | Graft Rejection - metabolism | Male | Receptors, Cell Surface - antagonists & inhibitors | Complement System Proteins - metabolism | Young Adult | Allografts | T-Lymphocytes - metabolism | Antigens, Surface - metabolism | Adult | Complement System Proteins - genetics | Female | Complement Pathway, Alternative - genetics | Disease Models, Animal | Antibodies, Monoclonal - pharmacology | Gene Expression Regulation | Rats | Receptors, Cell Surface - metabolism | Kidney Transplantation | Animals | Graft Survival - genetics | Complement Pathway, Alternative - immunology | T-Lymphocytes - immunology | Aged | CD59 Antigens - metabolism | Graft Rejection - immunology | Graft Rejection - genetics | Graft Survival - drug effects | Kidneys | Physiological aspects | Development and progression | Genetic aspects | Transplantation | Research | T cells | Risk factors | Graft rejection | Bone grafts | Complement receptors | Animal models | Nephrology | Complement component C3a | Transplants & implants | Syngeneic grafts | Complement component C5a | Antibodies | Staining | Lymphocytes T | CD59 antigen | Activation | Complement | Mental depression | Alternative pathway | Grafting | Urology | Proteins | Receptors | Immunology | Lymphocytes | Rodents | Surgery | University graduates | Creatinine | Antigens | Crry protein | Complement regulatory proteins | Gene expression | Survival | Membrane proteins | Medicine | Rejection | Complement component C1q | Pathology | Complement activation | Grafts | Complement component C4 | Complement component C3 | Medical prognosis | Membrane cofactor protein | Kidney transplantation | Apoptosis | Index Medicus
Journal Article
Journal Article
Biology of Blood and Marrow Transplantation, ISSN 1083-8791, 2016, Volume 23, Issue 1, pp. 126 - 133
Journal Article
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 12/2017, Volume 190, Issue 3, pp. 315 - 327
The role of macrophage infiltrates in oral mucosal acute graft‐ versus ‐host disease (AGVHD) remains unclear, although clinical studies suggest that macrophage... 
monocyte chemoattractant protein‐1 | CC chemokine receptor 2 | M1 macrophages | acute graft | versus | host disease | CD29 β 1 integrin | monocyte chemoattractant protein-1 | acute graft-versus-host disease | MIGRATION | MATRIX | CELLS | RECRUITMENT | CD29 1 integrin | IMMUNOLOGY | TRANSPLANTATION | INFLAMMATORY-BOWEL-DISEASE | GELATINASE-A | EXPRESSION | ALTERNATIVE ACTIVATION | CCR2 | Acute Disease | Macrophages - pathology | Chemokine CCL2 - immunology | Receptors, CCR2 - immunology | Mouth Mucosa - immunology | Rats, Inbred Lew | Rats | Male | Graft vs Host Disease - immunology | Graft vs Host Disease - pathology | Cell Movement - immunology | Animals | Female | Integrin beta1 - immunology | Mouth Mucosa - pathology | Laminin - immunology | Macrophages - microbiology | Matrix Metalloproteinase 2 - immunology | Immunohistochemistry | Laminin | Macrophages | Political corruption | Integrins | CC chemokine receptors | Graft-versus-host reaction | Tongue | Leukocyte migration | Mucosa | Reverse transcription | Matrix metalloproteinase | CD29 antigen | Assaying | CCR2 protein | Rodents | Metalloproteinase | Lesions | Spleen | Histology | Epithelium | Adhesion | Polymerase chain reaction | Monocyte chemoattractant protein | Correlation analysis | Infiltration | Immunofluorescence | Monocyte chemoattractant protein 1 | Index Medicus | acute graft‐versus‐host disease | Original
Journal Article
Journal Article
Transplantation, ISSN 0041-1337, 04/2017, Volume 101, Issue 4, pp. e75 - e85
BACKGROUNDRecipients of vascularized composite allografts require aggressive and lifelong immunosuppression, and because the surgery is usually performed in... 
SURGERY | ALLOTRANSPLANTATION | RECOGNITION | REGENERATION | TOLERANCE | TRANSPLANT | IMMUNOLOGY | HAND | TRANSPLANTATION | ISCHEMIA-REPERFUSION INJURY | ACUTE REJECTION | ALTERNATIVE PATHWAY | EXPRESSION | Receptor, Anaphylatoxin C5a - genetics | Composite Tissue Allografts - transplantation | Reperfusion Injury - etiology | Spleen - immunology | Composite Tissue Allografts - immunology | Recombinant Fusion Proteins - pharmacology | Cyclosporine - pharmacology | Male | Spleen - drug effects | Composite Tissue Allografts - drug effects | Complement C3 - deficiency | Chemotaxis, Leukocyte - drug effects | Time Factors | Hindlimb - transplantation | T-Lymphocytes - drug effects | Hindlimb - immunology | Models, Animal | Complement C3 - genetics | Immunosuppressive Agents - pharmacology | Reperfusion Injury - metabolism | Complement Activation - drug effects | Vascularized Composite Allotransplantation - adverse effects | Mice, Inbred C57BL | Genotype | Mice, Knockout | Phenotype | Animals | Receptors, G-Protein-Coupled - deficiency | Reperfusion Injury - prevention & control | Complement Inactivating Agents - pharmacology | Hindlimb - blood supply | Receptor, Anaphylatoxin C5a - deficiency | Reperfusion Injury - immunology | Hindlimb - drug effects | T-Lymphocytes - immunology | Mice, Inbred BALB C | Receptors, G-Protein-Coupled - genetics | Composite Tissue Allografts - blood supply | Graft Survival - drug effects | Index Medicus
Journal Article
Journal Article
American Journal of Transplantation, ISSN 1600-6135, 03/2013, Volume 13, Issue 3, pp. 663 - 675
Atypical hemolytic and uremic syndrome (aHUS) is a severe disease strongly associated with genetic abnormalities in the complement alternative pathway. In... 
Acute rejection | endothelial cell damage | mutations and polymorphisms in complement alternative pathway | thrombotic microangiopathy | atypical hemolytic and uremic syndrome | renal transplantation | SURGERY | FACTOR-B | HUS | FACTOR-I | FACTOR-H-AUTOANTIBODIES | TRANSPLANTATION | KIDNEY-TRANSPLANTATION | MUTATIONS | CD46 | PREDISPOSE | MEMBRANE COFACTOR PROTEIN | Recurrence | Genetic Testing | Prognosis | Humans | Middle Aged | Male | Young Adult | Hemolytic-Uremic Syndrome - genetics | Complement Factor B - genetics | Adult | Complement System Proteins - genetics | Female | Hemolytic-Uremic Syndrome - therapy | Retrospective Studies | Complement C3 - genetics | Biomarkers - metabolism | Risk Factors | Biomarkers - analysis | Kidney Transplantation | Mutation - genetics | Fibrinogen - genetics | Graft Survival - genetics | Adolescent | Membrane Cofactor Protein - genetics | Aged | Complement Factor H - genetics | Graft Rejection - genetics | Atypical Hemolytic Uremic Syndrome | Medical research | Kidneys | Genes | Organ transplant recipients | Medicine, Experimental | Genetic research | Transplantation | Clinical trials | Mutation | Transplants & implants | Drug therapy | Index Medicus | Biological Markers | Complement C3 | Life Sciences | Immunology | Fibrinogen | Hemolytic-Uremic Syndrome | Antigens, CD46 | Graft Survival | Complement Factor H | Complement System Proteins | Complement Factor B | Graft Rejection
Journal Article