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Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2016, Volume 36, Issue 1, pp. 86 - 96
OBJECTIVE—Comprehensive understanding of the mechanisms regulating angiogenesis might provide new strategies for angiogenic therapies for treating diverse... 
hematopoietic systems | endothelial cells | regeneration | hindlimb ischemia | TRANSCRIPTION FACTORS | ACTS DOWNSTREAM | COLLATERAL VESSEL DEVELOPMENT | LINEAGE ANALYSIS | CELL-SURVIVAL | MOUSE MODEL | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | GROWTH-FACTOR | HEMATOLOGY | TRANSGENIC MICE | Angiogenic Proteins - genetics | Hindlimb | Transcription Factors - deficiency | Male | Muscle, Skeletal - metabolism | Ischemia - genetics | Vascular Endothelial Growth Factor A - metabolism | Wound Healing | Recovery of Function | Vascular Endothelial Growth Factor Receptor-2 - genetics | Time Factors | Lentivirus - genetics | Ischemia - pathology | Disease Models, Animal | Muscle, Skeletal - blood supply | Gene Transfer Techniques | Angiogenic Proteins - deficiency | Signal Transduction | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Ischemia - metabolism | Ischemia - therapy | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Ischemia - physiopathology | Transcription Factors - genetics | Choroidal Neovascularization - physiopathology | Mice, Knockout | Skin - blood supply | Transcription Factors - metabolism | Regeneration | Phenotype | Animals | Choroidal Neovascularization - metabolism | Heterozygote | Muscle, Skeletal - pathology | Endothelial Cells - pathology | Genetic Vectors | Angiogenic Proteins - metabolism | Choroidal Neovascularization - genetics | Neovascularization, Physiologic | Index Medicus | VEGFR2 | injury-induced neovascularization | vascular regeneration | ER71 | ETV2 | Tie2-Cre | VEcadherin-Cre | FLK1
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e90921 - e90921
A striking feature of microvascular endothelial cells is their capacity to fuse and differentiate into tubular structures when grown in three-dimensional (3D)... 
IN-VITRO | SIGNALING PATHWAYS | ANGIOGENIC OLIGOSACCHARIDES | ACID | CANCER CELLS | MOLECULAR-WEIGHT HYALURONAN | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | ADHESION MOLECULES | RECEPTORS CD44 | UP-REGULATION | Hyaluronoglucosaminidase - metabolism | Humans | Male | NF-kappa B - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Gene Knockdown Techniques | Hyaluronan Receptors - metabolism | Telomerase - metabolism | Cell Differentiation | Microvessels - cytology | Membrane Fusion | Signal Transduction | Endothelial Cells - metabolism | Receptors, Chemokine - metabolism | Gene Expression Regulation | Gene Silencing | Cell Adhesion | Cell Adhesion Molecules - metabolism | GPI-Linked Proteins - metabolism | Protein Transport | Chemokine CXCL12 - metabolism | Endothelial Cells - cytology | Chemokine CXCL9 - metabolism | Hyaluronic Acid - metabolism | Cell Line, Tumor | Vacuoles - metabolism | Neovascularization, Physiologic | Translocation | Phosphorylation | CXCL12 protein | Cytokines | Blood vessels | Breast cancer | CXCR3 protein | Cell differentiation | Gene expression | CXCR4 protein | Endothelial cells | Mimicry | Hyaluronan | Receptors | CD44 antigen | Transcription activation | Collagen | In vivo methods and tests | Microvasculature | Auditory defects | Telomerase | Chemokines | Cancer | Index Medicus | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Cancer Cell, ISSN 1535-6108, 2008, Volume 13, Issue 6, pp. 507 - 518
Immune responses may arrest tumor growth by inducing tumor dormancy. The mechanisms leading to either tumor dormancy or promotion of multistage carcinogenesis... 
CELLCYCLE | METASTATIC MELANOMA | INTERFERON-GAMMA | DENDRITIC CELLS | ONCOLOGY | INTEGRIN EXPRESSION | ANGIOGENIC SWITCH | ENDOTHELIAL-CELLS | SKIN CARCINOGENESIS | PANCREATIC-ISLET TUMORS | NECROSIS-FACTOR-ALPHA | INNATE IMMUNITY | CELL BIOLOGY | Th1 Cells - pathology | Whole-Body Irradiation | Immunotherapy - methods | Cell Proliferation | Pancreatic Neoplasms - metabolism | Pancreatic Neoplasms - blood supply | Insulinoma - genetics | GTPase-Activating Proteins - metabolism | Interferon-gamma - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | CD4-Positive T-Lymphocytes - pathology | Th1 Cells - immunology | Integrin alphaVbeta3 - metabolism | Neovascularization, Pathologic - pathology | CD4-Positive T-Lymphocytes - immunology | Receptors, Tumor Necrosis Factor, Type I - deficiency | Time Factors | Cell Transformation, Neoplastic - genetics | Insulinoma - blood supply | Neovascularization, Pathologic - immunology | Antigens, Viral, Tumor - genetics | Insulinoma - immunology | Signal Transduction | Cell Survival | Pancreatic Neoplasms - pathology | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Mice, Transgenic | Pancreatic Neoplasms - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | Cell Transformation, Neoplastic - metabolism | Insulinoma - therapy | Mice, Inbred C3H | Mice, Knockout | Antigens, Viral, Tumor - metabolism | Cell Transformation, Neoplastic - immunology | Animals | CD4-Positive T-Lymphocytes - transplantation | Insulinoma - metabolism | Pancreatic Neoplasms - immunology | Insulinoma - pathology | Mice | GTPase-Activating Proteins - genetics | Blood Glucose - metabolism | Cell Transformation, Neoplastic - pathology | Cell Movement | Pancreatic Neoplasms - therapy | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 04/2015, Volume 35, Issue 4, pp. 865 - 876
Journal Article
Scientific Reports, ISSN 2045-2322, 02/2016, Volume 6, Issue 1, pp. 19857 - 19857
Through both gain-and loss-of-TTF-1 expression strategies, we show that TTF-1 positively regulates vascular endothelial growth factor (VEGF) and that the VEGF... 
SURVIVAL | MORPHOGENESIS | EPITHELIAL-CELLS | GM-CSF | VEGF | MULTIDISCIPLINARY SCIENCES | LUNG ADENOCARCINOMA | GENE-EXPRESSION | NKX2-1 | CANCER | ENDOTHELIAL GROWTH-FACTOR | Exosomes - metabolism | Angiogenic Proteins - genetics | Neovascularization, Physiologic - drug effects | Thyroid Nuclear Factor 1 | TOR Serine-Threonine Kinases - metabolism | Humans | Lung Neoplasms - metabolism | Culture Media, Conditioned - pharmacology | Lung Neoplasms - pathology | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor Receptor-2 - immunology | Vascular Endothelial Growth Factor A - genetics | Cell Hypoxia | Transcription Factors - secretion | Nuclear Proteins - secretion | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Cytokines - genetics | Real-Time Polymerase Chain Reaction | Lung Neoplasms - blood supply | A549 Cells | Promoter Regions, Genetic | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Cell Survival | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Indoles - toxicity | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Vascular Endothelial Growth Factor A - analysis | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Blotting, Western | Antibodies - pharmacology | Transcription Factors - metabolism | Up-Regulation - drug effects | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Pyrroles - toxicity | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Angiogenic Proteins - metabolism | RNA, Small Interfering - metabolism | TOR protein | Statistical analysis | Granulocyte-macrophage colony-stimulating factor | Lung | Lung cancer | Media | Rapamycin | Secretome | Exosomes | Macrophages | Angiogenesis | Hypoxia | Colony-stimulating factor | Vascular endothelial growth factor | Thyroid transcription factor 1 | Thyroid | Index Medicus
Journal Article
Oncogene, ISSN 0950-9232, 01/2013, Volume 32, Issue 4, pp. 502 - 513
Despite their apparent success in pre-clinical trials, metalloproteinase (MMP) inhibitors proved to be inefficacious in clinical settings. In an effort to... 
invasion | cathepsin | metalloproteinase | Myc | tumor | interleukin-1β | ANGIOGENIC SWITCH | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHASE-III | MULTISTAGE TUMORIGENESIS | CELL BIOLOGY | interleukin-1 beta | ONCOLOGY | MATRIX-METALLOPROTEINASE INHIBITORS | MARROW-DERIVED CELLS | GENETICS & HEREDITY | CAPILLARY ENDOTHELIAL-CELLS | EXTRACELLULAR-MATRIX | LYSOSOMAL CYSTEINE PROTEASES | MALIGNANT PROGRESSION | CATHEPSIN-B | Inflammation - pathology | Leukocytes - pathology | Pancreatic Neoplasms - metabolism | Cathepsin B - genetics | Pancreatic Neoplasms - blood supply | bcl-X Protein - genetics | GTPase-Activating Proteins - metabolism | Interleukin-1beta - genetics | Neuroendocrine Tumors - enzymology | Cysteine - genetics | Neovascularization, Pathologic - pathology | CD11b Antigen - genetics | Inflammation - metabolism | Matrix Metalloproteinase 9 - metabolism | Cell Transformation, Neoplastic - genetics | Interleukin-1beta - metabolism | Matrix Metalloproteinase 9 - genetics | Cysteine - metabolism | Disease Models, Animal | Neuroendocrine Tumors - pathology | Neoplasm Invasiveness | Neuroendocrine Tumors - metabolism | Mice, Inbred C57BL | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - enzymology | Cathepsin B - metabolism | Matrix Metalloproteinase 9 - deficiency | Cell Transformation, Neoplastic - metabolism | Proto-Oncogene Proteins c-myc - metabolism | Matrix Metalloproteinase Inhibitors - pharmacology | Animals | Inflammation - genetics | Leukocytes - drug effects | Neovascularization, Pathologic - genetics | Mice | Neovascularization, Pathologic - metabolism | Proto-Oncogene Proteins c-myc - genetics | CD11b Antigen - metabolism | Cell Transformation, Neoplastic - drug effects | GTPase-Activating Proteins - genetics | Cell Transformation, Neoplastic - pathology | bcl-X Protein - metabolism | Leukocytes - metabolism | Neuroendocrine Tumors - blood supply | Physiological aspects | Genetic aspects | Research | Metalloenzymes | Gene expression | Oncology | Cytokines | Proteases | Pancreatic cancer | Tumors | Index Medicus
Journal Article
Biological and Pharmaceutical Bulletin, ISSN 0918-6158, 2018, Volume 41, Issue 6, pp. 869 - 876
Pinelliae Rhizoma Praeparatum (PRP) as traditional Chinese medicine had been used for hepatic diseases in combinative forms. However, the effect of PRP was not... 
bile acids metabolic | Pinelliae Rhizoma Praeparatum | bile salt export pump | Mrp3 | nuclear factor erythroid 2-related factor 2 (Nrf2) | multidrug resistance-associated protein 2 (Mrp2) | Nuclear factor erythroid 2-related factor 2 (Nrf2) | Multidrug resistance-associated protein 2 (Mrp2) | Key words bile acids metabolic | Bile salt export pump | FXR | CELLS | CHOLESTASIS | FARNESOID-X RECEPTOR | FREE-RADICALS | HEPATOCARCINOGENESIS | CHOTO-SAN | LIVER | multidrug resistance associated protein 2 (Mrp2) | PHARMACOLOGY & PHARMACY | MICE | EXPRESSION | Liver - pathology | Cholestasis - metabolism | Plant Extracts - pharmacology | Liver - metabolism | Bile Acids and Salts - metabolism | Male | Pinellia | Cholestasis - chemically induced | Mice, Inbred ICR | Dose-Response Relationship, Drug | Plant Extracts - administration & dosage | Cholestasis - pathology | Animals | Liver - drug effects | ATP Binding Cassette Subfamily B Member 11 - metabolism | Acetaminophen | Chemical and Drug Induced Liver Injury - metabolism | NF-E2-Related Factor 2 - metabolism | Chemical and Drug Induced Liver Injury - pathology | Angiogenic Proteins - metabolism | Multidrug Resistance-Associated Proteins - metabolism | Oxidative stress | Bile acids | Liver diseases | Liver | Multidrug resistance | Medical treatment | Markers | Traditional Chinese medicine | Metabolism | Dosage | Antioxidants | Proteins | Herbal medicine | Acids | Rodents | Mice | Pretreatment | Transporter | Cholestasis | Injuries | Bile | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 04/2010, Volume 106, Issue 8, pp. 1425 - 1433
RATIONALE:Arteriovenous malformations (AVMs) result in anomalous direct blood flow between arteries and veins, bypassing the normal capillary bed. Depending on... 
Angiogenesis | Mouse models of cardiovascular disease | Endothelial cells | mouse models of cardiovascular disease | RETINAL VASCULATURE | endothelial cells | CARDIAC & CARDIOVASCULAR SYSTEMS | HEREDITARY HEMORRHAGIC TELANGIECTASIA | ENDOTHELIAL-CELL PROLIFERATION | MICE LACKING ENDOGLIN | angiogenesis | BETA RECEPTOR | RECEPTOR-LIKE KINASE-1 | MORPHOGENESIS | VESSELS | GENE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Cell Proliferation | Capillaries - pathology | Retinal Neovascularization - metabolism | Antigens, CD - genetics | Arteriovenous Malformations - physiopathology | Neovascularization, Pathologic - pathology | Intracellular Signaling Peptides and Proteins - deficiency | Endoglin | Microcirculation | Arteriovenous Malformations - pathology | Aging | Neovascularization, Pathologic - physiopathology | Retinal Neovascularization - genetics | Cadherins - genetics | Capillaries - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Endothelial Cells - metabolism | Mice, Transgenic | Regional Blood Flow | Mice, Knockout | Animals | Arteriovenous Malformations - genetics | Arteriovenous Malformations - metabolism | Neovascularization, Pathologic - genetics | Mice | Neovascularization, Pathologic - metabolism | Mutation | Endothelial Cells - pathology | Retinal Neovascularization - physiopathology | Angiogenic Proteins - metabolism | Integrases - genetics | Retinal Neovascularization - pathology | Index Medicus
Journal Article
British Journal of Cancer, ISSN 0007-0920, 07/2012, Volume 107, Issue 3, pp. 501 - 507
BACKGROUND: Our earlier reports demonstrated that membrane-bound semaphorin 5A (SEMA5A) is expressed in aggressive pancreatic cancer cells and tumours, and... 
semaphorin | angiogenic factors | pancreatic cancer | secreted SEMA5A | reduced tumour burden | tumour burden and metastasis | CANCER-CELLS | SEMA5A | LUNG | ANGIOGENESIS | GUIDANCE | IDENTIFICATION | GENE | ONCOLOGY | GROWTH | EXPRESSION | CARCINOMA | Interleukin-8 - genetics | Nerve Tissue Proteins - secretion | Pancreatic Neoplasms - metabolism | Humans | Cell Growth Processes - physiology | Male | Vascular Endothelial Growth Factor A - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Vascular Endothelial Growth Factor A - genetics | Extracellular Signal-Regulated MAP Kinases - genetics | Neovascularization, Pathologic - pathology | Phosphorylation - genetics | Neoplasm Micrometastasis | Angiogenesis Inducing Agents - metabolism | Membrane Proteins - metabolism | Interleukin-8 - metabolism | Tumor Cells, Cultured | Liver Neoplasms - secondary | Pancreatic Neoplasms - secretion | Membrane Proteins - secretion | Endothelial Cells - metabolism | Membrane Proteins - genetics | Neoplasm Invasiveness | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - genetics | Nerve Tissue Proteins - genetics | Disease Progression | Nerve Tissue Proteins - metabolism | Animals | Mice, Nude | Neovascularization, Pathologic - genetics | Mice | Mice, Inbred BALB C | Neovascularization, Pathologic - metabolism | Endothelial Cells - pathology | Index Medicus | Molecular Diagnostics
Journal Article
Journal Article