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Nature, ISSN 0028-0836, 07/2015, Volume 523, Issue 7560, pp. 347 - 351
Prostate cancer resistance to castration occurs because tumours acquire the metabolic capability of converting precursor steroids to 5... 
ANDROGEN RECEPTOR | 3-BETA-HYDROXYSTEROID DEHYDROGENASE | CYP17A1 INHIBITION | MECHANISM | TESTOSTERONE | RATIONALE | MULTIDISCIPLINARY SCIENCES | INCREASED SURVIVAL | CHEMOTHERAPY | EXPOSURE | ENZALUTAMIDE | Chromatin - metabolism | Prostatic Neoplasms - metabolism | Steroid 17-alpha-Hydroxylase - antagonists & inhibitors | Androgen Receptor Antagonists - therapeutic use | Humans | Receptors, Androgen - metabolism | Gene Expression Regulation, Neoplastic | Androgens - biosynthesis | Male | Androstenes - pharmacology | Androgen Receptor Antagonists - pharmacology | 3-Hydroxysteroid Dehydrogenases - antagonists & inhibitors | Biotransformation | Cell Division | 3-Oxo-5-alpha-Steroid 4-Dehydrogenase - metabolism | 3-Hydroxysteroid Dehydrogenases - metabolism | Prostatic Neoplasms - drug therapy | Dihydrotestosterone - metabolism | Phenylthiohydantoin - pharmacology | Prostatic Neoplasms - pathology | Androstenes - therapeutic use | 5-alpha Reductase Inhibitors - therapeutic use | Prostatic Neoplasms, Castration-Resistant - drug therapy | Phenylthiohydantoin - analogs & derivatives | Androgen Receptor Antagonists - metabolism | Xenograft Model Antitumor Assays | Androstenes - chemistry | Animals | Biosynthetic Pathways - drug effects | Survival Analysis | 5-alpha Reductase Inhibitors - pharmacology | Androstenes - metabolism | Prostatic Neoplasms - enzymology | Mice | Steroid 17-alpha-Hydroxylase - metabolism | 5-alpha Reductase Inhibitors - metabolism | Androgens - metabolism | Enzymes | Testosterone | Androgens | Metabolites | Ligands | Gene expression | Prostate cancer | Tumors
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Nature, ISSN 0028-0836, 05/2016, Volume 533, Issue 7604, pp. 547 - 551
Abiraterone blocks androgen synthesis and prolongs survival in patients with castration-resistant prostate cancer, which is otherwise driven by intratumoral... 
ACTIVATION | CYP17A1 INHIBITION | RAT | MULTIDISCIPLINARY SCIENCES | ANDROGEN-RECEPTOR | BIOLOGY | MEN | RESISTANCE | STEROID 5-ALPHA-REDUCTASE | DIHYDROTESTOSTERONE | DEHYDROGENASE | Physiological aspects | Prostate cancer | Care and treatment | Enzymes | Androgens | Metabolites | Oxidation | Metabolism | Gene expression | Steroids
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Clinical Cancer Research, ISSN 1078-0432, 05/2015, Volume 21, Issue 10, pp. 2315 - 2324
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FASEB Journal, ISSN 0892-6638, 2014, Volume 28, Issue 8, pp. 3745 - 3757
The mineralocorticoid receptor (MR) controls adipocyte function, but its role in the conversion of white adipose tissue (WAT) into thermogenic fat has not been... 
UCP1 | Obesity | Aldosterone | Metabolic syndrome | Uncoupling protein 1 | Adipogenesis | aldosterone | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | ADULT HUMANS | ORGAN | CELL BIOLOGY | metabolic syndrome | uncoupling protein 1 | GLUCOSE-HOMEOSTASIS | adipogenesis | IN-VIVO | BIOLOGY | GENE-EXPRESSION | DIFFERENTIATION | obesity | Inguinal Canal | Obesity - drug therapy | Adipogenesis - drug effects | Diet, High-Fat - adverse effects | Ion Channels - genetics | Adipocytes - cytology | Androstenes - pharmacology | Gene Expression Profiling | Mitochondrial Proteins - genetics | Adipocytes - drug effects | Autophagy - drug effects | Adipogenesis - physiology | Receptors, Mineralocorticoid - physiology | Receptors, Mineralocorticoid - drug effects | Glucose Intolerance - prevention & control | Spironolactone - pharmacology | Body Composition - drug effects | Glucose Intolerance - etiology | Cell Transdifferentiation - drug effects | Mineralocorticoid Receptor Antagonists - pharmacology | Macrolides - pharmacology | Female | Weight Gain - drug effects | Intra-Abdominal Fat - drug effects | Spironolactone - therapeutic use | Ion Channels - biosynthesis | Mice, Inbred C57BL | Cells, Cultured | Androstenes - therapeutic use | Aldosterone - pharmacology | Obesity - physiopathology | Mitochondrial Proteins - biosynthesis | Mineralocorticoid Receptor Antagonists - therapeutic use | Up-Regulation - drug effects | Animals | Obesity - prevention & control | Adipose Tissue, Brown - metabolism | Mice | Uncoupling Protein 1 | Adipose Tissue, White - drug effects
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