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Journal of Neuroscience, ISSN 0270-6474, 07/2012, Volume 32, Issue 28, pp. 9677 - 9689
Passive immunization against beta-amyloid (A beta) has become an increasingly desirable strategy as a therapeutic treatment for Alzheimer's disease (AD).... 
APP TRANSGENIC MICE | NATURAL OLIGOMERS | HUMAN IGG1 | ALZHEIMERS-DISEASE | PROTEIN-KINASE | LONG-TERM POTENTIATION | SYNAPTIC PLASTICITY | NEUROSCIENCES | PASSIVE-IMMUNIZATION | SECRETED OLIGOMERS | P38 MAP KINASE | Microglia - metabolism | Humans | Middle Aged | Male | Green Fluorescent Proteins - genetics | Neuroprotective Agents - metabolism | Alzheimer Disease - pathology | Neuroprotective Agents - pharmacology | Time Factors | Protein Binding - drug effects | Amyloid beta-Peptides - metabolism | Statistics, Nonparametric | Aged, 80 and over | Neurons - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Alzheimer Disease - immunology | Receptors, Chemokine - genetics | Plaque, Amyloid - immunology | Disease Models, Animal | Animals, Newborn | Rats | Mice, Transgenic | Mutation - genetics | Rats, Sprague-Dawley | Microscopy, Confocal | Plaque, Amyloid - metabolism | Mice | CX3C Chemokine Receptor 1 | Alzheimer Disease - blood | Immunoglobulin G - metabolism | Tumor Necrosis Factor-alpha - metabolism | Dose-Response Relationship, Immunologic | Cerebral Cortex - cytology | Dose-Response Relationship, Drug | Immunoglobulin G - pharmacology | Female | Neurons - drug effects | Peptide Fragments - metabolism | Double-Blind Method | Enzyme-Linked Immunosorbent Assay | Microglia - drug effects | Plaque, Amyloid - pathology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Alzheimer Disease - therapy | Cells, Cultured | Presenilin-1 - genetics | Hippocampus - cytology | Gene Expression Regulation - drug effects | Amyloid beta-Protein Precursor - genetics | Animals | Amyloid beta-Peptides - immunology | Aged
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2012, Volume 109, Issue 27, pp. 11025 - 11030
Journal Article
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 11/2008, Volume 86, Issue 11, pp. 1255 - 1267
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2014, Volume 34, Issue 38, pp. 12893 - 12903
Journal Article
Molecular Nutrition & Food Research, ISSN 1613-4125, 10/2014, Volume 58, Issue 10, pp. 1931 - 1940
Scope Dietary polyphenols are suggested to play a role in the prevention of Alzheimer's disease, of which accumulation of aggregated beta amyloid (Aβ) is a key... 
Amyloid β peptide | Caenorhabditis elegans | Alzheimer's disease | Autophagy | Proteasome | Unfolded protein response | ALZHEIMERS-DISEASE | MEDITERRANEAN DIET | FOOD SCIENCE & TECHNOLOGY | NEURODEGENERATION | MODEL | IMPAIRMENT | GENE-EXPRESSION | Amyloid beta peptide | PRECURSOR PROTEIN | STRESS | AGGREGATION | Humans | Caenorhabditis elegans Proteins - metabolism | Lysosomes - enzymology | Lysosomes - metabolism | RNA Interference | Alzheimer Disease - prevention & control | Amyloid beta-Peptides - genetics | Protein Aggregation, Pathological - prevention & control | Proteolysis | Amyloid beta-Peptides - metabolism | Quercetin - administration & dosage | Muscles - metabolism | Peptide Fragments - genetics | Disease Models, Animal | Nerve Tissue Proteins - antagonists & inhibitors | Paralysis - chemically induced | Peptide Fragments - metabolism | Protein Aggregation, Pathological - enzymology | Animals, Genetically Modified | Quercetin - therapeutic use | Unfolded Protein Response | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Amyloid beta-Peptides - antagonists & inhibitors | Antioxidants - therapeutic use | Animals | Caenorhabditis elegans Proteins - antagonists & inhibitors | Muscles - enzymology | Peptide Fragments - antagonists & inhibitors | Paralysis - prevention & control | Alzheimer Disease - metabolism | Antioxidants - administration & dosage | Proteasome Endopeptidase Complex - metabolism | Caenorhabditis elegans Proteins - genetics | Dietary Supplements | Protein Aggregation, Pathological - metabolism | Proteins | Analysis | Polyphenols | Genetic engineering | Mitochondrial DNA | Paralysis
Journal Article
European Journal of Nutrition, ISSN 1436-6207, 3/2016, Volume 55, Issue 2, pp. 741 - 747
Journal Article