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Investigative ophthalmology & visual science, ISSN 0146-0404, 2012, Volume 53, Issue 6, pp. 2968 - 2977
Journal Article
Journal Article
FASEB Journal, ISSN 0892-6638, 12/2012, Volume 26, Issue 12, pp. 4876 - 4885
Uterine spiral artery remodeling is required for successful human pregnancy; impaired remodeling is associated with pregnancy complications, including late... 
Proteases | Vascular smooth muscle cells | Angiogenic growth factors | TROPHOBLAST INVASION | GRANULATED LYMPHOCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | proteases | CELL BIOLOGY | angiogenic growth factors | EXTRAVILLOUS TROPHOBLAST | SMOOTH-MUSCLE | IN-VITRO | BIOLOGY | CARDIOVASCULAR-DISEASE | MATERNAL ARTERIES | GROWTH-FACTOR | vascular smooth muscle cells | EXPRESSION | FACTS | Immunohistochemistry | Muscle, Smooth, Vascular - metabolism | Decidua - cytology | Humans | Extracellular Matrix - metabolism | Culture Media, Conditioned - pharmacology | Interferon-gamma - metabolism | Angiopoietin-1 - metabolism | Angiopoietin-2 - pharmacology | Angiopoietin-2 - metabolism | Uterine Artery - physiology | Placenta - cytology | Myometrium - metabolism | Female | Trophoblasts - cytology | Uterine Artery - drug effects | Myocytes, Smooth Muscle - drug effects | Uterus - blood supply | Myocytes, Smooth Muscle - metabolism | Angiopoietin-1 - pharmacology | Cell Line | Trophoblasts - metabolism | Uterine Artery - cytology | Tissue Culture Techniques | Extracellular Matrix - drug effects | Cells, Cultured | Muscle, Smooth, Vascular - cytology | Killer Cells, Natural - physiology | Decidua - metabolism | Myometrium - cytology | Placenta - metabolism | Pregnancy | Trophoblasts - physiology | Vascular Endothelial Growth Factor C - metabolism | Uterus - cytology | Killer Cells, Natural - metabolism | Interferon-gamma - pharmacology | Culture Media, Conditioned - metabolism | Vascular Endothelial Growth Factor C - pharmacology
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2012, Volume 7, Issue 4, p. e35905
Hematopoietic progenitor CD133(+)/c-kit(+) cells have been shown to be involved in myocardial healing following myocardial infarction (MI). Previously we... 
CD31(+) CELLS | ENDOTHELIAL PROGENITOR CELLS | ISCHEMIA/REPERFUSION INJURY | ACTIVATION | ISCHEMIC VASCULAR-DISEASE | ANGIOGENIC ACTIVITY | BONE-MARROW | BIOLOGY | RECEPTOR | TOPCARE-AMI | EXPRESSION | Up-Regulation | Capillaries - pathology | Receptors, Notch - metabolism | Cardiomegaly - pathology | Angiopoietin-1 - metabolism | Antigens, CD - metabolism | Peptides - metabolism | Serrate-Jagged Proteins | Bone Marrow - metabolism | Myocardium - metabolism | Endomyocardial Fibrosis - complications | Myocardial Infarction - physiopathology | Capillaries - metabolism | Apelin | Jagged-1 Protein | Capillaries - physiopathology | Signal Transduction | Cardiomegaly - physiopathology | Myocardial Infarction - metabolism | Receptors, CXCR4 - metabolism | Chemokine CXCL12 - metabolism | Hematopoietic Stem Cells - cytology | Endomyocardial Fibrosis - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Cardiomegaly - metabolism | Cell Movement | Actins - metabolism | Glycoproteins - metabolism | Diabetes Mellitus, Type 2 - metabolism | Proto-Oncogene Proteins c-kit - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Myocardial Infarction - pathology | Endomyocardial Fibrosis - pathology | Membrane Proteins - metabolism | Heart Function Tests | Diabetes Mellitus, Type 2 - complications | Calcium-Binding Proteins - metabolism | Myocardium - pathology | Adipokines | AC133 Antigen | Cardiomegaly - complications | Myocardial Infarction - complications | Animals | Diabetes Mellitus, Type 2 - physiopathology | Bone Marrow - pathology | Receptor, Notch3 | Endomyocardial Fibrosis - physiopathology | Apoptosis | Advertising executives | Vascular endothelial growth factor | Adenoviruses | Heart attack | Myocardial infarction | Heart | Diabetic retinopathy | Heart attacks | Angiopoietin | Recovery of function | Smooth muscle | Cardiovascular disease | Recruitment | Angiogenesis | Toxicology | Cell growth | Ischemia | Data recovery | Rodents | Bone marrow | Repair | Heart diseases | Immunoglobulins | Diabetes mellitus | Coronary artery | Muscles | Pharmacology | c-Kit protein | Hemopoiesis | Studies | Ostomy | Coronary vessels | Fibrosis | Stem cells | Myocardium | Infarction | Diabetes | Laboratory animals
Journal Article
Stem Cells and Development, ISSN 1547-3287, 04/2014, Volume 23, Issue 7, pp. 741 - 754
In future, adipose-derived stem cells (ASC) might be used to treat neurological disorders. In this study, the neurotrophic and angiogenic properties of human... 
Original Research Reports | MEDICINE, RESEARCH & EXPERIMENTAL | AXONAL REGENERATION | ROOT GANGLIA NEURONS | TRANSCRIPTION FACTOR-3 ATF3 | SENSORY NEURONS | CELL & TISSUE ENGINEERING | TRANSPLANTATION | C-JUN PHOSPHORYLATION | IN-VITRO | NEURITE OUTGROWTH | GROWTH-FACTOR | SCHWANN-CELLS | STROMAL CELLS | HEMATOLOGY | Brain-Derived Neurotrophic Factor - genetics | Spinal Cord - metabolism | Humans | Nerve Regeneration - physiology | Adipose Tissue - cytology | Adipocytes - cytology | Culture Media, Conditioned - pharmacology | JNK Mitogen-Activated Protein Kinases - metabolism | Neurons - cytology | Ganglia, Spinal - cytology | Vascular Endothelial Growth Factor A - metabolism | Angiopoietin-1 - metabolism | GAP-43 Protein - genetics | Vascular Endothelial Growth Factor A - genetics | Stem Cell Transplantation | Adipose Tissue - metabolism | Glial Cell Line-Derived Neurotrophic Factor - metabolism | Mesenchymal Stromal Cells - cytology | Adult | Brain-Derived Neurotrophic Factor - metabolism | Cell Differentiation | Neurons - metabolism | JNK Mitogen-Activated Protein Kinases - genetics | Spinal Cord - cytology | Activating Transcription Factor 3 - genetics | Biomarkers - metabolism | Mesenchymal Stromal Cells - drug effects | Gene Expression | GAP-43 Protein - metabolism | Glial Cell Line-Derived Neurotrophic Factor - genetics | Mesenchymal Stromal Cells - metabolism | Rats | Activating Transcription Factor 3 - metabolism | Cell Lineage - physiology | Angiopoietin-1 - genetics | Animals | Adipocytes - metabolism | Adipocytes - transplantation | Adipose Tissue - drug effects | Neovascularization, Physiologic | Ganglia, Spinal - metabolism | Adipose tissues | Nervous system diseases | Care and treatment | Prognosis | Physiological aspects | Development and progression | Research | Neovascularization | Gene expression | Risk factors | Basic Medicine | Neurosciences | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Neurovetenskaper
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 17763 - 11
We investigated the effect of a peroxisome proliferator-activated receptor alpha (PPAR?alpha) agonist ophthalmic solution in wound healing using a rat corneal... 
GAMMA | PPAR-ALPHA | CELLS | LIPID-METABOLISM | COLLAGEN | TIE2 | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | FACTOR-KAPPA-B | EXPRESSION | BASEMENT-MEMBRANE | Eye Burns - drug therapy | Ophthalmic Solutions - pharmacology | Burns, Chemical - metabolism | Corneal Neovascularization - drug therapy | Collagen Type III - metabolism | Male | Angiopoietin-1 - metabolism | RNA, Messenger - metabolism | Angiopoietin-2 - metabolism | Corneal Injuries - metabolism | Myofibroblasts - metabolism | Cicatrix - metabolism | Fenofibrate - pharmacology | Cornea - drug effects | Alkalies - pharmacology | Neutrophils - metabolism | Wound Healing - drug effects | Disease Models, Animal | Endothelial Cells - metabolism | Neutrophils - drug effects | Rats | Myofibroblasts - drug effects | Up-Regulation - drug effects | Cornea - metabolism | Cicatrix - drug therapy | Macrophages - metabolism | Animals | Corneal Neovascularization - metabolism | Neovascularization, Pathologic - drug therapy | Burns, Chemical - drug therapy | Eye Burns - metabolism | Macrophages - drug effects | PPAR alpha - agonists | Vascular Endothelial Growth Factors - metabolism | Neovascularization, Pathologic - metabolism | Corneal Injuries - drug therapy | Cornea | Wound healing | Angiopoietin | Stroma | Collagen (type III) | Leukocytes (neutrophilic) | Inflammation | Gene expression | Macrophages | Endothelial cells | Vascularization | Fenofibrate | Rodents | Vascular endothelial growth factor
Journal Article
Journal Article
Neurochemistry International, ISSN 0197-0186, 2009, Volume 54, Issue 3, pp. 253 - 263
Blood–brain barrier (BBB) characteristics are induced and maintained by cross-talk between brain microvessel endothelial cells and neighbouring elements of the... 
Astrocytes | Pericytes | In vitro BBB model (rat) | Tight junction | Transendothelial electrical resistance | Co-culture | P-Glycoprotein | Drug permeability | Blood–brain barrier | Brain endothelial cells | Blood-brain barrier | DRUG | PERMEABILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | ANGIOPOIETIN-1 | INDUCTION | GLIAL-CELLS | NEUROSCIENCES | MESSENGER-RNA | IN-VITRO MODEL | CULTURES | EXPRESSION | ATP-Binding Cassette, Sub-Family B, Member 1 - metabolism | Pericytes - ultrastructure | Phosphoproteins - metabolism | Blood-Brain Barrier - ultrastructure | Occludin | Microcirculation - physiology | Endothelial Cells - ultrastructure | Membrane Proteins - metabolism | Capillaries - metabolism | Rhodamine 123 - pharmacokinetics | Tight Junctions - metabolism | Microscopy, Electron, Transmission | Capillaries - ultrastructure | Endothelial Cells - metabolism | Pericytes - metabolism | Cells, Cultured | Rats | Claudin-5 | Cerebral Arteries - ultrastructure | Membrane Potentials - physiology | Blood-Brain Barrier - metabolism | Astrocytes - ultrastructure | Tight Junctions - ultrastructure | Animals | Carrier Proteins - metabolism | Zonula Occludens-1 Protein | Cell Membrane Permeability - physiology | Cerebral Arteries - metabolism | Models, Biological | Cell Membrane Permeability - drug effects | ATP Binding Cassette Transporter, Sub-Family B - metabolism | Astrocytes - metabolism | Proteins | Drug resistance in microorganisms | Brain | Fluorescein | Physiological aspects | Glucose | Universities and colleges | Permeability | Dextrose | Endothelium
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2007, Volume 27, Issue 12, pp. 2619 - 2626
OBJECTIVE—Tie2 and its ligands, the angiopoietins (Ang), are required for embryonic and postnatal angiogenesis. Previous studies have demonstrated that Tie2 is... 
Phosphoinositide 3-kinase | Akt | Tie2 | VEGF | Endothelium | endothelium | ACTIVATION | ANGIOGENESIS | TYROSINE KINASE | SOLUBLE TIE | GROWTH-FACTOR LEVELS | HUMAN ENDOTHELIAL-CELLS | OVEREXPRESSING ANGIOPOIETIN-1 | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ANGIOPOIETIN RECEPTOR TIE-2 | EXPRESSION | phosphoinositide 3-kinase | PLASMA ANGIOPOIETIN-1 | NIH 3T3 Cells | Neovascularization, Physiologic - drug effects | Fibroblasts - enzymology | Phosphorylation | Tetradecanoylphorbol Acetate - pharmacology | Humans | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Angiopoietin-1 - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Angiopoietin-2 - metabolism | Transfection | Receptor, TIE-2 - blood | Peptide Fragments - blood | Receptor, TIE-2 - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Peptide Fragments - genetics | Fibroblasts - metabolism | Peptide Hydrolases - metabolism | PTEN Phosphohydrolase - genetics | Peptide Fragments - metabolism | Endothelial Cells - metabolism | Dipeptides - pharmacology | Cells, Cultured | Morpholines - pharmacology | PTEN Phosphohydrolase - metabolism | Imidazoles - pharmacology | Receptor, TIE-2 - genetics | Animals | Signal Transduction - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Matrix Metalloproteinase Inhibitors | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Matrix Metalloproteinases - metabolism | Endothelial Cells - pathology | Endothelial Cells - enzymology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Apoptosis | Endothelial Cells - drug effects
Journal Article
Journal Article
eLife, ISSN 2050-084X, 03/2015, Volume 2015, Issue 4, p. e05521
Hematopoietic stem cells (HSCs) are maintained by a perivascular niche in bone marrow but it is unclear whether the niche is reciprocally regulated by HSCs.... 
LONG-TERM | OVEREXPRESSING ANGIOPOIETIN-1 | SINUSOIDAL ENDOTHELIAL-CELLS | BONE-MARROW NICHE | BIOLOGY | SELF-RENEWAL | LYMPHOID PROGENITORS | MICE | DISTINCT ROLES | RECEPTOR TYROSINE KINASE | TIE2 RECEPTOR | Gamma Rays | Stem Cell Niche - radiation effects | Receptors, Leptin - genetics | Blood Vessels - metabolism | Male | Angiopoietin-1 - metabolism | Proto-Oncogene Proteins c-kit - metabolism | Capillary Permeability - radiation effects | Regeneration - radiation effects | Blood Vessels - radiation effects | Hematopoiesis - radiation effects | Regeneration - genetics | Nestin - genetics | Female | Integrases - metabolism | Proto-Oncogene Proteins c-kit - genetics | Osteoblasts - cytology | Megakaryocytes - cytology | Megakaryocytes - metabolism | Megakaryocytes - radiation effects | Hematopoietic Stem Cells - radiation effects | Stem Cell Niche - genetics | Osteoblasts - radiation effects | Signal Transduction | Bone Marrow Cells - cytology | Stromal Cells - metabolism | Gene Expression Regulation | Hematopoiesis - genetics | Mice, Transgenic | Hematopoietic Stem Cells - metabolism | Bone Marrow Cells - radiation effects | Blood Vessels - cytology | Nestin - metabolism | Angiopoietin-1 - genetics | Animals | Hematopoietic Stem Cells - cytology | Mice | Receptors, Leptin - metabolism | Osteoblasts - metabolism | Integrases - genetics | Bone Marrow Cells - metabolism | Stromal Cells - cytology | Stromal Cells - radiation effects | Angiopoietin | Radiation | Stem cell transplantation | Osteoblasts | c-Kit protein | Experiments | Hematopoietic stem cells | Endothelial cells | Stains & staining | Regeneration | Angiogenesis | Allografts | Clonal deletion | Microscopy | Stromal cells | Rodents | Stem cells | Bone marrow | Megakaryocytes
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2015, Volume 10, Issue 6, p. e0130357
Background The successful treatment of malignant gliomas remains a challenge despite the current standard of care, which consists of surgery, radiation and... 
SURVIVAL | ANTIANGIOGENIC THERAPY | GLIOBLASTOMA | HYPERGLYCEMIA | EDEMA | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | GROWTH | ANGIOPOIETIN-1 | PROMOTES | CANCER | Carbonic Anhydrases - genetics | Brain Neoplasms - pathology | Hypoxia - diet therapy | Cell Membrane Permeability | NF-kappa B - metabolism | Neovascularization, Pathologic - diet therapy | Brain Neoplasms - blood supply | Glioma - diet therapy | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Brain Neoplasms - metabolism | Immunoenzyme Techniques | Carbonic Anhydrases - metabolism | Glioma - metabolism | Hypoxia - metabolism | Neovascularization, Pathologic - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Glioma - pathology | Aquaporin 4 - genetics | Biomarkers, Tumor - metabolism | Female | Zonula Occludens-1 Protein - metabolism | Real-Time Polymerase Chain Reaction | Carbonic Anhydrase IX | Disease Models, Animal | Brain Neoplasms - diet therapy | Zonula Occludens-1 Protein - genetics | Diet, Ketogenic | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Matrix Metalloproteinase 2 - metabolism | Neoplasm Invasiveness | Mice, Inbred C57BL | RNA, Messenger - genetics | Reverse Transcriptase Polymerase Chain Reaction | Glioma - blood supply | Matrix Metalloproteinase 2 - genetics | Animals | NF-kappa B - genetics | Hypoxia - pathology | Aquaporin 4 - metabolism | Biomarkers, Tumor - genetics | Mice | Neovascularization, Pathologic - metabolism | Low-carbohydrate diet | Aquaporins | Models | Permeability | Intermediate filament proteins | Health aspects | Vascular endothelial growth factor | Aquaporin 4 | Vimentin | Brain | Ketogenesis | Brain tumors | Brain cancer | Radiation | Oncology | Neurosurgery | Matrix metalloproteinase | Cancer therapies | High carbohydrate diet | High fat diet | Proteins | Angiogenesis | Low carbohydrate diet | Surgery | Life sciences | Metalloproteinase | Vascular endothelial growth factor receptor 2 | Growth factors | Research centers | Low protein diet | Edema | Carbohydrates | Phenotypes | Invasiveness | Metabolism | Survival | Brain research | Diet | Glioma | Carbonic anhydrase | Medical prognosis | Low fat diet | Hypoxia | Critical path | Microvasculature | Temozolomide | Laboratory animals | High protein diet | Tumors | Cancer
Journal Article
Osteoarthritis and Cartilage, ISSN 1063-4584, 2013, Volume 21, Issue 12, pp. 1913 - 1923
Summary Objective The aim of this study was to investigate the link between the hypertrophic phenotype of chondrocytes and angiogenesis in osteoarthritis (OA)... 
Rheumatology | Angiogenesis | Bone sialoprotein | Chondrocyte hypertrophy | Osteoarthritis | RHEUMATOLOGY | METALLOPROTEINASES | OSTEOCHONDRAL JUNCTION | ARTICULAR-CARTILAGE | SIALOPROTEIN | ENDOTHELIAL-CELLS | GROWTH-FACTOR | BONE | DIFFERENTIATION | ORTHOPEDICS | EXPRESSION | ALGINATE BEADS | Osteopontin - genetics | Up-Regulation | Thrombospondins - genetics | Humans | Osteopontin - metabolism | Vascular Endothelial Growth Factor A - metabolism | Angiopoietin-1 - metabolism | Vascular Endothelial Growth Factor A - genetics | Osteoarthritis - physiopathology | Intercellular Signaling Peptides and Proteins - metabolism | Cartilage, Articular - metabolism | Thrombospondin 1 - genetics | Fibroblast Growth Factor 2 - metabolism | Neovascularization, Pathologic - physiopathology | Membrane Proteins - metabolism | Chondrocytes - metabolism | Endothelial Cells - physiology | Chondrocytes - pathology | Nerve Growth Factor - metabolism | Cartilage, Articular - cytology | Matrix Metalloproteinase 2 - metabolism | Membrane Proteins - genetics | Cells, Cultured | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Osteoarthritis - metabolism | Nerve Growth Factor - genetics | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion | Osteoarthritis - genetics | Angiopoietin-1 - genetics | Matrix Metalloproteinase 2 - genetics | Fibroblast Growth Factor 2 - genetics | Integrin-Binding Sialoprotein - genetics | Neovascularization, Pathologic - genetics | Neovascularization, Pathologic - metabolism | Thrombospondin 1 - metabolism | Integrin-Binding Sialoprotein - metabolism | Thrombospondins - metabolism | Cell Movement | Hypertrophy
Journal Article