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EMBO JOURNAL, ISSN 0261-4189, 10/2010, Volume 29, Issue 19, pp. 3408 - 3420
The amyloid peptides A beta(40) and A beta(42) of Alzheimer's disease are thought to contribute differentially to the disease process. Although A beta(42)... 
LONG-TERM | BIOCHEMISTRY & MOLECULAR BIOLOGY | oligomer | TERMINAL FRAGMENT | AMYLOID PRECURSOR PROTEIN | SYNAPTIC PLASTICITY | OLIGOMERS | CELL BIOLOGY | microelectrode array | neurotoxicity | IN-VIVO | MUTATIONS | beta-amyloid peptides | Alzheimer's disease | GAMMA-SECRETASE | MOLECULAR-BASIS | AGGREGATION
Journal Article
Archives of Oral Biology, ISSN 0003-9969, 03/2019, Volume 99, pp. 120 - 125
The aim of this investigation was to determine the circulating levels of amyloid beta (Aβ) peptides using the ( ) lipopolysaccharide (LPS) model to induce... 
Porphyromonas gingivalis | Cognitive decline | Lipopolysaccharide | Endothelial dysfunction | Periodontitis | Amyloid-beta peptides | WILD-TYPE | MODELS | ALZHEIMERS-DISEASE | DENTISTRY, ORAL SURGERY & MEDICINE | BLOOD-BRAIN-BARRIER | Peptides | Mitogens | Analysis | Amyloid beta-protein | Endothelium
Journal Article
Alzheimer's & Dementia: The Journal of the Alzheimer's Association, ISSN 1552-5260, 2009, Volume 5, Issue 1, pp. 18 - 29
Journal Article
American Journal of Psychiatry, ISSN 0002-953X, 5/2012, Volume 169, Issue 5, pp. 523 - 530
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2006, Volume 23, Issue 3, pp. 669 - 678
Prion (PrP) and amyloid-β (Aβ) peptides are involved in the neuronal loss that occurs in Prion disorders (PrD) and Alzheimer's disease (AD), respectively,... 
Oxidative stress | Prion peptide | Ca 2+ homeostasis | Prion disorders | Amyloid-β peptide | Alzheimer's disease | Endoplasmic reticulum | Apoptosis | homeostasis | prion peptide | peptide | ACTIVATION | CA2+ STORES | prion disorders | ALZHEIMERS-DISEASE | CALCIUM HOMEOSTASIS | ER STRESS | apoptosis | NEUROBLASTOMA-CELLS | NEUROSCIENCES | amyloid-beta | Ca2+ homeostasis | UNFOLDED PROTEIN RESPONSE | A-BETA | endoplasmic reticulum | oxidative stress | CASPASE-12 | Neurons - pathology | Prion Diseases - physiopathology | Reactive Oxygen Species - metabolism | Calcium Channels - metabolism | Rats, Wistar | Apoptosis - drug effects | Calcium - metabolism | Peptide Fragments - toxicity | Calcium Signaling - physiology | Oxidative Stress - physiology | Endoplasmic Reticulum - metabolism | Ryanodine Receptor Calcium Release Channel - metabolism | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Electron Transport Complex IV - metabolism | Caspases - metabolism | Peptides - metabolism | Peptides - toxicity | Endoplasmic Reticulum - drug effects | Amyloid beta-Peptides - metabolism | Inositol 1,4,5-Trisphosphate Receptors | Cerebral Cortex - drug effects | Homeostasis - drug effects | Neurons - drug effects | Calcium Channels - drug effects | Prions - metabolism | Alzheimer Disease - physiopathology | Peptide Fragments - metabolism | Amyloid beta-Peptides - toxicity | Receptors, Cytoplasmic and Nuclear - drug effects | Cells, Cultured | Rats | Neurotoxins - toxicity | Ryanodine Receptor Calcium Release Channel - drug effects | Prions - toxicity | Animals | Calcium Signaling - drug effects | Homeostasis - physiology | Alzheimer Disease - metabolism | Neurotoxins - metabolism | Apoptosis - physiology | Oxidative Stress - drug effects | Prion Diseases - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Cytochrome c | Inositol | Neurosciences | Corticosteroids | Synthesis | Peptides | Analysis | Lipids | Target marketing | Index Medicus
Journal Article