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Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 01/2014, Volume 20, Issue 2, pp. 281 - 294
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2011, Volume 31, Issue 8, pp. 1898 - 1907
OBJECTIVE—Circulating microparticles are increased in cardiovascular disease and may themselves promote oxidative stress and inflammation. Molecular mechanisms... 
endothelium | microparticles | signal transduction | angiotensin II | reactive oxygen species | NADPH OXIDASE | OXIDATIVE STRESS | SUPEROXIDE-PRODUCTION | HYPERTENSIVE PATIENTS | IN-VITRO | NAD(P)H OXIDASE | PLATELET-DERIVED MICROPARTICLES | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | CIRCULATING MICROPARTICLES | HEMATOLOGY | E-DEFICIENT MICE | Reactive Oxygen Species - metabolism | Tetrazoles - pharmacology | Apolipoproteins E - deficiency | Membrane Microdomains - metabolism | NADPH Oxidases - metabolism | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Male | Angiotensin II Type 1 Receptor Blockers - pharmacology | Endothelial Cells - ultrastructure | Biphenyl Compounds - pharmacology | Cell-Derived Microparticles - drug effects | rho-Associated Kinases - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Acetophenones - pharmacology | Blood Pressure - drug effects | Blood Pressure - physiology | Cell-Derived Microparticles - metabolism | Cell-Derived Microparticles - ultrastructure | Microscopy, Electron, Transmission | Angiotensin II - pharmacology | Angiotensin II - metabolism | Signal Transduction | Endothelial Cells - metabolism | Hypertrophy, Left Ventricular - etiology | Mice, Inbred C57BL | Cells, Cultured | Mice, Knockout | Animals | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Apolipoproteins E - genetics | Mice | Protein Kinase Inhibitors - pharmacology | Adaptor Proteins, Signal Transducing - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - drug effects
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2012, Volume 109, Issue 20, pp. 7929 - 7934
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 12/2010, Volume 335, Issue 3, pp. 572 - 579
Biased G protein-coupled receptor ligands engage subsets of the receptor signals normally stimulated by unbiased agonists. However, it is unclear whether... 
PATHWAYS | BIASED LIGANDS | ACTIVATION | INTERNALIZATION | ANTAGONIST | CONTRACTILITY | PHOSPHORYLATION | NITRIC-OXIDE SYNTHASE | KINASE | FUNCTIONAL SELECTIVITY | PHARMACOLOGY & PHARMACY | RNA, Small Interfering - genetics | Humans | Myocardial Contraction - physiology | Myocardial Contraction - drug effects | Arrestins - genetics | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Arrestins - metabolism | Receptor, Angiotensin, Type 1 - genetics | Drug Interactions | Transfection | HEK293 Cells | src-Family Kinases - metabolism | Cardiovascular Physiological Phenomena - drug effects | Ventricular Function, Left - physiology | Blood Pressure - drug effects | Nitric Oxide Synthase Type III - metabolism | Focal Adhesion Kinase 1 - metabolism | Binding, Competitive | Angiotensin II - pharmacology | Angiotensin II - metabolism | Myocytes, Cardiac - cytology | Ventricular Function, Left - drug effects | Rats | Angiotensin II - analogs & derivatives | Oligopeptides - metabolism | Receptor, Angiotensin, Type 1 - agonists | Animals | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | Proto-Oncogene Proteins c-jun - metabolism | beta-Arrestins | Cell Line, Tumor | Signal Transduction - physiology | Mice | Oligopeptides - pharmacology | GTP-Binding Proteins - metabolism
Journal Article
Hypertension, ISSN 0194-911X, 11/2013, Volume 62, Issue 5, pp. 886 - 892
Journal Article
Science, ISSN 0036-8075, 4/2011, Volume 332, Issue 6027, pp. 361 - 365
Angiotensin II (AngII) mediates progression of aortic aneurysm, but the relative contribution of its type 1 (AT1) and type 2 (AT2) receptors remains unknown.... 
Connective tissues | Receptors | Root growth | Medical treatment | REPORTS | Aneurysms | Placebos | Aorta | Mice | Aortic aneurysm | Marfan syndrome | PATHOGENESIS | ACTIVATION | MECHANISM | MULTIDISCIPLINARY SCIENCES | MARFAN-SYNDROME | MOUSE MODEL | GROWTH | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | EXPRESSION | Aortic Aneurysm - metabolism | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Aortic Rupture - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Enalapril - therapeutic use | MAP Kinase Signaling System | Marfan Syndrome - drug therapy | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Aortic Rupture - prevention & control | Angiotensin-Converting Enzyme Inhibitors - pharmacology | Aortic Aneurysm - pathology | Aortic Aneurysm - prevention & control | Disease Models, Animal | Receptor, Angiotensin, Type 2 - genetics | Angiotensin II - metabolism | Signal Transduction | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Losartan - pharmacology | Aortic Rupture - pathology | Disease Progression | Mice, Knockout | Animals | Enalapril - pharmacology | Receptor, Angiotensin, Type 2 - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Aortic Aneurysm - drug therapy | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Transforming Growth Factor beta - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Aortic aneurysms | Development and progression | Genetic aspects | Health aspects | Angiotensin | Signal transduction | Peptides | Cellular biology | Coronary vessels | Rodents
Journal Article
Journal Article