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Clinical Pharmacology & Therapeutics, ISSN 0009-9236, 07/2018, Volume 104, Issue 1, pp. 188 - 200
The accuracy of physiologically based pharmacokinetic (PBPK) model prediction in children, especially those younger than 2 years old, has not been... 
CLEARANCE | TISSUE DISTRIBUTION | IN-VIVO | VOLUME | ONTOGENY | PROTON PUMP INHIBITORS | PHARMACOLOGY & PHARMACY | PHARMACODYNAMICS | SIMULATION | DESLORATADINE | EXPOSURE | Humans | Anti-Asthmatic Agents - metabolism | Child, Preschool | Cytochrome P-450 Enzyme System - metabolism | Sufentanil - metabolism | Bronchodilator Agents - metabolism | Quinolines - pharmacokinetics | Theophylline - pharmacokinetics | Esomeprazole - metabolism | Anti-Inflammatory Agents, Non-Steroidal - metabolism | Ondansetron - metabolism | Child | Cytochrome P-450 CYP2C9 - metabolism | Histamine H1 Antagonists, Non-Sedating - pharmacokinetics | Infant, Newborn | Itraconazole - metabolism | Serotonin Antagonists - pharmacokinetics | Tramadol - pharmacokinetics | Theophylline - metabolism | Cytochrome P-450 CYP3A - metabolism | Models, Biological | Proton Pump Inhibitors - metabolism | Anti-Inflammatory Agents, Non-Steroidal - pharmacokinetics | Cytochrome P-450 CYP2D6 - metabolism | Serotonin Antagonists - metabolism | Analgesics, Opioid - metabolism | Diclofenac - metabolism | Lansoprazole - pharmacokinetics | Loratadine - pharmacokinetics | Sufentanil - pharmacokinetics | Infant | Anti-Asthmatic Agents - pharmacokinetics | Itraconazole - pharmacokinetics | Antifungal Agents - pharmacokinetics | Antifungal Agents - metabolism | Loratadine - metabolism | Cytochrome P-450 CYP2C8 - metabolism | Lansoprazole - metabolism | Acetates - metabolism | Esomeprazole - pharmacokinetics | Cytochrome P-450 CYP1A2 - metabolism | Ondansetron - pharmacokinetics | Tramadol - metabolism | Diclofenac - pharmacokinetics | Quinolines - metabolism | Pharmaceutical Preparations - metabolism | Proton Pump Inhibitors - pharmacokinetics | Bronchodilator Agents - pharmacokinetics | Analgesics, Opioid - pharmacokinetics | Cytochrome P-450 CYP2C19 - metabolism | Loratadine - analogs & derivatives | Histamine H1 Antagonists, Non-Sedating - metabolism | Pharmacokinetics | Acetates - pharmacokinetics
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, p. e47977
In recent years a growing debate is about whether botulinum neurotoxins are retrogradely transported from the site of injection. Immunodetection of cleaved... 
ACTIVATION | ACIDIC PROTEIN GFAP | RAT | TOXIN | MULTIDISCIPLINARY SCIENCES | NEURONS | AXONAL-TRANSPORT | NEUROEXOCYTOSIS | GLIAL-CELLS | EXPRESSION | SNAP-25 | Immunohistochemistry | Analgesics - pharmacology | Sciatic Nerve - injuries | Spinal Cord - drug effects | Spinal Cord - metabolism | Neuralgia - metabolism | Analgesics - metabolism | Male | Acetylcholine - metabolism | Glial Fibrillary Acidic Protein - metabolism | Botulinum Toxins, Type A - pharmacology | Hindlimb - physiopathology | Sciatic Nerve - metabolism | Biological Transport | Neuralgia - prevention & control | Neuromuscular Agents - pharmacology | Peripheral Nerves - metabolism | Schwann Cells - drug effects | Neuromuscular Agents - metabolism | Cells, Cultured | Nuclear Proteins - metabolism | Schwann Cells - metabolism | Nerve Tissue Proteins - metabolism | Microscopy, Confocal | Animals | Hindlimb - drug effects | Cell Proliferation - drug effects | Mice | Synaptosomal-Associated Protein 25 - metabolism | Botulinum Toxins, Type A - metabolism | CD11b Antigen - metabolism | Hindlimb - metabolism | Astrocytes - metabolism | Ganglia, Spinal - metabolism | Care and treatment | Analysis | Pain | Cell proliferation | Spinal cord | Neurosciences | Complement receptor 3 | Neurobiology | SNAP receptors | Biology | Neuropathy | Proteins | Biological effects | Dorsal root ganglia | Analgesics | Botulinum Toxin Type A | Animal tissues | Fibroblasts | Neurotoxin A | Horns | Growth factors | Nerve endings | CD11b antigen | Neurotoxins | Pain perception | Astrocytes | Glial fibrillary acidic protein | Schwann cells | Injection | Retrograde transport | Ganglia | Quality of life | Councils | SNAP-25 protein | Acetylcholine | Toxins | Transport | Immunofluorescence | Sciatic nerve | Combinatorial analysis
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2015, Volume 125, Issue 2, pp. 539 - 550
In contrast to microbially triggered inflammation, mechanisms promoting sterile inflammation remain poorly understood. Damage-associated molecular patterns... 
MEDICINE, RESEARCH & EXPERIMENTAL | DANGER SIGNALS | STERILE INFLAMMATION | CHROMATIN PROTEIN HMGB1 | IN-VIVO | SEPTIC SHOCK | MOBILITY GROUP BOX-1 | RECEPTOR | DEFICIENT MICE | CELL-DEATH | TRANSGENIC MICE | Acetaminophen - adverse effects | Inflammation - chemically induced | Leukocyte Elastase - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Tumor Necrosis Factor-alpha - genetics | Analgesics, Non-Narcotic - pharmacology | Hepatocytes - pathology | Hepatocytes - metabolism | Neutrophil Infiltration | fas Receptor - metabolism | Necrosis - pathology | HMGB1 Protein - genetics | Shock, Septic - metabolism | Inflammation - metabolism | Acetaminophen - pharmacology | Analgesics, Non-Narcotic - adverse effects | HMGB1 Protein - metabolism | fas Receptor - genetics | Chemical and Drug Induced Liver Injury - pathology | Neutrophils - metabolism | Receptor for Advanced Glycation End Products | Neutrophils - pathology | Shock, Septic - genetics | Shock, Septic - pathology | Lipopolysaccharides - toxicity | Macrophages - pathology | Shock, Septic - chemically induced | Leukocyte Elastase - genetics | Necrosis - metabolism | Chemical and Drug Induced Liver Injury - genetics | Mice, Knockout | Necrosis - chemically induced | Macrophages - metabolism | Animals | Chemical and Drug Induced Liver Injury - metabolism | Inflammation - genetics | Mice | Necrosis - genetics | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Physiological aspects | Causes of | Inflammation | Genetic aspects | Cell death | Necrosis | Chromosomal proteins | Mass spectrometry | Analysis | Proteins | Studies | Rodents | Mortality | Gangrene | Homeostasis | Mitochondrial DNA | Laboratory animals | Deoxyribonucleic acid--DNA | Apoptosis
Journal Article
Neuron, ISSN 0896-6273, 06/2018, Volume 98, Issue 5, pp. 963 - 976.e5
Journal Article
Spine, ISSN 0362-2436, 10/2011, Volume 36, Issue 21, pp. e1372 - e1383
Study Design. Descriptive and mechanistic investigation of the anti-inflammatory and anticatabolic effect of resveratrol in intervertebral discs (IVDs) in... 
nucleus pulposusmediated back pain | NF-κB | MAP kinases | anti-inflammatory | anticatabolic | radiculopathy | toll-like receptor | resveratrol | INTERVERTEBRAL DISCS | NECROSIS-FACTOR-ALPHA | NERVE ROOT | CYTOKINE EXPRESSION | CLINICAL NEUROLOGY | EPIDURAL APPLICATION | BACK-PAIN | NF-kappa B | LUMBAR DISC HERNIATION | nucleus pulposus-mediated back pain | TNF-ALPHA | ORTHOPEDICS | NF-KAPPA-B | MOUSE SKIN | Wine | Analgesics - pharmacology | Interleukin-8 - genetics | Toll-Like Receptor 2 - genetics | Radiculopathy - genetics | Humans | Middle Aged | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | Stilbenes - pharmacology | Pain - metabolism | RNA, Messenger - metabolism | Young Adult | Radiculopathy - drug therapy | Pain - genetics | Time Factors | Interleukin-1beta - metabolism | Toll-Like Receptor 2 - drug effects | Pain - etiology | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Disease Models, Animal | Interleukin-6 - genetics | Anti-Inflammatory Agents - pharmacology | Matrix Metalloproteinase 13 - genetics | Rats | Matrix Metalloproteinase 3 - metabolism | Toll-Like Receptor 2 - metabolism | Rats, Sprague-Dawley | Matrix Metalloproteinase 13 - metabolism | Signal Transduction - drug effects | Matrix Metalloproteinase 1 - metabolism | Matrix Metalloproteinase 3 - genetics | Sirtuin 1 - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Pain - immunology | Extracellular Signal-Regulated MAP Kinases - metabolism | Intervertebral Disc - immunology | Dose-Response Relationship, Drug | Radiculopathy - complications | Pain - drug therapy | Inflammation Mediators - metabolism | Resveratrol | Adult | Female | Radiculopathy - metabolism | Matrix Metalloproteinase 1 - genetics | Cells, Cultured | Intervertebral Disc - metabolism | Gene Expression Regulation - drug effects | Animals | Aged | Intervertebral Disc - drug effects | Pain Measurement | Radiculopathy - immunology
Journal Article
Molecular and Cellular Proteomics, ISSN 1535-9476, 05/2015, Volume 14, Issue 5, pp. 1400 - 1410
The data-independent acquisition (DIA) approach has recently been introduced as a novel mass spectrometric method that promises to combine the high content... 
DRUG-INDUCED HEPATOTOXICITY | ACCURATE | PEPTIDE IDENTIFICATION | BIOCHEMICAL RESEARCH METHODS | VALIDATION | NORMALIZATION | ABSOLUTE QUANTIFICATION | PROTEINS | MASS-SPECTROMETRY | DISCOVERY | Glutamate Formimidoyltransferase - metabolism | Oncogene Proteins - genetics | Glutamate Formimidoyltransferase - genetics | Proteome - genetics | Humans | Analgesics, Non-Narcotic - pharmacology | Voltage-Dependent Anion Channel 2 - genetics | Amidinotransferases - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Hepatocytes - metabolism | Peroxiredoxin VI - genetics | Peroxiredoxin VI - metabolism | Liver - drug effects | Acetaminophen - pharmacology | Proteolysis | Peroxiredoxin VI - analysis | Annexin A2 - analysis | Ammonia-Lyases - analysis | Proteomics - methods | Amidinotransferases - analysis | Oncogene Proteins - analysis | Intracellular Signaling Peptides and Proteins - genetics | Hepatocytes - drug effects | Gene Expression | Glutamate Formimidoyltransferase - analysis | Voltage-Dependent Anion Channel 2 - analysis | Ammonia-Lyases - genetics | Tissue Culture Techniques | Liver - metabolism | Intracellular Signaling Peptides and Proteins - analysis | Oncogene Proteins - metabolism | Ammonia-Lyases - metabolism | Proteome - analysis | Voltage-Dependent Anion Channel 2 - metabolism | Amidinotransferases - metabolism | Protein Deglycase DJ-1 | Annexin A2 - genetics | Annexin A2 - metabolism | Proteome - metabolism | Trypsin - chemistry | Peptides - analysis | Index Medicus | Research
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 03/2013, Volume 33, Issue 13, pp. 5603 - 5611
Visceral afferents expressing transient receptor potential (TRP) channels TRPV1 and TRPA1 are thought to be required for neurogenic inflammation and... 
ACTIVATION | SUBPOPULATIONS | PHOSPHORYLATION | SENSITIZATION | PROTEIN-KINASE | HYPERALGESIA | EXPRESSION | NEUROSCIENCES | SENSORY NEURONS | ERK | Calcium - metabolism | Male | Nodose Ganglion - metabolism | RNA, Messenger - metabolism | Amidines - metabolism | Time Factors | TRPV Cation Channels - antagonists & inhibitors | Pancreatitis, Chronic - drug therapy | Exploratory Behavior - drug effects | Monocytes - pathology | Pain - etiology | Sensory Receptor Cells - metabolism | Transient Receptor Potential Channels - metabolism | Calcitonin Gene-Related Peptide - metabolism | Neutrophil Infiltration - drug effects | Disease Models, Animal | Injections, Intraperitoneal | Pancreas - pathology | Analgesics, Opioid - therapeutic use | Pancreas - metabolism | Pain - pathology | Disease Progression | Pancreatitis, Chronic - pathology | Analysis of Variance | Transient Receptor Potential Channels - genetics | Ganglia, Spinal - pathology | Mice | Transient Receptor Potential Channels - antagonists & inhibitors | Oximes - therapeutic use | Pancreatitis, Chronic - chemically induced | Monocytes - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | TRPV Cation Channels - metabolism | Antigens, Differentiation - metabolism | Pyridines - therapeutic use | Sensory Receptor Cells - pathology | Pain - prevention & control | Mice, Inbred C57BL | Ceruletide - toxicity | Pancreas - drug effects | Pain Measurement - drug effects | TRPV Cation Channels - genetics | Gene Expression Regulation - drug effects | Nodose Ganglion - pathology | Animals | Sensory Receptor Cells - drug effects | Pancreatitis, Chronic - complications | TRPA1 Cation Channel | Morphine - therapeutic use | Ganglia, Spinal - metabolism | Peroxidase - metabolism
Journal Article
Journal Article
Hepatology, ISSN 0270-9139, 08/2012, Volume 56, Issue 2, pp. 735 - 746
Journal Article