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PLoS Genetics, ISSN 1553-7390, 01/2014, Volume 10, Issue 1, p. e1004102
Journal Article
Nature, ISSN 0028-0836, 02/2011, Volume 470, Issue 7333, pp. 214 - 220
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2015, Volume 290, Issue 25, pp. 15799 - 15811
Background: MicroRNA-21 (miR-21) is an oncomiR in human hepatocellular carcinoma and is highly expressed in liver, but its regulation is uncharacterized.... 
METHYL-BETA-CYCLODEXTRIN | MIGRATION | gene transcription | VARIANT ER-ALPHA-36 | microRNA biogenesis | microRNA (miRNA) | estrogen receptor | hormones | BIOCHEMISTRY & MOLECULAR BIOLOGY | GPR30 | PROLIFERATION | G-protein-coupled receptor (GPCR) | BREAST-CANCER CELLS | EPIDERMAL-GROWTH-FACTOR | PROSTATE-CANCER | GENE-EXPRESSION | UP-REGULATION | Transcription, Genetic - drug effects | Receptors, Estrogen - metabolism | Adjuvants, Immunologic - pharmacology | Receptors, G-Protein-Coupled - metabolism | Transcription Factor AP-1 - genetics | Humans | Receptors, Androgen - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | Transcription Factor AP-1 - metabolism | MAP Kinase Signaling System - genetics | Carcinoma, Hepatocellular - genetics | Mitogen-Activated Protein Kinase 1 - genetics | src-Family Kinases - metabolism | Estrogen Receptor alpha - metabolism | Female | Liver Neoplasms - pathology | Gene Expression Regulation, Neoplastic - drug effects | Gene Expression Regulation, Neoplastic - genetics | Receptors, Estrogen - genetics | Liver Neoplasms - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Response Elements | Dehydroepiandrosterone - pharmacology | Proto-Oncogene Proteins c-jun - genetics | Proto-Oncogene Proteins c-fos - metabolism | MicroRNAs - biosynthesis | Hep G2 Cells | Phosphatidylinositol 3-Kinases - genetics | Estrogen Receptor alpha - genetics | MAP Kinase Signaling System - drug effects | RNA, Neoplasm - biosynthesis | Receptors, Androgen - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Proto-Oncogene Proteins c-jun - metabolism | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | RNA, Neoplasm - genetics | Proto-Oncogene Proteins c-fos - genetics | MicroRNAs - genetics | Receptors, G-Protein-Coupled - genetics | Transcription, Genetic - genetics | src-Family Kinases - genetics | Carcinoma, Hepatocellular - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus | Signal Transduction
Journal Article
Journal Article
Nature Genetics, ISSN 1061-4036, 05/2018, Volume 50, Issue 5, pp. 645 - 651
Journal Article
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 09/2015, Volume 33, Issue 25, pp. 2789 - 2796
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 02/2014, Volume 53, Issue 2, pp. 85 - 97
As a link between inflammation and cancer has been reported in many studies, we established an in vitro model of prostatic inflammation to investigate the loss... 
androgen | loss of p53 | inflammation | DNA damage | ROS | prostate | NFkB | Loss of p53 | Androgen | Inflammation | Prostate | SURVIVAL | OXIDATIVE STRESS | CANCER CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUTATHIONE-PEROXIDASE | CARCINOGENESIS | ONCOLOGY | NKX3.1 | CARCINOMAS | LINK | HOMEOBOX GENE | EXPRESSION | Proto-Oncogene Proteins c-mdm2 - genetics | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Antioxidants - metabolism | Homeodomain Proteins - metabolism | Humans | Transcriptional Activation - drug effects | Tumor Necrosis Factor-alpha - genetics | Apoptosis - genetics | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | Prostatitis - metabolism | Inflammation - metabolism | Matrix Metalloproteinase 9 - metabolism | Phosphorylation - genetics | Acetylcysteine - analogs & derivatives | Interleukin-1beta - metabolism | Matrix Metalloproteinase 9 - genetics | U937 Cells | Prostate-Specific Antigen - metabolism | Phosphorylation - drug effects | Interleukin-6 - metabolism | Prostate-Specific Antigen - genetics | Glutathione Peroxidase - metabolism | Lysine - analogs & derivatives | Interleukin-6 - genetics | Down-Regulation - genetics | Glutathione Peroxidase - genetics | Prostatitis - drug therapy | Androgens - genetics | Macrophages - metabolism | Receptors, Androgen - genetics | Signal Transduction - drug effects | Cell Line, Tumor | Androgens - metabolism | Tumor Necrosis Factor-alpha - metabolism | Prostatic Neoplasms - metabolism | Receptors, Androgen - metabolism | I-kappa B Proteins - metabolism | I-kappa B Proteins - genetics | Prostatitis - pathology | Tumor Suppressor Protein p53 - genetics | Cell Differentiation - genetics | Prostatic Neoplasms - genetics | Inflammation Mediators - metabolism | Proto-Oncogene Proteins c-mdm2 - metabolism | Prostatic Neoplasms - pathology | Kallikreins - genetics | Tumor Suppressor Protein p53 - metabolism | Antioxidants - pharmacology | Transcription Factors - genetics | Down-Regulation - drug effects | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Up-Regulation - drug effects | Lysine - pharmacology | NF-kappa B - genetics | Cell Differentiation - drug effects | Acetylcysteine - pharmacology | Inflammation - genetics | Macrophages - drug effects | Kallikreins - metabolism | Ubiquitin | Analysis | Kallikrein | Hormones, Sex | Tumor proteins | Mitogens | Androgens | Cytokines | Molecular biology | Gene expression | Prostate cancer | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2013, Volume 8, Issue 5, p. e63925
Breast Cancer (BCa) genome-wide association studies revealed allelic frequency differences between cases and controls at index single nucleotide polymorphisms... 
COMMON VARIANTS | DIFFERENTIAL EXPRESSION | PROTEIN | DATABASE | MULTIDISCIPLINARY SCIENCES | ANDROGEN-RECEPTOR | GENE-EXPRESSION | SUSCEPTIBILITY LOCUS | REGULATORY ELEMENTS | DATA SETS | GENOME-WIDE ASSOCIATION | Endonucleases - genetics | Gene Frequency - genetics | Genetic Predisposition to Disease - genetics | Humans | Risk Factors | Cells, Cultured | Linkage Disequilibrium - genetics | Exons - genetics | Risk | Transcription Factors - genetics | Breast Neoplasms - genetics | Epigenesis, Genetic - genetics | Transcription Initiation Site | Alleles | Polymorphism, Single Nucleotide - genetics | Female | Response Elements - genetics | Genome-Wide Association Study - methods | Chromatin - genetics | Quantitative Trait Loci - genetics | Chromatin | Oncology, Experimental | Genes | Genomics | Breast cancer | Genomes | Genetic transcription | Research | DNA binding proteins | Risk factors | Quantitative genetics | Genetic aspects | Single nucleotide polymorphisms | Health aspects | Cancer | Transcription factors | Disease | Exons | Linkage disequilibrium | Single-nucleotide polymorphism | Annotations | Genetics | Deoxyribonucleic acid--DNA | Sequences | Health risks | Preventive medicine | Gene expression | Disease control | Loci | Quantitative trait loci | Disease prevention | Studies | Enhancers | Breast | Mutation | Health risk assessment | Prostate | Deoxyribonucleic acid | DNA
Journal Article