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Science, ISSN 0036-8075, 03/2012, Volume 335, Issue 6075, pp. 1503 - 1506
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 10/2017, Volume 54, Issue 8, pp. 6507 - 6522
Alzheimer’s disease (AD) is associated with reduced insulin level and impairment of insulin receptor (IR) signaling in the brain, which correlates to amyloid... 
Neurology | post synaptic neurotoxicity | Neurosciences | Glial activation | Biomedicine | Neurobiology | Insulin receptor dysfunction | Amyloidogenic protein expression | Intranasal insulin | Cell Biology | OXIDATIVE STRESS | ALZHEIMERS-DISEASE | IMPROVES MEMORY | FACTOR-KAPPA-B | DEPENDENT PATHWAY | KINASE-B | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | A-BETA | CENTRAL-NERVOUS-SYSTEM | DEGRADING ENZYME | Peptide Fragments | Phosphorylation | Streptozocin | Cerebral Cortex | Glycogen Synthase Kinase 3 | Glycogen Synthase Kinase 3 beta | Male | Irs1 protein, rat | Hypoglycemic Agents | NF-kappa B | Aspartic Acid Endopeptidases | Amyloid Precursor Protein Secretases | Amyloid beta-Peptides | Insulin Receptor Substrate Proteins | Bace protein, rat | amyloid beta-protein (1-42) | Cytokines | Signal Transduction | Astrocytes | Down-Regulation | Rats | Inflammation | Administration, Intranasal | Insulin | Microglia | Index Medicus | Proto-Oncogene Proteins c-akt | Memory Disorders | Animals | Receptor, Insulin | Spatial Memory | glycogen synthase kinase 3 alpha | Hippocampus | Amyloidogenesis | Neuroprotection | Brain | Cerebral cortex | Mental disorders | Memory | Medical services | Impairment | Activation | Neuronal-glial interactions | Inflammatory diseases | Neurotoxicity | Cell activation | Rodents | Amyloid | Drug therapy | Alzheimer's disease | Translocation | b-Site APP cleaving enzyme 1 | Neurodegenerative diseases | Neurons | Glial fibrillary acidic protein | Cortex | Caspase | Hypoglycemia | Pathology | Tumor necrosis factor | Interleukin 10 | Cyclooxygenase-2 | b-Site APP cleaving enzyme | Alzheimers disease | Microglia - metabolism | Memory Disorders - metabolism | NF-kappa B - metabolism | Hippocampus - drug effects | Insulin Receptor Substrate Proteins - metabolism | Cerebral Cortex - metabolism | Inflammation - metabolism | Hypoglycemic Agents - administration & dosage | Inflammation - drug therapy | Amyloid beta-Peptides - metabolism | Cerebral Cortex - drug effects | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Hypoglycemic Agents - therapeutic use | Astrocytes - drug effects | Peptide Fragments - metabolism | Cytokines - metabolism | Microglia - drug effects | Insulin - administration & dosage | Down-Regulation - drug effects | Glycogen Synthase Kinase 3 - metabolism | Glycogen Synthase Kinase 3 beta - metabolism | Hippocampus - metabolism | Amyloid Precursor Protein Secretases - metabolism | Memory Disorders - drug therapy | Spatial Memory - drug effects | Signal Transduction - drug effects | Aspartic Acid Endopeptidases - metabolism | Receptor, Insulin - metabolism | Insulin - therapeutic use | Astrocytes - metabolism | Proteins | Medical research | Medicine, Experimental | Amyloidosis
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2014, Volume 9, Issue 4, pp. e94287 - e94287
The development of an effective therapy for Alzheimer's disease (AD) is a major challenge to biomedical sciences. Because much of early AD pathophysiology... 
APP TRANSGENIC MICE | BASAL FOREBRAIN NEURONS | MEMORY DEFICITS | MULTIDISCIPLINARY SCIENCES | ACETYLCHOLINE-RELEASE | COGNITIVE IMPAIRMENT | APPSWE/PS1DE9 MOUSE MODEL | KINASE 1 ALK1 | RAT SEPTAL NEURONS | GROWTH-FACTOR-I | IMPAIRED NEUROGENESIS | Alzheimer Disease - complications | Growth Differentiation Factor 2 - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Gliosis - physiopathology | Nerve Growth Factors - metabolism | Glial Fibrillary Acidic Protein - metabolism | Neurogenesis | Choline O-Acetyltransferase - metabolism | Alzheimer Disease - pathology | Gliosis - pathology | Amyloid beta-Peptides - metabolism | Hippocampus - enzymology | Amyloidosis - complications | Disease Models, Animal | Biomarkers - metabolism | Alzheimer Disease - physiopathology | Amyloidosis - pathology | Plaque, Amyloid - pathology | Cholinergic Neurons - metabolism | Activin Receptors, Type I - metabolism | Amyloidosis - physiopathology | Mice, Transgenic | Neuropeptides - metabolism | Hippocampus - pathology | Gliosis - complications | Insulin-Like Growth Factor II - metabolism | Plaque, Amyloid - complications | Hippocampus - metabolism | Animals | Plaque, Amyloid - physiopathology | Insulin-Like Growth Factor II - administration & dosage | Alzheimer Disease - metabolism | Mice | Cholinergic Neurons - pathology | Care and treatment | Physiological aspects | Fluorescence | Nerve growth factor | Acetylcholine | Amyloidosis | Diagnosis | Health aspects | Risk factors | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 11/2014, Volume 515, Issue 7526, pp. 274 - 278
Journal Article
Neuropharmacology, ISSN 0028-3908, 11/2016, Volume 110, Issue Pt A, pp. 519 - 529
Cannabinoid CB receptors (CB Rs) are emerging as important therapeutic targets in brain disorders that typically involve neurometabolic alterations. We here... 
Positron emission tomography (PET) | Anandamide (AEA) | Cerebral glucose uptake | β-amyloid | Cannabinoid CB2 receptor | Cyclooxygenase-2 (COX-2) | Cannabinoid CB | receptor | ACTIVATED PROTEIN-KINASE | ENDOCANNABINOID SYSTEM | KNOCKOUT MICE | ARRIVE GUIDELINES | NEUROSCIENCES | beta-amyloid | MOUSE HIPPOCAMPUS | METABOLISM | IN-VIVO | PHARMACOLOGY & PHARMACY | LONG-TERM POTENTIATION | SELECTIVE AGONISTS | EXPRESSION | Cannabinoid Receptor Modulators - pharmacology | Endocannabinoids - metabolism | Brain - diagnostic imaging | Receptor, Cannabinoid, CB2 - agonists | Aging - drug effects | Male | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Amyloidosis - diagnostic imaging | Brain - metabolism | Arachidonic Acids - metabolism | Neurons - metabolism | Hydroxyethylrutoside | Neurons - drug effects | Astrocytes - drug effects | Cyclooxygenase 2 Inhibitors - pharmacology | Tissue Culture Techniques | Mice, Inbred C57BL | Cells, Cultured | Alzheimer Disease - drug therapy | Mice, Transgenic | Amyloidosis - drug therapy | Polyunsaturated Alkamides - metabolism | Brain - drug effects | Receptor, Cannabinoid, CB2 - metabolism | Animals | Alzheimer Disease - metabolism | Cyclooxygenase 2 - metabolism | Glucose - metabolism | Alzheimer Disease - diagnostic imaging | Amyloid beta-Protein Precursor | Amyloidosis - metabolism | Nootropic Agents - pharmacology | Aging - metabolism | Astrocytes - metabolism | Index Medicus
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 12/2016, Volume 53, Issue 10, pp. 6730 - 6744
Our earlier studies showed that insulin receptor (IR) dysfunction along with neuroinflammation and amyloidogenesis played a major role in streptozotocin... 
Neurology | Neurotrophic factor | Neurosciences | Biomedicine | Memantine | Insulin receptor signaling | Astrocytes | Neurobiology | Amyloidogenic protein expression | Streptozotocin | Cell Biology | AMYLOID BETA-PROTEIN | NMDA RECEPTOR | ALZHEIMERS-DISEASE | NEUROSCIENCES | COGNITIVE DECLINE | PEPTIDE LEVELS | IN-VIVO | RAT-BRAIN | EXPRESSION | CELL-LINE | TRANSGENIC MICE | Phosphorylation | Nervous System | Oxidative Stress | Streptozocin | Cyclooxygenase 2 | Nitrites | Glycogen Synthase Kinase 3 | Glycogen Synthase Kinase 3 beta | Nitric Oxide Synthase Type II | Irs1 protein, rat | NF-kappa B | Insulin Receptor Substrate Proteins | Amyloid | Signal Transduction | Down-Regulation | Insulysin | Rats | Inflammation | Index Medicus | Animals | Nerve Growth Factors | Receptor, Insulin | Fluorescent Antibody Technique | Biomarkers | Cell Line, Tumor | glycogen synthase kinase 3 alpha | RNA, Messenger | Amyloid beta-Protein Precursor | Studies | Proteins | Insulin | Amyloidogenesis | Glial cell line-derived neurotrophic factor | Neurodegenerative diseases | memantine | Toxicity | Glutamic acid receptors (ionotropic) | Glial fibrillary acidic protein | N-Methyl-D-aspartic acid receptors | Glutamic acid receptors | Neurotrophic factors | Stress | Amyloid precursor protein | Nuclear transport | Brain-derived neurotrophic factor | Insulin receptors | Alzheimer's disease | Inflammation - pathology | Astrocytes - pathology | Nitrites - metabolism | NF-kappa B - metabolism | Nerve Growth Factors - metabolism | Insulin Receptor Substrate Proteins - metabolism | RNA, Messenger - metabolism | Inflammation - metabolism | Amyloid - metabolism | Nervous System - pathology | Amyloid beta-Protein Precursor - metabolism | Phosphorylation - drug effects | Biomarkers - metabolism | Astrocytes - drug effects | Insulysin - metabolism | Memantine - pharmacology | RNA, Messenger - genetics | Down-Regulation - drug effects | Glycogen Synthase Kinase 3 - metabolism | Glycogen Synthase Kinase 3 beta - metabolism | Signal Transduction - drug effects | Nerve Growth Factors - genetics | Cyclooxygenase 2 - metabolism | Receptor, Insulin - metabolism | Oxidative Stress - drug effects | Astrocytes - metabolism | Nitric Oxide Synthase Type II - metabolism | COX-2 inhibitors | Methyl aspartate | Amyloid beta-protein | Amyloidosis | Intermediate filament proteins | Nervous system agents
Journal Article
Aging Cell, ISSN 1474-9718, 12/2015, Volume 14, Issue 6, pp. 1067 - 1074
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 09/2011, Volume 15, Issue 5, pp. 1167 - 1178
Cerebral amyloid angiopathy (CAA) is frequently observed in Alzheimer's disease (AD) and is characterized by deposition of amyloid beta (Aβ) in leptomeningeal... 
Forum Original Research Communication | PATHOGENESIS | NADPH OXIDASE | PROTEIN | OXYGEN SPECIES MEDIATE | PERMEABILITY | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOTHELIAL-CELLS | ENDOCRINOLOGY & METABOLISM | PATHOLOGY | PEPTIDE | BASEMENT-MEMBRANE | Endothelial Cells | Reactive Oxygen Species | Oxidative Stress | Humans | Male | TJP1 protein, human | Occludin | Antioxidants | Phosphoproteins | CYBB protein, human | Tight Junctions | Amyloid beta-Peptides | Aged, 80 and over | Female | Membrane Glycoproteins | Capillaries | Advanced Glycosylation End Product-Specific Receptor | Cell Line | OCLN protein, human | Down-Regulation | Gene Expression Regulation | Microglia | Index Medicus | Membrane Proteins | Zonula Occludens-1 Protein | Cerebral Amyloid Angiopathy | Aged | RNA, Messenger | Blood-Brain Barrier | NADPH Oxidase | Brain | Advanced glycosylation end products | Tight junctions | Neurodegenerative diseases | Cortex | Data processing | Cytotoxicity | beta -Amyloid | Zonula occludens-1 protein | Endothelial cells | NAD(P)H oxidase | Cerebral amyloid angiopathy | Blood-brain barrier | Meninges | Amyloid | Alzheimer's disease | Microglia - metabolism | Reactive Oxygen Species - metabolism | Capillaries - pathology | Membrane Glycoproteins - metabolism | NADPH Oxidases - metabolism | Cerebral Amyloid Angiopathy - metabolism | Membrane Proteins - metabolism | Cerebral Amyloid Angiopathy - genetics | Capillaries - metabolism | Tight Junctions - metabolism | Amyloid beta-Peptides - toxicity | Endothelial Cells - metabolism | Membrane Proteins - genetics | RNA, Messenger - genetics | Receptor for Advanced Glycation End Products - metabolism | Antioxidants - pharmacology | Phosphoproteins - genetics | Down-Regulation - drug effects | NADPH Oxidase 2 | Down-Regulation - genetics | Blood-Brain Barrier - metabolism | Gene Expression Regulation - drug effects | Cerebral Amyloid Angiopathy - pathology | Oxidative Stress - drug effects | Tight Junctions - pathology | Endothelial Cells - drug effects | Amyloid beta-protein | Physiological aspects | Development and progression | Amyloidosis | Research
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 11/2015, Volume 33, Issue 32, pp. 3750 - 3758
Purpose In contrast to Hodgkin lymphoma and systemic anaplastic large-cell lymphoma, CD30 expression of malignant lymphocytes in mycosis fungoides (MF) and... 
STAGING SYSTEM | SURVIVAL | DEXAMETHASONE | LIGHT-CHAIN AMYLOIDOSIS | ONCOLOGY | PRIMARY SYSTEMIC AMYLOIDOSIS | HIGH-DOSE MELPHALAN | BORTEZOMIB | STEM-CELL TRANSPLANTATION | AL AMYLOIDOSIS | CARDIAC BIOMARKERS | Immunohistochemistry | biomarkers | Mycosis fungoides | Macrophages | Peripheral neuropathy | Survival | Blood | CD163 antigen | CD30 antigen | Sezary syndrome | Lymphocytes | Anaplastic large-cell lymphoma | Hodgkin's disease | Prognosis | Skin Neoplasms - drug therapy | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - administration & dosage | Immunoconjugates - administration & dosage | Antigens, CD - metabolism | Mycosis Fungoides - pathology | Mycosis Fungoides - metabolism | Research Personnel | Antineoplastic Agents - adverse effects | Aged, 80 and over | Biomarkers, Tumor - metabolism | Immunoconjugates - therapeutic use | Sezary Syndrome - drug therapy | Adult | Female | Sezary Syndrome - pathology | Immunoconjugates - adverse effects | Severity of Illness Index | Skin Neoplasms - pathology | Drug Administration Schedule | Risk Factors | Cooperative Behavior | Receptors, Cell Surface - metabolism | Ki-1 Antigen - metabolism | Mycosis Fungoides - drug therapy | Skin Neoplasms - metabolism | CD8 Antigens - metabolism | Sezary Syndrome - metabolism | Antigens, Differentiation, Myelomonocytic - metabolism | Aged | Neoplasm Staging | Index Medicus
Journal Article
Journal of Immunology, ISSN 0022-1767, 06/2011, Volume 186, Issue 11, pp. 6119 - 6128
Serum amyloid A (SAA) is an acute-phase protein, the serum levels of which can increase up to 1000-fold during inflammation. SAA has a pathogenic role in... 
cathepsins | Inflammation | TLR4 protein | Macrophages | adaptor proteins | Purine P2X receptors | Caspase-1 | Serum levels | Cell activation | Acute phase substances | TLR2 protein | Interleukin 1 | Cathepsin B | Toll-like receptors | Purine P2 receptors | Amyloidosis | Amyloid | Cardiovascular diseases | Tumor Necrosis Factor-alpha - metabolism | Inflammasomes - metabolism | Toll-Like Receptor 2 - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Serum Amyloid A Protein - pharmacology | Humans | Tumor Necrosis Factor-alpha - genetics | Caspase 1 - metabolism | Serum Amyloid A Protein - genetics | Interleukin-1beta - genetics | Receptors, Purinergic P2X7 - genetics | Dose-Response Relationship, Drug | RNA Interference | Interleukin-1beta - metabolism | Interleukin-1beta - secretion | Cell Line | Serum Amyloid A Protein - metabolism | Dipeptides - pharmacology | Mice, Inbred C57BL | Tumor Necrosis Factor-alpha - secretion | Cells, Cultured | Cathepsin B - antagonists & inhibitors | Toll-Like Receptor 4 - genetics | Cathepsin B - metabolism | Macrophages - cytology | Recombinant Proteins - pharmacology | Toll-Like Receptor 2 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Toll-Like Receptor 4 - metabolism | Blotting, Western | Inflammasomes - genetics | Mice, Knockout | Carrier Proteins - genetics | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Signal Transduction - drug effects | Caspase 1 - genetics | Receptors, Purinergic P2X7 - metabolism | Macrophages - drug effects | Mice | Index Medicus | Abridged Index Medicus
Journal Article