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Journal of andrology, ISSN 0196-3635
Journal
1977, ISBN 9780387079417, Volume 10., xi, 178
Book
Science, ISSN 0036-8075, 5/2009, Volume 324, Issue 5928, pp. 787 - 790
Metastatic prostate cancer is treated with drugs that antagonize androgen action, but most patients progress to a more aggressive form of the disease called... 
COS cells | Androgens | Cell lines | Agonists | Oncology | Reports | Androgen antagonists | Prostate cancer | Heterologous transplantation | Tumors | Vehicles | NONSTEROIDAL ANTIANDROGENS | STRUCTURAL BASIS | AFFINITY | MULTIDISCIPLINARY SCIENCES | ANDROGEN-RECEPTOR | RESISTANCE | ANTAGONISM | LIGAND | MODEL | BICALUTAMIDE | Transcription, Genetic - drug effects | Nitriles - pharmacology | Phenylthiohydantoin - therapeutic use | Humans | Receptors, Androgen - metabolism | Biological Availability | Male | Antineoplastic Agents - therapeutic use | Tosyl Compounds - pharmacology | Antineoplastic Agents - metabolism | Cell Nucleus - metabolism | Receptors, Androgen - chemistry | Antineoplastic Agents - pharmacokinetics | Antineoplastic Agents - pharmacology | Gene Expression Regulation, Neoplastic - drug effects | Prostatic Neoplasms - drug therapy | Androgen Antagonists - pharmacokinetics | Phenylthiohydantoin - pharmacology | Nitriles - metabolism | Prostatic Neoplasms - pathology | Androgen Antagonists - metabolism | Androgen Antagonists - pharmacology | Anilides - metabolism | DNA - metabolism | Phenylthiohydantoin - analogs & derivatives | Xenograft Model Antitumor Assays | Animals | Receptors, Androgen - genetics | Anilides - pharmacology | Tosyl Compounds - metabolism | Androgen Antagonists - therapeutic use | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Phenylthiohydantoin - metabolism | Drug Screening Assays, Antitumor | Phenylthiohydantoin - pharmacokinetics | Care and treatment | Antiandrogens | Dosage and administration | Drug therapy | Methods | Cancer | Chemotherapy | Pharmacology | Binding sites
Journal Article
2006, 2nd ed., Contemporary endocrinology, ISBN 1588296636, xviii, 466
This updated, second edition is a comprehensive review of the latest research findings on the etiology, pathophysiology, clinical manifestations, and... 
Polycystic ovary syndrome | Androgens | Adrenal glands | Adrenocortical Hyperfunction | Hyperandrogenism | Metabolism | Physiological effect | Diseases | Medicine & Public Health | Endocrinology
Book
Molecular and Cellular Endocrinology, ISSN 0303-7207, 09/2012, Volume 360, Issue 1-2, pp. 3 - 13
Journal Article
Endocrinology, ISSN 0013-7227, 09/2013, Volume 154, Issue 9, pp. 3294 - 3304
Gonadal steroids are potent regulators of adult neurogenesis. We previously reported that androgens, such as testosterone (T) and dihydrotestosterone (DHT),... 
SPINAL NUCLEUS | FEMALE RATS | TESTOSTERONE | ENDOCRINOLOGY & METABOLISM | PROMOTES SURVIVAL | CELL-PROLIFERATION | GRANULE CELLS | HIPPOCAMPAL NEUROGENESIS | NEWBORN NEURONS | BRAIN | INSENSITIVITY | Castration - adverse effects | Neuroprotective Agents - therapeutic use | Neurons - pathology | Microtubule-Associated Proteins - metabolism | Neuroprotective Agents - antagonists & inhibitors | Receptors, Androgen - metabolism | Testosterone Propionate - antagonists & inhibitors | Male | Neurons - cytology | Neural Stem Cells - cytology | Hormone Replacement Therapy | Neuroprotective Agents - metabolism | Neuroprotective Agents - pharmacology | Dentate Gyrus - drug effects | Androgens - therapeutic use | Receptors, Androgen - chemistry | Neurogenesis - drug effects | Neurons - metabolism | Neurons - drug effects | Androgen Antagonists - toxicity | Mutant Proteins - antagonists & inhibitors | Drug Resistance | Biomarkers - metabolism | Cell Survival - drug effects | Testosterone Propionate - pharmacology | Dentate Gyrus - metabolism | Androgen-Insensitivity Syndrome - drug therapy | Neural Stem Cells - drug effects | Rats | Mutant Proteins - metabolism | Neuropeptides - metabolism | Neural Stem Cells - pathology | Rats, Sprague-Dawley | Dentate Gyrus - cytology | Animals | Receptors, Androgen - genetics | Androgens - pharmacology | Signal Transduction - drug effects | Androgen-Insensitivity Syndrome - metabolism | Androgen-Insensitivity Syndrome - chemically induced | Androgens - chemistry | Androgens - metabolism | Dentate Gyrus - pathology | Neural Stem Cells - metabolism | Testosterone Propionate - therapeutic use | Mutant Proteins - agonists
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 07/2017, Volume 377, Issue 4, pp. 338 - 351
Journal Article
The Lancet Oncology, ISSN 1470-2045, 09/2017, Volume 18, Issue 9, pp. e510 - e510
Journal Article
Nature, ISSN 0028-0836, 07/2015, Volume 523, Issue 7560, pp. 347 - 351
Prostate cancer resistance to castration occurs because tumours acquire the metabolic capability of converting precursor steroids to 5... 
ANDROGEN RECEPTOR | 3-BETA-HYDROXYSTEROID DEHYDROGENASE | CYP17A1 INHIBITION | MECHANISM | TESTOSTERONE | RATIONALE | MULTIDISCIPLINARY SCIENCES | INCREASED SURVIVAL | CHEMOTHERAPY | EXPOSURE | ENZALUTAMIDE | Chromatin - metabolism | Prostatic Neoplasms - metabolism | Steroid 17-alpha-Hydroxylase - antagonists & inhibitors | Androgen Receptor Antagonists - therapeutic use | Humans | Receptors, Androgen - metabolism | Gene Expression Regulation, Neoplastic | Androgens - biosynthesis | Male | Androstenes - pharmacology | Androgen Receptor Antagonists - pharmacology | 3-Hydroxysteroid Dehydrogenases - antagonists & inhibitors | Biotransformation | Cell Division | 3-Oxo-5-alpha-Steroid 4-Dehydrogenase - metabolism | 3-Hydroxysteroid Dehydrogenases - metabolism | Prostatic Neoplasms - drug therapy | Dihydrotestosterone - metabolism | Phenylthiohydantoin - pharmacology | Prostatic Neoplasms - pathology | Androstenes - therapeutic use | 5-alpha Reductase Inhibitors - therapeutic use | Prostatic Neoplasms, Castration-Resistant - drug therapy | Phenylthiohydantoin - analogs & derivatives | Androgen Receptor Antagonists - metabolism | Xenograft Model Antitumor Assays | Androstenes - chemistry | Animals | Biosynthetic Pathways - drug effects | Survival Analysis | 5-alpha Reductase Inhibitors - pharmacology | Androstenes - metabolism | Prostatic Neoplasms - enzymology | Mice | Steroid 17-alpha-Hydroxylase - metabolism | 5-alpha Reductase Inhibitors - metabolism | Androgens - metabolism | Enzymes | Testosterone | Androgens | Metabolites | Ligands | Gene expression | Prostate cancer | Tumors
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 07/2017, Volume 377, Issue 4, pp. 352 - 360
Journal Article