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PLoS ONE, ISSN 1932-6203, 01/2013, Volume 8, Issue 1, p. e53950
Journal Article
Nature Medicine, ISSN 1078-8956, 02/2017, Volume 23, Issue 2, pp. 200 - 212
Heritable thoracic aortic aneurysms and dissections (TAAD), including Marfan syndrome (MFS), currently lack a cure, and causative mutations have been... 
MEDICINE, RESEARCH & EXPERIMENTAL | INFLAMMATORY ANEURYSM | BIOCHEMISTRY & MOLECULAR BIOLOGY | THORACIC AORTA | ANGIOTENSIN-II | CELL BIOLOGY | SYNTHASE | DISINTEGRIN | IN-VIVO | DISSECTIONS | ANEURYSM FORMATION | EXPRESSION | PROGRESSION | Aneurysm, Dissecting - genetics | Aortic Aneurysm, Thoracic - genetics | Aortic Aneurysm - metabolism | ADAMTS1 Protein - genetics | Humans | Middle Aged | Immunoblotting | Male | Aorta - metabolism | Gene Knockdown Techniques | Nitric Oxide Synthase Type II - antagonists & inhibitors | Adult | Female | Fibrillin-1 - genetics | Real-Time Polymerase Chain Reaction | ADAMTS1 Protein - metabolism | Disease Models, Animal | NG-Nitroarginine Methyl Ester - pharmacology | Aneurysm, Dissecting - metabolism | Aorta - drug effects | Enzyme Inhibitors - pharmacology | Marfan Syndrome - genetics | Haploinsufficiency | Animals | Nitric Oxide Synthase Type II - genetics | Aortic Aneurysm, Thoracic - metabolism | Marfan Syndrome - metabolism | Aged | Mice | Aortic Aneurysm - genetics | Nitric Oxide - metabolism | Nitric Oxide Synthase Type II - metabolism | Blood circulation disorders | Molecular targeted therapy | Care and treatment | Proteases | Innovations | Development and progression | Genetic aspects | Oxidoreductases | Health aspects | Aneurysms | Cardiovascular disease | Genetic disorders | Rodents | Nitric oxide
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 09/2015, Volume 309, Issue 5, pp. H906 - H917
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology: Journal of the American Heart Association, ISSN 1079-5642, 03/2004, Volume 24, Issue 3, pp. 429 - 434
ABSTRACT—Many mouse models of abdominal aortic aneurysms have been developed that use a diverse array of methods for producing the disease, including genetic... 
Aneurysms | Mice | ACCELERATED ATHEROSCLEROSIS | VASCULAR-LESIONS | ANGIOTENSIN-II | HYPERCHOLESTEROLEMIA | mice | PERIARTERIAL APPLICATION | ARTERIOSCLEROSIS | TISSUE INHIBITOR | PERIPHERAL VASCULAR DISEASE | LDL RECEPTOR-DEFICIENT | HEMATOLOGY | EXPRESSION | aneurysms | TRANSGENIC MICE | Mice, Knockout - genetics | Aortic Aneurysm, Abdominal - chemically induced | Hyperlipidemias - genetics | Matrix Metalloproteinases - genetics | Angiotensin II - genetics | Aortic Aneurysm, Abdominal - prevention & control | Apolipoproteins E - deficiency | Species Specificity | Humans | Matrix Metalloproteinases - deficiency | Nitric Oxide Synthase - genetics | Aortic Aneurysm, Abdominal - blood | Mice, Mutant Strains - genetics | Nitric Oxide Synthase Type II | Renin - genetics | Pancreatic Elastase - toxicity | Receptors, LDL - deficiency | Female | Models, Animal | Angiotensin II - toxicity | Protease Inhibitors - therapeutic use | Extracellular Matrix Proteins - metabolism | Hyperlipidemias - complications | Disease Models, Animal | Calcium Chloride - toxicity | Receptors, LDL - genetics | Genetic Predisposition to Disease | Extracellular Matrix Proteins - genetics | Nitric Oxide Synthase Type III | Aortic Aneurysm, Abdominal - genetics | Cholesterol - metabolism | Animals | Genetic Engineering | Nitric Oxide Synthase - deficiency | Aortic Aneurysm, Abdominal - physiopathology | Apolipoproteins E - genetics | Matrix Metalloproteinase Inhibitors | Matrix Metalloproteinases - physiology
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2015, Volume 90, pp. 59 - 69
Abstract Delineation of mechanisms underlying the regulation of fibrosis-related genes in the heart is an important clinical goal as cardiac fibrosis is a... 
Cardiovascular | NF-κB | Cardiac fibroblasts | Collagen type I | Angiotensin II | p38 MAPK | Discoidin domain receptor 2 | P38 MAPK | TRANSCRIPTIONAL ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTEIN-COUPLED RECEPTORS | DDR2 REGULATES PROLIFERATION | NECROSIS-FACTOR-ALPHA | TYROSINE KINASES | CELL BIOLOGY | NF-kappa B | HEPATIC STELLATE CELLS | MYOCARDIAL FIBROSIS | SMOOTH-MUSCLE-CELLS | NF-KAPPA-B | DEPENDENT ACTIVATION | Protein Kinase C - genetics | RNA, Small Interfering - genetics | Angiotensin II - genetics | Reactive Oxygen Species - metabolism | NADPH Oxidases - metabolism | Type C Phospholipases - metabolism | Male | NF-kappa B - metabolism | Discoidin Domain Receptors | Collagen Type I - genetics | Protein Kinase C - metabolism | Mitogen-Activated Protein Kinase 1 - genetics | Myocardium - metabolism | NADPH Oxidases - genetics | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Transcription, Genetic | p38 Mitogen-Activated Protein Kinases - metabolism | Fibroblasts - metabolism | Angiotensin II - pharmacology | Collagen Type I - metabolism | Angiotensin II - metabolism | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Receptors, Mitogen - antagonists & inhibitors | Collagen Type I - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Rats | p38 Mitogen-Activated Protein Kinases - genetics | Receptors, Mitogen - metabolism | Receptor Protein-Tyrosine Kinases - metabolism | Rats, Sprague-Dawley | Myocardium - cytology | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | NF-kappa B - genetics | Receptor Protein-Tyrosine Kinases - genetics | Fibroblasts - drug effects | Type C Phospholipases - genetics | Fibroblasts - cytology | Primary Cell Culture | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism | Receptors, Mitogen - genetics | Oxidases | Memory (Computers) | Analysis | Collagen | Genes | Angiotensin | Genetic research | Gene expression
Journal Article
Journal Article
Peptides, ISSN 0196-9781, 07/2011, Volume 32, Issue 7, pp. 1551 - 1565
► The renin-angiotensin system is now evolving as an endocrine, paracrine and intracrine system. ► The renin/ACE/Ang II/AT receptor axis is the most recognized... 
Angiotensin converting enzyme 2 (ACE2) | Angiotensin II (Ang II) | Mas receptor | (Pro)renin receptor (PRR) | AT1 receptor signaling | Insulin-regulated aminopeptidase (IRAP) | receptor signaling | ANG-II | AT RECEPTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | INWARD CALCIUM CURRENT | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | BLOOD-PRESSURE RESPONSES | FACTOR-KAPPA-B | VACUOLAR H+-ATPASE | VASCULAR SMOOTH-MUSCLE | INTRACELLULAR FLUORESCENT FUSION | PROXIMAL TUBULE CELLS | ENDOCRINOLOGY & METABOLISM | PHARMACOLOGY & PHARMACY | AT receptor signaling | Blood Pressure | Cystinyl Aminopeptidase - genetics | Kidney Diseases - physiopathology | Receptors, Angiotensin - metabolism | Renin - metabolism | Angiotensin II - genetics | Cystinyl Aminopeptidase - metabolism | Kidney - pathology | Receptors, G-Protein-Coupled - metabolism | Humans | Peptidyl-Dipeptidase A - genetics | Kidney - metabolism | Renin - genetics | Peptidyl-Dipeptidase A - metabolism | Kidney - physiopathology | Kidney Diseases - metabolism | Proto-Oncogene Proteins - metabolism | Angiotensin II - metabolism | Rats, Transgenic | Signal Transduction | Kidney Diseases - pathology | Rats | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Renin-Angiotensin System - physiology | Angiotensin I - metabolism | Receptors, Angiotensin - genetics | Animals | Angiotensin I - genetics | Mice | Receptors, G-Protein-Coupled - genetics | Renin-angiotensin system | Phosphotransferases | Angiotensin | Angiotensin converting enzyme | Enzymes | Endocrine systems | Receptors | Kidneys | Physiology | Blood pressure | Kinases | Diseases | insulin-regulated aminopeptidase (IRAP) | angiotensin II (Ang II)
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2013, Volume 70, pp. 9 - 18
Abstract Cardiac fibrosis is a substantial problem in managing multiple forms of heart disease. Fibrosis results from an unrestrained tissue repair process... 
Cardiovascular | TGFβ | Serum response factor | TRP channel | Extracellular matrix | Angiotensin II | Fibrosis | CARDIAC & CARDIOVASCULAR SYSTEMS | TGF-BETA | SMOOTH-MUSCLE ACTIN | EPICARDIUM-DERIVED CELLS | IMPROVES CARDIAC-FUNCTION | PROTEIN-KINASE ACTIVATION | CELL BIOLOGY | GROWTH-FACTOR-BETA | HEPATIC STELLATE CELLS | INDUCED PULMONARY-FIBROSIS | TGF beta | HEAVY-CHAIN SMEMB | SERUM-RESPONSE FACTOR | Serum Response Factor - genetics | Angiotensin II - genetics | Myofibroblasts - physiology | Heart - physiology | Humans | Actins - metabolism | Extracellular Matrix - metabolism | Fibrosis - metabolism | Extracellular Matrix - chemistry | Actins - genetics | Cell Differentiation | Heart - physiopathology | Angiotensin II - metabolism | Fibrosis - physiopathology | Endothelin-1 - genetics | Endothelin-1 - metabolism | Serum Response Factor - metabolism | Gene Expression Regulation | Myofibroblasts - cytology | Mechanotransduction, Cellular | Transforming Growth Factor beta - genetics | Mitogen-Activated Protein Kinases - genetics | Fibrosis - pathology | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinases - metabolism | Transforming growth factors | Bone morphogenetic proteins | Endothelin | Stem cells | Angiotensin | Muscle proteins | Heart diseases | Mitogens | Smooth muscle alpha-actin | Rho signaling | Myofibroblasts | Mechanical tension
Journal Article