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Science, ISSN 0036-8075, 05/2016, Volume 352, Issue 6286, pp. 712 - 716
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, p. 24
Neuroinflammation caused by local deposits of A beta(42) in the brain is key for the pathogenesis and progression of Alzheimer's disease. However, inflammation... 
STROKE | SYSTEM | IMMUNE-RESPONSE | ENERGY HOMEOSTASIS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | NEURODEGENERATION | MECHANISMS | MODEL | BRAIN | COMMUNICATION | Antimicrobial Cationic Peptides - immunology | Alzheimer Disease - complications | Brain - microbiology | Drosophila melanogaster - immunology | Hemocytes - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | MAP Kinase Kinase 4 - immunology | Drosophila Proteins - immunology | Dysbiosis - microbiology | Enterobacteriaceae Infections - complications | Alzheimer Disease - pathology | Cell Movement - immunology | Enterobacteriaceae Infections - immunology | Hemocytes - pathology | Dysbiosis - immunology | Gastrointestinal Tract - immunology | Enterobacteriaceae - immunology | Leukocyte Reduction Procedures | Amyloid beta-Peptides - genetics | Peptide Fragments - immunology | Tumor Necrosis Factor-alpha - immunology | Hemocytes - microbiology | Alzheimer Disease - immunology | Antimicrobial Cationic Peptides - genetics | Peptide Fragments - genetics | Enterobacteriaceae - growth & development | Disease Models, Animal | Alzheimer Disease - microbiology | Microbiota - immunology | Signal Transduction | Enterobacteriaceae Infections - microbiology | Gene Expression Regulation | Gastrointestinal Tract - microbiology | Enterobacteriaceae Infections - pathology | Dysbiosis - complications | Disease Progression | Drosophila melanogaster - microbiology | Animals | Brain - pathology | MAP Kinase Kinase 4 - genetics | Amyloid beta-Peptides - immunology | Drosophila Proteins - genetics | Dysbiosis - pathology | Brain - immunology
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 12/2013, Volume 94, Issue 6, pp. 1309 - 1315
Adenosine signaling augments IL‐10‐induced STAT‐3 dependent gene expression in macrophages, suggesting its role in M2c polarization. The alternatively... 
TIMP‐1 | arginase | alternative activation | Arginase | TIMP-1 | Alternative activation | CELLS | ACTIVATION | RECEPTOR AGONISTS | IL-10 PRODUCTION | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | CELL BIOLOGY | TIMP-1 PRODUCTION | A(2B) | INFLAMMATION | TNF-ALPHA | HEMATOLOGY | EXPRESSION | Analgesics - pharmacology | Serum Amyloid A Protein - genetics | Receptor, Adenosine A2B - genetics | Interleukin-13 - genetics | Phosphorylation - genetics | Adenosine A2 Receptor Agonists - pharmacology | Serum Amyloid A Protein - biosynthesis | Phosphorylation - immunology | Phosphorylation - drug effects | Interleukin-6 - metabolism | STAT3 Transcription Factor - genetics | Interleukin-6 - genetics | Macrophage Activation - immunology | Arginase - genetics | Signal Transduction - genetics | Adenosine - pharmacology | Interleukin-13 - metabolism | Receptor, Adenosine A2B - metabolism | Mice, Knockout | Interleukin-4 - immunology | Macrophages - metabolism | Signal Transduction - drug effects | Interleukin-10 - genetics | Interleukin-6 - immunology | Mice | STAT3 Transcription Factor - immunology | Tissue Inhibitor of Metalloproteinase-1 - immunology | Interleukin-10 - immunology | Macrophage Activation - genetics | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Interleukin-13 - immunology | Arginase - biosynthesis | Analgesics - immunology | Signal Transduction - immunology | Arginase - immunology | Receptor, Adenosine A2B - immunology | Interleukin-10 - metabolism | Interleukin-4 - genetics | Macrophages - immunology | STAT3 Transcription Factor - metabolism | Cell Line | Adenosine - immunology | Interleukin-4 - metabolism | Gene Expression Regulation | Macrophages - cytology | Serum Amyloid A Protein - immunology | Tissue Inhibitor of Metalloproteinase-1 - genetics | Animals | Aminopyridines - pharmacology | Macrophage Activation - drug effects | Inflammation, Extracellular Mediators, & Effector Molecules
Journal Article
Journal of Clinical Endocrinology and Metabolism, ISSN 0021-972X, 05/2017, Volume 102, Issue 5, pp. 1468 - 1477
Context: Increasing evidences suggest a correlation between gut and type 1 diabetes (T1D). Objective: The objective of this study is to evaluate the gut... 
INCREASED INTESTINAL PERMEABILITY | HEALTHY-CHILDREN | DIFFERS | FECAL MICROBIOTA | DISEASE | ENDOCRINOLOGY & METABOLISM | CELL AUTOIMMUNITY | GUT MICROBIOTA | MELLITUS | ONSET | Receptors, CCR2 - genetics | Serum Amyloid P-Component - immunology | Receptors, CCR2 - immunology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Transcriptome | Child, Preschool | Male | Monocyte Chemoattractant Proteins - immunology | Vascular Endothelial Growth Factor A - genetics | Antigens, CD - genetics | Interleukin-4 Receptor alpha Subunit - genetics | Young Adult | Celiac Disease - microbiology | Cyclooxygenase 2 - genetics | Intestinal Mucosa - immunology | Diabetes Mellitus, Type 1 - microbiology | Tumor Necrosis Factor-alpha - immunology | Child | Real-Time Polymerase Chain Reaction | Duodenum - immunology | Vascular Endothelial Growth Factor A - immunology | C-Reactive Protein - genetics | Intestinal Mucosa - microbiology | Reverse Transcriptase Polymerase Chain Reaction | Monocyte Chemoattractant Proteins - genetics | Chemokine CCL22 - genetics | Chemokine CCL19 - genetics | Adolescent | RNA, Ribosomal, 16S - genetics | C-Reactive Protein - immunology | Antigens, CD - immunology | Infant | Case-Control Studies | Duodenum - microbiology | Chemokine CCL22 - immunology | Celiac Disease - immunology | Adult | Female | Diabetes Mellitus, Type 1 - immunology | Chemokine CCL19 - immunology | Cyclooxygenase 2 - immunology | Antigens, Differentiation, Myelomonocytic - immunology | Diabetes Mellitus, Type 1 - genetics | Gastrointestinal Microbiome - genetics | Interleukin-4 Receptor alpha Subunit - immunology | Antigens, Differentiation, Myelomonocytic - genetics | Serum Amyloid P-Component - genetics | Aged | Immunohistochemistry | CC chemokine receptors | rRNA 16S | CCL19 protein | Pathogenesis | Mucosa | Microbiomes | Diabetes mellitus (insulin dependent) | Macrophages | Small intestine | Gene sequencing | CCL22 protein | Microbiota | CCR2 protein | Intestine | Interleukin 4 receptors | Digestive tract | Duodenum | Digestive system | Abnormalities | Diabetes mellitus | Inflammation | Tumor necrosis factor-α | Gene expression | Ribonucleic acid--RNA | Disease control | Patients | Celiac disease | Biopsy | Infiltration | Diabetes | Cyclooxygenase-2 | Autoimmune diseases | Monocyte chemoattractant protein 1
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2013, Volume 190, Issue 5, pp. 2241 - 2251
Journal Article
Molecular and Cellular Neuroscience, ISSN 1044-7431, 2005, Volume 29, Issue 3, pp. 381 - 393
‘Protective autoimmunity’ refers to a well-controlled anti-self response that helps the body resist neurodegeneration. The response is mediated by autoimmune T... 
PROINFLAMMATORY CYTOKINE | NEUROPROTECTIVE AUTOIMMUNITY | MULTIPLE-SCLEROSIS | ALZHEIMERS-DISEASE | MACROPHAGES | GLUTAMATE | CENTRAL-NERVOUS-SYSTEM | SELF-ANTIGEN | NEUROSCIENCES | T-CELLS | GROWTH-FACTOR-I | Amyloid beta-Peptides - pharmacology | Gliosis - therapy | Transcriptional Activation - drug effects | Gliosis - physiopathology | Hippocampus - drug effects | Lipopolysaccharides - antagonists & inhibitors | Insulin-Like Growth Factor I - drug effects | Neurodegenerative Diseases - immunology | Lipopolysaccharides - immunology | Histocompatibility Antigens Class II - drug effects | Insulin-Like Growth Factor I - immunology | Interleukin-4 - pharmacology | Lymphocyte Activation - immunology | Encephalitis - physiopathology | Microglia - immunology | Autoimmunity - immunology | T-Lymphocytes - drug effects | Tumor Necrosis Factor-alpha - immunology | Gliosis - immunology | Organ Culture Techniques | Animals, Newborn | Cell Line | Encephalitis - therapy | Hippocampus - immunology | Microglia - drug effects | Cells, Cultured | Rats | Encephalitis - immunology | Neurodegenerative Diseases - therapy | Down-Regulation - drug effects | Rats, Sprague-Dawley | Interleukin-4 - immunology | Amyloid beta-Peptides - antagonists & inhibitors | Animals | Histocompatibility Antigens Class II - immunology | Neurodegenerative Diseases - physiopathology | Tumor Necrosis Factor-alpha - drug effects | Down-Regulation - immunology | Lymphocyte Activation - drug effects | Interferon-gamma - immunology | Lipopolysaccharides - pharmacology | Amyloid beta-Peptides - immunology | T-Lymphocytes - immunology | Transcriptional Activation - physiology | Hippocampus - physiopathology | Autoimmunity - drug effects | Interferon-gamma - pharmacology
Journal Article
American Journal of Kidney Diseases, ISSN 0272-6386, 2015, Volume 66, Issue 5, pp. 756 - 767
Background Kidney diseases associated with immunoglobulin M (IgM) monoclonal gammopathy are poorly described, with few data for patient outcomes and renal... 
Nephrology | tubular disorder | membranoproliferative glomerulonephritis | amyloidosis | kidney biopsy | cast nephropathy | Monoclonal gammopathy | renal response | Waldenström magroglobulinemia | Fanconi syndrome | B-cell lymphoproliferative disorder | IgM | MULTIPLE-MYELOMA | IMMUNOLOGICAL FEATURES | WALDENSTROMS MACROGLOBULINEMIA | RENAL SIGNIFICANCE | DEPOSITION DISEASE | Waldenstrom magroglobulinemia | HEAVY-CHAIN | INTERNATIONAL WORKSHOP | LIGHT-CHAIN AMYLOIDOSIS | UNDETERMINED SIGNIFICANCE | UROLOGY & NEPHROLOGY | NEPHROTIC SYNDROME | Lymphoma, B-Cell - complications | Humans | Middle Aged | Kidney Neoplasms - immunology | Lymphoproliferative Disorders - complications | Male | Lymphoproliferative Disorders - immunology | Immunoglobulin M - immunology | Waldenstrom Macroglobulinemia - pathology | Nephritis, Interstitial - pathology | Paraproteinemias - pathology | Waldenstrom Macroglobulinemia - complications | Amyloidosis - immunology | Lymphoma, B-Cell - immunology | Aged, 80 and over | Adult | Female | Amyloidosis - etiology | Retrospective Studies | Acute Kidney Injury - immunology | Kidney Neoplasms - complications | Acute Kidney Injury - etiology | Antibodies, Monoclonal - immunology | Paraproteinemias - immunology | Glomerulonephritis, Membranoproliferative - pathology | Amyloidosis - pathology | Acute Kidney Injury - pathology | Kidney Diseases - pathology | Nephrotic Syndrome - pathology | Paraproteinemias - complications | Kidney Diseases - immunology | Nephrotic Syndrome - immunology | Waldenstrom Macroglobulinemia - immunology | Glomerulonephritis, Membranoproliferative - immunology | B-Lymphocytes - immunology | Lymphoma, B-Cell - pathology | Amyloid - immunology | Nephritis, Interstitial - etiology | Kidney Neoplasms - pathology | Aged | Kidney Diseases - etiology | Glomerulonephritis, Membranoproliferative - etiology | Nephritis, Interstitial - immunology | Nephrotic Syndrome - etiology | Cohort Studies | Life Sciences | Immunology
Journal Article
Nature Immunology, ISSN 1529-2908, 02/2010, Volume 11, Issue 2, pp. 155 - 161
Journal Article