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1. Full Text One step towards dementia prevention
by Frisoni, Giovanni B and Jessen, Frank
The Lancet Neurology, ISSN 1474-4422, 04/2018, Volume 17, Issue 4, pp. 294 - 295
A Lancet Commission reviewed potentially modifiable risk factors for dementia, such as little education in early life, hypertension, obesity, and hearing loss... 
SUBJECTIVE COGNITIVE DECLINE | PRECLINICAL ALZHEIMERS-DISEASE | CLINICAL PROGRESSION | CLINICAL NEUROLOGY | Neuroimaging | Brain | Amyloidosis | Cognition | Alzheimer Disease | Humans | Studies | Disease prevention | Pathology | Neurodegeneration | Memory | Cognitive ability | Dementia disorders | Biomarkers | Electroencephalography | Alzheimers disease | Dementia | Index Medicus
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2. Full Text Prevention of Transthyretin Amyloid Disease by Changing Protein Misfolding Energetics
by Per Hammarström and R. Luke Wiseman and Evan T. Powers and Jeffery W. Kelly
Science, ISSN 0036-8075, 1/2003, Volume 299, Issue 5607, pp. 713 - 716
Genetic evidence suggests that inhibition of amyloid fibril formation by small molecules should be effective against amyloid diseases. Known amyloid inhibitors... 
Stoichiometry | Molecules | Proteostasis deficiencies | pH | Reports | Biochemistry | Amyloids | Kinetics | Preventive medicine | Solar fibrils | Monomers | FIBRIL FORMATION | INHIBITORS | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | Amyloidosis - prevention & control | Prealbumin - genetics | Protein Subunits | Humans | Protein Folding | Prealbumin - antagonists & inhibitors | Thermodynamics | Prealbumin - metabolism | Protein Denaturation | Protein Structure, Quaternary | Prealbumin - chemistry | Amyloidosis - metabolism | Hydrogen-Ion Concentration | Suppression, Genetic | Prevention | Chemical inhibitors | Amyloidosis | Research | Proteins | Genetics | Index Medicus
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3. Full Text Prevention of lipohypertrophy
by Kalra, Sanjay and Kumar, Arun and Gupta, Yashdeep
Journal of the Pakistan Medical Association, ISSN 0030-9982, 07/2016, Volume 66, Issue 7, pp. 910 - 911
Lipohypertrophy is an important insulin injection site reaction, which has clinically significant sequelae, including a greater risk of glycaemic variability... 
Fit guidelines | Insulin site reactions | Insulin technique | Needle reuse | Amyloidosis | FITTER guidelines | Insulin | Lipotrophy | MEDICINE, RESEARCH & EXPERIMENTAL | MEDICINE, GENERAL & INTERNAL | FIT guidelines | Drug-Related Side Effects and Adverse Reactions - prevention & control | Preventive Health Services - methods | Humans | Diabetes Mellitus - drug therapy | Drug-Related Side Effects and Adverse Reactions - etiology | Insulin - administration & dosage | Injections, Subcutaneous - adverse effects | Subcutaneous Fat - pathology | Injections, Subcutaneous - methods | Hypoglycemic Agents - administration & dosage | Subcutaneous Fat - drug effects | Hypoglycemic Agents - adverse effects | Insulin - adverse effects | Drug-Related Side Effects and Adverse Reactions - diagnosis | Hypertrophy | Index Medicus
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4. Full Text Atrophy Rates in Asymptomatic Amyloidosis: Implications for Alzheimer Prevention Trials
by Andrews, K. Abigail and Modat, Marc and Macdonald, Kate E and Yeatman, Tom and Cardoso, M. Jorge and Leung, Kelvin K and Barnes, Josephine and Villemagne, Victor L and Rowe, Christopher C and Fox, Nick C and Ourselin, Sebastien and Schott, Jonathan M and Lifestyle Flagship Study Ageing and Australian Imaging Biomarkers and Australian Imaging Biomarkers, Lifestyle Flagship Study of Ageing and Lifestyle Flagship Study of Ageing and the Australian Imaging Biomarkers
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e58816 - e58816
There is considerable interest in designing therapeutic studies of individuals at risk of Alzheimer disease (AD) to prevent the onset of symptoms. Cortical... 
MRI | BIOMARKER SIGNATURE | HIPPOCAMPAL ATROPHY | DEPOSITION | MULTIDISCIPLINARY SCIENCES | DISEASE | VOLUME | A-BETA | MILD COGNITIVE IMPAIRMENT | PITTSBURGH COMPOUND-B | BRAIN | Humans | Cognitive Dysfunction | Organ Size | Amyloidosis - diagnosis | Male | Positron-Emission Tomography | Alzheimer Disease - diagnosis | Atrophy | Magnetic Resonance Imaging | Plaque, Amyloid | Alzheimer Disease - prevention & control | Apolipoproteins E - genetics | Aged, 80 and over | Brain - pathology | Female | Aged | Advertising executives | Brain | Medical research | PET imaging | Clinical trials | Medicine, Experimental | Amyloidosis | Research | Drug therapy | Alzheimer's disease | Health aspects | Elderly people | Neuroimaging | Nuclear medicine | Load distribution | Nuclear magnetic resonance--NMR | Emission analysis | Cognitive ability | Ventricles (cerebral) | Prevention | Mathematical analysis | Older people | Tomography | Bioindicators | Age | Plaques | Apolipoprotein E4 | Medical imaging | Neurodegenerative diseases | Cortex | Feasibility studies | Positron emission | Health risks | Disease prevention | Studies | Pathology | Neurology | Brain research | Magnetic resonance imaging | Correlation analysis | Biomarkers | β-Amyloid | In vivo methods and tests | Ventricle | Positron emission tomography | Elderly | Hippocampus | Geriatrics | Dementia | Index Medicus | Nuclear magnetic resonance | NMR
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5. Full Text Addressing mechanism of fibrillization/aggregation and its prevention in presence of osmolytes: Spectroscopic and calorimetric approach
by Choudhary, Sinjan and Choudhary, Sinjan and Kishore, Nand and Kishore, Nand
PLoS ONE, ISSN 1932-6203, 2014, Volume 9, Issue 8, pp. e104600 - e104600
Understanding the mechanism of protein fibrillization/aggregation and its prevention is the basis of development of therapeutic strategies for amyloidosis. An... 
PRION PROTEIN | FIBRILLATION | IN-VITRO | AMYLOID FIBRILS | INHIBITION | PROTEIN AGGREGATION | STABILITY | MULTIDISCIPLINARY SCIENCES | LYSOZYME | ALPHA-SYNUCLEIN | THIOFLAVIN T-BINDING | Protein Aggregates | Animals | Osmolar Concentration | Muramidase - chemistry | Chickens | Spectrometry, Fluorescence | Titrimetry | Calorimetry, Differential Scanning | Thiazoles - chemistry | Amyloid - chemistry | Kinetics | Proline | Calorimetry | Amyloidosis | Health aspects | Analysis | Lysozyme | Fluorescence spectroscopy | Nucleation | Molecular structure | Fluorescence | Biochemistry | Agglomeration | Heat measurement | Stains & staining | Prevention | Fibrillogenesis | Protein folding | Elongation | Quantitative analysis | Differential scanning calorimetry | Hydration | Fibrils | Titration calorimetry | Electron microscopy | L-Proline | Trimethylamine | Transmission electron microscopy | Microscopy | Morphology | Sarcosine | Titration | Index Medicus
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6. Full Text Prevention of age-related spatial memory deficits in a transgenic mouse model of Alzheimer's disease by chronic Ginkgo biloba treatment
by Stackman, Robert W and Eckenstein, Felix and Frei, Balz and Kulhanek, Doris and Nowlin, Jessica and Quinn, Joseph F
Experimental Neurology, ISSN 0014-4886, 2003, Volume 184, Issue 1, pp. 510 - 520
Alzheimer's disease (AD) is characterized by cognitive decline and deposition of β-amyloid (Aβ) plaques in cortex and hippocampus. A transgenic mouse AD model... 
Oxidative stress | Amyloid | Antioxidant | Alzheimer | Spatial learning | Hippocampus | Spatial memory | Tg2576 | antioxidant | EXTRACT EGB 761 | hippocampus | spatial memory | GLOBAL BRAIN ISCHEMIA | spatial learning | AMYLOID-BETA-PROTEIN | HEALTHY-YOUNG VOLUNTEERS | GINSENG COMBINATION | NEUROSCIENCES | amyloid | RAT HIPPOCAMPAL CELLS | PHOSPHOLIPID BREAKDOWN | IN-VIVO | A-BETA | oxidative stress | Amyloidosis - prevention & control | Amyloidosis - pathology | Oxidation-Reduction | Plaque, Amyloid - pathology | Aging - psychology | Mice, Transgenic | Maze Learning - drug effects | Ginkgo biloba | Nerve Tissue Proteins - metabolism | Pregnancy | Memory Disorders - prevention & control | Animals | Space Perception - drug effects | Amyloid beta-Peptides - metabolism | Female | Brain Chemistry | Mice | Phytotherapy | Alzheimer Disease - psychology | Index Medicus
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7. Full Text PREVENTION OF CEREBRAL AMYLOID ANGIOPATHY IN A MOUSE MODEL OF HEREDITARY CEREBRAL HEMORRAGE WITH AMYLOIDOSIS-DUTCH TYPE
by Schelle, Juliane and Kaeser, Stephan A and Wegenast-Braun, Bettina M and Obermüller, Ulrike and Fritschi, Sarah K and Neumann, Ulf and Shimshek, Derya R and Staufenbiel, Matthias and Jucker, Mathias
Alzheimer's & Dementia: The Journal of the Alzheimer's Association, ISSN 1552-5260, 07/2017, Volume 13, Issue 7, pp. P626 - P627
Prevention | Nervous system diseases | Amyloidosis
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8. Full Text Risk Factor Analysis May Provide Clues to Diarrhea Prevention in Outdoor‐Housed Rhesus Macaques (Macaca mulatta)
by PRONGAY, KAMM and PARK, BYUNG and MURPHY, STEPHANIE J
American Journal of Primatology, ISSN 0275-2565, 08/2013, Volume 75, Issue 8, pp. 872 - 882
Seventy-five percent of rhesus macaques at national primate research centers are housed outside. Annually, 15-39% of these animals experience diarrhea and... 
mortality | risk | rhesus | morbidity | classification | diarrhea | Diarrhea | Risk | Rhesus | Mortality | Morbidity | Classification | NONHUMAN-PRIMATES | SHIGELLA-FLEXNERI | COLONIES | ZOOLOGY | AMYLOIDOSIS | JUVENILE | ADENOVIRUSES | INFECTION | MONKEYS | ASSOCIATION | CAMPYLOBACTER | Data Interpretation, Statistical | Risk Factors | Diarrhea - prevention & control | Diarrhea - veterinary | Male | Macaca mulatta | Monkey Diseases - prevention & control | Animals | Housing, Animal | Aging | Animal Husbandry | Female | Odds Ratio | Prevention | Rhesus monkey | Health risk assessment | Health aspects | Risk factors | Methods | Index Medicus | tree
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9. Full Text Alzheimer’s Disease: New Data Highlight Nonneuronal Cell Types and the Necessity for Presymptomatic Prevention Strategies
by Gandy, Sam
Biological Psychiatry, ISSN 0006-3223, 2014, Volume 75, Issue 7, pp. 553 - 557
Despite compelling genetic evidence indicating that cerebral amyloidosis can be, at least sometimes, the primary cause of Alzheimer’s disease (AD), clinical... 
Psychiatry | systems biology | neurodegeneration | tauopathy | amyloidosis | Amyloid plaques | microglia
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10. Full Text Regulation of Forkhead Transcription Factor FoxO3a Contributes to Calorie Restriction-induced Prevention of Alzheimer's Disease-type Amyloid Neuropathology and Spatial Memory Deterioration
by Qin, Weiping and Zhao, Wei and Ho, Lap and Wang, Jun and Walsh, Kenneth and Gandy, Sam and Pasinetti, Giulio Maria
Annals of the New York Academy of Sciences, ISSN 0077-8923, 12/2008, Volume 1147, Issue 1, pp. 335 - 347
Forkhead transcription factor FoxO3a, also known as DAF-16 in Caenorhabditis elegans, is a key regulator of the insulin receptor (IR)/insulin-like growth... 
calorie restriction | Forkhead transcription factor FoxO3a | Alzheimer's disease | amyloid-β peptide | amyloid‐β peptide | Amyloid-β peptide | Calorie restriction | DEACETYLASE | MULTIDISCIPLINARY SCIENCES | BIM | CELL-SURVIVAL | amyloid-beta peptide | SIRT1 | LONGEVITY | SIGNALING PATHWAY | INSULIN-RESISTANCE | MICE | TRANSGENIC MOUSE MODEL | MODULATION | Immunohistochemistry | Cricetinae | Phosphorylation | Enzyme-Linked Immunosorbent Assay | Signal Transduction | Caloric Restriction | Mice, Transgenic | Forkhead Transcription Factors - physiology | Alzheimer Disease | Blotting, Western | Brain - metabolism | Insulin - metabolism | Memory Disorders | Animals | Amyloid - metabolism | Female | Mice | Forkhead Box Protein O3 | Prevention | Medical colleges | Neurosciences | Peptides | Analysis | Amyloid beta-protein | Amyloidosis | Gene expression | Index Medicus
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11. Full Text Systematic analysis of time-dependent neural effects of soluble amyloid [beta] oligomers in culture and in vivo: Prevention by scyllo-inositol
by Jin, Ming and Selkoe, Dennis J
Neurobiology of Disease, ISSN 0969-9961, 10/2015, Volume 82, p. 152
Alzheimer's disease (AD) is currently being addressed by intensive investment in pre-clinical and clinical research on the amyloid hypothesis, but concern... 
Proteins | Oligomers | Prevention | Inositol | Analysis | Amyloidosis | Glutamate | Alzheimer's disease | Immunohistochemistry | Brain | Medical research | Medical colleges | Nervous system diseases | Neurons | Amyloid beta-protein | Medicine, Experimental
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12. Full Text The flavonoid quercetin ameliorates Alzheimer's disease pathology and protects cognitive and emotional function in aged triple transgenic Alzheimer's disease model mice
by Sabogal-Guáqueta, Angélica Maria and Muñoz-Manco, Juan Ignacio and Ramírez-Pineda, Jose R and Lamprea-Rodriguez, Marisol and Osorio, Edison and Cardona-Gómez, Gloria Patricia
Neuropharmacology, ISSN 0028-3908, 06/2015, Volume 93, pp. 134 - 145
Alzheimer's disease (AD) is the most common senile dementia in the world. Although important progress has been made in understanding the pathogenesis of AD,... 
Neuroprotection | Tauopathy | β-amyloidosis | Cognitive function | Alzheimer's disease | Quercetin | CELLS | OXIDATIVE STRESS | MICROGLIAL ACTIVATION | DOWN-REGULATION | DOPAMINERGIC-NEURONS | TAU PATHOLOGY | NEUROSCIENCES | ENTORHINAL CORTEX | IN-VITRO | MOUSE MODEL | A-BETA | PHARMACOLOGY & PHARMACY | beta-amyloidosis | Mood Disorders - prevention & control | Alzheimer Disease - complications | Neuroglia - pathology | Alzheimer Disease - pathology | Brain - metabolism | Mood Disorders - etiology | Cognition Disorders - prevention & control | tau Proteins - genetics | Cognition Disorders - etiology | Reaction Time - drug effects | Amyloid beta-Peptides - metabolism | Aging | Gliosis - prevention & control | Gliosis - etiology | Disease Models, Animal | Drug Administration Schedule | Quercetin - therapeutic use | Alzheimer Disease - drug therapy | Presenilin-1 - genetics | Mice, Transgenic | Reaction Time - genetics | Maze Learning - drug effects | Brain - drug effects | Nerve Tissue Proteins - metabolism | Antioxidants - therapeutic use | Amyloid beta-Protein Precursor - genetics | Animals | Brain - pathology | Mice | Alzheimer Disease - genetics | Models | Genetic engineering | Amyloidosis | Analysis | Neurophysiology | Index Medicus
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13. Full Text Toward the Treatment and Prevention of Alzheimer’s Disease: Rational Strategies and Recent Progress
by Gandy, Sam and DeKosky, Steven T
Annual review of medicine, ISSN 0066-4219, 2013, Volume 64, pp. 367 - 383
Alzheimer’s disease (AD) is the major cause of late-life brain failure. In the past 25 years, autosomal dominant forms of AD were found to be primariy... 
neurodegeneration | dementia | amyloidosis | memory disorder | cognitive decline
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14. Full Text AA Amyloidosis in Vaccinated Growing Chickens
by Murakami, T and Inoshima, Y and Sakamoto, E and Fukushi, H and Sakai, H and Yanai, T and Ishiguro, N
Journal of Comparative Pathology, ISSN 0021-9975, 08/2013, Volume 149, Issue 2-3, pp. 291 - 297
Systemic amyloid-A (AA) amyloidosis in birds occurs most frequently in waterfowl such as Pekin ducks. In chickens, AA amyloidosis is observed as amyloid... 
chicken | AA amyloidosis | hepatitis | vaccination | Hepatitis | Chicken | Vaccination | ARTHROPATHY | SUSCEPTIBILITY | WATERFOWL | AVIAN AMYLOIDOSIS | INDUCTION | PATHOLOGY | PATHOGENESIS | RABBITS | FIBRILS | VETERINARY SCIENCES | SYSTEMIC AMYLOIDOSIS | Immunohistochemistry | Poultry Diseases - pathology | Amyloidosis - pathology | Atadenovirus | Bacterial Vaccines - adverse effects | Poultry Diseases - etiology | Vaccination - adverse effects | Salmonella Infections - prevention & control | Viral Vaccines - adverse effects | Mycoplasma Infections - prevention & control | Amyloidosis - veterinary | Adenoviridae Infections - prevention & control | Incidence | Newcastle Disease - prevention & control | Animals | Amyloidosis - epidemiology | Chickens | Amyloidosis - etiology | Common Cold - prevention & control | Poultry Diseases - epidemiology | Mycoplasma gallisepticum | Amyloidosis | Livestock farms | Health aspects | Index Medicus
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15. Full Text All-trans retinoic acid (ATRA) prevents lipopolysaccharide-induced neuroinflammation, amyloidogenesis and memory impairment in aged rats
by Behairi, Nassima and Belkhelfa, Mourad and Rafa, Hayet and Labsi, Moussa and Deghbar, Nahla and Bouzid, Nora and Mesbah-Amroun, Hamida and Touil-Boukoffa, Chafia
Journal of Neuroimmunology, ISSN 0165-5728, 2016, Volume 300, pp. 21 - 29
Abstract We aimed to investigate preventive effects of All-trans retinoic acid (ATRA) on a lipopolysaccharide (LPS)-induced aged neuroinflammation model. We... 
Neurology | Allergy and Immunology | Aging | Neuroinflammation | All-trans retinoic acid | Alzheimer's disease | Inducible nitric oxide synthase | ALZHEIMERS-DISEASE | IMMUNOLOGY | NEUROSCIENCES | LPS | IN-VITRO | INFLAMMATION | MOUSE MODEL | DIMETHYL-SULFOXIDE | TNF-ALPHA | EX-VIVO | EXPRESSION | BRAIN | Neuroprotective Agents - therapeutic use | Inflammation - pathology | Nitric Oxide Synthase Type II - biosynthesis | Rats, Wistar | Aging - drug effects | Memory Disorders - metabolism | Male | Alzheimer Disease - pathology | Brain - metabolism | Inflammation - metabolism | Neuroprotective Agents - pharmacology | Alzheimer Disease - prevention & control | Memory Disorders - pathology | Lipopolysaccharides - toxicity | Amyloid beta-Peptides - biosynthesis | Rats | Peptide Fragments - biosynthesis | Aging - pathology | Brain - drug effects | Amyloid beta-Peptides - antagonists & inhibitors | Memory Disorders - prevention & control | Animals | Tretinoin - therapeutic use | Peptide Fragments - antagonists & inhibitors | Alzheimer Disease - metabolism | Brain - pathology | Inflammation - prevention & control | Aging - metabolism | Prevention | Memory | Nitric oxide | Amyloidosis | Mitogens | Tretinoin | Index Medicus
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