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International journal of molecular sciences, ISSN 1422-0067, 2017, Volume 18, Issue 7, p. 1414
...) and histone acetyltransferases (HAT). HDAC inhibitors induce cancer cell cycle arrest, differentiation and cell death, reduce angiogenesis and modulate immune response... 
Histone deacetylase inhibitors | Anti-angiogenic effect | Cell cycle arrest | Drug combinations | Histone deacetylases | Autophagy | Apoptosis | Cancer | HDAC INHIBITORS | autophagy | SUBEROYLANILIDE HYDROXAMIC ACID | HUMAN LEUKEMIA-CELLS | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR-SUPPRESSOR GENES | apoptosis | VALPROIC ACID | cell cycle arrest | anti-angiogenic effect | CHEMISTRY, MULTIDISCIPLINARY | drug combinations | CELL LUNG-CANCER | EPITHELIAL-MESENCHYMAL TRANSITION | PHASE-II TRIAL | LONG NONCODING RNA | histone deacetylases | cancer | histone deacetylase inhibitors | Immunomodulation - drug effects | Apoptosis - drug effects | Humans | Angiogenesis Inhibitors - pharmacology | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Antineoplastic Agents - therapeutic use | Clinical Trials as Topic | Autophagy - drug effects | Acetylation - drug effects | Animals | Signal Transduction - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Cell Cycle Checkpoints - drug effects | Angiogenesis Inhibitors - therapeutic use | Histone Deacetylase Inhibitors - pharmacology | Antineoplastic Agents - pharmacology | Epigenesis, Genetic - drug effects | Histone Deacetylase Inhibitors - therapeutic use | Gene Expression Regulation, Neoplastic - drug effects | Drug Evaluation, Preclinical
Journal Article
Drug design, development and therapy, ISSN 1177-8881, 2015, Volume 9, pp. 4479 - 4499
.... There are several pharmacological approaches to block TGF-beta signaling, such as monoclonal antibodies, vaccines, antisense oligonucleotides, and small molecule inhibitors. Galunisertib... 
LY2157299 | Clinical trials | TGF-β | TGF-βRI kinase inhibitor | ALK5 | Galunisertib | Cancer | CHEMISTRY, MEDICINAL | RECEPTOR-TYPE-II | MESENCHYMAL TRANSITION | BREAST-CANCER | TGF-beta | HEPATOCELLULAR-CARCINOMA | TGF-beta RI kinase inhibitor | PLASMA-LEVELS | THERAPEUTIC TARGET | REGULATORY T-CELLS | PHARMACOLOGY & PHARMACY | cancer | clinical trials | TUMOR-GROWTH | galunisertib | I KINASE INHIBITOR | Phosphorylation | Pyrazoles - therapeutic use | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - administration & dosage | Protein Kinase Inhibitors - adverse effects | Molecular Targeted Therapy | Quinolines - administration & dosage | Quinolines - pharmacokinetics | Protein Kinase Inhibitors - chemistry | Pyrazoles - chemistry | Antineoplastic Agents - adverse effects | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Antineoplastic Agents - pharmacokinetics | Molecular Structure | Heart Diseases - chemically induced | Pyrazoles - pharmacokinetics | Protein-Serine-Threonine Kinases - metabolism | Pyrazoles - adverse effects | Protein Kinase Inhibitors - pharmacokinetics | Drug Administration Schedule | Administration, Oral | Quinolines - chemistry | Smad2 Protein - metabolism | Neoplasms - enzymology | Treatment Outcome | Antineoplastic Agents - chemistry | Drug Discovery | Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Protein Kinase Inhibitors - administration & dosage | Pyrazoles - administration & dosage | Receptors, Transforming Growth Factor beta - antagonists & inhibitors | Receptors, Transforming Growth Factor beta - metabolism | Signal Transduction - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Quinolines - therapeutic use | Neoplasms - pathology | Quinolines - adverse effects | Antimitotic agents | Cellular signal transduction | Transforming growth factors | Antineoplastic agents | Health aspects | Testing | Animal models | Transcription factors | Laboratories | Toxicity | Transforming growth factor-b | Oligonucleotides | Glioblastoma | Hepatocellular carcinoma | Metastasis | Vaccines | Kinases | Drug resistance | Dosage | Cancer therapies | Anticancer properties | Metastases | Proteins | Angiogenesis | Signal transduction | Pancreatic carcinoma | Smad2 protein | Fibroblasts | Physiology | Tumorigenesis | Colon | Growth factors | Cytokines | Melanoma | Antisense oligonucleotides | Immunosurveillance | Pharmacology | Breast cancer | Lung carcinoma | Patients | Biological activity | Signaling | Inhibitors | Pancreatic cancer | Monoclonal antibodies | Ligands | Antitumor activity | Prostate cancer | Tumors
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 05/2011, Volume 286, Issue 21, pp. 18633 - 18640
Journal Article
Cellular and molecular life sciences : CMLS, ISSN 1420-9071, 2008, Volume 66, Issue 7, pp. 1163 - 1177
Journal Article
Journal Article
CA: a cancer journal for clinicians, ISSN 0007-9235, 2010, Volume 60, Issue 4, pp. 222 - 243
... Because angiogenesis is a key process in tumor growth, and a limited process in healthy adults, developing angiogenesis inhibitors is a desirable anticancer target for which few... 
TYROSINE KINASE INHIBITORS | CELL LUNG-CANCER | PHASE-III TRIAL | FARNESYL-PROTEIN TRANSFERASE | FACTOR 1-ALPHA | ONCOLOGY | ENDOTHELIAL-GROWTH-FACTOR | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | FACTOR EXPRESSION | VASCULAR-PERMEABILITY FACTOR | TUMOR-GROWTH | Neoplasms - metabolism | Nucleic Acid Synthesis Inhibitors - pharmacology | Humans | Receptor Protein-Tyrosine Kinases - physiology | Antibodies, Monoclonal - therapeutic use | Receptors, Notch - antagonists & inhibitors | Protein Binding - drug effects | Angiogenesis Inhibitors - therapeutic use | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Neovascularization, Pathologic - prevention & control | Farnesyltranstransferase - antagonists & inhibitors | Thioredoxins - antagonists & inhibitors | Nucleic Acid Synthesis Inhibitors - therapeutic use | Basic Helix-Loop-Helix Transcription Factors - physiology | Antibodies, Monoclonal - pharmacology | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Angiogenesis Inhibitors - pharmacology | Receptors, Notch - physiology | Neoplasms - blood supply | Basic Helix-Loop-Helix Transcription Factors - antagonists & inhibitors | Neoplasms - drug therapy | Signal Transduction - drug effects | Cell Transformation, Neoplastic | HSP90 Heat-Shock Proteins - antagonists & inhibitors | Intracellular Signaling Peptides and Proteins - physiology | Complications and side effects | Care and treatment | Ligands (Biochemistry) | Physiological aspects | Development and progression | Dosage and administration | Angiogenesis inhibitors | Neovascularization | Identification and classification | Growth factors | Cancer | Angiogenesis | Pharmacology | FDA approval | Drug therapy | Kinases | Tumors | hypoxia inducible factor (HIF) | kinase inhibitors | growth factors | angiogenesis inhibitors | VEGF | anti-angiogenesis
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 2016, Volume 17, Issue 9, pp. 1534 - 1534
Journal Article
Journal Article
Journal of Medicinal Chemistry, ISSN 0022-2623, 07/2011, Volume 54, Issue 13, pp. 4559 - 4580
Sixty-one phenyl 4-(2-oxoimidazolidin-1-yl)benzenesulfonates (PIB-SOs) and 13 of their tetrahydro-2-oxopyrimidin-1(2H)-yl analogues (PPB-SOs) were prepared and... 
BETA-TUBULIN | COLCHICINE-BINDING SITE | POTENTIAL ANTINEOPLASTIC AGENTS | CHEMISTRY, MEDICINAL | PHASE-I TRIAL | ALKYLATING-AGENTS | COVALENT BINDING | ANTIMICROTUBULE AGENTS | CELL-CYCLE PROGRESSION | UREA DERIVATIVES | NUCLEAR TRANSLOCATION | Neovascularization, Physiologic - drug effects | Benzene Derivatives - chemical synthesis | Arylsulfonates - chemical synthesis | Tubulin Modulators - pharmacology | Humans | Imidazolidines - pharmacology | Drug Resistance, Neoplasm | Transplantation, Heterologous | Stilbenes - chemistry | Stilbenes - pharmacology | Tubulin Modulators - chemical synthesis | Molecular Mimicry | Chorioallantoic Membrane - drug effects | Drug Design | Imidazolidines - chemistry | Binding Sites | Angiogenesis Inhibitors - chemical synthesis | Benzene Derivatives - pharmacology | Quantitative Structure-Activity Relationship | Chorioallantoic Membrane - blood supply | Tubulin Modulators - chemistry | Imidazolidines - chemical synthesis | Angiogenesis Inhibitors - pharmacology | Colchicine - metabolism | Models, Molecular | Chick Embryo | Arylsulfonates - pharmacology | Animals | Arylsulfonates - chemistry | Cell Line, Tumor | Cell Proliferation - drug effects | Benzene Derivatives - chemistry | Cell Cycle - drug effects | Angiogenesis Inhibitors - chemistry | Drug Screening Assays, Antitumor | Index Medicus | Medicinal Chemistry | Organic chemistry | Chemical Sciences | Coordination chemistry | Radiochemistry
Journal Article