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aminohydrolases - genetics (461) 461
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adenosine (67) 67
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sequence alignment (62) 62
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formate-tetrahydrofolate ligase - genetics (61) 61
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crystal-structure (53) 53
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enzyme stability (43) 43
mutagenesis, site-directed (43) 43
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1. Full Text Dementia revealed: Novel chromosome 6 locus for Late-onset alzheimer disease provides genetic evidence for Folate-pathway abnormalities
by Naj, Adam C and Beecham, Gary W and Martin, Eden R and Gallins, Paul J and Powell, Eric H and Konidari, Ioanna and Whitehead, Patrice L and Cai, Guiqing and Haroutunian, Vahram and Scott, William K and Vance, Jeffery M and Slifer, Michael A and Gwirtsman, Harry E and Gilbert, John R and Haines, Jonathan L and Buxbaum, Joseph D and Pericak-Vance, Margaret A
PLoS Genetics, ISSN 1553-7390, 09/2010, Volume 6, Issue 9, p. e1001130
Genome-wide association studies (GWAS) of late-onset Alzheimer disease (LOAD) have consistently observed strong evidence of association with polymorphisms in... 
TYPE-4 ALLELE | POPULATION | IDENTIFIES VARIANTS | RISK-FACTORS | COMMON DISEASES | SUSCEPTIBILITY | GENETICS & HEREDITY | TOTAL HOMOCYSTEINE | MISSENSE MUTATIONS | GENOME-WIDE ASSOCIATION | APOLIPOPROTEIN-E | Genome-Wide Association Study | Reproducibility of Results | Demography | Humans | Genetic Loci - genetics | Databases, Genetic | Male | Multienzyme Complexes - genetics | Aminohydrolases - genetics | Dementia - genetics | Chromosomes, Human, Pair 6 - genetics | Metabolic Networks and Pathways - genetics | Base Pairing - genetics | Folic Acid - metabolism | Formate-Tetrahydrofolate Ligase - genetics | Polymorphism, Single Nucleotide - genetics | Female | Aged | Alzheimer Disease - genetics | Methylenetetrahydrofolate Dehydrogenase (NADP) - genetics | Care and treatment | DNA replication | Genetic aspects | Single nucleotide polymorphisms | Research | Alzheimer's disease | Identification and classification | Health aspects | Folic acid | Studies | Genetics | Alzheimers disease | Genes
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2. Full Text Tel1(ATM)-mediated interference suppresses clustered meiotic double-strand-break formation
by Garcia, V and Gray, S and Allison, RM and Cooper, TJ and Neale, MJ
NATURE, ISSN 0028-0836, 04/2015, Volume 520, Issue 7545, pp. 114 - U277
Meiotic recombination is a critical step in gametogenesis for many organisms, enabling the creation of genetically diverse haploid gametes. In each meiotic... 
YEAST | RECOMBINATION | RESPONSES | REPAIR | MUTANTS | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | HOMOLOG | ATM | SACCHAROMYCES-CEREVISIAE | Chromatin - metabolism | Protein-Serine-Threonine Kinases - deficiency | Saccharomyces cerevisiae - genetics | Pyrophosphatases - genetics | 3-Isopropylmalate Dehydrogenase - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Aminohydrolases - genetics | DNA Breaks, Double-Stranded | Alcohol Oxidoreductases - genetics | Intracellular Signaling Peptides and Proteins - deficiency | Endodeoxyribonucleases - metabolism | Genes, Fungal - genetics | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Chromatin - chemistry | Endodeoxyribonucleases - antagonists & inhibitors | Saccharomyces cerevisiae Proteins - antagonists & inhibitors | Protein-Serine-Threonine Kinases - genetics | Saccharomyces cerevisiae Proteins - genetics | Saccharomyces cerevisiae - cytology | Meiosis - genetics | Chromosomes, Fungal - genetics | Saccharomyces cerevisiae Proteins - metabolism | Homologous Recombination - genetics | Saccharomyces cerevisiae - enzymology | Chromatin - genetics
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3. Full Text The Epitranscriptome and Innate Immunity
by O’Connell, Mary A and Mannion, Niamh M and Keegan, Liam P
PLoS Genetics, ISSN 1553-7390, 2015, Volume 11, Issue 12, p. e1005687
Our knowledge of the variety and abundances of RNA base modifications is rapidly increasing. Modified bases have critical roles in tRNAs, rRNAs, translation,... 
STRUCTURAL INSIGHTS | DOUBLE-STRANDED-RNA | SUBSTRATE-SPECIFICITY | ADENOSINE-DEAMINASE | EDITING ENZYME ADAR1 | I-LIKE RECEPTORS | GENETICS & HEREDITY | RIG-I | MAMMALIAN ADARS | LACTIS GAMMA-TOXIN | PRE-MESSENGER-RNA | Inosine - genetics | RNA-Binding Proteins - genetics | RNA Processing, Post-Transcriptional - genetics | Adenosine Deaminase - genetics | RNA Editing - genetics | Humans | RNA, Messenger - genetics | Immunity, Innate - genetics | Transcriptome - genetics | Aminohydrolases - genetics | Adenosine - genetics | Genetic aspects | Immune response | Genetic transcription | Observations | Enzymes | Adenosine | Writers | Funding | Genes | Editing | Roles | Proteins | Insects | Deoxyribonucleic acid | DNA | DNA methylation | Epigenetics | Sensors
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4. Full Text Evidence for selection at HIV host susceptibility genes in a West Central African human population
by Zhao, Kai and Ishida, Yasuko and Oleksyk, Taras K and Winkler, Cheryl A and Roca, Alfred L
BMC Evolutionary Biology, ISSN 1471-2148, 2012, Volume 12, Issue 1, pp. 237 - 237
Background: HIV-1 derives from multiple independent transfers of simian immunodeficiency virus (SIV) strains from chimpanzees to human populations. We... 
Single nucleotide polymorphisms | Mbuti pygmies | Biaka pygmies | HIV dependency factors | VARIANTS | AIDS | RESTRICTION | FOAMY VIRUS | EVOLUTIONARY BIOLOGY | GENETICS & HEREDITY | CHEMOKINE RECEPTOR | RETROVIRUS | PYGMY HUNTER-GATHERERS | INFECTION | DIVERSITY | PROGRESSION | African Continental Ancestry Group - ethnology | Humans | Endosomal Sorting Complexes Required for Transport - genetics | Molecular Sequence Data | Mutation, Missense | Receptors, Virus - genetics | Aminohydrolases - genetics | Africa, Central | Simian Acquired Immunodeficiency Syndrome - virology | Ethnic Groups - genetics | HIV-1 - physiology | Simian Acquired Immunodeficiency Syndrome - transmission | African Continental Ancestry Group - genetics | Receptors, Chemokine - genetics | Genetic Predisposition to Disease - genetics | Pan troglodytes - virology | HIV Infections - genetics | HIV Infections - virology | Selection, Genetic | Genotype | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Sequence Analysis, DNA | Simian Immunodeficiency Virus - physiology | Cullin Proteins - genetics | Host-Pathogen Interactions | Carrier Proteins - genetics | Animals | Polymorphism, Single Nucleotide | HLA-C Antigens - genetics | Africa, Western | Receptors, CXCR6 | Genetic susceptibility | Demographic aspects | Physiological aspects | Genetic aspects | Research | HIV infection | Chemokine receptors | Risk factors | Epidemics | United States | Genes | Genomics | HLA histocompatibility antigens | Development and progression | Disease susceptibility | HIV (Viruses) | Histocompatibility antigens | Comparative analysis | Health aspects | Biotechnology industry | Medical research | Genetics | Cancer | Antiviral agents | CCR5 protein | Immunodeficiency | Genomes | Single-nucleotide polymorphism | Population genetics | Infection | Acquired immune deficiency syndrome | genomics | Histocompatibility antigen HLA | Mutation | Budding | pandemics | Genotypes
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5. Full Text Functional Trade-Offs in Promiscuous Enzymes Cannot Be Explained by Intrinsic Mutational Robustness of the Native Activity
by Kaltenbach, Miriam and Emond, Stephane and Hollfelder, Florian and Tokuriki, Nobuhiko
PLoS Genetics, ISSN 1553-7390, 10/2016, Volume 12, Issue 10, p. e1006305
The extent to which an emerging new function trades off with the original function is a key characteristic of the dynamics of enzyme evolution. Various cases... 
EVOLVABILITY | CATALYTIC PROMISCUITY | SPECIFICITY | PROTEIN EVOLUTION | GENETICS & HEREDITY | SUPERFAMILY | DEGRADATION | POPULATIONS | DIVERSITY | DIRECTED EVOLUTION | S-TRIAZINE | Enzymes - chemistry | Sequence Deletion | Hydrolases - genetics | Aminohydrolases - chemistry | Carboxylic Ester Hydrolases - chemistry | Phosphoric Triester Hydrolases - genetics | Selection, Genetic | Aminohydrolases - genetics | Point Mutation | Genetic Fitness | Enzymes - genetics | Amino Acid Substitution - genetics | Carboxylic Ester Hydrolases - genetics | Hydrolases - chemistry | Models, Genetic | Phosphoric Triester Hydrolases - chemistry | Evolution, Molecular | Physiological aspects | Genetic research | Enzymes | Evolution | Genetic aspects | Research | Laboratories | Funding | Colleges & universities | Academic libraries | Investigations | Hypotheses | Councils | Plasmids | Mutation | Acquisitions & mergers | Adaptation
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6. Full Text Human APOBEC3 Induced Mutation of Human Immunodeficiency Virus Type-1 Contributes to Adaptation and Evolution in Natural Infection
by Kim, Eun-Young and Lorenzo-Redondo, Ramon and Little, Susan J and Chung, Yoon-Seok and Phalora, Prabhjeet K and Maljkovic Berry, Irina and Archer, John and Penugonda, Sudhir and Fischer, Will and Richman, Douglas D and Bhattacharya, Tanmoy and Malim, Michael H and Wolinsky, Steven M
PLoS Pathogens, ISSN 1553-7366, 2014, Volume 10, Issue 7, pp. e1004281 - e1004281
Human APOBEC3 proteins are cytidine deaminases that contribute broadly to innate immunity through the control of exogenous retrovirus replication and... 
HIGH-THROUGHPUT | REVERSE-TRANSCRIPTASE | CD4(+) T-CELLS | MICROBIOLOGY | VIF | SELECTIVE PRESSURE | VIROLOGY | DNA | IN-VIVO | ANTIVIRAL ACTIVITY | CYTIDINE DEAMINATION | HIV-1 INFECTION | PARASITOLOGY | Genome, Viral | Virus Replication - genetics | Cytidine Deaminase - genetics | HIV Infections - genetics | HIV Infections - virology | Humans | Molecular Sequence Data | Phylogeny | Aminohydrolases - genetics | HIV-1 - genetics | Mutation - genetics | Sequence Homology, Nucleic Acid | Biological Evolution | APOBEC-3G Deaminase | HIV Infections - immunology | Adaptation, Physiological - genetics | Base Sequence | Polymerase Chain Reaction | Genetic Variation - genetics | High-Throughput Nucleotide Sequencing | DNA, Viral - genetics | Cytosine Deaminase - genetics | Physiological aspects | Cellular proteins | Genetic aspects | Research | Gene mutations | HIV infection | Index Medicus | Haplotypes | Peptides | Genes | Colleges & universities | Editing | Defense mechanisms | Infections | Genomes | Genetic diversity | RNA polymerase | Proteins | Error correction & detection | HIV | Deoxyribonucleic acid | Mutation | Human immunodeficiency virus | Binding sites
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7. Full Text Natural Polymorphisms in Human APOBEC3H and HIV-1 Vif Combine in Primary T Lymphocytes to Affect Viral G-to-A Mutation Levels and Infectivity
by Refsland, Eric W and Hultquist, Judd F and Luengas, Elizabeth M and Ikeda, Terumasa and Shaban, Nadine M and Law, Emily K and Brown, William L and Reilly, Cavan and Emerman, Michael and Harris, Reuben S
PLoS Genetics, ISSN 1553-7390, 11/2014, Volume 10, Issue 11, p. e1004761
The Vif protein of HIV-1 allows virus replication by degrading several members of the host-encoded APOBEC3 family of DNA cytosine deaminases. Polymorphisms in... 
CBF-BETA | G->A HYPERMUTATION | GENETIC-VARIATION | ANTIRETROVIRAL ACTIVITY | CYTIDINE DEAMINASE APOBEC3H | RESTRICTION FACTORS | IN-VIVO | GENETICS & HEREDITY | ANTIVIRAL ACTIVITY | HUMAN-IMMUNODEFICIENCY-VIRUS | TYPE-1 | Haplotypes | Cell Line | HIV-1 - pathogenicity | Virus Replication - genetics | HIV Infections - genetics | HIV Infections - virology | Humans | vif Gene Products, Human Immunodeficiency Virus - genetics | Aminohydrolases - genetics | CD4-Positive T-Lymphocytes - pathology | HIV-1 - genetics | Polymorphism, Genetic | HIV Infections - pathology | Mutation | CD4-Positive T-Lymphocytes - virology | Gene mutations | Lymphocytes | Genetic research | Genetic aspects | Research | Host-virus relationships | HIV (Viruses) | Virus research | Health aspects | Genetic polymorphisms | Proteins | Confidence intervals | Genes | Deoxyribonucleic acid | DNA | Viruses | Infections | Viral infections
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8. Full Text Deletion of Mthfd1l causes embryonic lethality and neural tube and craniofacial defects in mice
by Jessica Momb and Jordan P. Lewandowski and Joshua D. Bryant and Rebecca Fitch and Deborah R. Surman and Steven A. Vokes and Dean R. Appling
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2013, Volume 110, Issue 2, pp. 549 - 554
Maternal supplementation with folic acid is known to reduce the incidence of neural tube defects (NTDs) by as much as 70%. Despite the strong clinical link... 
Enzymes | Phenotypes | Mitochondria | Dams | Sodium | Exons | Neural tube defects | Alleles | Formates | Embryos | CELLS | MESSENGER-RNA | MOUSE MUTANTS | RAT EMBRYOS | MULTIDISCIPLINARY SCIENCES | DEPENDENT METHYLENETETRAHYDROFOLATE DEHYDROGENASE | METHENYLTETRAHYDROFOLATE CYCLOHYDROLASE | CLOSURE | MITOCHONDRIAL C-1-TETRAHYDROFOLATE SYNTHASE | FOLIC ACID METABOLISM | KNOCKOUT MICE | Methylenetetrahydrofolate Dehydrogenase (NADP) - deficiency | Aminohydrolases - deficiency | Embryonic Development - genetics | Formate-Tetrahydrofolate Ligase - deficiency | Neural Tube Defects - genetics | Genotype | Immunoblotting | DNA Primers - genetics | Multienzyme Complexes - genetics | Aminohydrolases - genetics | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Multienzyme Complexes - deficiency | Animals | Formates - pharmacology | Gene Deletion | Formate-Tetrahydrofolate Ligase - genetics | Metabolic Networks and Pathways - physiology | Mice | Embryonic Development - drug effects | Formates - administration & dosage | Abnormalities, Multiple - genetics | Craniofacial Abnormalities - genetics | Methylenetetrahydrofolate Dehydrogenase (NADP) - genetics | Embryonic development | Phenotype | Neural tube | Physiological aspects | Genetic aspects | Research | Gene expression | Health aspects | Biological Sciences
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9. Full Text Genetic polymorphism rs6922269 in the MTHFD1L gene is associated with survival and baseline active vitamin B12 levels in post-acute coronary syndromes patients
by Palmer, Barry R and Slow, Sandy and Ellis, Katrina L and Pilbrow, Anna P and Skelton, Lorraine and Frampton, Chris M and Palmer, Suetonia C and Troughton, Richard W and Yandle, Tim G and Doughty, Rob N and Whalley, Gillian A and Lever, Michael and George, Peter M and Chambers, Stephen T and Ellis, Chris and Richards, A. Mark and Cameron, Vicky A
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e89029
Background and Aims: The methylene-tetrahydrofolate dehydrogenase (NADP+ dependent) 1-like (MTHFD1L) gene is involved in mitochondrial tetrahydrofolate... 
MORTALITY | STROKE | GENOTYPE | MULTIDISCIPLINARY SCIENCES | DISEASE | ACUTE MYOCARDIAL-INFARCTION | RISK | Myocardial Infarction - blood | Myocardial Infarction - genetics | Myocardial Infarction - mortality | Prognosis | Follow-Up Studies | Genetic Association Studies | Humans | Acute Coronary Syndrome - mortality | Genotype | Multienzyme Complexes - genetics | Vitamin B 12 - blood | Aminohydrolases - genetics | Acute Coronary Syndrome - blood | Polymorphism, Genetic | Time Factors | Alleles | Survival Analysis | Biomarkers | Formate-Tetrahydrofolate Ligase - genetics | Acute Coronary Syndrome - genetics | Methylenetetrahydrofolate Dehydrogenase (NADP) - genetics | Cohort Studies | Medical research | Hospital patients | Patient outcomes | Mortality | Genes | Research | Heart attack | Risk factors | Vitamins | Genetic polymorphisms | Analysis | Physiological aspects | Medicine, Experimental | Genetic aspects | Dihydrofolate reductase | Homocysteine | Myocardial infarction | Immunoassay | Disorders | Cardiovascular disease | Vitamin B12 | Multivariate analysis | Gene polymorphism | Mitochondria | NADP | Heart diseases | Genotypes | Deoxyribonucleic acid--DNA | Tetrahydrofolic acid | Coronary artery | Health risks | Metabolism | Risk analysis | Coronary artery disease | Patients | Survival | Cyanocobalamin | Studies | Infarction | Cardiovascular diseases | Health risk assessment | Polymorphism | Deoxyribonucleic acid | DNA
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10. Full Text Folate cycle enzyme MTHFD1L confers metabolic advantages in hepatocellular carcinoma
by Lee, Derek and Xu, Iris Ming-Jing and Chiu, David Kung-Chun and Lai, Robin Kit-Ho and Tse, Aki Pui-Wah and Li, Lynna Lan and Law, Cheuk-Ting and Tsang, Felice Ho-Ching and Wei, Larry Lai and Chan, Cerise Yuen-Ki and Wong, Chun-Ming and Ng, Irene Oi-Lin and Wong, Carmen Chak-Lui
Journal of Clinical Investigation, ISSN 0021-9738, 05/2017, Volume 127, Issue 5, pp. 1856 - 1872
Cancer cells preferentially utilize glucose and glutamine, which provide macromolecules and antioxidants that sustain rapid cell division. Metabolic... 
MEDICINE, RESEARCH & EXPERIMENTAL | SERINE BIOSYNTHESIS | OXIDATIVE STRESS | NRF2 | NEURAL-TUBE | CELL-PROLIFERATION | METASTATIC NICHE FORMATION | CANCER | ONE-CARBON METABOLISM | GENOME | PROGRESSION | Liver Neoplasms - genetics | Humans | Multienzyme Complexes - metabolism | Multienzyme Complexes - genetics | Carcinoma, Hepatocellular - enzymology | Aminohydrolases - genetics | Neoplasm Proteins - metabolism | Hep G2 Cells | Formate-Tetrahydrofolate Ligase - metabolism | Carcinoma, Hepatocellular - genetics | NF-E2-Related Factor 2 - metabolism | Carcinoma, Hepatocellular - pathology | Folic Acid - metabolism | Formate-Tetrahydrofolate Ligase - genetics | Aminohydrolases - metabolism | Liver Neoplasms - pathology | NF-E2-Related Factor 2 - genetics | Folic Acid - genetics | Liver Neoplasms - enzymology | Neoplasm Proteins - genetics | Methylenetetrahydrofolate Dehydrogenase (NADP) - genetics | Methylenetetrahydrofolate Dehydrogenase (NADP) - metabolism | Growth | Cancer cells | Genetic aspects | Models | Research | Hepatoma | Folic acid | Antioxidants | Enzymes | Metabolites | Nucleotides | Glutamine
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11. Full Text Folate network genetic variation, plasma homocysteine, and global genomic methylation content: a genetic association study
by Wernimont, Susan M and Clark, Andrew G and Stover, Patrick J and Wells, Martin T and Litonjua, Augusto A and Weiss, Scott T and Gaziano, J Michael and Tucker, Katherine L and Baccarelli, Andrea and Schwartz, Joel and Bollati, Valentina and Cassano, Patricia A
BMC Medical Genetics, ISSN 1471-2350, 11/2011, Volume 12, Issue 1, pp. 150 - 150
Background: Sequence variants in genes functioning in folate-mediated one-carbon metabolism are hypothesized to lead to changes in levels of homocysteine and... 
METAANALYSIS | POLYMORPHISMS | DNA METHYLATION | S-ADENOSYLHOMOCYSTEINE | GENETICS & HEREDITY | CARDIOVASCULAR-DISEASE | VETERANS | MTHFR | CORONARY-HEART-DISEASE | ONE-CARBON METABOLISM | VASCULAR-DISEASE | Humans | Long Interspersed Nucleotide Elements - genetics | Middle Aged | Male | Aminohydrolases - genetics | Gene Regulatory Networks | Cardiovascular Diseases - genetics | Genetic Variation | DNA Methylation | Methylenetetrahydrofolate Reductase (NADPH2) - genetics | Aged, 80 and over | Adult | Alu Elements - genetics | Methylenetetrahydrofolate Dehydrogenase (NADP) - genetics | Genetic Association Studies | Vitamin B 6 - metabolism | Sarcosine Dehydrogenase - genetics | Homocysteine - blood | Genotype | Multienzyme Complexes - genetics | Phenotype | Formate-Tetrahydrofolate Ligase - genetics | Aged | Polymorphism, Single Nucleotide | Folic Acid - genetics | Measurement | Genetic aspects | Research | Homocysteine | Metabolism | Methylation | Folic acid | DNA methylation | Womens health | Vitamin B | Genes | Genomics | Deoxyribonucleic acid--DNA
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12. Full Text Stable Transfection of the Diplomonad Parasite Spironucleus salmonicida
by Jon Jerlström-Hultqvist and Elin Einarsson and Staffan G. Svärd and Teknisk-naturvetenskapliga vetenskapsområdet and Biologiska sektionen and Uppsala universitet and Institutionen för cell- och molekylärbiologi and Mikrobiologi
Eukaryotic Cell, ISSN 1535-9778, 11/2012, Volume 11, Issue 11, pp. 1353 - 1361
Classifications Services EC Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit... 
GENE | INTESTINAL PARASITE | PURIFICATION | MICROBIOLOGY | EUKARYOTES | BARKHANUS | MONOCLONAL-ANTIBODIES | PROTEIN FUSIONS | GIARDIA-LAMBLIA | VORTENS | GENOME | Kanamycin Kinase - metabolism | Porins - metabolism | Acyltransferases - metabolism | Aminohydrolases - genetics | Acyltransferases - genetics | Hemagglutinins - metabolism | Glutathione Transferase - genetics | Transfection - methods | Chromosomes - genetics | Gentamicins - pharmacology | Cloning, Molecular | Diplomonadida - drug effects | Escherichia coli - metabolism | Organisms, Genetically Modified - metabolism | Plasmids - genetics | Chromosomes - metabolism | Inhibitory Concentration 50 | Nucleosides - pharmacology | Puromycin - pharmacology | Organisms, Genetically Modified - genetics | Recombinant Proteins - metabolism | Promoter Regions, Genetic | Porins - genetics | Genetic Vectors - metabolism | Glutathione Transferase - metabolism | Recombinant Proteins - genetics | Diplomonadida - metabolism | Genetic Markers |