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Publication DateEnvironmental Health Perspectives, ISSN 0091-6765, 8/2011, Volume 119, Issue 8, pp. 1142 - 1148
Background: The large and increasing number of chemicals released into the environment demands more efficient and cost-effective approaches for assessing...
Chemical hazards | Receptors | Genomics | Environmental health | Agonists | Ligands | Cytotoxicity | Libraries | Environmental agencies | Research | Nuclear receptors | Chemical genomics | qHTS | Assay performance | Tox21 | assay performance | TOXICITY | ANTAGONISM | IDENTIFICATION | ESTROGEN-RECEPTOR | nuclear receptors | ENVIRONMENTAL SCIENCES | IN-VITRO | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | EVOLUTION | DISEASE | ASSAYS | SUPERFAMILY | HEALTH | chemical genomics | cytotoxicity | Peroxisome Proliferator-Activated Receptors - agonists | Cell Line | Retinoid X Receptor alpha - metabolism | Peroxisome Proliferator-Activated Receptors - antagonists & inhibitors | Humans | Orphan Nuclear Receptors - agonists | Receptors, Androgen - metabolism | Receptors, Cytoplasmic and Nuclear - agonists | Orphan Nuclear Receptors - metabolism | PPAR gamma - metabolism | Receptors, Thrombin - antagonists & inhibitors | Retinoid X Receptor alpha - agonists | Fragile X Mental Retardation Protein | Orphan Nuclear Receptors - antagonists & inhibitors | Retinoid X Receptor alpha - antagonists & inhibitors | PPAR gamma - antagonists & inhibitors | Receptors, Thrombin - agonists | Liver X Receptors | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Peroxisome Proliferator-Activated Receptors - metabolism | Receptors, Thrombin - metabolism | Estrogen Receptor alpha - metabolism | Environmental Pollutants - toxicity | Receptors, Cytoplasmic and Nuclear - metabolism | Environmental aspects | Physiological aspects | Cell receptors | Health aspects | Chemicals | Index Medicus
Chemical hazards | Receptors | Genomics | Environmental health | Agonists | Ligands | Cytotoxicity | Libraries | Environmental agencies | Research | Nuclear receptors | Chemical genomics | qHTS | Assay performance | Tox21 | assay performance | TOXICITY | ANTAGONISM | IDENTIFICATION | ESTROGEN-RECEPTOR | nuclear receptors | ENVIRONMENTAL SCIENCES | IN-VITRO | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | EVOLUTION | DISEASE | ASSAYS | SUPERFAMILY | HEALTH | chemical genomics | cytotoxicity | Peroxisome Proliferator-Activated Receptors - agonists | Cell Line | Retinoid X Receptor alpha - metabolism | Peroxisome Proliferator-Activated Receptors - antagonists & inhibitors | Humans | Orphan Nuclear Receptors - agonists | Receptors, Androgen - metabolism | Receptors, Cytoplasmic and Nuclear - agonists | Orphan Nuclear Receptors - metabolism | PPAR gamma - metabolism | Receptors, Thrombin - antagonists & inhibitors | Retinoid X Receptor alpha - agonists | Fragile X Mental Retardation Protein | Orphan Nuclear Receptors - antagonists & inhibitors | Retinoid X Receptor alpha - antagonists & inhibitors | PPAR gamma - antagonists & inhibitors | Receptors, Thrombin - agonists | Liver X Receptors | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Peroxisome Proliferator-Activated Receptors - metabolism | Receptors, Thrombin - metabolism | Estrogen Receptor alpha - metabolism | Environmental Pollutants - toxicity | Receptors, Cytoplasmic and Nuclear - metabolism | Environmental aspects | Physiological aspects | Cell receptors | Health aspects | Chemicals | Index Medicus
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Loading RightsEnvironmental Health Perspectives, ISSN 0091-6765, 8/2009, Volume 117, Issue 8, pp. 1210 - 1218
Background: An increasing number of studies are reporting the existence of polybrominated diphenyl ethers (PBDEs) and their hydroxylated (HO) and methoxylated...
Hormone receptors | Hormone metabolism | Receptors | Dose response relationship | Cellular metabolism | Halogenated diphenyl ethers | Plasmids | Congeners | Flame retardants | Research | Ethers | Estrogen receptor | Androgen receptor | Glucocorticoid receptor | Reporter gene assay | Brominated diphenyl ether | Thyroid hormone receptor | Chinese hamster ovary cells | glucocorticoid receptor | ESTROGEN-RECEPTORS | estrogen receptor | THYROID-HORMONE | ALPHA | brominated diphenyl ether | reporter gene assay | BETA | ENVIRONMENTAL SCIENCES | IN-VITRO | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | androgen receptor | thyroid hormone receptor | BROMINATED FLAME RETARDANTS | REPORTER GENE ASSAYS | 200 PESTICIDES | TOXICOLOGY | EXPOSURE | Androgen Receptor Antagonists | Cricetinae | Receptors, Glucocorticoid - antagonists & inhibitors | Cricetulus | Humans | Thyroid Hormone Receptors beta - antagonists & inhibitors | Estrogen Receptor alpha - antagonists & inhibitors | Thyroid Hormone Receptors alpha - antagonists & inhibitors | Receptors, Cytoplasmic and Nuclear - agonists | Halogenated Diphenyl Ethers - chemistry | Receptors, Glucocorticoid - agonists | Halogenated Diphenyl Ethers - metabolism | Androgens | Animals | Estrogen Receptor alpha - agonists | Estrogen Receptor beta - agonists | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Thyroid Hormone Receptors beta - agonists | Molecular Structure | Thyroid Hormone Receptors alpha - agonists | Estrogen Receptor beta - antagonists & inhibitors | CHO Cells | Receptors, Cytoplasmic and Nuclear - metabolism | Physiological aspects | Metabolites | Health aspects | Polybrominated biphenyls
Hormone receptors | Hormone metabolism | Receptors | Dose response relationship | Cellular metabolism | Halogenated diphenyl ethers | Plasmids | Congeners | Flame retardants | Research | Ethers | Estrogen receptor | Androgen receptor | Glucocorticoid receptor | Reporter gene assay | Brominated diphenyl ether | Thyroid hormone receptor | Chinese hamster ovary cells | glucocorticoid receptor | ESTROGEN-RECEPTORS | estrogen receptor | THYROID-HORMONE | ALPHA | brominated diphenyl ether | reporter gene assay | BETA | ENVIRONMENTAL SCIENCES | IN-VITRO | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | androgen receptor | thyroid hormone receptor | BROMINATED FLAME RETARDANTS | REPORTER GENE ASSAYS | 200 PESTICIDES | TOXICOLOGY | EXPOSURE | Androgen Receptor Antagonists | Cricetinae | Receptors, Glucocorticoid - antagonists & inhibitors | Cricetulus | Humans | Thyroid Hormone Receptors beta - antagonists & inhibitors | Estrogen Receptor alpha - antagonists & inhibitors | Thyroid Hormone Receptors alpha - antagonists & inhibitors | Receptors, Cytoplasmic and Nuclear - agonists | Halogenated Diphenyl Ethers - chemistry | Receptors, Glucocorticoid - agonists | Halogenated Diphenyl Ethers - metabolism | Androgens | Animals | Estrogen Receptor alpha - agonists | Estrogen Receptor beta - agonists | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Thyroid Hormone Receptors beta - agonists | Molecular Structure | Thyroid Hormone Receptors alpha - agonists | Estrogen Receptor beta - antagonists & inhibitors | CHO Cells | Receptors, Cytoplasmic and Nuclear - metabolism | Physiological aspects | Metabolites | Health aspects | Polybrominated biphenyls
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Loading RightsNeurotoxicity Research, ISSN 1029-8428, 1/2017, Volume 31, Issue 1, pp. 77 - 89
Dibutyl phthalate (di-n-butyl phthalate, DBP) is one of the most commonly used phthalate esters. DBP is widely used as a plasticizer in a variety of household...
Neurochemistry | Neurosciences | ERβ | ERα | Neurobiology | AhR | Phthalate | PPARγ | Cell Biology | Neurology | Biomedicine | DBP | Neuron | Pharmacology/Toxicology | Apoptosis | ANDROGEN RECEPTOR | STEM-CELLS | DI(2-ETHYLHEXYL) PHTHALATE | PPAR gamma | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | ER beta | DEVELOPMENTAL TOXICITY | IN-VITRO | INDOOR AIR | N-BUTYL PHTHALATE | GENE-EXPRESSION | EXPOSURE | ER alpha | Neurons - pathology | Reactive Oxygen Species - metabolism | Receptors, Aryl Hydrocarbon - antagonists & inhibitors | Apoptosis - drug effects | Caspase 3 - metabolism | Cerebral Cortex - pathology | PPAR gamma - metabolism | RNA, Messenger - metabolism | Cerebral Cortex - metabolism | Dose-Response Relationship, Drug | Estrogen Receptor beta - metabolism | Time Factors | Receptors, Aryl Hydrocarbon - metabolism | Estrogen Receptor alpha - metabolism | Neurons - metabolism | Cerebral Cortex - drug effects | Neurons - drug effects | Estrogen Receptor beta - antagonists & inhibitors | Cell Survival - physiology | Cell Survival - drug effects | Cells, Cultured | Estrogen Receptor alpha - antagonists & inhibitors | Animals | PPAR gamma - antagonists & inhibitors | Mice | Apoptosis - physiology | Dibutyl Phthalate - toxicity | Consumer goods | RNA | Neurons | Estrogen | Phenols | Esters | Original
Neurochemistry | Neurosciences | ERβ | ERα | Neurobiology | AhR | Phthalate | PPARγ | Cell Biology | Neurology | Biomedicine | DBP | Neuron | Pharmacology/Toxicology | Apoptosis | ANDROGEN RECEPTOR | STEM-CELLS | DI(2-ETHYLHEXYL) PHTHALATE | PPAR gamma | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | ER beta | DEVELOPMENTAL TOXICITY | IN-VITRO | INDOOR AIR | N-BUTYL PHTHALATE | GENE-EXPRESSION | EXPOSURE | ER alpha | Neurons - pathology | Reactive Oxygen Species - metabolism | Receptors, Aryl Hydrocarbon - antagonists & inhibitors | Apoptosis - drug effects | Caspase 3 - metabolism | Cerebral Cortex - pathology | PPAR gamma - metabolism | RNA, Messenger - metabolism | Cerebral Cortex - metabolism | Dose-Response Relationship, Drug | Estrogen Receptor beta - metabolism | Time Factors | Receptors, Aryl Hydrocarbon - metabolism | Estrogen Receptor alpha - metabolism | Neurons - metabolism | Cerebral Cortex - drug effects | Neurons - drug effects | Estrogen Receptor beta - antagonists & inhibitors | Cell Survival - physiology | Cell Survival - drug effects | Cells, Cultured | Estrogen Receptor alpha - antagonists & inhibitors | Animals | PPAR gamma - antagonists & inhibitors | Mice | Apoptosis - physiology | Dibutyl Phthalate - toxicity | Consumer goods | RNA | Neurons | Estrogen | Phenols | Esters | Original
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Loading RightsChemical Reviews, ISSN 0009-2665, 09/2011, Volume 111, Issue 9, pp. 5701 - 5722
DICARBA-CLOSO-DODECABORANE | ANDROGEN RECEPTOR ANTAGONISTS | POLYCYCLIC CAGE COMPOUNDS | FACTOR-ALPHA PRODUCTION | LIGAND-BINDING DOMAINS | NEUTRON-CAPTURE THERAPY | SUBSTANCE-P OCTAPEPTIDES | KINASE C-DELTA | L-ORTHO-CARBORANYLALANINE | CHEMISTRY, MULTIDISCIPLINARY | ESTROGEN-RECEPTOR | Tumor Necrosis Factor-alpha - metabolism | Receptors, Purinergic - chemistry | Receptors, Estrogen - metabolism | Receptors, Estrogen - agonists | Thymidine Kinase - chemistry | Humans | Receptors, Androgen - metabolism | Receptors, Estrogen - antagonists & inhibitors | Receptors, Retinoic Acid - metabolism | Receptors, Thrombin - chemistry | Drug Discovery | Receptors, Retinoic Acid - chemistry | Receptors, Purinergic - metabolism | Proteins - metabolism | Receptors, Estrogen - chemistry | Tumor Necrosis Factor-alpha - chemistry | Protein Kinase C - metabolism | Protein Kinase C - chemistry | Thymidine Kinase - metabolism | Receptors, Thrombin - metabolism | Ligands | Receptors, Androgen - chemistry | Proteins - chemistry | Boron Compounds | Receptors | Estrogen | Research | Hydrophobicity | Chemical properties | Structure | DNA-ligand interactions | Polycyclic compounds | Tretinoin
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Loading RightsAnnual Review of Physiology, ISSN 0066-4278, 03/2009, Volume 72, Issue 1, pp. 247 - 272
As ligand-regulated transcription factors, the nuclear hormone receptors are nearly ideal drug targets, with internal pockets that bind to hydrophobic,...
Nuclear receptor | Protein-ligand interactions | Drug discovery | protein-ligand interactions | PHYSIOLOGY | ESTROGEN-RECEPTOR | RAY CRYSTAL-STRUCTURE | GLUCOCORTICOID-RECEPTOR | drug discovery | HUMAN ANDROGEN RECEPTOR | nuclear receptor | FARNESOID-X-RECEPTOR | REV-ERB-ALPHA | PPAR-GAMMA | LIGAND-BINDING DOMAIN | THYROID-HORMONE RECEPTOR | STRUCTURE-BASED DESIGN | Receptors, Thyroid Hormone - drug effects | Amino Acid Sequence | Protein Structure, Secondary | Receptors, Cytoplasmic and Nuclear - drug effects | Humans | Drug Therapy | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - physiology | Receptors, Steroid - drug effects | Nuclear Proteins - chemistry | Drug Discovery | Receptors, Thyroid Hormone - physiology | Receptors, Estrogen - drug effects | Animals | Receptors, Steroid - physiology | Receptors, Cytoplasmic and Nuclear - chemistry | Lipid Metabolism - drug effects | Nuclear Proteins - physiology | Lipid Metabolism - physiology | Receptors, Estrogen - physiology | Drugs | Hormone receptors | Drug delivery systems | Ligand binding (Biochemistry) | Research | Vehicles
Nuclear receptor | Protein-ligand interactions | Drug discovery | protein-ligand interactions | PHYSIOLOGY | ESTROGEN-RECEPTOR | RAY CRYSTAL-STRUCTURE | GLUCOCORTICOID-RECEPTOR | drug discovery | HUMAN ANDROGEN RECEPTOR | nuclear receptor | FARNESOID-X-RECEPTOR | REV-ERB-ALPHA | PPAR-GAMMA | LIGAND-BINDING DOMAIN | THYROID-HORMONE RECEPTOR | STRUCTURE-BASED DESIGN | Receptors, Thyroid Hormone - drug effects | Amino Acid Sequence | Protein Structure, Secondary | Receptors, Cytoplasmic and Nuclear - drug effects | Humans | Drug Therapy | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - physiology | Receptors, Steroid - drug effects | Nuclear Proteins - chemistry | Drug Discovery | Receptors, Thyroid Hormone - physiology | Receptors, Estrogen - drug effects | Animals | Receptors, Steroid - physiology | Receptors, Cytoplasmic and Nuclear - chemistry | Lipid Metabolism - drug effects | Nuclear Proteins - physiology | Lipid Metabolism - physiology | Receptors, Estrogen - physiology | Drugs | Hormone receptors | Drug delivery systems | Ligand binding (Biochemistry) | Research | Vehicles
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Loading RightsInternational Urology and Nephrology, ISSN 0301-1623, 3/2016, Volume 48, Issue 3, pp. 325 - 333
To compare the expression levels of androgen receptor (AR), oestrogen receptor α (ERα)and oestrogen receptor β (ERβ) in human prostate with various degrees of...
Nephrology | Medicine & Public Health | Prostate volume | Androgen receptor | Benign prostatic hyperplasia | Urology | Oestrogen receptor | LOCALIZATION | TESTOSTERONE | MODULATORS | HYPERPLASIA | CANCER | BENIGN | URINARY-TRACT SYMPTOMS | MEN | UROLOGY & NEPHROLOGY | EXPRESSION | HORMONES | Immunohistochemistry | Receptors, Estrogen - blood | Prospective Studies | Follow-Up Studies | Humans | Middle Aged | Organ Size | Immunoblotting | Male | Biomarkers - blood | Disease Progression | Prostatic Hyperplasia - diagnosis | Prostate - diagnostic imaging | Prostatic Hyperplasia - blood | Aged | Receptors, Androgen - blood | Testosterone | Glycoproteins | Pituitary hormones | Comparative analysis | Estrogen | Luteinizing hormone
Nephrology | Medicine & Public Health | Prostate volume | Androgen receptor | Benign prostatic hyperplasia | Urology | Oestrogen receptor | LOCALIZATION | TESTOSTERONE | MODULATORS | HYPERPLASIA | CANCER | BENIGN | URINARY-TRACT SYMPTOMS | MEN | UROLOGY & NEPHROLOGY | EXPRESSION | HORMONES | Immunohistochemistry | Receptors, Estrogen - blood | Prospective Studies | Follow-Up Studies | Humans | Middle Aged | Organ Size | Immunoblotting | Male | Biomarkers - blood | Disease Progression | Prostatic Hyperplasia - diagnosis | Prostate - diagnostic imaging | Prostatic Hyperplasia - blood | Aged | Receptors, Androgen - blood | Testosterone | Glycoproteins | Pituitary hormones | Comparative analysis | Estrogen | Luteinizing hormone
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Loading RightsThe EMBO Journal, ISSN 0261-4189, 10/2000, Volume 19, Issue 20, pp. 5406 - 5417
Treatment of human prostate carcinoma‐derived LNCaP cells with androgen or oestradiol triggers simultaneous association of androgen receptor and oestradiol...
cross‐talk | androgen receptor | Src association | oestradiol receptor | Oestradiol receptor | Androgen receptor | Cross-talk | HUMAN ESTROGEN-RECEPTOR | LIGAND-INDEPENDENT ACTIVATION | ACTIVATED PROTEIN-KINASE | cross-talk | TYROSINE KINASE | MAP KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HORMONE RECEPTORS | CELL BIOLOGY | IN-VITRO | MCF-7 CELLS | NONGENOMIC ACTIONS | ESTRADIOL-RECEPTOR | Humans | Receptors, Androgen - metabolism | Estrogen Antagonists - pharmacology | Male | Recombinant Fusion Proteins - metabolism | src Homology Domains - drug effects | Transfection | Protein Binding - drug effects | src-Family Kinases - metabolism | Steroids - pharmacology | Tumor Cells, Cultured | Metribolone - pharmacology | Estradiol - pharmacology | Receptors, Estradiol - metabolism | Prostatic Neoplasms - pathology | Androgen Antagonists - pharmacology | Cell Division - drug effects | Macromolecular Substances | Animals | MAP Kinase Signaling System - drug effects | Receptors, Androgen - genetics | Androgens - pharmacology | Receptors, Estradiol - genetics | Models, Biological | Recombinant Fusion Proteins - genetics | src-Family Kinases - genetics | COS Cells
cross‐talk | androgen receptor | Src association | oestradiol receptor | Oestradiol receptor | Androgen receptor | Cross-talk | HUMAN ESTROGEN-RECEPTOR | LIGAND-INDEPENDENT ACTIVATION | ACTIVATED PROTEIN-KINASE | cross-talk | TYROSINE KINASE | MAP KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | HORMONE RECEPTORS | CELL BIOLOGY | IN-VITRO | MCF-7 CELLS | NONGENOMIC ACTIONS | ESTRADIOL-RECEPTOR | Humans | Receptors, Androgen - metabolism | Estrogen Antagonists - pharmacology | Male | Recombinant Fusion Proteins - metabolism | src Homology Domains - drug effects | Transfection | Protein Binding - drug effects | src-Family Kinases - metabolism | Steroids - pharmacology | Tumor Cells, Cultured | Metribolone - pharmacology | Estradiol - pharmacology | Receptors, Estradiol - metabolism | Prostatic Neoplasms - pathology | Androgen Antagonists - pharmacology | Cell Division - drug effects | Macromolecular Substances | Animals | MAP Kinase Signaling System - drug effects | Receptors, Androgen - genetics | Androgens - pharmacology | Receptors, Estradiol - genetics | Models, Biological | Recombinant Fusion Proteins - genetics | src-Family Kinases - genetics | COS Cells
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Loading RightsNature Reviews Molecular Cell Biology, ISSN 1471-0072, 11/2016, Volume 17, Issue 12, pp. 783 - 797
Steroid hormone receptors mediate numerous crucial biological processes and are classically thought to function as transcriptional regulators in the nucleus....
BREAST-CANCER CELLS | GROWTH-FACTOR RECEPTOR | ANDROGEN RECEPTOR | MEMBRANE ESTROGEN-RECEPTORS | ACTIVATED PROTEIN-KINASE | GLUCOCORTICOID-RECEPTOR | XENOPUS-LAEVIS OOCYTES | HORMONE RECEPTOR | PROGESTERONE-RECEPTOR | PLASMA-MEMBRANE | CELL BIOLOGY | Animals | Signal Transduction | Receptors, Steroid - physiology | Humans | Gene Expression Regulation | Receptors, Cytoplasmic and Nuclear - physiology | Cell Nucleus | Protein Transport | Cell receptors | Cellular signal transduction | Genetic aspects | Disease susceptibility | Properties | Health aspects
BREAST-CANCER CELLS | GROWTH-FACTOR RECEPTOR | ANDROGEN RECEPTOR | MEMBRANE ESTROGEN-RECEPTORS | ACTIVATED PROTEIN-KINASE | GLUCOCORTICOID-RECEPTOR | XENOPUS-LAEVIS OOCYTES | HORMONE RECEPTOR | PROGESTERONE-RECEPTOR | PLASMA-MEMBRANE | CELL BIOLOGY | Animals | Signal Transduction | Receptors, Steroid - physiology | Humans | Gene Expression Regulation | Receptors, Cytoplasmic and Nuclear - physiology | Cell Nucleus | Protein Transport | Cell receptors | Cellular signal transduction | Genetic aspects | Disease susceptibility | Properties | Health aspects
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Loading RightsCancer, ISSN 0008-543X, 01/2012, Volume 118, Issue 1, pp. 157 - 163
BACKGROUND: The current study was performed to investigate the potential impact of tumor cell expression of estrogen receptor-a (ER-a), progesterone receptor...
radiotherapy | estrogen receptor‐α | androgen receptor | progesterone receptor | non‐small cell lung cancer | non-small cell lung cancer | estrogen receptor-α | SURVIVAL | ONCOLOGY | OXYGENATION | PO HISTOGRAPHY | HEMOGLOBIN LEVELS | estrogen receptor-a | ADVANCED HEAD | RADIATION-THERAPY | Carcinoma, Non-Small-Cell Lung - radiotherapy | Prognosis | Humans | Middle Aged | Carcinoma, Non-Small-Cell Lung - metabolism | Male | Carcinoma, Non-Small-Cell Lung - mortality | Receptors, Androgen - biosynthesis | Female | Aged | Estrogen Receptor alpha - biosynthesis | Retrospective Studies | Biomarkers, Tumor - biosynthesis | Receptors, Progesterone - biosynthesis
radiotherapy | estrogen receptor‐α | androgen receptor | progesterone receptor | non‐small cell lung cancer | non-small cell lung cancer | estrogen receptor-α | SURVIVAL | ONCOLOGY | OXYGENATION | PO HISTOGRAPHY | HEMOGLOBIN LEVELS | estrogen receptor-a | ADVANCED HEAD | RADIATION-THERAPY | Carcinoma, Non-Small-Cell Lung - radiotherapy | Prognosis | Humans | Middle Aged | Carcinoma, Non-Small-Cell Lung - metabolism | Male | Carcinoma, Non-Small-Cell Lung - mortality | Receptors, Androgen - biosynthesis | Female | Aged | Estrogen Receptor alpha - biosynthesis | Retrospective Studies | Biomarkers, Tumor - biosynthesis | Receptors, Progesterone - biosynthesis
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Loading RightsToxicology and Applied Pharmacology, ISSN 0041-008X, 2006, Volume 216, Issue 1, pp. 44 - 54
Several pesticides and fungicides commonly used to control agricultural and indoor pests are highly suspected to display endocrine-disrupting effects in...
Vinclozolin | Mineralocorticoid receptor | Androgen receptor | Glucocorticoid receptor | Estrogen receptors | Progesterone receptor | Fungicide | Reporter cell lines | REPORTER CELL-LINES | glucocorticoid receptor | ANTIANDROGENIC ACTIVITY | MALE-RAT | FUNGICIDE VINCLOZOLIN | PROGESTERONE-RECEPTORS | mineralocorticoid receptor | GENE ASSAYS | vinclozolin | estrogen receptors | progesterone receptor | ENVIRONMENTAL ESTROGENS | IN-VITRO | ENDOCRINE DISRUPTORS | androgen receptor | SEXUAL-DIFFERENTIATION | PHARMACOLOGY & PHARMACY | TOXICOLOGY | reporter cell lines | fungicide | Receptors, Mineralocorticoid - genetics | Androgen Receptor Antagonists | Receptors, Glucocorticoid - antagonists & inhibitors | Luciferases - metabolism | Receptors, Steroid - metabolism | Gene Expression - drug effects | Receptors, Mineralocorticoid - metabolism | Humans | Receptors, Androgen - metabolism | Anilides - chemistry | Tritium | Receptors, Glucocorticoid - metabolism | Receptors, Progesterone - genetics | Receptors, Steroid - antagonists & inhibitors | Receptors, Progesterone - antagonists & inhibitors | Luciferases - genetics | Receptors, Progesterone - metabolism | Dose-Response Relationship, Drug | Dexamethasone - pharmacology | Progesterone Congeners - pharmacology | Metribolone - pharmacology | Mineralocorticoid Receptor Antagonists | Estradiol - pharmacology | Carbamates - pharmacology | Cell Survival - drug effects | Oxazoles - metabolism | Androgen Antagonists - pharmacology | Anilides - metabolism | Aldosterone - pharmacology | Testosterone Congeners - pharmacology | Binding, Competitive - drug effects | Receptors, Androgen - genetics | Receptors, Steroid - genetics | Receptors, Glucocorticoid - genetics | Cell Line, Tumor | Promegestone - pharmacology | Oxazoles - pharmacology | Corticosteroids | Progesterone | Fungicides | Plant metabolites | Estrogen | ANIMALS | ESTROGENS | METABOLITES | FUNGICIDES | GLUCOCORTICOIDS | PROGESTERONE | ERBIUM SULFIDES | 60 APPLIED LIFE SCIENCES | LUCIFERASE | RISK ASSESSMENT | LIGANDS | ANDROGENS | RECEPTORS
Vinclozolin | Mineralocorticoid receptor | Androgen receptor | Glucocorticoid receptor | Estrogen receptors | Progesterone receptor | Fungicide | Reporter cell lines | REPORTER CELL-LINES | glucocorticoid receptor | ANTIANDROGENIC ACTIVITY | MALE-RAT | FUNGICIDE VINCLOZOLIN | PROGESTERONE-RECEPTORS | mineralocorticoid receptor | GENE ASSAYS | vinclozolin | estrogen receptors | progesterone receptor | ENVIRONMENTAL ESTROGENS | IN-VITRO | ENDOCRINE DISRUPTORS | androgen receptor | SEXUAL-DIFFERENTIATION | PHARMACOLOGY & PHARMACY | TOXICOLOGY | reporter cell lines | fungicide | Receptors, Mineralocorticoid - genetics | Androgen Receptor Antagonists | Receptors, Glucocorticoid - antagonists & inhibitors | Luciferases - metabolism | Receptors, Steroid - metabolism | Gene Expression - drug effects | Receptors, Mineralocorticoid - metabolism | Humans | Receptors, Androgen - metabolism | Anilides - chemistry | Tritium | Receptors, Glucocorticoid - metabolism | Receptors, Progesterone - genetics | Receptors, Steroid - antagonists & inhibitors | Receptors, Progesterone - antagonists & inhibitors | Luciferases - genetics | Receptors, Progesterone - metabolism | Dose-Response Relationship, Drug | Dexamethasone - pharmacology | Progesterone Congeners - pharmacology | Metribolone - pharmacology | Mineralocorticoid Receptor Antagonists | Estradiol - pharmacology | Carbamates - pharmacology | Cell Survival - drug effects | Oxazoles - metabolism | Androgen Antagonists - pharmacology | Anilides - metabolism | Aldosterone - pharmacology | Testosterone Congeners - pharmacology | Binding, Competitive - drug effects | Receptors, Androgen - genetics | Receptors, Steroid - genetics | Receptors, Glucocorticoid - genetics | Cell Line, Tumor | Promegestone - pharmacology | Oxazoles - pharmacology | Corticosteroids | Progesterone | Fungicides | Plant metabolites | Estrogen | ANIMALS | ESTROGENS | METABOLITES | FUNGICIDES | GLUCOCORTICOIDS | PROGESTERONE | ERBIUM SULFIDES | 60 APPLIED LIFE SCIENCES | LUCIFERASE | RISK ASSESSMENT | LIGANDS | ANDROGENS | RECEPTORS
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Loading RightsAMERICAN JOURNAL OF CHINESE MEDICINE, ISSN 0192-415X, 2017, Volume 45, Issue 6, pp. 1147 - 1156
The first record of ginseng use dates back over two millennia, and ginseng is now popular in more than 35 countries. Ginsenosides are the pharmacological...
3T3-L1 ADIPOCYTES | PANAX-GINSENG | Estrogen Receptor | ACTIVATES ESTROGEN-RECEPTOR | Glucocorticoid Receptor | Nuclear Hormone Receptors | Review | Panax ginseng | HUMAN ENDOTHELIAL-CELLS | Androgen Receptor | MEDICINE, GENERAL & INTERNAL | IN-VITRO | GLUCOCORTICOID-RECEPTOR | RED GINSENG | SIGNALING PATHWAY | INTEGRATIVE & COMPLEMENTARY MEDICINE | ANDROGEN RECEPTORS | PPAR-GAMMA | Peroxisome Proliferator-Activated Receptor | Peroxisome Proliferator-Activated Receptors - physiology | Obesity - drug therapy | Plant Extracts - pharmacology | Cardiovascular Diseases - drug therapy | Humans | Male | Ginsenosides - pharmacology | Ginsenosides - therapeutic use | MAP Kinase Signaling System | Receptors, Androgen - drug effects | Female | Phytotherapy | Receptors, Glucocorticoid - drug effects | Receptors, Androgen - physiology | Receptors, Cytoplasmic and Nuclear - drug effects | Nervous System Diseases - drug therapy | Plant Extracts - isolation & purification | Receptors, Cytoplasmic and Nuclear - physiology | Neoplasms - drug therapy | Receptors, Estrogen - drug effects | Animals | Panax - chemistry | Peroxisome Proliferator-Activated Receptors - drug effects | Ginsenosides - isolation & purification | Plant Extracts - therapeutic use | Receptors, Glucocorticoid - physiology | Receptors, Estrogen - physiology | Hormone receptors | Nervous system diseases | Care and treatment | Research
3T3-L1 ADIPOCYTES | PANAX-GINSENG | Estrogen Receptor | ACTIVATES ESTROGEN-RECEPTOR | Glucocorticoid Receptor | Nuclear Hormone Receptors | Review | Panax ginseng | HUMAN ENDOTHELIAL-CELLS | Androgen Receptor | MEDICINE, GENERAL & INTERNAL | IN-VITRO | GLUCOCORTICOID-RECEPTOR | RED GINSENG | SIGNALING PATHWAY | INTEGRATIVE & COMPLEMENTARY MEDICINE | ANDROGEN RECEPTORS | PPAR-GAMMA | Peroxisome Proliferator-Activated Receptor | Peroxisome Proliferator-Activated Receptors - physiology | Obesity - drug therapy | Plant Extracts - pharmacology | Cardiovascular Diseases - drug therapy | Humans | Male | Ginsenosides - pharmacology | Ginsenosides - therapeutic use | MAP Kinase Signaling System | Receptors, Androgen - drug effects | Female | Phytotherapy | Receptors, Glucocorticoid - drug effects | Receptors, Androgen - physiology | Receptors, Cytoplasmic and Nuclear - drug effects | Nervous System Diseases - drug therapy | Plant Extracts - isolation & purification | Receptors, Cytoplasmic and Nuclear - physiology | Neoplasms - drug therapy | Receptors, Estrogen - drug effects | Animals | Panax - chemistry | Peroxisome Proliferator-Activated Receptors - drug effects | Ginsenosides - isolation & purification | Plant Extracts - therapeutic use | Receptors, Glucocorticoid - physiology | Receptors, Estrogen - physiology | Hormone receptors | Nervous system diseases | Care and treatment | Research
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