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American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 09/2011, Volume 301, Issue 3, pp. C559 - C565
Deliu E, Tica AA, Motoc D, Brailoiu GC, Brailoiu E. Intracellular angiotensin II activates rat myometrium... 
Calcium imaging | Microinjection | Cytosolic calcium concentration | Endoplasmic reticulum | CYCLIC ADP-RIBOSE | PHYSIOLOGY | ANG-II | LIVER LYSOSOMES | RECEPTOR | microinjection | METABOLIC-CLEARANCE | BLOOD-PRESSURE | UTERINE | CELL BIOLOGY | SIGNAL-TRANSDUCTION | RENAL ENDOSOMES | SMOOTH-MUSCLE-CELLS | calcium imaging | cytosolic calcium concentration | endoplasmic reticulum | Myometrium - drug effects | Type C Phospholipases - metabolism | Brefeldin A - pharmacology | Angiotensin II Type 1 Receptor Blockers - pharmacology | Autophagy - drug effects | Endosomes - metabolism | Angiotensin II Type 2 Receptor Blockers - pharmacology | Macrocyclic Compounds - pharmacology | Saralasin - pharmacology | Endoplasmic Reticulum - drug effects | Estrenes - pharmacology | Myometrium - metabolism | Inositol 1,4,5-Trisphosphate Receptors - antagonists & inhibitors | Ryanodine - pharmacology | Angiotensin II - pharmacology | Pyridines - administration & dosage | Endocytosis - drug effects | Enzyme Inhibitors - pharmacology | Losartan - pharmacology | Saralasin - administration & dosage | Rats | Imidazoles - pharmacology | Piperazines - pharmacology | Rats, Sprague-Dawley | Receptor, Angiotensin, Type 2 - metabolism | Signal Transduction - drug effects | Calcium Signaling - drug effects | Models, Biological | Receptor, Angiotensin, Type 1 - metabolism | Oxazoles - pharmacology | Losartan - administration & dosage | Angiotensin II - administration & dosage | Calcium Signaling - physiology | Endoplasmic Reticulum - metabolism | Imidazoles - administration & dosage | Type C Phospholipases - antagonists & inhibitors | Lysosomes - metabolism | Inositol 1,4,5-Trisphosphate Receptors - metabolism | Pyrrolidinones - pharmacology | Macrolides - pharmacology | NADP - analogs & derivatives | Female | NADP - metabolism | Heparin - pharmacology | Angiotensin II - metabolism | Arsenicals - pharmacology | Cells, Cultured | Sirolimus - pharmacology | Myometrium - cytology | Animals | Egtazic Acid - pharmacology | Uterus - cytology | Signal Transduction - physiology | Pyridines - pharmacology | Carbolines - pharmacology | Myometrium | Cell receptors | Calcium channels | Genetic aspects | Properties | Angiotensin | Receptors and Signal Transduction
Journal Article
Circulation Research, ISSN 0009-7330, 12/2009, Volume 105, Issue 12, pp. 1204 - 1212
RATIONALE:Angiotensin (Ang) II–induced apoptosis was reported to be mediated by different signaling molecules... 
Reactive oxygen species | CaMKII | Angiotensin II | Apoptosis | CARDIAC & CARDIOVASCULAR SYSTEMS | P38 MAPK | INHIBITION PROTECTS | apoptosis | VENTRICULAR MYOCYTES | CELL-DEATH | PHOSPHOLAMBAN PHOSPHORYLATION | HEART-DISEASE | METHIONINE OXIDATION | angiotensin II | reactive oxygen species | PERIPHERAL VASCULAR DISEASE | INDEPENDENT ACTIVATION | HEMATOLOGY | Free Radical Scavengers - pharmacology | Tiopronin - pharmacology | Reactive Oxygen Species - metabolism | Rats, Wistar | Species Specificity | Apoptosis - drug effects | Calcium - metabolism | NADPH Oxidases - metabolism | Peptides - genetics | Myocytes, Cardiac - enzymology | Time Factors | Egtazic Acid - analogs & derivatives | p38 Mitogen-Activated Protein Kinases - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Cats | Calmodulin - metabolism | Angiotensin II - metabolism | Cell Survival | NADPH Oxidases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Rats | Mice, Transgenic | Imidazoles - pharmacology | Chelating Agents - pharmacology | Onium Compounds - pharmacology | Sulfonamides - pharmacology | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Peptides - pharmacology | Myocytes, Cardiac - pathology | Animals | Egtazic Acid - pharmacology | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Enzyme Activation | Oxidative Stress - drug effects | Benzylamines - pharmacology | Calmodulin - antagonists & inhibitors
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2011, Volume 31, Issue 8, pp. 1898 - 1907
...). METHODS AND RESULTS—Microparticle formation from angiotensin II (Ang II)–stimulated ECs and apolipoprotein E mice was assessed by annexin V or by CD144 staining and electron microscopy... 
endothelium | microparticles | signal transduction | angiotensin II | reactive oxygen species | NADPH OXIDASE | OXIDATIVE STRESS | SUPEROXIDE-PRODUCTION | HYPERTENSIVE PATIENTS | IN-VITRO | NAD(P)H OXIDASE | PLATELET-DERIVED MICROPARTICLES | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | CIRCULATING MICROPARTICLES | HEMATOLOGY | E-DEFICIENT MICE | Reactive Oxygen Species - metabolism | Tetrazoles - pharmacology | Apolipoproteins E - deficiency | Membrane Microdomains - metabolism | NADPH Oxidases - metabolism | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Male | Angiotensin II Type 1 Receptor Blockers - pharmacology | Endothelial Cells - ultrastructure | Biphenyl Compounds - pharmacology | Cell-Derived Microparticles - drug effects | rho-Associated Kinases - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Acetophenones - pharmacology | Blood Pressure - drug effects | Blood Pressure - physiology | Cell-Derived Microparticles - metabolism | Cell-Derived Microparticles - ultrastructure | Microscopy, Electron, Transmission | Angiotensin II - pharmacology | Angiotensin II - metabolism | Signal Transduction | Endothelial Cells - metabolism | Hypertrophy, Left Ventricular - etiology | Mice, Inbred C57BL | Cells, Cultured | Mice, Knockout | Animals | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Apolipoproteins E - genetics | Mice | Protein Kinase Inhibitors - pharmacology | Adaptor Proteins, Signal Transducing - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - drug effects
Journal Article
The Journal of physiology, ISSN 0022-3751, 2016, Volume 594, Issue 23, pp. 7027 - 7047
Key points Candesartan, an inverse agonist of the type 1 angiotensin II receptor (AT1R... 
type 1 angiotensin II receptor | cytoskeleton | mechanotransduction | actin stress fibres | vascular smooth muscle cell signalling | myogenic response | protein kinase C | RHO-ASSOCIATED KINASE | INTRAVASCULAR PRESSURE | PHYSIOLOGY | CEREBRAL PARENCHYMAL ARTERIOLES | RESISTANCE ARTERIES | PROTEIN-COUPLED RECEPTORS | MYOSIN PHOSPHORYLATION | STRETCH-ACTIVATION | CYTOSKELETON REORGANIZATION | NEUROSCIENCES | VASCULAR SMOOTH-MUSCLE | LIGHT-CHAIN PHOSPHORYLATION | Arterioles - physiology | Antihypertensive Agents - pharmacology | Receptor, Angiotensin, Type 1 - physiology | Tetrazoles - pharmacology | Captopril - pharmacology | Maleimides - pharmacology | Male | Angiotensin II Type 1 Receptor Blockers - pharmacology | Muscle Development | Receptor, Angiotensin, Type 1 - genetics | Diglycerides - pharmacology | Indoles - pharmacology | Muscle Fibers, Skeletal - physiology | Vasoconstrictor Agents - pharmacology | Protein Kinase C - physiology | Abdominal Muscles - physiology | Cells, Cultured | Losartan - pharmacology | Imidazoles - pharmacology | Protein Kinase C - antagonists & inhibitors | Arterioles - drug effects | Pressure | Rats, Sprague-Dawley | Vasoconstriction - drug effects | Animals | Benzimidazoles - pharmacology | Pyridines - pharmacology | Actins - physiology | Atomic force microscopy | Actin | Angiotensin | Polymerization | Muscles | G proteins | Muscle proteins | Protein kinases | Arteries | Membrane proteins | Cardiovascular Physiology | Skeletal Muscle | Cardiovascular | Research Paper
Journal Article
Journal Article
Gastroenterology, ISSN 0016-5085, 2000, Volume 118, Issue 6, pp. 1149 - 1156
Background & Aims: Circulating levels of angiotensin II (ANGII), a powerful vasoconstrictor factor, are frequently increased in chronic liver diseases... 
FAT-STORING CELLS | RAT-LIVER | GLOMERULAR MESANGIAL CELLS | DEPENDENT CONTRACTILITY | NITRIC-OXIDE | GROWTH-FACTOR | ITO CELLS | GASTROENTEROLOGY & HEPATOLOGY | SMOOTH-MUSCLE CELLS | PORTAL-HYPERTENSION | ALDOSTERONE SYSTEM | Receptor, Angiotensin, Type 1 | Receptors, Angiotensin - metabolism | Receptor, Angiotensin, Type 2 | Antihypertensive Agents - pharmacology | Iodine Radioisotopes | Liver - enzymology | Humans | Penicillamine - analogs & derivatives | Sodium Nitrite - pharmacology | Liver - drug effects | Thymidine - pharmacology | Nitric Oxide Donors - pharmacology | Radioligand Assay | Thymidine - metabolism | NG-Nitroarginine Methyl Ester - pharmacology | Vasoconstrictor Agents - pharmacology | Dinoprostone - pharmacology | Angiotensin II - pharmacology | Angiotensin II - metabolism | Cell Size - drug effects | Penicillamine - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Losartan - pharmacology | Rats | Imidazoles - pharmacology | Cell Division - drug effects | Indomethacin - pharmacology | Cyclooxygenase Inhibitors - pharmacology | Animals | Vasoconstrictor Agents - metabolism | Liver - cytology | Pyridines - pharmacology | Mitogen-Activated Protein Kinases - metabolism | Cell research | Liver diseases | Platelet-derived growth factor | Prostaglandins E | Nitric oxide | Angiotensin | Physiological aspects | Bookbinding | DNA synthesis | Peptide hormones | Protein kinases
Journal Article
Circulation Research: Journal of the American Heart Association, ISSN 0009-7330, 09/2002, Volume 91, Issue 5, pp. 406 - 413
ABSTRACT—Angiotensin II (Ang II)–stimulated hypertrophy of vascular smooth muscle cells is mediated by reactive oxygen species (ROS) derived from NAD(P)H oxidases... 
Reactive oxygen species | Vascular smooth muscle | Angiotensin II | Rac | NAD(P)H oxidase | NADPH-OXIDASE | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | vascular smooth muscle | PHOSPHATIDYLINOSITOL 3-KINASE | GUANOSINE TRIPHOSPHATASES | SUPEROXIDE ANION PRODUCTION | GROWTH-FACTOR RECEPTOR | angiotensin II | reactive oxygen species | ENDOTHELIAL-CELLS | REQUIRES | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | SMOOTH-MUSCLE CELLS | Reactive Oxygen Species - metabolism | Muscle, Smooth, Vascular - metabolism | Maleimides - pharmacology | rac GTP-Binding Proteins - metabolism | Male | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Flow Cytometry | Time Factors | Protein Kinase C - metabolism | src-Family Kinases - metabolism | Indoles - pharmacology | Chromones - pharmacology | NADH, NADPH Oxidoreductases - metabolism | NADH, NADPH Oxidoreductases - drug effects | Muscle, Smooth, Vascular - drug effects | Pyrazoles - pharmacology | NADPH Oxidases | Angiotensin II - pharmacology | ErbB Receptors - metabolism | src-Family Kinases - antagonists & inhibitors | Cells, Cultured | Enzyme Inhibitors - pharmacology | Morpholines - pharmacology | Rats | Protein Kinase C - antagonists & inhibitors | Pyrimidines - pharmacology | Muscle, Smooth, Vascular - cytology | Rats, Sprague-Dawley | Hydrogen Peroxide - metabolism | Animals | Androstadienes - pharmacology | Signal Transduction - drug effects | Wortmannin
Journal Article
Drug metabolism and disposition, ISSN 1521-009X, 2012, Volume 40, Issue 9, pp. 1744 - 1756
Interindividual variability in activity of uptake transporters is evident in vivo, yet limited data exist in vitro, confounding in vitro-in vivo extrapolation.... 
DRUG TRANSPORTERS | IN-VITRO CLEARANCE | CRYOPRESERVED HUMAN HEPATOCYTES | HMG-COA REDUCTASE | PHARMACOLOGY & PHARMACY | ANION TRANSPORTING POLYPEPTIDES | HEPATIC-UPTAKE | HEALTHY-VOLUNTEERS | METABOLIC ENZYMES | RECEPTOR ANTAGONIST | PREDICTION | Antihypertensive Agents - pharmacology | Tetrazoles - pharmacology | Species Specificity | Valsartan | Hypoglycemic Agents - metabolism | Benzoates - metabolism | Humans | Antihypertensive Agents - metabolism | Hepatocytes - metabolism | Angiotensin II Type 1 Receptor Blockers - pharmacology | Angiotensin II Type 1 Receptor Blockers - metabolism | Organic Anion Transporters - metabolism | Pyrimidines - metabolism | Quinolines - pharmacology | Tetrazoles - metabolism | Carbamates - metabolism | Dose-Response Relationship, Drug | Fluorobenzenes - pharmacology | Drug Interactions | Hydroxymethylglutaryl-CoA Reductase Inhibitors - metabolism | Biological Transport | Piperidines - pharmacology | Hepatocytes - drug effects | Carbamates - pharmacology | Pravastatin - pharmacology | Fluorobenzenes - metabolism | Piperidines - metabolism | Pravastatin - metabolism | Valine - analogs & derivatives | Rats | Rosuvastatin Calcium | Pyrimidines - pharmacology | Sulfonamides - pharmacology | Hypoglycemic Agents - pharmacology | Quinolines - metabolism | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Valine - metabolism | Models, Biological | Benzimidazoles - metabolism | Sulfonamides - metabolism | Benzoates - pharmacology | Benzimidazoles - pharmacology | Kinetics | Valine - pharmacology | Organic Anion Transporters - drug effects
Journal Article
Hypertension, ISSN 0194-911X, 09/2012, Volume 60, Issue 3, pp. 722 - 729
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 05/2011, Volume 300, Issue 5, pp. 1181 - 1192
... (NO) synthase, thereby reducing NO production. Elevated angiotensin II (ANG II) is a key component of endothelial dysfunction in many cardiovascular diseases and has been linked to elevated arginase activity... 
Endothelial nitric oxide synthase dysfunction | PHYSIOLOGY | BLOOD-PRESSURE | P38 MAP KINASE | CELL BIOLOGY | INCREASED ARGINASE ACTIVITY | NITRIC-OXIDE PRODUCTION | INHIBITION | MOUSE MODEL | endothelial nitric oxide synthase dysfunction | CARDIOVASCULAR-DISEASE | SMOOTH-MUSCLE-CELLS | HYPERTENSIVE-RATS | CONTRIBUTES | Phosphorylation | Endothelial Cells | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Endothelium, Vascular - drug effects | Endothelium, Vascular - enzymology | GTP-Binding Protein alpha Subunits, G12-G13 - antagonists & inhibitors | Simvastatin - pharmacology | Angiotensin II Type 1 Receptor Blockers - pharmacology | rho-Associated Kinases - antagonists & inhibitors | Cattle | Arginase - antagonists & inhibitors | Amides - pharmacology | Cell Line | rho-Associated Kinases - physiology | Angiotensin II - pharmacology | p38 Mitogen-Activated Protein Kinases - physiology | RNA, Small Interfering - pharmacology | Endothelium, Vascular - physiopathology | Enzyme Inhibitors - pharmacology | GTP-Binding Protein alpha Subunits, Gq-G11 - antagonists & inhibitors | Imidazoles - pharmacology | Animals | Signal Transduction - drug effects | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Signal Transduction - physiology | Mice | Pyridines - pharmacology | Arginase - physiology | Angiotensin II - physiology | Boronic Acids - pharmacology | Abnormalities | Angiotensin | Physiological aspects | Vascular endothelium | Genetic aspects | Research | Mitogen-activated protein kinases | Index Medicus | Vascular Biology
Journal Article
Psychopharmacology (Berlin, Germany), ISSN 1432-2072, 2012, Volume 226, Issue 2, pp. 357 - 369
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/1996, Volume 97, Issue 8, pp. 1916 - 1923
We tested the hypothesis that angiotensin II-induced hypertension is associated with an increase in vascular . O-2... 
Hypertension | Superoxide | Angiotensin II | Nitric oxide |