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Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 01/2008, Volume 35, Issue 1, pp. 29 - 34
SUMMARY • The aim of the present study was to determine whether inhibition of dipeptidyl peptidase IV (DPP IV) elevates arterial blood pressure and whether any... 
3-N-[(2S,3S)-2-amino-3-methylpentanoyl]-1,3-thiazolidine | N2-(diphenylacetyl)-N-[(4-hydroxyphenyl)methyl]-d-arginine amide | P32/98 | Wistar-Kyoto rat | neuropeptide Y | dipeptidyl peptidase IV | spontaneously hypertensive rat | Y1 receptor | BIBP 3226 | peptide YY | 3‐N‐[(2S,3S)‐2‐amino‐3‐methylpentanoyl]‐1,3‐thiazolidine | Wistar‐Kyoto rat | N2‐(diphenylacetyl)‐N‐[(4‐hydroxyphenyl)methyl]‐d‐arginine amide | BIBP 3226 | Neuropeptide Y | Spontaneously hypertensive rat | N2-(diphenylacetyl)-N-[(4-hydroxyphenyl)methyl]-D- arginine amide | Peptide YY | Dipeptidyl peptidase IV | Pharmacology & Pharmacy | Physiology | Life Sciences & Biomedicine | Science & Technology | Antihypertensive Agents - pharmacology | Dipeptidyl Peptidase 4 - metabolism | Captopril - pharmacology | Hydralazine - pharmacology | Dipeptidyl-Peptidase IV Inhibitors - therapeutic use | Rats, Inbred WKY | Receptors, Neuropeptide Y - metabolism | Hypertension - drug therapy | Ganglionic Blockers - pharmacology | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | Arginine - analogs & derivatives | Dose-Response Relationship, Drug | Drug Interactions | Receptors, Neuropeptide Y - drug effects | Pentanoic Acids - pharmacology | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Blood Pressure - drug effects | Hypertension - enzymology | Hypertension - genetics | Thiazolidines - pharmacology | Disease Models, Animal | Chlorisondamine - pharmacology | Rats, Inbred SHR | Vasodilator Agents - pharmacology | Rats | Antihypertensive Agents - therapeutic use | Hypertension - physiopathology | Animals | Arginine - pharmacology | Receptors, Neuropeptide Y, drug effects | Antihypertensive Agents, therapeutic use | Hypertension, enzymology | Arginine, pharmacology | Hypertension, drug therapy | Arginine, analogs and derivatives | Hypertension, genetics | Pentanoic Acids, pharmacology | Captopril, pharmacology | Vasodilator Agents, pharmacology | Ganglionic Blockers, pharmacology | Hypertension, physiopathology | Blood Pressure, drug effects | Dipeptidyl-Peptidase IV Inhibitors, therapeutic use | Angiotensin-Converting Enzyme Inhibitors, pharmacology | Antihypertensive Agents, pharmacology | Chlorisondamine, pharmacology | Receptors, Neuropeptide Y, metabolism | Hydralazine, pharmacology | Dipeptidyl-Peptidase IV Inhibitors, pharmacology | Thiazolidines, pharmacology | Antigens, CD26, metabolism | Antigens, CD26, antagonists and inhibitors | Hypertension | Blood pressure | Captopril | Analysis | Index Medicus
Journal Article
Molecular Pharmacology, ISSN 0026-895X, 03/2008, Volume 73, Issue 3, pp. 789 - 800
In addition to being an important receptor in leukocyte activation and mobilization, CCR5 is the essential coreceptor for human immunodeficiency virus (HIV). A... 
Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Anti-HIV Agents - pharmacology | Benzoates - metabolism | Humans | Receptors, CCR5 - genetics | Piperazines - chemistry | Pyrimidines - metabolism | Receptors, CCR5 - metabolism | Piperidines - pharmacology | Amides - metabolism | Anti-HIV Agents - metabolism | Radioligand Assay | Binding Sites | Amides - pharmacology | Anti-HIV Agents - classification | Amino Acid Sequence | Piperidines - metabolism | Models, Molecular | Pyrimidines - pharmacology | HIV Fusion Inhibitors - pharmacology | Piperazines - pharmacology | Triazoles - metabolism | Anti-HIV Agents - chemistry | Amides - chemistry | Benzoates - pharmacology | Hydrophobic and Hydrophilic Interactions | Receptors, CCR5 - chemistry | Quaternary Ammonium Compounds - pharmacology | Cricetulus | Triazoles - chemistry | Benzoates - chemistry | Cyclohexanes - pharmacology | Molecular Sequence Data | Piperazines - metabolism | Quaternary Ammonium Compounds - chemistry | Pyrimidines - chemistry | Cyclohexanes - metabolism | Transfection | Inhibitory Concentration 50 | Cyclohexanes - chemistry | Molecular Structure | Membrane Fusion - drug effects | Spiro Compounds - chemistry | CCR5 Receptor Antagonists | CHO Cells | Protein Structure, Tertiary | Cricetinae | Piperidines - chemistry | Mutagenesis, Site-Directed | Protein Structure, Secondary | HIV-1 - drug effects | Static Electricity | Sequence Homology, Amino Acid | Triazoles - pharmacology | Animals | Quaternary Ammonium Compounds - metabolism | Spiro Compounds - pharmacology | Spiro Compounds - metabolism | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 10/2013, Volume 110, Issue 43, pp. 17253 - 17258
Phenotypic polarization of macrophages is regulated by a milieu of cues in the local tissue microenvironment. Although much is known about how soluble factors... 
Phenotypes | Cell growth | Cytokines | Stem cells | Cell lines | Actins | Physiological regulation | Macrophages | Endothelial cells | Cells | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Mannose-Binding Lectins - metabolism | Arginase - metabolism | Lectins, C-Type - immunology | Cell Shape - immunology | Myosin-Light-Chain Kinase - antagonists & inhibitors | Cytochalasin D - pharmacology | Lectins, C-Type - metabolism | Arginase - immunology | Interleukin-4 - pharmacology | Flow Cytometry | Doxorubicin - analogs & derivatives | Inflammation Mediators - metabolism | Female | Immunophenotyping - methods | Cell Polarity - drug effects | Doxorubicin - metabolism | Macrophages - immunology | Cytokines - immunology | Amides - pharmacology | Biomarkers - metabolism | Inflammation Mediators - immunology | Cell Polarity - immunology | Cytokines - metabolism | Myosin-Light-Chain Kinase - metabolism | Mice, Inbred C57BL | Biomarkers - analysis | Cells, Cultured | Receptors, Cell Surface - metabolism | Macrophages - cytology | Interleukin-13 - pharmacology | Receptors, Cell Surface - immunology | Azepines - pharmacology | Cell Shape - drug effects | Macrophages - metabolism | Animals | Mannose-Binding Lectins - immunology | Lipopolysaccharides - pharmacology | Mice | Pyridines - pharmacology | Doxorubicin - immunology | Interferon-gamma - pharmacology | Microscopy, Fluorescence | Physiological aspects | Microbial genetics | Phenotype | Genetic aspects | Research | Index Medicus | Biological Sciences | Physical Sciences
Journal Article
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 12/2018, Volume 22, Issue 12, pp. 6122 - 6133
Interleukin (IL)‐33 is a member of the IL‐1 family and is able to act cardioprotective. The aim of this study was to investigate the regulation of IL‐33 by... 
cardiac cells | interleukin‐33 | bisphosphonates | pleiotropic | statins | interleukin-33 | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | Lovastatin - pharmacology | Apoptosis - drug effects | Humans | Simvastatin - pharmacology | Fluvastatin - pharmacology | Monoterpenes - pharmacology | Heart Diseases - diet therapy | Myocardium - metabolism | Thiazolidines - pharmacology | Cytokines - genetics | Amides - pharmacology | Mevalonic Acid - pharmacology | Pravastatin - pharmacology | Myocardium - pathology | rhoA GTP-Binding Protein | Gene Expression Regulation - drug effects | Bridged Bicyclo Compounds, Heterocyclic - pharmacology | Interleukin-33 - genetics | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Cyclohexenes - pharmacology | Heart - drug effects | Myocytes, Cardiac - metabolism | Heart Diseases - drug therapy | Pyridines - pharmacology | Diphosphonates - pharmacology | Interleukins | Statins | Cardiovascular agents | Heart | Interleukin | Lovastatin | mRNA | Myocytes | Pravastatin | Kinases | Bisphosphonates | Proteins | Lipophilic | Alendronic acid | Mevalonic acid | Fluvastatin | Fibroblasts | Cytokines | Simvastatin | RhoA protein | Cardiomyocytes | Ibandronic acid | Patients | Inhibitors | latrunculin B | Atorvastatin | Reductase | Index Medicus | Original
Journal Article
Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 10/2005, Volume 32, Issue 10, pp. 817 - 824
SUMMARY 1. Rho-kinase (ROK) stimulation represents a key step in the maintenance of agonist-induced contraction, an effect counteracted by nitric oxide (NO)... 
Rho-kinase | nitric oxide | coeliac artery | Rho guanine nucleotide exchange factors | RhoA | Rho‐kinase | Coeliac artery | Nitric oxide | Pharmacology & Pharmacy | Physiology | Life Sciences & Biomedicine | Science & Technology | Celiac Artery - physiology | Gene Expression - drug effects | Nitric Oxide Synthase - antagonists & inhibitors | Male | rhoA GTP-Binding Protein - metabolism | Guanylate Cyclase - antagonists & inhibitors | Nitroprusside - pharmacology | RNA, Messenger - metabolism | Dose-Response Relationship, Drug | Quinoxalines - pharmacology | Phenylephrine - pharmacology | Oxadiazoles - pharmacology | rho-Associated Kinases | Protein-Serine-Threonine Kinases - metabolism | Amides - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Vasoconstrictor Agents - pharmacology | Guanine Nucleotide Exchange Factors - genetics | Vasodilator Agents - pharmacology | RNA, Messenger - genetics | Enzyme Inhibitors - pharmacology | Intracellular Signaling Peptides and Proteins | Rats | Rho Guanine Nucleotide Exchange Factors | Potassium - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Rats, Sprague-Dawley | Blotting, Western | Celiac Artery - drug effects | Indomethacin - pharmacology | Cyclooxygenase Inhibitors - pharmacology | Animals | Cyclic GMP - metabolism | Signal Transduction - physiology | Pyridines - pharmacology | Vasodilation - drug effects | In Vitro Techniques | Celiac Artery - metabolism | NG-Nitroarginine Methyl Ester, pharmacology | Nitric Oxide Synthase, antagonists and inhibitors | Quinoxalines, pharmacology | Enzyme Inhibitors, pharmacology | In Vitro | Vasodilator Agents, pharmacology | Cyclooxygenase Inhibitors, pharmacology | Pyridines, pharmacology | Guanine Nucleotide Exchange Factors, genetics | Vasoconstrictor Agents, pharmacology | RNA, Messenger, genetics | Celiac Artery, drug effects | Potassium, pharmacology | Protein-Serine-Threonine Kinases, metabolism | Amides, pharmacology | Vasodilation, drug effects | Gene Expression, drug effects | Nitroprusside, pharmacology | rhoA GTP-Binding Protein, metabolism | Oxadiazoles, pharmacology | Phenylephrine, pharmacology | Guanylate Cyclase, antagonists and inhibitors | Signal Transduction, physiology | Cyclic GMP, metabolism | Indomethacin, pharmacology | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 12/2008, Volume 28, Issue 51, pp. 13985 - 13994
Nicotine stimulates the activity of mesolimbic dopamine neurons, which is believed to mediate the rewarding and addictive properties of tobacco use.... 
Peroxisome proliferator-activated receptor | Electrophysiology | Fatty acid amide hydrolase | Patch clamp | Dopamine neurons | Endocannabinoids | Nicotine | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | PPAR alpha - drug effects | Cannabinoid Receptor Antagonists | Protein-Tyrosine Kinases - metabolism | Injections, Intraventricular | Male | Ventral Tegmental Area - cytology | Nicotine - pharmacology | Receptors, Cannabinoid - metabolism | Piperidines - pharmacology | Benzamides - pharmacology | Neurons - metabolism | Oleic Acids - pharmacology | Palmitic Acids - pharmacology | Neurons - drug effects | Dopamine - metabolism | Organ Culture Techniques | Carbamates - pharmacology | Pyrazoles - pharmacology | Amidohydrolases - metabolism | Ventral Tegmental Area - drug effects | Appetite Depressants - pharmacology | Enzyme Inhibitors - pharmacology | Rats | Amidohydrolases - antagonists & inhibitors | Enzyme Activation - drug effects | Ethanolamines | Nicotine - antagonists & inhibitors | Rats, Sprague-Dawley | Arachidonic Acids - pharmacology | Patch-Clamp Techniques | Animals | Rimonabant | Ventral Tegmental Area - metabolism | PPAR alpha - metabolism | Lipoxygenase Inhibitors - pharmacology | Index Medicus | dopamine neurons | nicotine | patch clamp | fatty acid amide hydrolase | electrophysiology | endocannabinoids | peroxisome proliferator-activated receptor
Journal Article
American Journal of Kidney Diseases, ISSN 0272-6386, 2010, Volume 55, Issue 5, pp. 928 - 940
Journal Article