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PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e52287
Elevated plasma concentrations of lipoprotein(a) are associated with increased risk for atherothrombotic diseases. Apolipoprotein(a), the unique glycoprotein... 
LIPOPROTEIN(A) | MATRIX METALLOPROTEINASES | LYSINE-BINDING SITE | RISK-FACTOR | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | FIBRIN FRAGMENT-E | NITRIC-OXIDE | ATHEROSCLEROSIS | TUMOR-GROWTH | MAMMALIAN-CELLS | Apolipoproteins A - metabolism | Cell Line | Neovascularization, Physiologic - drug effects | Apolipoproteins A - genetics | Human Umbilical Vein Endothelial Cells - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Tissue Plasminogen Activator - metabolism | Humans | Kringles - genetics | Angiogenesis Inhibitors - pharmacology | Glycosylation | Recombinant Proteins - pharmacology | Human Umbilical Vein Endothelial Cells - physiology | Apolipoproteins A - pharmacology | HEK293 Cells | Fibrinolysin - pharmacology | Urokinase-Type Plasminogen Activator - pharmacology | Vascular Endothelial Growth Factor A - pharmacology | Angiopoietin-1 - pharmacology | Lysine | Thrombin | Thrombolytic drugs | Apolipoproteins | Vascular endothelial growth factor | Lipoprotein A | Endothelium | Angiopoietin | Serine | Homology | Biochemistry | Plasmin | Proenzymes | Cell adhesion & migration | Angiogenesis | Urokinase | Cell growth | Proteolysis | Atherosclerosis | Extracellular matrix | Physiology | Inhibition | Recombinant | Glycoprotein | Health risks | Permeability | Endothelial cells | U-Plasminogen activator | Lipoproteins | Glycosidases | Serine proteinase | Umbilical cord | Angiogenesis inhibitors | Angiostatin | Binding sites | Cancer | Veins & arteries
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2007, Volume 27, Issue 12, pp. 2619 - 2626
OBJECTIVE—Tie2 and its ligands, the angiopoietins (Ang), are required for embryonic and postnatal angiogenesis. Previous studies have demonstrated that Tie2 is... 
Phosphoinositide 3-kinase | Akt | Tie2 | VEGF | Endothelium | endothelium | ACTIVATION | ANGIOGENESIS | TYROSINE KINASE | SOLUBLE TIE | GROWTH-FACTOR LEVELS | HUMAN ENDOTHELIAL-CELLS | OVEREXPRESSING ANGIOPOIETIN-1 | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ANGIOPOIETIN RECEPTOR TIE-2 | EXPRESSION | phosphoinositide 3-kinase | PLASMA ANGIOPOIETIN-1 | NIH 3T3 Cells | Neovascularization, Physiologic - drug effects | Fibroblasts - enzymology | Phosphorylation | Tetradecanoylphorbol Acetate - pharmacology | Humans | Phosphatidylinositol 3-Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Angiopoietin-1 - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Angiopoietin-2 - metabolism | Transfection | Receptor, TIE-2 - blood | Peptide Fragments - blood | Receptor, TIE-2 - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Peptide Fragments - genetics | Fibroblasts - metabolism | Peptide Hydrolases - metabolism | PTEN Phosphohydrolase - genetics | Peptide Fragments - metabolism | Endothelial Cells - metabolism | Dipeptides - pharmacology | Cells, Cultured | Morpholines - pharmacology | PTEN Phosphohydrolase - metabolism | Imidazoles - pharmacology | Receptor, TIE-2 - genetics | Animals | Signal Transduction - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Matrix Metalloproteinase Inhibitors | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Matrix Metalloproteinases - metabolism | Endothelial Cells - pathology | Endothelial Cells - enzymology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Apoptosis | Endothelial Cells - drug effects
Journal Article
FASEB Journal, ISSN 0892-6638, 12/2012, Volume 26, Issue 12, pp. 4876 - 4885
Uterine spiral artery remodeling is required for successful human pregnancy; impaired remodeling is associated with pregnancy complications, including late... 
Proteases | Vascular smooth muscle cells | Angiogenic growth factors | TROPHOBLAST INVASION | GRANULATED LYMPHOCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | proteases | CELL BIOLOGY | angiogenic growth factors | EXTRAVILLOUS TROPHOBLAST | SMOOTH-MUSCLE | IN-VITRO | BIOLOGY | CARDIOVASCULAR-DISEASE | MATERNAL ARTERIES | GROWTH-FACTOR | vascular smooth muscle cells | EXPRESSION | FACTS | Immunohistochemistry | Muscle, Smooth, Vascular - metabolism | Decidua - cytology | Humans | Extracellular Matrix - metabolism | Culture Media, Conditioned - pharmacology | Interferon-gamma - metabolism | Angiopoietin-1 - metabolism | Angiopoietin-2 - pharmacology | Angiopoietin-2 - metabolism | Uterine Artery - physiology | Placenta - cytology | Myometrium - metabolism | Female | Trophoblasts - cytology | Uterine Artery - drug effects | Myocytes, Smooth Muscle - drug effects | Uterus - blood supply | Myocytes, Smooth Muscle - metabolism | Angiopoietin-1 - pharmacology | Cell Line | Trophoblasts - metabolism | Uterine Artery - cytology | Tissue Culture Techniques | Extracellular Matrix - drug effects | Cells, Cultured | Muscle, Smooth, Vascular - cytology | Killer Cells, Natural - physiology | Decidua - metabolism | Myometrium - cytology | Placenta - metabolism | Pregnancy | Trophoblasts - physiology | Vascular Endothelial Growth Factor C - metabolism | Uterus - cytology | Killer Cells, Natural - metabolism | Interferon-gamma - pharmacology | Culture Media, Conditioned - metabolism | Vascular Endothelial Growth Factor C - pharmacology
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 02/2008, Volume 214, Issue 2, pp. 491 - 503
Endothelial cells are constantly exposed to high or low shear stress in arteries and veins by the flowing blood. Angiopoietin‐2 (Ang‐2) is acting as a critical... 
OXIDATIVE STRESS | PHYSIOLOGY | RECEPTOR TYROSINE KINASES | ANGIOGENESIS | FLUID SHEAR-STRESS | NITRIC-OXIDE SYNTHASE | GROWTH-FACTOR | FOXO TRANSCRIPTION FACTORS | EXPRESSION | HUMAN ENDOTHELIAL-CELLS | TIE2 RECEPTOR | CELL BIOLOGY | Humans | Rifabutin - analogs & derivatives | RNA, Messenger - analysis | Stress, Mechanical | Culture Media, Serum-Free | Endothelium, Vascular - drug effects | Male | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Angiopoietin-2 - metabolism | Nitric Oxide - analysis | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Forkhead Transcription Factors - metabolism | Triazenes - pharmacology | Receptor, TIE-2 - metabolism | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Umbilical Veins - cytology | Vascular Endothelial Growth Factor A - pharmacology | Angiopoietin-1 - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Morpholines - pharmacology | Cell Separation - methods | Lactams, Macrocyclic - pharmacology | Regional Blood Flow | Nitric Oxide Synthase Type III - antagonists & inhibitors | Benzoquinones - pharmacology | Animals | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Forkhead Transcription Factors - analysis | Proto-Oncogene Proteins c-akt - analysis | Femoral Artery - physiology | Forkhead Box Protein O1 | Mice | Nitrites - analysis | Angiopoietin-2 - secretion | Dactinomycin - pharmacology | Protein-Tyrosine Kinases - antagonists & inhibitors
Journal Article
Biomaterials, ISSN 0142-9612, 2013, Volume 34, Issue 36, pp. 9201 - 9209
Journal Article
Journal Article
Inflammopharmacology, ISSN 0925-4692, 10/2014, Volume 22, Issue 5, pp. 305 - 317
Rheumatoid arthritis is a chronic inflammatory disease characterized by overproduction of inflammatory mediators along with undermined oxidative defensive... 
Adjuvant-induced arthritis | Oxidative stress | Dermatology | Rheumatology | Angiopoietin-1 | Evening primrose oil | Allergology | Angiogenesis | Biomedicine | Immunology | TNF-α | NSAIDs | Gastroenterology | Pharmacology/Toxicology | RHEUMATOID-ARTHRITIS | COLLAGEN-INDUCED ARTHRITIS | TNF-alpha | RATS | NECROSIS-FACTOR-ALPHA | GAMMA-LINOLENIC ACID | MODEL | IMMUNOLOGY | SYNOVIAL FIBROBLASTS | TOXICOLOGY | MODULATION | ENDOTHELIAL GROWTH-FACTOR | ESSENTIAL FATTY-ACIDS | Inflammation - pathology | Arthritis, Experimental - drug therapy | Rats, Wistar | Tumor Necrosis Factor-alpha - blood | Male | Aspirin - administration & dosage | Neovascularization, Pathologic - pathology | Arthritis, Experimental - pathology | Arthritis, Rheumatoid - drug therapy | Inflammation - drug therapy | Linoleic Acids - administration & dosage | Lipid Peroxidation - drug effects | Angiopoietin-1 - blood | Anti-Inflammatory Agents - administration & dosage | Aspirin - pharmacology | Plant Oils - administration & dosage | Drug Therapy, Combination | Linoleic Acids - pharmacology | gamma-Linolenic Acid - administration & dosage | Pyrazoles - pharmacology | Plant Oils - pharmacology | Administration, Oral | Anti-Inflammatory Agents - pharmacology | Rats | Antioxidants - pharmacology | Celecoxib | Sulfonamides - pharmacology | Disease Progression | Arthritis, Rheumatoid - pathology | Pyrazoles - administration & dosage | Animals | Neovascularization, Pathologic - drug therapy | Antioxidants - administration & dosage | gamma-Linolenic Acid - pharmacology | Oxidative Stress - drug effects | Sulfonamides - administration & dosage | Rheumatoid factor | Enzymes | Aspirin | Pharmacy | Linolenic acids | Development and progression | Arthritis | Superoxide | Glycoproteins
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2002, Volume 99, Issue 17, pp. 11205 - 11210
Journal Article
Journal Article
Journal Article
Cell Stem Cell, ISSN 1934-5909, 01/2009, Volume 4, Issue 1, pp. 62 - 72
Journal Article
PLoS One, ISSN 1932-6203, 2011, Volume 6, Issue 8, p. e23448
Background: Angiopoietin-2 (Ang-2) is associated with lung injury in ALI/ARDS. As endothelial activation by thrombin plays a role in the permeability of acute... 
INDUCED INCREASE | TIE2 | IN-VIVO | JUNCTIONS | KINASE | BIOLOGY | BARRIER FUNCTION | ACUTE LUNG INJURY | VE-CADHERIN | GENE-THERAPY | VASCULAR-PERMEABILITY | Angiopoietins - pharmacology | Cadherins - metabolism | Angiopoietin-2 - genetics | Humans | Immunoblotting | rhoA GTP-Binding Protein - metabolism | Angiopoietin-1 - metabolism | Angiopoietin-2 - pharmacology | Angiopoietin-2 - metabolism | Antigens, CD - metabolism | Thrombin - pharmacology | Time Factors | Coagulants - pharmacology | Receptor, TIE-2 - metabolism | Phosphorylation - drug effects | Endothelial Cells - physiology | Angiopoietin-1 - pharmacology | Angiopoietins - metabolism | Endothelial Cells - metabolism | Cells, Cultured | Electric Impedance | Recombinant Proteins - pharmacology | Angiopoietin-1 - genetics | Cell Membrane Permeability - drug effects | Angiopoietins - genetics | Lung - blood supply | rac1 GTP-Binding Protein - metabolism | Endothelial Cells - drug effects | Acute respiratory distress syndrome | Thrombin | Permeability | G proteins | Endothelium | Phosphorylation | Intensive care | Angiopoietin | Cell junctions | Activation | Kinases | Experiments | Angiogenesis | Cell activation | Cell growth | Rodents | Physiology | Vascular endothelial growth factor | Medical research | RhoA protein | Rac1 protein | Inflammation | Cadherin | Endothelial cells | Lungs | Nitric oxide | Ligands | Microvasculature | Kinetics | Destabilization | Electrical junctions
Journal Article