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1. Full Text Amphiregulin is not required for mammary fate reprogramming
Journal of cell science, ISSN 0021-9533, 06/2017, Volume 130, Issue 12, pp. e1202 - e1202
Amphiregulin
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2. Full Text Amphiregulin-Producing Pathogenic Memory T Helper 2 Cells Instruct Eosinophils to Secrete Osteopontin and Facilitate Airway Fibrosis
by Morimoto, Yuki and Hirahara, Kiyoshi and Kiuchi, Masahiro and Wada, Tomoko and Ichikawa, Tomomi and Kanno, Toshio and Okano, Mikiko and Kokubo, Kota and Onodera, Atsushi and Sakurai, Daiju and Okamoto, Yoshitaka and Nakayama, Toshinori
Immunity (Cambridge, Mass.), ISSN 1074-7613, 07/2018, Volume 49, Issue 1, pp. 134 - 150.e6
amphiregulin | pathogenic memory Th2 cells | airway fibrosis | osteopontin | eosinophil | IL-33 | EGFR | ECRS | Life Sciences & Biomedicine | Immunology | Science & Technology | Rhinitis - immunology | Transcription, Genetic - drug effects | ErbB Receptors - metabolism | Immunomodulation | Pulmonary Fibrosis - immunology | Osteopontin - metabolism | Th2 Cells - immunology | Eosinophils - immunology | Signal Transduction - immunology | Amphiregulin - biosynthesis | Amphiregulin - pharmacology | Animals | Sinusitis - immunology | Interleukin-33 - metabolism | Amphiregulin - metabolism | Sinusitis - pathology | Female | Mice | Rhinitis - pathology | Immunologic Memory - immunology | Amphiregulin - immunology | Disease Models, Animal | Medical colleges | Epidermal growth factor | Interleukins | Analysis | Memory | Fibrosis | T cells | Cells | Asthma | Ontology | Disease | Pathogenesis | Rhinosinusitis | Genes | Interleukin | Lymphocytes T | Amphiregulin | Immunity | Inflammatory diseases | Proteins | Respiratory tract | Airway management | Interleukin 5 | Lymphocytes | Biocompatibility | Respiratory tract diseases | Subpopulations | Antigens | Immunological memory | Memory cells | Statistical analysis | Cytokines | Epidermal growth factor receptors | Leukocytes (eosinophilic) | Osteopontin | Principal components analysis | Inflammation | Variance analysis | CD4 antigen | Inventors | Lungs | Collagen | Software | Polyps | Eosinophils | Index Medicus
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3. Full Text TGFα and amphiregulin paracrine network promotes resistance to EGFR blockade in colorectal cancer cells
by Hobor, Sebastijan and Van Emburgh, Beth O and Crowley, Emily and Misale, Sandra and Di Nicolantonio, Federica and Bardelli, Alberto
Clinical cancer research, ISSN 1078-0432, 12/2014, Volume 20, Issue 24, pp. 6429 - 6438
Life Sciences & Biomedicine | Oncology | Science & Technology | Amphiregulin - secretion | Paracrine Communication | Colorectal Neoplasms - genetics | Humans | Culture Media, Conditioned - pharmacology | Drug Resistance, Neoplasm | Transforming Growth Factor alpha - secretion | Protein Kinase Inhibitors - administration & dosage | Amphiregulin - pharmacology | Antibodies, Monoclonal, Humanized - administration & dosage | MAP Kinase Signaling System - drug effects | Antibodies, Monoclonal, Humanized - pharmacology | Colorectal Neoplasms - drug therapy | Amphiregulin - metabolism | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Mutation | Cetuximab | Genes, ras | Transforming Growth Factor alpha - metabolism | Colorectal Neoplasms - metabolism | Transforming Growth Factor alpha - pharmacology | Index Medicus
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4. Full Text Amphiregulin contained in NSCLC-exosomes induces osteoclast differentiation through the activation of EGFR pathway
by Taverna, Simona and Pucci, Marzia and Giallombardo, Marco and Di Bella, Maria Antonietta and Santarpia, Mariacarmela and Reclusa, Pablo and Gil-Bazo, Ignacio and Rolfo, Christian and Alessandro, Riccardo
Scientific reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 3170 - 14
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | RNA, Small Interfering - genetics | RANK Ligand - metabolism | Coculture Techniques | Humans | Lung Neoplasms - metabolism | ErbB Receptors - genetics | Gene Expression Regulation, Neoplastic | Bone Neoplasms - secondary | Exosomes - pathology | Lung Neoplasms - pathology | Bone Neoplasms - metabolism | Biological Transport | Amphiregulin - metabolism | Bone Neoplasms - genetics | Cell Differentiation | Amphiregulin - genetics | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Osteoclasts - pathology | ErbB Receptors - metabolism | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Osteoclasts - metabolism | RANK Ligand - genetics | Animals | Cell Line, Tumor | RAW 264.7 Cells | Exosomes - chemistry | Mice | Primary Cell Culture | RNA, Small Interfering - metabolism | Intercellular signalling | Epidermal growth factor receptors | Proteolytic enzymes | Non-small cell lung carcinoma | Metastasis | Tumor cell lines | Amphiregulin | Osteolysis | Cell interactions | Exosomes | Osteoblasts | Metastases | Osteoclastogenesis | Epidermal growth factor | Proteolysis | Osteoclasts | Colon | TRANCE protein | Cancer | Index Medicus
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5. Full Text Hypoxia-inducible factor 2-alpha-dependent induction of amphiregulin dampens myocardial ischemia-reperfusion injury
by Koeppen, Michael and Lee, Jae W and Seo, Seong-Wook and Brodsky, Kelley S and Kreth, Simone and Yang, Ivana V and Buttrick, Peter M and Eckle, Tobias and Eltzschig, Holger K
Nature communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 816 - 13
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | RNA, Small Interfering - genetics | Humans | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Male | Myocardial Reperfusion Injury - pathology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Microarray Analysis | Myocardium - metabolism | Amphiregulin - metabolism | Integrases - metabolism | Myosins - metabolism | Myocardial Reperfusion Injury - genetics | Amphiregulin - genetics | Cell Line | Basic Helix-Loop-Helix Transcription Factors - genetics | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Gene Expression Regulation | Mice, Transgenic | Myocardium - pathology | Basic Helix-Loop-Helix Transcription Factors - antagonists & inhibitors | Myosins - genetics | Myocardial Reperfusion Injury - metabolism | Myocytes, Cardiac - pathology | Amphiregulin - pharmacology | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | Mice | Integrases - genetics | Myocardial Reperfusion Injury - prevention & control | RNA, Small Interfering - metabolism | Fibroblast growth factor 2 | Heart | Hypoxia-inducible factor 1 | Animal models | Transcription factors | Myocardial ischemia | Cardiomyocytes | Pharmacology | Myocytes | Amphiregulin | Coronary artery disease | Signaling | Hypoxia-inducible factor 1a | Reperfusion | DNA microarrays | Ischemia | Rodents | Animal tissues | Hypoxia | Heart diseases | Index Medicus
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6. Full Text Amphiregulin
by Berasain, Carmen and Avila, Matías A
Seminars in cell & developmental biology, ISSN 1084-9521, 04/2014, Volume 28, pp. 31 - 41
Epidermal growth factor receptor (EGFR) | Chemoresistance | Proliferation | Amphiregulin | Survival | Cell signaling | Cancer | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Cell Biology | Amphiregulin - genetics | Neoplasms - metabolism | Receptor, Epidermal Growth Factor - metabolism | Animals | Humans | Cell Proliferation - physiology | Amphiregulin - metabolism | Signal Transduction - physiology | Index Medicus
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7. Full Text Amphiregulin enhances VEGF-A production in human chondrosarcoma cells and promotes angiogenesis by inhibiting miR-206 via FAK/c-Src/PKCδ pathway
by Wang, Chao-Qun and Huang, Yu-Wen and Wang, Shih-Wei and Huang, Yuan-Li and Tsai, Chun-Hao and Zhao, Yong-Ming and Huang, Bi-Fei and Xu, Guo-Hong and Fong, Yi-Chin and Tang, Chih-Hsin
Cancer letters, ISSN 0304-3835, 2016, Volume 385, pp. 261 - 270
Hematology, Oncology and Palliative Medicine | Angiogenesis | Amphiregulin | VEGF-A | miR-206 | Life Sciences & Biomedicine | Oncology | Science & Technology | Focal Adhesion Kinase 1 - genetics | Humans | Gene Expression Regulation, Neoplastic | Neovascularization, Pathologic | Male | MicroRNAs - metabolism | Vascular Endothelial Growth Factor A - metabolism | Endothelial Progenitor Cells - enzymology | Vascular Endothelial Growth Factor A - genetics | Bone Neoplasms - pathology | Chondrosarcoma - blood supply | Dose-Response Relationship, Drug | Transfection | RNA Interference | Time Factors | Bone Neoplasms - blood supply | src-Family Kinases - metabolism | Amphiregulin - metabolism | Chondrosarcoma - genetics | Chondrosarcoma - pathology | Bone Neoplasms - genetics | Focal Adhesion Kinase 1 - metabolism | Vascular Endothelial Growth Factor A - pharmacology | Chorioallantoic Membrane - blood supply | Amphiregulin - genetics | Bone Neoplasms - enzymology | Signal Transduction | Chick Embryo | Protein Kinase C-delta - metabolism | Amphiregulin - pharmacology | Animals | Chondrosarcoma - enzymology | Endothelial Progenitor Cells - pathology | Mice, Nude | Cell Line, Tumor | Mice, Inbred BALB C | MicroRNAs - genetics | Neoplasm Staging | src-Family Kinases - genetics | Protein Kinase C-delta - genetics | Index Medicus
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8. Full Text Amphiregulin triggered epidermal growth factor receptor activation confers in vivo crizotinib‐resistance of EML4‐ALK lung cancer and circumvention by epidermal growth factor receptor inhibitors
by Taniguchi, Hirokazu and Takeuchi, Shinji and Fukuda, Koji and Nakagawa, Takayuki and Arai, Sachiko and Nanjo, Shigeki and Yamada, Tadaaki and Yamaguchi, Hiroyuki and Mukae, Hiroshi and Yano, Seiji
Cancer science, ISSN 1347-9032, 01/2017, Volume 108, Issue 1, pp. 53 - 60
lung cancer | crizotinib‐resistance | EML4‐ALK | Amphiregulin | epidermal growth factor receptor | EML4-ALK | crizotinib-resistance | Life Sciences & Biomedicine | Oncology | Science & Technology | Erlotinib Hydrochloride | Lung Neoplasms - drug therapy | RNA, Small Interfering - genetics | Amphiregulin - deficiency | Pleural Effusion | Microtubule-Associated Proteins - metabolism | Humans | Lung Neoplasms - metabolism | ErbB Receptors - genetics | Lung Neoplasms - pathology | Male | Anaplastic Lymphoma Kinase | Amphiregulin - metabolism | Pleural Neoplasms | Disease Models, Animal | Pyrazoles - pharmacology | Amphiregulin - genetics | Carcinoma, Non-Small-Cell Lung - pathology | Crizotinib | ErbB Receptors - antagonists & inhibitors | ErbB Receptors - metabolism | Carcinoma, Non-Small-Cell Lung - metabolism | Cell Cycle Proteins - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Serine Endopeptidases - metabolism | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Drug Resistance, Neoplasm - drug effects | Index Medicus | Original
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9. Full Text Multiple myeloma-derived exosomes are enriched of amphiregulin (AREG) and activate the epidermal growth factor pathway in the bone microenvironment leading to osteoclastogenesis
by Raimondo, Stefania and Saieva, Laura and Vicario, Emanuela and Pucci, Marzia and Toscani, Denise and Manno, Mauro and Raccosta, Samuele and Giuliani, Nicola and Alessandro, Riccardo
Journal of hematology and oncology, ISSN 1756-8722, 01/2019, Volume 12, Issue 1, pp. 2 - 2
Bone disease | Amphiregulin | Exosomes | Epidermal growth factor receptor | Interleukin 8 | Multiple myeloma | Oncology | Life Sciences & Biomedicine | Hematology | Science & Technology | Amphiregulin - genetics | Exosomes - metabolism | ErbB Receptors - metabolism | Antibodies, Monoclonal - pharmacology | Humans | ErbB Receptors - genetics | Tumor Microenvironment | Epidermal Growth Factor - metabolism | Interleukin-8 - antagonists & inhibitors | Multiple Myeloma - metabolism | Cell Adhesion | Osteoclasts - metabolism | Mesenchymal Stem Cells | Multiple Myeloma - pathology | Animals | Cell Line, Tumor | Ligands | RAW 264.7 Cells | Cell Differentiation | Mice | Osteoblasts - metabolism | Osteogenesis | Amphiregulin - immunology | Monoclonal antibodies | Epidermal growth factor | Interleukins | RNA | Cell differentiation | Stem cells | Plasma | Motility | Mesenchyme | Leukemia | Malignancy | Metastasis | Bone diseases | Kinases | Osteolysis | CD14 antigen | Cell adhesion & migration | Cell adhesion | Bone marrow | Cytokines | Epidermal growth factor receptors | Gene expression | Osteoclastogenesis | Osteoblastogenesis | Stromal cells | Cancer | Index Medicus
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