X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (3593) 3593
Book Review (775) 775
Publication (474) 474
Book Chapter (34) 34
Conference Proceeding (10) 10
Book / eBook (3) 3
Dissertation (3) 3
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (3404) 3404
humans (2679) 2679
animals (1908) 1908
female (1341) 1341
angiogenesis (1215) 1215
male (919) 919
mice (881) 881
endothelial growth-factor (772) 772
oncology (745) 745
expression (730) 730
vascular endothelial growth factor (670) 670
angiogenic proteins - metabolism (654) 654
angiogenic factors (630) 630
cancer (620) 620
adult (542) 542
cell biology (504) 504
vascular endothelial growth factor a - metabolism (500) 500
pregnancy (474) 474
cells, cultured (452) 452
middle aged (442) 442
biochemistry & molecular biology (422) 422
vegf (390) 390
analysis (372) 372
cell line, tumor (358) 358
aged (356) 356
angiogenic proteins - genetics (342) 342
rats (340) 340
signal transduction (340) 340
neovascularization (338) 338
immunohistochemistry (335) 335
cells (329) 329
tumors (326) 326
research (322) 322
in-vivo (320) 320
gene expression (316) 316
neovascularization, pathologic - metabolism (303) 303
angiogenic factor (297) 297
endothelial cells - metabolism (296) 296
article (294) 294
endothelium (294) 294
apoptosis (287) 287
disease models, animal (286) 286
cell proliferation (284) 284
endothelial-cells (284) 284
preeclampsia (283) 283
in-vitro (280) 280
proteins (275) 275
hypoxia (274) 274
neovascularization, physiologic (271) 271
peripheral vascular disease (270) 270
time factors (268) 268
metastasis (250) 250
rna, messenger - metabolism (250) 250
mice, inbred c57bl (249) 249
growth (242) 242
angiogenic switch (240) 240
pharmacology & pharmacy (236) 236
physiological aspects (230) 230
neovascularization, pathologic (228) 228
obstetrics & gynecology (227) 227
health aspects (223) 223
inflammation (223) 223
angiogenesis inhibitors - pharmacology (222) 222
growth-factor (222) 222
medicine, research & experimental (220) 220
up-regulation (213) 213
vascular endothelial growth factor a - genetics (210) 210
prognosis (209) 209
physiology (207) 207
abridged index medicus (205) 205
gene-expression (204) 204
endocrinology & metabolism (201) 201
care and treatment (200) 200
growth factors (192) 192
hematology (189) 189
neovascularization, physiologic - drug effects (189) 189
cell proliferation - drug effects (188) 188
reverse transcriptase polymerase chain reaction (186) 186
anti-angiogenic therapy (183) 183
case-control studies (183) 183
endothelial cells (182) 182
tumor angiogenesis (181) 181
blotting, western (179) 179
enzyme-linked immunosorbent assay (179) 179
rodents (179) 179
angiogenesis inhibitors - therapeutic use (176) 176
hypertension (175) 175
mice, nude (174) 174
breast-cancer (173) 173
neovascularization, pathologic - pathology (173) 173
neovascularization, pathologic - drug therapy (167) 167
activation (163) 163
cardiac & cardiovascular systems (163) 163
development and progression (163) 163
medicine & public health (159) 159
pathology (159) 159
medicine (158) 158
reproductive biology (157) 157
cell line (155) 155
neoplasms - metabolism (155) 155
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (3588) 3588
French (7) 7
Russian (4) 4
Chinese (3) 3
German (2) 2
Bulgarian (1) 1
Japanese (1) 1
Polish (1) 1
Spanish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


FASEB journal : official publication of the Federation of American Societies for Experimental Biology, ISSN 0892-6638, 04/2015, Volume 29, Issue 4, pp. 1290 - 1298
Journal Article
PLoS Biology, ISSN 1544-9173, 08/2016, Volume 14, Issue 8, pp. e1002529 - e1002529
AGGF1 is an angiogenic factor with therapeutic potential to treat coronary artery disease (CAD) and myocardial infarction (MI). However, the underlying... 
MYOCARDIAL-INFARCTION | KLIPPEL-TRENAUNAY-SYNDROME | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOTHELIAL-CELLS | GROWTH | SUSCEPTIBILITY | BIOLOGY | INJURY | SPECIFICATION | IDENTIFICATION | TUMORIGENESIS | ASSOCIATION | Angiogenic Proteins - genetics | Neovascularization, Physiologic - drug effects | Heart Diseases - metabolism | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Autophagy - physiology | Autophagy-Related Protein 5 - genetics | Autophagy - drug effects | Human Umbilical Vein Endothelial Cells - physiology | Macrolides - pharmacology | Autophagy - genetics | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Angiogenic Proteins - pharmacology | Recombinant Proteins - metabolism | Cell Line | Human Umbilical Vein Endothelial Cells - drug effects | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Recombinant Proteins - pharmacology | Muscle, Smooth, Vascular - cytology | Blotting, Western | Mice, Knockout | Animals | Myocytes, Cardiac - drug effects | Neovascularization, Pathologic - drug therapy | Autophagy-Related Protein 5 - metabolism | Beclin-1 - genetics | Myocytes, Cardiac - metabolism | Neovascularization, Pathologic - genetics | Heart Diseases - drug therapy | Neovascularization, Pathologic - metabolism | Beclin-1 - metabolism | Angiogenic Proteins - metabolism | Heart Diseases - genetics | Proteins | Angiogenesis | Heart attacks | Rodents | Surgery | Coronary vessels | Cardiovascular disease | Kinases | Autophagy | Growth factors | Apoptosis | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 4/2016, Volume 113, Issue 16, pp. 4470 - 4475
Glioblastomas (GBMs) rapidly become refractory to anti-VEGF therapies. We previously demonstrated that ectopic overexpression of angiopoietin-2 (Ang-2)... 
Anti-angiogenic therapy | Colony-stimulating factor 1 | Tumor immunity | Tumor microenvironment | Macrophage | anti-angiogenic therapy | ANGIOPOIETIN-2 FUNCTIONS | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | macrophage | BEVACIZUMAB PLUS IRINOTECAN | RANDOMIZED-TRIAL | ANTIBODY | tumor immunity | KINASE INHIBITOR | ANTIANGIOGENIC THERAPY | PHASE-II TRIAL | colony-stimulating factor 1 | tumor microenvironment | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Macrophages - pathology | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Neovascularization, Pathologic - pathology | Neoplasms, Experimental - pathology | Macrophages - metabolism | Animals | Antibodies, Neoplasm - pharmacology | Receptors, Vascular Endothelial Growth Factor - metabolism | Ribonuclease, Pancreatic - metabolism | Neovascularization, Pathologic - drug therapy | Glioblastoma - pathology | Cell Line, Tumor | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Glioblastoma - metabolism | Mice | Neovascularization, Pathologic - metabolism | Neoplasms, Experimental - metabolism | Glioblastoma - drug therapy | Quinazolines - pharmacology | Ribonuclease, Pancreatic - antagonists & inhibitors | Neoplasms, Experimental - drug therapy | Drug Screening Assays, Antitumor | Survival analysis | Gene expression | Kinases | Cells | Tumors | Cancer | Index Medicus | Biological Sciences
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2015, Volume 125, Issue 4, pp. 1497 - 1508
Synaptic plasticity is the ability of synapses to modulate the strength of neuronal connections; however, the molecular factors that regulate this feature are... 
DENDRITIC SPINES | MEDICINE, RESEARCH & EXPERIMENTAL | EMERGING ROLES | MEMORY | EXCITATORY SYNAPSES | APOPTOTIC CELLS | RECEPTOR | BRAIN ANGIOGENESIS INHIBITOR-1 | LONG-TERM POTENTIATION | PSD-95 | POSTSYNAPTIC DENSITY | Disks Large Homolog 4 Protein | Guanylate Kinases - genetics | Angiogenic Proteins - genetics | Memory Disorders - physiopathology | Synaptic Transmission - physiology | Memory Disorders - genetics | Humans | Nerve Tissue Proteins - deficiency | Angiogenic Proteins - physiology | Learning Disorders - physiopathology | Proto-Oncogene Proteins c-mdm2 - physiology | Recombinant Fusion Proteins - metabolism | Spatial Learning - physiology | Brain - blood supply | Membrane Proteins - deficiency | Neuronal Plasticity - genetics | Ubiquitination | Neuronal Plasticity - physiology | Neurons - ultrastructure | HEK293 Cells | Long-Term Potentiation - physiology | Membrane Proteins - metabolism | Learning Curve | Protein Structure, Tertiary | Nerve Tissue Proteins - physiology | Angiogenic Proteins - deficiency | Maze Learning - physiology | Membrane Proteins - genetics | Mice, Inbred C57BL | Hippocampus - pathology | Nerve Tissue Proteins - genetics | Mice, Knockout | Protein Interaction Mapping | Guanylate Kinases - deficiency | Animals | Guanylate Kinases - metabolism | Learning Disorders - genetics | Mice | Protein Processing, Post-Translational | Hippocampus - physiopathology | Cell receptors | Nervous system diseases | Neural transmission | Development and progression | Genetic aspects | Hippocampus (Brain) | Health aspects | Proteins | Angiogenesis | Rodents | Memory | Cloning | Software | Behavior | Gene expression | Index Medicus | Abridged Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2011, Volume 31, Issue 5, pp. 1141 - 1150
Journal Article
Circulation, ISSN 0009-7322, 06/2013, Volume 127, Issue 25, pp. 2514 - 2522
BACKGROUND—The exact etiology of preeclampsia is unknown, but there is growing evidence of an imbalance in angiogenic growth factors and abnormal placentation.... 
fms-like tyrosine kinase-1 | placental growth factor | hydrogen sulfide | angiogenesis | vascular endothelial growth factor | preeclampsia | fetal development | CARDIAC & CARDIOVASCULAR SYSTEMS | SOLUBLE ENDOGLIN | RISK | ANGIOGENIC FACTORS | WOMEN | SERUM CONCENTRATIONS | TYROSINE KINASE-1 | PREGNANCIES | VASORELAXANT | PERIPHERAL VASCULAR DISEASE | GROWTH-FACTOR | MOUSE PLACENTA | Endothelium, Vascular - cytology | Hydrogen Sulfide - metabolism | Pregnancy Complications, Cardiovascular - physiopathology | Pregnancy, Animal | Glycine - analogs & derivatives | Glycine - adverse effects | Humans | Cystathionine gamma-Lyase - metabolism | Pregnancy Proteins - metabolism | Organothiophosphorus Compounds - pharmacology | Pre-Eclampsia - physiopathology | Antigens, CD - metabolism | Hypertension - etiology | Placenta Diseases - etiology | Young Adult | Endoglin | Pregnancy Complications, Cardiovascular - metabolism | Fetal Development - physiology | Hypertension - chemically induced | Adult | Alkynes - pharmacology | Female | Pre-Eclampsia - metabolism | Organ Culture Techniques | Disease Models, Animal | Fetal Development - drug effects | Mice, Inbred C57BL | Cells, Cultured | Morpholines - pharmacology | Receptors, Cell Surface - metabolism | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Placenta Diseases - physiopathology | Hypertension - metabolism | Hydrogen Sulfide - antagonists & inhibitors | Placenta - drug effects | Placenta - metabolism | Pregnancy | Neovascularization, Physiologic - physiology | Placenta - physiopathology | Animals | Glycine - pharmacology | Endothelium, Vascular - metabolism | Adolescent | Placenta Growth Factor | Mice | Placenta Diseases - metabolism | Alkynes - adverse effects | Pregnancy Complications, Cardiovascular - etiology | Index Medicus | Abridged Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 02/2012, Volume 31, Issue 6, pp. 728 - 738
Fibulin-2 (FBLN2) has been identified as a candidate tumor-suppressor gene in nasopharyngeal carcinoma (NPC). Originally identified through a chromosome 3 NotI... 
tumor suppressor | nasopharyngeal carcinoma | anti-angiogenic | fibulin-2 | methylation | VEGF | METASTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | IDENTIFICATION | ESOPHAGEAL | CANCER | BREAST | CELL BIOLOGY | ONCOLOGY | COPY NUMBER | GENETICS & HEREDITY | EXTRACELLULAR-MATRIX PROTEIN | CHROMOSOME 3P21.3 | EXPRESSION | PROGRESSION | Nasopharyngeal Neoplasms - genetics | Nasopharyngeal Neoplasms - metabolism | Carcinoma | Cell Proliferation | Humans | Gene Expression Regulation, Neoplastic | Molecular Sequence Data | Transplantation, Heterologous | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Nasopharyngeal Neoplasms - pathology | DNA Methylation | Protein Isoforms - metabolism | Base Sequence | Tumor Suppressor Proteins - genetics | Extracellular Matrix Proteins - metabolism | Calcium-Binding Proteins - metabolism | Cell Line | Nasopharyngeal Carcinoma | Tumor Suppressor Proteins - metabolism | Matrix Metalloproteinase 2 - metabolism | Neoplasm Invasiveness | Extracellular Matrix Proteins - genetics | Tumor Burden | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Matrix Metalloproteinase 2 - genetics | Animals | Mice, Nude | Cell Line, Tumor | Neovascularization, Pathologic - genetics | Mice | Neovascularization, Pathologic - metabolism | Calcium-Binding Proteins - genetics | Cell Movement | Protein Isoforms - genetics | Physiological aspects | Tumor suppressor genes | Genetic aspects | Research | Nasopharyngeal cancer | Gene expression | Vascular endothelial growth factor | Proteins | DNA methylation | Angiogenesis | Head & neck cancer | Cellular biology | Cell proliferation | Nasopharyngeal carcinoma | chromosome 3 | Extracellular matrix | genomics | Chromosome deletion | Matrix metalloproteinase | Cell migration | Tumors | Index Medicus
Journal Article
Developmental Biology, ISSN 0012-1606, 07/2013, Volume 379, Issue 1, pp. 38 - 52
( ), a gene highly conserved across the animal kingdom, encodes for a transmembrane protein that mediates Wnt ligand secretion. Wls is expressed in developing... 
Lung development | Wnt | Epithelium | Wntless | Endothelium | MULTIPLE MESENCHYMAL LINEAGES | BRANCHING MORPHOGENESIS | CELL PROLIFERATION | EMBRYONIC LUNG | DEVELOPMENTAL BIOLOGY | BETA-CATENIN | ANGIOGENIC FACTORS | IN-VITRO | ENDOTHELIAL-CELLS | MOUSE LUNG | Immunohistochemistry | Receptor, TIE-2 | Intracellular Signaling Peptides and Proteins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Wnt-5a Protein | Vascular Endothelial Growth Factor A - genetics | Wnt Proteins - metabolism | Embryo, Mammalian - metabolism | Wnt Proteins - genetics | Gene Expression Regulation, Developmental | Gene Deletion | Cell Death | Cell Differentiation | Lung - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Wnt Signaling Pathway | SOX9 Transcription Factor - metabolism | Lung - pathology | Epithelium - metabolism | Receptors, G-Protein-Coupled | Embryonic Development | Receptor Protein-Tyrosine Kinases - metabolism | Mice, Knockout | Animals | Embryo, Mammalian - embryology | Receptor Protein-Tyrosine Kinases - genetics | Endothelium, Vascular - metabolism | Lung - embryology | Mesoderm - metabolism | Mice | In Vitro Techniques | Lung - blood supply | SOX9 Transcription Factor - genetics | Genetic disorders | Wildlife conservation | Vascular endothelial growth factor | Stem cells | Index Medicus | endothelium | epithelium | lung development
Journal Article
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2012, Volume 181, Issue 4, pp. 1250 - 1270
Hyaluronan is activated by fragmentation and controls inflammation and fibroplasia during wound repair and diseases (eg, cancer). Hyaluronan-binding peptides... 
Pathology | REGULATED KINASE | CANCER-CELLS | DIABETIC MICE | ANGIOGENIC OLIGOSACCHARIDES | RECEPTOR RHAMM | BINDING PROTEINS | CELL LOCOMOTION | FOCAL ADHESION KINASE | PATHOLOGY | MEDIATED MOTILITY RHAMM | MYOFIBROBLAST DIFFERENTIATION | Inflammation - pathology | Molecular Weight | Extracellular Matrix Proteins - deficiency | Cell Count | Humans | Transforming Growth Factor beta1 - metabolism | Hyaluronan Receptors - chemistry | Tenascin - metabolism | Neovascularization, Pathologic - pathology | Inflammation - metabolism | Peptides - metabolism | Protein Binding - drug effects | Hyaluronan Receptors - metabolism | Female | Collagen - biosynthesis | Wound Healing - drug effects | Extracellular Matrix Proteins - metabolism | Skin - pathology | Fibroblasts - metabolism | Extracellular Matrix Proteins - chemistry | Macrophages - pathology | Rats | Recombinant Proteins - pharmacology | Fibroblasts - pathology | Peptides - pharmacology | Binding, Competitive - drug effects | Cell Movement - drug effects | Macrophages - metabolism | Peptides - isolation & purification | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Fibroblasts - drug effects | Hyaluronic Acid - metabolism | Fibrosis | Macrophages - drug effects | Mice | Skin - drug effects | Index Medicus | Abridged Index Medicus | Regular
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2012, Volume 122, Issue 1, pp. 178 - 191
Journal Article