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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2016, Volume 36, Issue 8, pp. 1587 - 1597
OBJECTIVE—Abdominal aortic aneurysm (AAA) is a vascular disorder characterized by chronic inflammation of the aortic wall. Low concentrations of vitamin D3 are... 
endothelial cells inflammation | chemokines | calcitriol | abdominal aortic aneurysm | dilatation | OXIDATIVE STRESS | D DEFICIENCY | 1-ALPHA,25-DIHYDROXYVITAMIN D-3 | MATRIX METALLOPROTEINASES | REGULATORY T-CELLS | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | 1,25-DIHYDROXYVITAMIN D-3 | HEMATOLOGY | NF-KAPPA-B | ENDOTHELIAL GROWTH-FACTOR | Aortic Aneurysm, Abdominal - chemically induced | Aortic Aneurysm, Abdominal - prevention & control | Retinoid X Receptor alpha - metabolism | Apolipoproteins E - deficiency | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Male | Vascular Endothelial Growth Factor A - metabolism | Receptors, Calcitriol - genetics | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | Matrix Metalloproteinase 9 - metabolism | Transfection | Chemotaxis, Leukocyte - drug effects | RNA Interference | Aneurysm, Dissecting - chemically induced | Aorta, Abdominal - pathology | Angiotensin II | Aneurysm, Dissecting - prevention & control | Aorta, Abdominal - metabolism | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Genetic Predisposition to Disease | Human Umbilical Vein Endothelial Cells - drug effects | Aneurysm, Dissecting - metabolism | Macrophages - pathology | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Cells, Cultured | Aneurysm, Dissecting - pathology | Receptors, Calcitriol - metabolism | Mice, Knockout | Macrophages - metabolism | Phenotype | Animals | Signal Transduction - drug effects | Receptors, Calcitriol - agonists | Apolipoproteins E - genetics | Calcitriol - pharmacology | Retinoid X Receptor alpha - genetics | Ligands | Macrophages - drug effects | Chemokines - metabolism | Mitogen-Activated Protein Kinases - metabolism
Journal Article
Circulation Research, ISSN 0009-7330, 04/2015, Volume 116, Issue 8, pp. 1448 - 1461
Thoracic aortic diseases that involve progressive enlargement, acute dissection, or rupture are influenced by the hemodynamic loads and mechanical properties... 
Elastic fibers | Actomyosin | Marfan syndrome | Focal adhesion | focal adhesion | LIGHT-CHAIN KINASE | ASCENDING AORTA | DEVELOPING MOUSE AORTA | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOTENSIN-II | GROWTH-FACTOR-BETA | actomyosin | MARFAN-SYNDROME | ARTERIAL-WALL | elastic fibers | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | HEMATOLOGY | EHLERS-DANLOS-SYNDROME | Aneurysm, Dissecting - genetics | Aortic Aneurysm, Thoracic - genetics | Humans | Stress, Mechanical | Aortic Rupture - metabolism | Aneurysm, Dissecting - physiopathology | Aortic Aneurysm, Thoracic - pathology | Aorta, Thoracic - pathology | Extracellular Matrix Proteins - metabolism | Aortic Rupture - physiopathology | Genetic Predisposition to Disease | Aneurysm, Dissecting - metabolism | Extracellular Matrix Proteins - genetics | Aorta, Thoracic - metabolism | Aortic Rupture - pathology | Aneurysm, Dissecting - pathology | Aneurysm, Dissecting - therapy | Aorta, Thoracic - physiopathology | Aortic Aneurysm, Thoracic - physiopathology | Disease Progression | Mechanotransduction, Cellular | Biomechanical Phenomena | Phenotype | Animals | Aortic Aneurysm, Thoracic - metabolism | Aortic Aneurysm, Thoracic - therapy | Aortic Rupture - genetics | Aortic Rupture - therapy | Hemodynamics | familial thoracic aortic aneurysms and dissections | mechanosensing
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2017, Volume 37, Issue 1, pp. 26 - 34
The importance of maintaining contractile function in aortic smooth muscle cells (SMCs) is evident by the fact that heterozygous mutations in the major... 
mutation | thoracic aorta | aortic aneurysm | aortic dissection | vascular smooth muscle cells | Marfan syndrome | LIGHT-CHAIN KINASE | ACTIN POLYMERIZATION | DOMAIN-STRUCTURE | TGFBR2 MUTATIONS | MYOSIN HEAVY-CHAIN | BETA RECEPTOR | MARFAN-SYNDROME | MOUSE MODEL | DISEASE | PERIPHERAL VASCULAR DISEASE | PATENT DUCTUS-ARTERIOSUS | HEMATOLOGY | Myosin-Light-Chain Kinase - genetics | Aneurysm, Dissecting - genetics | Aortic Aneurysm, Thoracic - genetics | Genetic Testing | Muscle, Smooth, Vascular - metabolism | Pulsatile Flow | Humans | Actins - metabolism | Myocytes, Smooth Muscle - pathology | Myosin Heavy Chains - genetics | Cyclic GMP-Dependent Protein Kinase Type I - genetics | Muscle, Smooth, Vascular - physiopathology | Myosin Heavy Chains - metabolism | Actins - genetics | Cyclic GMP-Dependent Protein Kinase Type I - metabolism | Aneurysm, Dissecting - physiopathology | Aortic Aneurysm, Thoracic - pathology | Dilatation, Pathologic | Myocytes, Smooth Muscle - metabolism | Calcium-Binding Proteins - metabolism | Vasoconstriction - genetics | Elastin - metabolism | Aneurysm, Dissecting - metabolism | Myosin-Light-Chain Kinase - metabolism | Heredity | Aneurysm, Dissecting - pathology | Genetic Markers | Aortic Aneurysm, Thoracic - physiopathology | Mechanotransduction, Cellular | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Aortic Aneurysm, Thoracic - metabolism | Mutation | Calcium-Binding Proteins - genetics | elastin-contractile unit | Contractile Function | Vascular Biology | Aneurysm | Genetics | vascular smooth muscle cell | Aortic Dissection | thoracic
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2018, Volume 38, Issue 8, pp. 1796 - 1805
Journal Article
Nature Genetics, ISSN 1061-4036, 07/2012, Volume 44, Issue 8, pp. 916 - 921
Journal Article
Circulation, ISSN 0009-7322, 06/2014, Volume 129, Issue 25, pp. 2661 - 2672
Background-Increased production of reactive oxygen species (ROS) throughout the vascular wall is a feature of cardiovascular disease states, but therapeutic... 
Aorta | Reactive oxygen species | Angiotensin II | Dissection | NADPH oxidase | Cyclophilin A | CARDIAC & CARDIOVASCULAR SYSTEMS | VASCULAR OXIDATIVE STRESS | cyclophilin A | ATHEROSCLEROSIS | ANGIOTENSIN-II | dissection | SUPPLEMENTATION | angiotensin II | reactive oxygen species | PERIPHERAL VASCULAR DISEASE | aorta | HYPERTENSION | E-DEFICIENT MICE | ANEURYSM FORMATION | NOX1 | TRANSGENIC MICE | Aneurysm, Dissecting - epidemiology | Reactive Oxygen Species - metabolism | Aortic Aneurysm - metabolism | Membrane Glycoproteins - metabolism | Muscle, Smooth, Vascular - metabolism | NADPH Oxidases - metabolism | Disease Susceptibility - etiology | Disease Susceptibility - metabolism | Male | Cyclophilins - metabolism | Aortic Aneurysm - etiology | NADPH Oxidases - genetics | Angiotensin II - adverse effects | Aneurysm, Dissecting - etiology | Disease Models, Animal | Aneurysm, Dissecting - metabolism | Signal Transduction | Aortic Aneurysm - epidemiology | Mice, Inbred C57BL | Mice, Transgenic | Cyclophilins - genetics | NADPH Oxidase 2 | Membrane Glycoproteins - genetics | Animals | Disease Susceptibility - epidemiology | Endothelium, Vascular - metabolism | Mice | Matrix Metalloproteinases - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Dissecting aneurysm | Physiological aspects | Disease susceptibility | Research | Endothelium
Journal Article
Circulation Research, ISSN 0009-7330, 02/2015, Volume 116, Issue 4, pp. 612 - 623
RATIONALE:In-hospital outcomes are generally acceptable in patients with type B dissection; however, some patients present with undesirable complications, such... 
Aortic dissection | Inflammation | Angiotensin II | Chemokines | Neutrophils | Interleukin-6 | CHEMOKINE RECEPTOR CXCR2 | ASCENDING AORTA | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTECTION | PORCINE CORONARY-ARTERIES | ANGIOTENSIN-II | INTERNATIONAL REGISTRY | chemokines | interleukin-6 | inflammation | angiotensin II | ANEURYSMS | HISTOLOGIC-CHANGES | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | aortic dissection | neutrophils | Chemotaxis, Leukocyte | Humans | Middle Aged | Male | Adventitia - metabolism | Time Factors | Aortic Rupture - diagnostic imaging | Chemokine CXCL1 - metabolism | Dilatation, Pathologic | Interleukin-6 - metabolism | Neutrophils - metabolism | Granulocyte Colony-Stimulating Factor - metabolism | Neutrophil Infiltration - drug effects | Disease Models, Animal | Aminopropionitrile - analogs & derivatives | Acute Disease | Interleukin-6 - genetics | Signal Transduction | Antibodies, Monoclonal - pharmacology | Neutrophils - drug effects | Aorta, Thoracic - metabolism | Mice, Knockout | Chemokine CXCL12 - metabolism | Aneurysm, Dissecting - diagnostic imaging | Aorta, Thoracic - diagnostic imaging | Aortic Aneurysm, Thoracic - diagnostic imaging | Neutrophil Activation - drug effects | Adventitia - diagnostic imaging | Aortic Aneurysm, Thoracic - drug therapy | Aortic Aneurysm, Thoracic - chemically induced | Aortic Rupture - metabolism | Receptors, Interleukin-8B - antagonists & inhibitors | Interleukin-6 - blood | Aneurysm, Dissecting - chemically induced | Aortic Rupture - prevention & control | Inflammation Mediators - metabolism | Female | Interleukin-8 - blood | Aneurysm, Dissecting - metabolism | Mice, Inbred C57BL | Aneurysm, Dissecting - drug therapy | Animals | Aortic Aneurysm, Thoracic - metabolism | Neutrophils - transplantation | Receptors, Interleukin-8B - metabolism | Aged | Aortic Rupture - chemically induced | Aortography
Journal Article
Journal of Vascular Surgery, ISSN 0741-5214, 12/2018, Volume 68, Issue 6, pp. 48S - 59S.e1
Osteoclastogenic activation of macrophages (OCG) occurs in human abdominal aortic aneurysms (AAAs) and in calcium chloride-induced degenerative AAAs in mice,... 
SYSTEM | SURGERY | PERIPHERAL VASCULAR DISEASE | ANEURYSM | INFLAMMATION | Aortic Aneurysm, Abdominal - chemically induced | Aortic Aneurysm, Abdominal - prevention & control | RANK Ligand - metabolism | Muscle, Smooth, Vascular - metabolism | Myocytes, Smooth Muscle - pathology | Male | Aorta, Abdominal - drug effects | Aortic Aneurysm, Abdominal - metabolism | RANK Ligand - immunology | STAT5 Transcription Factor - metabolism | Matrix Metalloproteinase 9 - metabolism | Aneurysm, Dissecting - chemically induced | Cell Transdifferentiation - drug effects | Janus Kinase 2 - metabolism | Aorta, Abdominal - pathology | Angiotensin II | Myocytes, Smooth Muscle - drug effects | Aneurysm, Dissecting - prevention & control | Aorta, Abdominal - metabolism | Myocytes, Smooth Muscle - metabolism | Aortic Aneurysm, Abdominal - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Osteoclasts - pathology | Aneurysm, Dissecting - metabolism | Macrophages - pathology | Signal Transduction | Osteogenesis - drug effects | Antibodies, Neutralizing - pharmacology | Aneurysm, Dissecting - pathology | Mice, Knockout, ApoE | Osteoclasts - metabolism | RANK Ligand - genetics | Macrophages - metabolism | Muscle, Smooth, Vascular - pathology | Animals | RANK Ligand - antagonists & inhibitors | Macrophages - drug effects | RAW 264.7 Cells | Mice | Osteoclasts - drug effects | Immunohistochemistry | Phosphatases | Analysis | Angiotensin | Aneurysms | Macrophages | Apolipoproteins | Calcium chloride
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 02/2017, Volume 23, Issue 2, pp. 200 - 212
Heritable thoracic aortic aneurysms and dissections (TAAD), including Marfan syndrome (MFS), currently lack a cure, and causative mutations have been... 
MEDICINE, RESEARCH & EXPERIMENTAL | INFLAMMATORY ANEURYSM | BIOCHEMISTRY & MOLECULAR BIOLOGY | THORACIC AORTA | ANGIOTENSIN-II | CELL BIOLOGY | SYNTHASE | DISINTEGRIN | IN-VIVO | DISSECTIONS | ANEURYSM FORMATION | EXPRESSION | PROGRESSION | Aneurysm, Dissecting - genetics | Aortic Aneurysm, Thoracic - genetics | Aortic Aneurysm - metabolism | ADAMTS1 Protein - genetics | Humans | Middle Aged | Immunoblotting | Male | Aorta - metabolism | Gene Knockdown Techniques | Nitric Oxide Synthase Type II - antagonists & inhibitors | Adult | Female | Fibrillin-1 - genetics | Real-Time Polymerase Chain Reaction | ADAMTS1 Protein - metabolism | Disease Models, Animal | NG-Nitroarginine Methyl Ester - pharmacology | Aneurysm, Dissecting - metabolism | Aorta - drug effects | Enzyme Inhibitors - pharmacology | Marfan Syndrome - genetics | Haploinsufficiency | Animals | Nitric Oxide Synthase Type II - genetics | Aortic Aneurysm, Thoracic - metabolism | Marfan Syndrome - metabolism | Aged | Mice | Aortic Aneurysm - genetics | Nitric Oxide - metabolism | Nitric Oxide Synthase Type II - metabolism | Blood circulation disorders | Molecular targeted therapy | Care and treatment | Proteases | Innovations | Development and progression | Genetic aspects | Oxidoreductases | Health aspects | Aneurysms | Cardiovascular disease | Genetic disorders | Rodents | Nitric oxide
Journal Article