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Journal Article
PLoS ONE, ISSN 1932-6203, 08/2008, Volume 3, Issue 8, pp. e2899 - e2899
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 07/2012, Volume 122, Issue 2, pp. 321 - 332
Journal Article
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2006, Volume 23, Issue 3, pp. 669 - 678
Prion (PrP) and amyloid-β (Aβ) peptides are involved in the neuronal loss that occurs in Prion disorders (PrD) and Alzheimer's disease (AD), respectively,... 
Oxidative stress | Prion peptide | Ca 2+ homeostasis | Prion disorders | Amyloid-β peptide | Alzheimer's disease | Endoplasmic reticulum | Apoptosis | homeostasis | prion peptide | peptide | ACTIVATION | CA2+ STORES | prion disorders | ALZHEIMERS-DISEASE | CALCIUM HOMEOSTASIS | ER STRESS | apoptosis | NEUROBLASTOMA-CELLS | NEUROSCIENCES | amyloid-beta | Ca2+ homeostasis | UNFOLDED PROTEIN RESPONSE | A-BETA | endoplasmic reticulum | oxidative stress | CASPASE-12 | Neurons - pathology | Prion Diseases - physiopathology | Reactive Oxygen Species - metabolism | Calcium Channels - metabolism | Rats, Wistar | Apoptosis - drug effects | Calcium - metabolism | Peptide Fragments - toxicity | Calcium Signaling - physiology | Oxidative Stress - physiology | Endoplasmic Reticulum - metabolism | Ryanodine Receptor Calcium Release Channel - metabolism | Cerebral Cortex - metabolism | Cerebral Cortex - physiopathology | Electron Transport Complex IV - metabolism | Caspases - metabolism | Peptides - metabolism | Peptides - toxicity | Endoplasmic Reticulum - drug effects | Amyloid beta-Peptides - metabolism | Inositol 1,4,5-Trisphosphate Receptors | Cerebral Cortex - drug effects | Homeostasis - drug effects | Neurons - drug effects | Calcium Channels - drug effects | Prions - metabolism | Alzheimer Disease - physiopathology | Peptide Fragments - metabolism | Amyloid beta-Peptides - toxicity | Receptors, Cytoplasmic and Nuclear - drug effects | Cells, Cultured | Rats | Neurotoxins - toxicity | Ryanodine Receptor Calcium Release Channel - drug effects | Prions - toxicity | Animals | Calcium Signaling - drug effects | Homeostasis - physiology | Alzheimer Disease - metabolism | Neurotoxins - metabolism | Apoptosis - physiology | Oxidative Stress - drug effects | Prion Diseases - metabolism | Receptors, Cytoplasmic and Nuclear - metabolism | Cytochrome c | Inositol | Neurosciences | Corticosteroids | Synthesis | Peptides | Analysis | Lipids | Target marketing | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 2012, Volume 485, Issue 7400, pp. 651 - 655
Extracellular plaques of amyloid-beta and intraneuronal neurofibrillary tangles made from tau are the histopathological signatures of