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Current Pharmaceutical Design, ISSN 1381-6128, 12/2007, Volume 13, Issue 35, pp. 3545 - 3558
Malignant glioma represents one of the most lethal and angiogenic cancers. Angiogenesis is a fundamental process of blood vessel growth that is a hallmark of... 
Angiogenesis | Biomarker | Glioma | VEGF | Glioblastoma | Cancer stem cell | Kinase inhibitor | Bevacizumab | RECOMBINANT INTERFERON-BETA | angiogenesis | NEWLY-DIAGNOSED GLIOBLASTOMA | biomarker | METASTATIC COLORECTAL-CANCER | kinase inhibitor | ANTI-VEGF ANTIBODY | PHASE-II TRIAL | glioblastoma | cancer stem cell | BRAIN-TUMOR CONSORTIUM | bevacizumab | glioma | TYROSINE KINASE INHIBITOR | PHARMACOLOGY & PHARMACY | RECURRENT GLIOBLASTOMA-MULTIFORME | HIGH-GRADE GLIOMAS | ENDOTHELIAL GROWTH-FACTOR | Humans | Drug Resistance, Neoplasm | Angiopoietins - antagonists & inhibitors | Brain Neoplasms - blood supply | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Integrins - metabolism | Brain Neoplasms - metabolism | Brain Neoplasms - surgery | Glioma - metabolism | Stem Cell Transplantation | Fibroblast Growth Factors - metabolism | Hepatocyte Growth Factor - antagonists & inhibitors | Receptors, Vascular Endothelial Growth Factor - metabolism | Antineoplastic Agents - adverse effects | Protein Kinase C - metabolism | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Neovascularization, Pathologic - prevention & control | Receptor, TIE-2 - metabolism | Antineoplastic Agents - pharmacology | Glioma - surgery | Angiogenesis Inhibitors - adverse effects | Receptor, TIE-2 - antagonists & inhibitors | Angiopoietins - metabolism | Antibodies, Monoclonal - pharmacology | Angiogenesis Inhibitors - pharmacology | Combined Modality Therapy | Protein Kinase C - antagonists & inhibitors | Brain Neoplasms - drug therapy | Receptor Protein-Tyrosine Kinases - metabolism | Hepatocyte Growth Factor - metabolism | Glioma - blood supply | Animals | Signal Transduction - drug effects | Angiogenic Proteins - antagonists & inhibitors | Neovascularization, Pathologic - metabolism | Protein Kinase Inhibitors - pharmacology | Fibroblast Growth Factors - antagonists & inhibitors | Angiogenic Proteins - metabolism | Biomarkers, Pharmacological - metabolism | Glioma - drug therapy | Integrins - antagonists & inhibitors
Journal Article
Developmental Cell, ISSN 1534-5807, 10/2017, Volume 43, Issue 1, pp. 71 - 82.e6
Journal Article
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2002, Volume 99, Issue 17, pp. 11393 - 11398
Vascular endothelial growth factor (VEGF) plays a critical role during normal embryonic angiogenesis and also in the pathological angiogenesis that occurs in a... 
Angiogenesis | Biological Sciences | Receptors | Humans | Antibodies | Monoclonal antibodies | Animal traps | Pharmacokinetics | Dosage | Tumors | Cancer | REGRESSION | INHIBITION | METASTASIS | ANGIOPOIETINS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MONOCLONAL-ANTIBODY | TUMOR ANGIOGENESIS | NEOVASCULARIZATION | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Vascular Endothelial Growth Factor A | Phosphorylation | Vascular Endothelial Growth Factors | Melanoma, Experimental - drug therapy | Antineoplastic Agents - therapeutic use | Endothelial Growth Factors - pharmacology | Endothelial Growth Factors - immunology | Endothelium, Vascular - physiology | Vascular Endothelial Growth Factor Receptor-1 | Immunoglobulin Constant Regions - genetics | Rhabdomyosarcoma - blood supply | Bone Neoplasms - blood supply | Cell Division | Extracellular Matrix - physiology | Lymphokines - antagonists & inhibitors | Drug Design | Protein Engineering | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Rhabdomyosarcoma - drug therapy | Antineoplastic Agents - pharmacology | Bone Neoplasms - drug therapy | Lymphokines - pharmacology | Proto-Oncogene Proteins - antagonists & inhibitors | Melanoma, Experimental - blood supply | Umbilical Veins | Endothelial Growth Factors - antagonists & inhibitors | Animals | Immunoglobulin G - genetics | Lymphokines - immunology | Mice | Mice, Inbred BALB C | 3T3 Cells | Growth | Physiological aspects | Causes of | Neovascularization | Vascular endothelial growth factor | Diseases
Journal Article
International Immunopharmacology, ISSN 1567-5769, 2009, Volume 9, Issue 6, pp. 767 - 773
Recent data reported that chitosan reduces high-fat (HF) diet-induced obesity in mice without describing the metabolic consequences of such an effect. The aim... 
Adipocytokine | Obesity | Chitosan | Adiposity | Mushrooms | BILE-ACID | CHOLESTEROL | HIGH-FAT DIET | RATS | HYPERINSULINEMIA | IMMUNOLOGY | METABOLISM | INSULIN-RESISTANCE | INFLAMMATION | LIVER | PHARMACOLOGY & PHARMACY | ADIPOSE-TISSUE | Leptin - antagonists & inhibitors | Obesity - diet therapy | Liver - pathology | Interleukin-6 - antagonists & inhibitors | Leptin - metabolism | Male | Angiopoietins - antagonists & inhibitors | Insulin - blood | Muscles - pathology | Angiopoietin-like 4 Protein | Liver - drug effects | Lipids - blood | Chemokine CCL2 - metabolism | Muscles - metabolism | Interleukin-6 - metabolism | Angiopoietins - metabolism | Resistin - metabolism | Liver - metabolism | Mice, Inbred C57BL | Chitosan - administration & dosage | Adipokines - metabolism | Adipokines - antagonists & inhibitors | Obesity - pathology | Animals | Anticholesteremic Agents - therapeutic use | Chitosan - chemistry | Diet | Muscles - drug effects | Mice, Obese | Mice | Chemokine CCL2 - antagonists & inhibitors | Resistin - antagonists & inhibitors | Dietary Supplements | Anticholesteremic Agents - pharmacology | Agaricales - chemistry | Proteolipids | Lipoproteins | Leptin | Dietary supplements | Physiological aspects | Lipoprotein lipase | Glucose | Blood lipoproteins | Glucose tolerance tests | Fatty acids | Dextrose | Carnitine
Journal Article