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Psychopharmacology (Berlin, Germany), ISSN 1432-2072, 11/2018, Volume 236, Issue 1, pp. 531 - 543
Environmental stimuli, or cues, associated with the use of drugs such as cocaine are one of the primary drivers of relapse... 
Reconsolidation | Self-administration | Neurosciences | Phosphorylation | Biomedicine | Extinction | Memory | Proteomics | Cocaine | Pharmacology/Toxicology | Psychiatry | Pharmacology & Pharmacy | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Amygdala - physiopathology | Recurrence | Amygdala - drug effects | Extinction, Psychological - drug effects | Male | Cocaine-Related Disorders - physiopathology | Motivation - drug effects | Self Administration | Basolateral Nuclear Complex - physiopathology | Phosphoproteins | Extinction, Psychological - physiology | Basolateral Nuclear Complex - drug effects | Cues | Nucleus Accumbens - physiopathology | Receptors, GABA-B - drug effects | Rats | Receptors, GABA-B - physiology | Mental Recall - drug effects | Rats, Sprague-Dawley | Association Learning - physiology | Association Learning - drug effects | Animals | Motivation - physiology | Signal Transduction - drug effects | Mental Recall - physiology | Signal Transduction - physiology | Nucleus Accumbens - drug effects | Psychological aspects | Extinction (Psychology) | Nucleus accumbens | Cell interaction | Physiological aspects | Research | Neurological research | γ-Aminobutyric acid B receptors | Drug abuse | Amygdala | Serine | Mass spectroscopy | Pharmacology | Kinases | Environmental effects | Proteins | Drug self-administration | Syntaxin | Signaling | Sensory integration | Cascades | Rodents | Syntaxin 1 | Extinction behavior | Nuclei (cytology) | Mass spectrometry | Index Medicus | memory | extinction | proteomics | cocaine | self-administration | reconsolidation | phosphorylation
Journal Article
Molecular neurobiology, ISSN 0893-7648, 12/2016, Volume 53, Issue 10, pp. 6774 - 6785
Stress derived from an adverse environment during brain development could contribute to psychiatric disorders. To study the influence of stress occurring at... 
Neurology | Neurosciences | Early life stress | Emotional disorders | Biomedicine | Neurobiology | Valproic acid | Cell Biology | Microglia | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Microglia - metabolism | Neurons - pathology | Amygdala - drug effects | Prenatal Exposure Delayed Effects - psychology | Depression - pathology | Cell Count | Male | Gene Expression Profiling | Hippocampus - drug effects | Depression - genetics | RNA, Messenger - metabolism | Emotions - drug effects | Prolactin - metabolism | Cell Differentiation - genetics | Prolactin - genetics | Social Behavior | Microglia - pathology | Female | Neurons - metabolism | Neurons - drug effects | Valproic Acid - adverse effects | Astrocytes - drug effects | Microglia - drug effects | RNA, Messenger - genetics | Hippocampus - pathology | Rats, Sprague-Dawley | Amygdala - pathology | Anxiety - pathology | Cell Shape - drug effects | Gene Expression Regulation - drug effects | Pregnancy | Prenatal Exposure Delayed Effects - genetics | Anxiety - genetics | Injections | Animals | Valproic Acid - administration & dosage | Cell Differentiation - drug effects | Cell Proliferation - drug effects | Prenatal Exposure Delayed Effects - pathology | Astrocytes - metabolism | Brain | Medical colleges | Divalproex | Neurons | Development and progression | Stress (Psychology) | Social aspects | Gene expression | Cell differentiation | Mental illness | Stress | Emotions | Index Medicus
Journal Article
Journal of Neural Transmission, ISSN 0300-9564, 12/2011, Volume 118, Issue 12, pp. 1703 - 1716
...) is a novel proprietary, selective, orally bioavailable mGluR5 NAM. MRZ-8676 (8.33, 25 and 75 mg/kg) showed a high efficacy in the rat model of LID, with the maximal effect size reaching... 
Neurology | Neurosciences | Microdialysis | Pain | Medicine & Public Health | Anxiety | Pharmacology/Toxicology | Levodopa-induced dyskinesia | Psychiatry | mGluR5 | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Excitatory Amino Acid Antagonists - therapeutic use | Calcium - metabolism | Fear - drug effects | Motor Activity - drug effects | Male | Receptors, Metabotropic Glutamate - metabolism | Monoamine Oxidase Inhibitors - administration & dosage | Adrenergic Agents - toxicity | Brain - metabolism | Dopamine Agents - adverse effects | Dose-Response Relationship, Drug | Quinolones - chemistry | Time Factors | Dyskinesia, Drug-Induced - drug therapy | Cognition Disorders - etiology | Quinolones - therapeutic use | Parkinson Disease, Secondary - drug therapy | Receptor, Metabotropic Glutamate 5 | Excitatory Amino Acid Antagonists - chemistry | Parkinson Disease, Secondary - chemically induced | Tandem Mass Spectrometry - methods | Disease Models, Animal | Dyskinesia, Drug-Induced - complications | Allosteric Regulation - drug effects | Dyskinesia, Drug-Induced - etiology | Conditioning (Psychology) - drug effects | Picolinic Acids - administration & dosage | Rats | Psychomotor Performance - drug effects | Cognition Disorders - drug therapy | Dyskinesia, Drug-Induced - pathology | Maze Learning - drug effects | Rats, Sprague-Dawley | Brain - drug effects | Oxidopamine - toxicity | Rotarod Performance Test | Animals | Analysis of Variance | Levodopa - adverse effects | In Vitro Techniques | Pain Measurement | Receptors, Metabotropic Glutamate - antagonists & inhibitors | Analgesics | Glutamate | Analysis | Formaldehyde | Antianxiety agents | Movement disorders | Index Medicus | Drugs | Basal ganglia | Pain perception | Spinal cord | Amygdala | Septum | Motor task performance | Neuromodulation | Anxiolytics | Emotions | Fear conditioning | Learning | Allosteric properties | Sensory neurons | Glutamic acid receptors (metabotropic) | Dorsal horn | Dyskinesia
Journal Article